Littérature scientifique sur le sujet « Diseased red cell »
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Articles de revues sur le sujet "Diseased red cell"
Yang, Lan, Shiqi Huang, Zhirong Zhang, Zhenmi Liu et Ling Zhang. « Roles and Applications of Red Blood Cell-Derived Extracellular Vesicles in Health and Diseases ». International Journal of Molecular Sciences 23, no 11 (25 mai 2022) : 5927. http://dx.doi.org/10.3390/ijms23115927.
Texte intégralCHEN, YONG, JIYE CAI et JINGXIAN ZHAO. « DISEASED RED BLOOD CELLS STUDIED BY ATOMIC FORCE MICROSCOPY ». International Journal of Nanoscience 01, no 05n06 (octobre 2002) : 683–88. http://dx.doi.org/10.1142/s0219581x02000899.
Texte intégralMattè, Alessandro, Enrica Federti, Elena Tibaldi, Maria Luisa Di Paolo, Giovanni Bisello, Mariarita Bertoldi, Andrea Carpentieri et al. « Tyrosine Phosphorylation Modulates Peroxiredoxin-2 Activity in Normal and Diseased Red Cells ». Antioxidants 10, no 2 (1 février 2021) : 206. http://dx.doi.org/10.3390/antiox10020206.
Texte intégralAoki, T., et T. Inoue. « Glycophorin in red blood cell membranes of healthy and diseased carp, Cyprinus carpio L. » Journal of Fish Diseases 34, no 7 (18 mai 2011) : 573–76. http://dx.doi.org/10.1111/j.1365-2761.2011.01262.x.
Texte intégralLi, He, Lu Lu, Xuejin Li, Pierre A. Buffet, Ming Dao, George E. Karniadakis et Subra Suresh. « Mechanics of diseased red blood cells in human spleen and consequences for hereditary blood disorders ». Proceedings of the National Academy of Sciences 115, no 38 (6 septembre 2018) : 9574–79. http://dx.doi.org/10.1073/pnas.1806501115.
Texte intégralWilkes, Mark C., Aya Shibuya et Kathleen M. Sakamoto. « Signaling Pathways That Regulate Normal and Aberrant Red Blood Cell Development ». Genes 12, no 10 (19 octobre 2021) : 1646. http://dx.doi.org/10.3390/genes12101646.
Texte intégralNayani, Karthik, Arthur A. Evans, Saverio E. Spagnolie et Nicholas L. Abbott. « Dynamic and reversible shape response of red blood cells in synthetic liquid crystals ». Proceedings of the National Academy of Sciences 117, no 42 (2 octobre 2020) : 26083–90. http://dx.doi.org/10.1073/pnas.2007753117.
Texte intégralLi, He, Dimitrios Papageorgiou, Hung-Yu Chang, Lu Lu, Jun Yang et Yixiang Deng. « Synergistic Integration of Laboratory and Numerical Approaches in Studies of the Biomechanics of Diseased Red Blood Cells ». Biosensors 8, no 3 (10 août 2018) : 76. http://dx.doi.org/10.3390/bios8030076.
Texte intégralPolák, J., M. Jokes et Marie Ulrychová. « Cell wall disintegration consistently found in tissues of reversion diseased red currant cv. Heinemann’s rote spätlesse ». Biologia Plantarum 27, no 6 (novembre 1985) : 462–64. http://dx.doi.org/10.1007/bf02894718.
Texte intégralProdanovic, Radisa, Ivan Vujanac, Danijela Kirovski, Vojin Ivetic, Bozidar Savic, Milenko Zutic, Branislav Kureljusic et Oliver Radanovic. « Paratuberculosis in breeding stock of red Holstein cows ». Veterinarski glasnik 65, no 3-4 (2011) : 179–90. http://dx.doi.org/10.2298/vetgl1104179p.
Texte intégralThèses sur le sujet "Diseased red cell"
Cytlak, Urszula Malgorzata. « Phosphatidylserine exposure in red blood cells from patients with sickle cell disease ». Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708601.
Texte intégralBarber, Latorya Arnold. « The Activity of Lipid Transport Proteins in Normal and Sickle Red Blood Cells ». University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1243353188.
Texte intégralAl, Balushi Halima. « Novel approach towards pathogenesis and treatment of sickle cell disease ». Thesis, University of Cambridge, 2019. https://www.repository.cam.ac.uk/handle/1810/288739.
Texte intégralLizarralde, Iragorri Maria. « Impact of mechanical and oxidative stress on red blood cell properties in sickle cell disease ». Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCC324.
Texte intégralThe red blood cell is a simple cell with one of the most important functions in the organism, that is fulfilling the gas exchange function and delivering oxygen to the tissues. It is a highly elastic biconcave disk thanks to a network of specific skeletal and membrane proteins. The function and structure of the red cell are altered in several human pathologies like hemoglobinopathies and membrane disorders. Sickle cell disease is a genetic hereditary disorder characterized by abnormal hemoglobin that polymerizes under hypoxic conditions leading to the sickling and alteration of circulating red cells. The hallmarks of sickle cell disease are hemolytic anemia and painful vaso-occlusive crises due to the obstruction of fine capillaries.With the aim of better understanding the mechanisms behind these clinical manifestations we investigated the mechanical and adhesive properties of red blood cells from patients with sickle cell disease by assessing 1) the impact of repeated mechanical stress on red cell survival using a microfluidic device that mimic human microcirculation, and 2) the role of oxidative stress in the activation of erythroid adhesion proteins.We designed a microfluidic device that allowed us to show that mechanical stress is a critical parameter underlying intravascular hemolysis in sickle cell disease and that high intracellular levels of fetal hemoglobin protect against lysis. Furthermore, we found that treatment with hydroxyurea protects red blood cells from lysis upon mechanical stress even in the absence of fetal hemoglobin expression. On the other hand, we investigated the structure and function of the erythroid adhesion protein Lu/BCAM under oxidative conditions using biochemical and imaging approaches. We observed that oxidative stress activates the adhesive function of Lu/BCAM through post-translational modifications that alter its membrane distribution. We describe a novel mechanism that affects Lu/BCAM cis-interactions at the cell surface that might account for the abnormal adhesion of sickle red cells to laminin in the absence of phosphorylation events.In conclusion, we developed a microfluidic device replicating the physiological dimensions of human microvessels that allows assessing previously unexplored cellular characteristics in sickle cell disease. We show that repeated mechanical stress is partly responsible for hemolysis in patients with sickle cell disease, which might contribute to the high levels of oxidative stress because of free heme in the circulation. Our work demonstrates the importance of the mechanical dimension in the blockade of small capillaries and the critical contribution of oxidative stress in the abnormal adhesion of red cells in this disease. Improving red cell deformability and targeting oxidative stress to inhibit red cell adhesion would be promising strategies to target the main hallmarks of this pathology and alleviate the disease burden
Simionato, Greta [Verfasser]. « The influence of hypoxia in erythropoiesis and morphology of red blood cells in sickle cell disease and hereditary spherocytosis. / Greta Simionato ». Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2020. http://d-nb.info/1221599666/34.
Texte intégralBoa-Amponsem, Kwame Jr. « Genetics, humoral immunoresponsiveness, and disease resistance in chickens ». Diss., Virginia Tech, 1998. http://hdl.handle.net/10919/30580.
Texte intégralPh. D.
Panknin, Christina Monika [Verfasser]. « Characterization of red blood cell functions in health and coronary artery disease / Christina Monika Panknin ». Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2017. http://d-nb.info/1136421815/34.
Texte intégralKucukal, Erdem. « BIOMIMETIC MICROFLUIDIC PLATFORMS FOR MONITORING CELLULAR INTERACTIONS IN MICROSCALE FLOW ». Case Western Reserve University School of Graduate Studies / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1576231265150031.
Texte intégralClaveria, Pizarro Viviana Andrea. « Flow of healthy and sickle red blood cells in microcirculatory conditions : clustering process and self-margination phenomenon ». Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTS081/document.
Texte intégralI experimentally characterized the clustering formation of healthy and sickle red blood cells (RBCs) flowing through straight micro-capillaries. The effect of aggregation was also investigated. I found that cluster formation under physiological conditions is most likely caused by a combination of hydrodynamic and macromolecule-induced interactions. Macromolecule-induced interactions are not fully overcome by shear stresses within the physiological range, and they contribute to cluster stability. Moreover, I found that a pronounced bimodal distribution of the cell-to-cell distances in the hydrodynamic clusters is produced.Additionally, I investigated experimentally the collective behavior of oxygenated sickle RBCs and their distribution along cylindrical micro-capillaries with diameters comparable to a human venule or arteriole. I have shown that there is a heterogeneous distribution of RBCs according to their density: low-density cells tend to stay closer to the center of the channel, while most dense cells (also more rigid) self-marginated under defined conditions. Aggregation seems to inhibit self-margination depending on the aggregative factor and patient: dextran allows self-margination in some patients and inhibits it in others. Plasma inhibits self-margination of cells in all cases, highlighting the importance of the plasma proteins and adhesive molecules in the aggregation phenomena
El, Hoss Sara. « Novel insights into the role of fetal hemoglobin in spleen function, red cell survival and ineffective erythropoiesis in sickle cell disease ». Thesis, Université de Paris (2019-....), 2019. https://theses.md.univ-paris-diderot.fr/ELHOSS_Sara_va2_20190924.pdf.
Texte intégralSickle cell disease (SCD) is caused by a single point mutation in the β-globin gene generating sickle hemoglobin (HbS). Hypoxia drives HbS polymerization that is responsible for red blood cell (RBC) sickling and reduced deformability. In SCD, splenic dysfunction results in life-threatening complications, particularly in early childhood. During the course of the disease, the spleen functionally declines and anatomically disappears, although with great individual variability depending on modulating genetic and environmental factors. The key modulator of disease severity is fetal hemoglobin (HbF), as the presence of HbF inhibits HbS polymerization, thus delaying and preventing severe complications, ameliorating patients’ quality of life and increasing survival. There is a rather well characterized hetero cellular concentration of HbF and distribution in circulating RBCs but the role of HbF during erythropoiesis, is poorly documented. With the aim of better understanding the role of HbF in spleen function, red cell survival and ineffective erythropoiesis we investigated 1) the natural history of spleen dysfunction in SCD children, 2) the cellular expression and distribution of HbF in SCD children, in untreated patients and patients treated with Hydroxycarbamide and 3) ineffective erythropoiesis and the role of HbF during terminal erythropoiesis.We developed a flow cytometry high-throughput method to measure splenic filtration function and showed that splenic loss of function is present very early in life at 3-6 months in SCD children and further declines with age. We also highlighted that irreversibly sickled cells (ISCs) are a potential contributor to acute splenic sequestration (ASS) which in turn results in further loss of splenic function. In the second part of this work, we set up an original approach to determine HbF distribution per cell. Using a longitudinal cohort of patients treated with hydroxycarbamide (HC - an inducer of HbF), we showed that HC has a global positive impact on RBCs, by not only increasing HbF content but also by increasing the volume of all RBCs independent of HbF. We moreover showed that High F-cells are a more precise marker of HC efficacy. In the last part of the thesis, we showed for the first time clear evidence of ineffective erythropoiesis in SCD and revealed a new role of HbF during terminal erythropoiesis protecting erythroblasts from apoptosis. In conclusion, this work shows that HbF has an additional beneficial effect in SCD by not only conferring a preferential survival of F-cells in the circulation but also by decreasing ineffective erythropoiesis. Importantly, it suggests that the delay in hemoglobin switch in SCD might be also due to an enrichment in F-erythroblasts during terminal erythroid differentiation occurring very early in infancy, shortly after birth
Livres sur le sujet "Diseased red cell"
Bernhardt, Ingolf, et J. Clive Ellory, dir. Red Cell Membrane Transport in Health and Disease. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05181-8.
Texte intégralBernhardt, Ingolf. Red Cell Membrane Transport in Health and Disease. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003.
Trouver le texte intégralR, Bridges Kenneth, et Pearson Howard A, dir. Anemias and other red cell disorders. New York : McGraw-Hill, 2007.
Trouver le texte intégralTsuyoshi, Ohnishi S., et Ohnishi Tomoko, dir. Membrane abnormalities in sickle cell disease and in other red blood cell disorders. Boca Raton, Fla : CRC Press, 1994.
Trouver le texte intégralMalaria resistance or susceptibility in red cells disorders. Hauppauge, NY : Nova Science, 2009.
Trouver le texte intégral1949-, Agre Peter, et Cartron Jean Pierre, dir. Protein blood group antigens of the human red cell : Structure, function, and clinical significance. Baltimore : Johns Hopkins University Press, 1992.
Trouver le texte intégralThe match : "savior siblings" and one family's battle to heal their daughter. Boston : Beacon Press, 2010.
Trouver le texte intégralLichtman, Marshall A., et Josef T. Prchal. Red Cell and Its Diseases. McGraw-Hill Education, 2021.
Trouver le texte intégralProvan, Drew, Trevor Baglin, Inderjeet Dokal et Johannes de Vos. Red cell disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0002.
Texte intégralProvan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, Banu Kaya et Angela Theodoulou. Red cell disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0002_update_001.
Texte intégralChapitres de livres sur le sujet "Diseased red cell"
Brovelli, Augusta, et Giampaolo Minetti. « Red Cell Ageing ». Dans Red Cell Membrane Transport in Health and Disease, 673–90. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05181-8_29.
Texte intégralBrugnara, Carlo. « Sickle Cell Disease ». Dans Red Cell Membrane Transport in Health and Disease, 549–67. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05181-8_23.
Texte intégralBien, Christian G., Christian E. Elger, Ali R. Afzal, Sirajedin Natah, Ritva Häyrinen-Immonen, Yrjö Konttinen, George S. Zubenko et al. « Red Cell Pyruvate Kinase Deficiency ». Dans Encyclopedia of Molecular Mechanisms of Disease, 1809–10. Berlin, Heidelberg : Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_1526.
Texte intégralOrbach, Hedi, Gisele Zandman Goddard, Asher Winder et Yehuda Shoenfeld. « Acquired Adult Pure Red Cell Aplasia ». Dans Diagnostic Criteria in Autoimmune Diseases, 525–28. Totowa, NJ : Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_96.
Texte intégralTauler Riera, Pedro, Maurizio Volterrani, Ferdinando Iellamo, Francesco Fallo, Andrea Ermolao, William J. Kraemer, Nicholas A. Ratamess, Avery Faigenbaum, Andrew Philp et Keith Baar. « Red Blood Cell Rheological Properties ». Dans Encyclopedia of Exercise Medicine in Health and Disease, 750. Berlin, Heidelberg : Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4491.
Texte intégralPatrinos, George P., et Frank G. Grosveld. « Transgenic Models of Red Cell Disorders ». Dans Red Cell Membrane Transport in Health and Disease, 643–71. Berlin, Heidelberg : Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05181-8_28.
Texte intégralTauler Riera, Pedro, Maurizio Volterrani, Ferdinando Iellamo, Francesco Fallo, Andrea Ermolao, William J. Kraemer, Nicholas A. Ratamess, Avery Faigenbaum, Andrew Philp et Keith Baar. « Red Blood Cell Capillary Transit Time ». Dans Encyclopedia of Exercise Medicine in Health and Disease, 750. Berlin, Heidelberg : Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2950.
Texte intégralKing, Karen E., et Paul M. Ness. « Red Cell Transfusions in Patients with Hematologic Malignancies ». Dans Neoplastic Diseases of the Blood, 1139–52. Cham : Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-64263-5_53.
Texte intégralKing, Karen E., et Paul M. Ness. « Red Cell Transfusions in Patients with Hematologic Malignancies ». Dans Neoplastic Diseases of the Blood, 1251–63. New York, NY : Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3764-2_55.
Texte intégralJones, D. B. « The Origin of the Reed-Sternberg Cell ». Dans Lymphoproliferative Diseases, 91–106. Dordrecht : Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0739-3_7.
Texte intégralActes de conférences sur le sujet "Diseased red cell"
Liu, Jia, Yuhao Qiang et E. Du. « Measurement of Electrical Properties of Sickle Cells From Electrical Impedance of Cell Suspension ». Dans ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-71734.
Texte intégralFerrant, A., N. Leners, E. K. Gini, J. P. Osselaerey et J. Sonnet. « EFFECT OF PIRACETAM ON THE MEAN INTRASPLENIC RED CELL TRANSIT TIME (MST) IN SICKLE CELL DISEASE AND SICKLE CELL THALASSAEMIA ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644215.
Texte intégralKuroda, H., M. Iribe, M. Matsubara, M. Watanabe et T. Sanada. « Image Analysis of Human Nailfold Capillary With High Speed Digital Video Capillaroscopy ». Dans ASME/JSME 2007 5th Joint Fluids Engineering Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/fedsm2007-37464.
Texte intégralHabib, GS, W. Saliba et P. Froom. « FRI0129 Lupus and pure red cell aplasia ». Dans Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.164.
Texte intégralGalpayage Dona, Kalpani Nisansala Udeni, Jia Liu, Yuhao Qiang, E. Du et A. W. C. Lau. « Electrical Equivalent Circuit Model of Sickle Cell ». Dans ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-70677.
Texte intégralShaked, Natan T., Lisa L. Satterwhite, Marilyn J. Telen, George A. Truskey et Adam Wax. « Dynamic quantitative microscopy and nanoscopy of red blood cells in sickle cell disease ». Dans SPIE BiOS, sous la direction de Jose-Angel Conchello, Carol J. Cogswell, Tony Wilson et Thomas G. Brown. SPIE, 2012. http://dx.doi.org/10.1117/12.907659.
Texte intégralErjavec, Vladimira, et Alenka Nemec Svete. « Brachycephalic Dogs with Brachycephalic Obstructive Airway Syndrome Have Increased Variability in Red Blood Cell Size ». Dans Socratic Lectures 7. University of Lubljana Press, 2022. http://dx.doi.org/10.55295/psl.2022.d8.
Texte intégralCHEN, YONG, JIYE CAI et JINGXIAN ZHAO. « DISEASED RED BLOOD CELLS STUDIED BY ATOMIC FORCE MICROSCOPY ». Dans Proceedings of the Asian Symposium on Nanotechnology and Nanoscience 2002. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812796714_0054.
Texte intégralFarhat, Hassan, et Joon Sang Lee. « The Study of RBC Deformation in Capillaries With a Lattice Boltzmann Method for Surfactant Covered Droplets ». Dans ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-12629.
Texte intégralDintenfass, L. « VERY LARGE AND COMPACT AGGREGATES OF RED CELLS IN HEART DISEASE AND CANCER : POSSIBLY AN ANALOGOUS ROLE IN THE MICROCIRCULATION AS PLATELET AGGREGATES ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644213.
Texte intégralRapports d'organisations sur le sujet "Diseased red cell"
Banks, H. T., Karen M. Bliss et Hien Tran. Modeling Red Blood Cell and Iron Dynamics in Patients with Chronic Kidney Disease. Fort Belvoir, VA : Defense Technical Information Center, février 2012. http://dx.doi.org/10.21236/ada556965.
Texte intégralDavidson, Irit, Hsing-Jien Kung et Richard L. Witter. Molecular Interactions between Herpes and Retroviruses in Dually Infected Chickens and Turkeys. United States Department of Agriculture, janvier 2002. http://dx.doi.org/10.32747/2002.7575275.bard.
Texte intégralFicht, Thomas, Gary Splitter, Menachem Banai et Menachem Davidson. Characterization of B. Melinensis REV 1 Attenuated Mutants. United States Department of Agriculture, décembre 2000. http://dx.doi.org/10.32747/2000.7580667.bard.
Texte intégralHorwitz, Benjamin, et Nicole M. Donofrio. Identifying unique and overlapping roles of reactive oxygen species in rice blast and Southern corn leaf blight. United States Department of Agriculture, janvier 2017. http://dx.doi.org/10.32747/2017.7604290.bard.
Texte intégralHovav, Ran, Peggy Ozias-Akins et Scott A. Jackson. The genetics of pod-filling in peanut under water-limiting conditions. United States Department of Agriculture, janvier 2012. http://dx.doi.org/10.32747/2012.7597923.bard.
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