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Auteur : Grafiati
Publié le 10 mars 2023

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1

Cristini, Carlo, et Amelia Belloni Sonzogni. « Psicopatologia e invecchiamento nell'opera di Marcello Cesa-Bianchi ». RICERCHE DI PSICOLOGIA, no 1 (mai 2021) : 191–215. http://dx.doi.org/10.3280/rip1-2021oa11626.

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In età senile, le più frequenti condizioni che implicano una sofferenza psichica e che acquisiscono spesso connotazioni psicopatologiche riguardano il disadattamento, le ansie e le paure, la depressione e il declino cognitivo. Sono ambiti esistenziali e clinici che il Professor Marcello Cesa-Bianchi ha più volte trattato nella sua lunga esperienza scientifica. Fra i suoi primi lavori si annovera in particolare il disadattamento dell'anziano rilevato e analizzato in diversi contesti esperienziali.Se ansie, paure e depressione – oltre al disadattamento – si registrano anche in altri periodi della vita, in vecchiaia vengono ad assumere peculiari caratteristiche.Inoltre il processo di invecchiamento comporta il rischio della comparsa di una demenza, nelle sue varie forme. Il declino cognitivo – senza naturalmente ignorare le sue manifestazioni cliniche e la sua ancora incognita eziologia – viene considerato nella sua multidimensionalità e ipercomplessità evidenziando soprattutto le dinamiche affettive, relazionali e creative.
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Cuadrao Zavalela, Luis. « Envejecimiento Celular - Alzheimer ». Odontología Sanmarquina 8, no 1 (14 mai 2014) : 39. http://dx.doi.org/10.15381/os.v8i1.3136.

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Históricamente, El término enfermedad de Alzheimer se usó si el padecimiento empezaba antes de los 65 años de edad, y demencia senil si lo hacía después. No obstante en la actualidad, enfermedad de Alzheimer se refiere a demencia relacionada con los cambios anatomopatológicos característicos independiente de la edad de principio .EI examen macroscópico del cerebro muestra ensanchamiento de los surcos cerebrales, por lo general mas pronunciado en las regiones frontal y temporal. las características microscópicas importantes de la enfermedad de alzheimer son masas neurofibrilares , placas seniles, degeneración granulovacuolar y cuerpos de Hirano.
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Ohtomo, E. « Senile dementia. » Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 30, no 1 (1993) : 16–22. http://dx.doi.org/10.3143/geriatrics.30.16.

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&NA;. « Senile Dementia ». Journal of Clinical Psychopharmacology 5, no 1 (février 1985) : 62. http://dx.doi.org/10.1097/00004714-198502000-00034.

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Reisberg, Barry. « Senile Dementia ». Alzheimer Disease & ; Associated Disorders 1, no 1 (1987) : 63. http://dx.doi.org/10.1097/00002093-198701000-00064.

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Ball, Melvyn J. « Senile Dementia ». Journal of Neuropathology and Experimental Neurology 44, no 2 (mars 1985) : 218–19. http://dx.doi.org/10.1097/00005072-198503000-00014.

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Liu, Xin, DeRen Hou, FangBo Lin, Jing Luo, JingWen Xie, Yan Wang et Yi Tian. « The role of neurovascular unit damage in the occurrence and development of Alzheimer’s disease ». Reviews in the Neurosciences 30, no 5 (26 juillet 2019) : 477–84. http://dx.doi.org/10.1515/revneuro-2018-0056.

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Abstract Alzheimer’s disease (AD) is a neurodegenerative disease with progressive cognitive impairment. It is the most common type of senile dementia, accounting for 65%–70% of senile dementia [Alzheimer’s Association (2016). 2016 Alzheimer’s disease facts and figures. Alzheimers Dement. 12, 459–509]. At present, the pathogenesis of AD is still unclear. It is considered that β-amyloid deposition, abnormal phosphorylation of tau protein, and neurofibrillary tangles are the basic pathological changes of AD. However, the role of neurovascular unit damage in the pathogenesis of AD has been attracting more and more attention in recent years. The composition of neurovascular unit and the role of neurovascular unit damage in the occurrence and development of AD were reviewed in this paper.
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8

Tanaka, Atsushi, Hideyuki Okuzumi, Iwao Kobayashi, Norio Murai, Kenichi Meguro et Takashi Nakamura. « Gait Disturbance of Patients with Vascular and Alzheimer-Type Dementias ». Perceptual and Motor Skills 80, no 3 (juin 1995) : 735–38. http://dx.doi.org/10.2466/pms.1995.80.3.735.

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The gaits of 15 patients with senile dementia of the Alzheimer type and 15 with vascular dementia were compared with those of 15 healthy control subjects. Patients with senile dementia showed significantly slower velocity and shorter step length than the healthy controls, and those with vascular dementia exhibited a reduction on these two variables relative to patients with senile dementia of the Alzheimer type.
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Cacabelos, Ramón, Hisayoshi Niigawa, Yoshiaki Ikemura, Yuji Yanagi, Shigemi Tanaka, María D. Rodríguez-Arnao, Antonio Gómez-Pan et Tsuyoshi Nishimura. « GHRH-induced GH response in patients with senile dementia of the Alzheimer type ». Acta Endocrinologica 117, no 3 (mars 1988) : 295–301. http://dx.doi.org/10.1530/acta.0.1170295.

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Abstract. To clarify the functional state of the somatotropinergic system at the hypothalamo-hypophyseal level in senile dementia of the Alzheimer type, the GHRH test was performed in three groups of subjects: a) healthy elderly subjects; b) early onset senile dementia patients; and c) late onset senile dementia patients. Intravenous administration of GHRH(1–44)NH2 (100 μg) elicited a marked plasma GH response with a maximum peak (709.54 ± 259.0 pmol/l; P < 0.005) 60 min after injection in patients with early onset senile dementia, but no significant response was detected in the other two groups. Electroencephalographic recording showed that GHRH modifies brain bioelectrical activity, decreasing frequency (0.52 ± 0.15 Hz) and increasing amplitude (8.25 ± 4.5 μV) of the electroencephalogram basic rhythm. The evaluation of mental performance and behaviour with a battery of different tests for mental assessment revealed that GHRH induces transient clinical changes in psychomotor behaviour. According to these results, it seems likely that the somatostatin deficiency reported in senile dementia of the Alzheimer type may account for the enhanced GHRH-induced GH response observed in patients with early onset senile dementia. In consequence, the GHRH test might constitute a useful antemortem marker for senile dementia of the Alzheimer type if the present results can be replicated in early stages of the disease.
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Heinemann, Uta, Joanna Gawinecka, Christian Schmidt et Inga Zerr. « Differential Diagnosis of Rapid Progressive Dementia ». European Neurological Review 5, no 2 (2010) : 21. http://dx.doi.org/10.17925/enr.2010.05.02.21.

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There is a broad range of diseases underlying dementia, of which Alzheimer’s disease is the most frequent in senile and pre-senile dementia. While senile dementia is predominantly caused by neurodegenerative or vascular disorders, in early-onset dementia other conditions are more relevant. Autoimmune, metabolic and genetic reasons should be evaluated, as well as toxic causes. A list of mutations associated with dementia is provided in this article. A higher proportion of potentially reversible conditions in pre-senile dementia highlights the value of detailed evaluation. Lumbar puncture is important in the diagnostic process to detect inflammatory changes, but dementia markers such as Aβ1–42 are also helpful in differential diagnosis. The value of cerebrospinal fluid markers for differential diagnosis is discussed in this article.
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Philpot, Michael P. « Understanding Senile Dementia ». Behaviour Research and Therapy 26, no 4 (1988) : 364. http://dx.doi.org/10.1016/0005-7967(88)90101-5.

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Jellinger, Kurt A., et Christian Bancher. « Senile Dementia with Tangles (Tangle Predominant Form of Senile Dementia) ». Brain Pathology 8, no 2 (5 avril 2006) : 367–76. http://dx.doi.org/10.1111/j.1750-3639.1998.tb00160.x.

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NAKAMURA, SHIGENOBU. « Senile Dementia and Presenile Dementia ». Tohoku Journal of Experimental Medicine 161, Supplement (1990) : 49–60. http://dx.doi.org/10.1620/tjem.161.supplement_49.

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14

Breitner, John C. S. « What should we do if we were wrong and Alzheimer was right ? » International Psychogeriatrics 26, no 1 (18 octobre 2013) : 3–6. http://dx.doi.org/10.1017/s1041610213001749.

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For more than 2,000 years cognitive decline and dementia were considered a part of aging, like graying of hair, wrinkling of skin, or (more recently) reduction of pulmonary capacity or glomerular filtration rate. Thus, our recent predecessors considered “senile dementia” a normal consequence of the aging process. Their confidence increased after Martin Roth and colleagues added specificity to the concept by differentiating it within the broader category of “senile psychosis” (which, consequently, became obsolete). A diagnosis of “Alzheimer's pre-senile dementia,” by contrast, was reserved for people with earlier onset of dementia.
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15

Kay, David W. K., Donald P. Forster et Andrew J. Newens. « Long-term survival, place of death, and death certification in clinically diagnosed pre-senile dementia in northern England ». British Journal of Psychiatry 177, no 2 (août 2000) : 156–62. http://dx.doi.org/10.1192/bjp.177.2.156.

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BackgroundInformation on survival and cause of death in pre-senile dementia is scarce and the organisation of services controversial.AimsTo study survival, place of death and death certification in pre-senile dementia.MethodPatients aged 45–64 were identified from hospital and community sources in the Northern health region (1985–89) and classified as having pre-senile dementia of Alzheimer type (PDAT) or pre-senile vascular dementia (PVD) by applying an algorithm to case notes. Deaths were ascertained from the National Health Service Central Registry (NHSCR) to 31 December 1998. Survival analysis was performed using the SPSS/PC program, and expected survival calculated from life tables.ResultsMedian survival time from diagnosis was 6.08 years and did not differ significantly in PDAT and PVD, or by age or gender; 19.3% of deaths occurred at home, 24.5% in nursing or residential homes and 56.3% in hospital; 72.4% of the death certificates mentioned dementia or Alzheimer's disease; 15.4% were still alive.ConclusionsPre-senile dementia has a variable but usually chronic course, requiring appropriate planning and services.
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Okuzumi, Hideyuki, Atsushi Tanaka, Kouichi Haishi, Ken-Ichi Meguro, Hideki Yamazaki, Iwao Kobayashi et Takashi Nakamura. « Characteristics of Postural Control and Locomotion of Patients with Vascular and Alzheimer-Type Dementias ». Perceptual and Motor Skills 84, no 1 (février 1997) : 16–18. http://dx.doi.org/10.2466/pms.1997.84.1.16.

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The postural control and locomotion of patients with Alzheimer-type senile dementia ( n = 20) or vascular dementia ( n = 20) were investigated. Patients with both types of senile dementia, especially patients with vascular dementia, generally showed lower scores than healthy old people ( n = 53). Moreover, deterioration of locomotion was more remarkable.
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Wallesch, C. W. « Alzheimer-Krankheit und senile Demenz ». Fortschritte der Neurologie · Psychiatrie 79, no 03 (mars 2011) : 137. http://dx.doi.org/10.1055/s-0029-1246086.

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18

IGATA, AKIHIRO. « Problems on senile dementia. » Nihon Naika Gakkai Zasshi 84, no 3 (1995) : 424–27. http://dx.doi.org/10.2169/naika.84.424.

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McLoughlin, I. J. « Zinc in Senile Dementia ». British Journal of Psychiatry 150, no 3 (mars 1987) : 409. http://dx.doi.org/10.1192/s0007125000106191.

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20

Passeri, Mario, Domenico Cucinotta, Manuel De Mello, Guido Storchi, Romeo Roncucci et Kathleen Bizière. « MINAPRINE FOR SENILE DEMENTIA ». Lancet 325, no 8432 (avril 1985) : 824. http://dx.doi.org/10.1016/s0140-6736(85)91488-6.

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Fox, P. J. « Alzheimer's and senile dementia ». Neurology 37, no 8 (1 août 1987) : 1435. http://dx.doi.org/10.1212/wnl.37.8.1435.

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Amaducci, L. A., W. A. Rocca et B. S. Schoenberg. « Alzheimer's and senile dementia ». Neurology 37, no 8 (1 août 1987) : 1435. http://dx.doi.org/10.1212/wnl.37.8.1435-a.

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Kenn, Chris, et Elizabeth Gibb. « Zinc in Senile Dementia ». British Journal of Psychiatry 150, no 6 (juin 1987) : 875. http://dx.doi.org/10.1192/bjp.150.6.875b.

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O'Connor, Thomas St James. « Ministry without a Future : A Pastoral Care Approach to Patients with Senile Dementia ». Journal of Pastoral Care 46, no 1 (mars 1992) : 5–12. http://dx.doi.org/10.1177/002234099204600103.

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Describes the nature of senile dementia and discusses how different ways of thinking, relating, and using traditional religious symbols are needed by the pastoral caregiver in order to offer effective pastoral care to these patients. Focuses on two specific cases to illustrate the author's clinical experiences with senile dementia patients.
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Mallet, Louise, et Jirair Kuyumjian. « Indomethacin-Induced Behavioral Changes in an Elderly Patient with Dementia ». Annals of Pharmacotherapy 32, no 2 (février 1998) : 201–3. http://dx.doi.org/10.1345/aph.17203.

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OBJECTIVE: To describe a case of behavioral changes induced by indomethacin in an elderly man with a history of senile dementia of the Alzheimer type. CASE SUMMARY: A 92-year-old white man with a history of senile dementia of the Alzheimer type, glaucoma, and constipation was treated with indomethacin for an episode of pseudogout. After six doses of indomethacin 25 mg, the patient became very agitated, confused, and was physically and verbally aggressive to the nurses. Indomethacin was discontinued and the patient recovered after receiving haloperidol 0.5 mg/d po over a period of 10 days. DISCUSSION: Only a few cases of psychosis have been reported in the literature concerning the use of indomethacin in patients over the age of 60 years. The mechanism by which indomethacin causes psychosis is not well understood, but it is postulated that the acute psychosis may be related to the similarity between the molecular structure of indomethacin and serotonin. Both indomethacin and serotonin have an indolic moiety, which may explain the development of acute psychosis in this patient. CONCLUSIONS: Healthcare providers should be aware that patients with a history of dementia who are receiving indomethacin may be at risk of developing severe behavior problems along with gastrointestinal and renal adverse effects. OBJETIVO: Informar un caso de cambios en conducta inducidos por indometacina en un envejeciente varón con historial de demencia senil del tipo Alzheimer. RESUMEN DEL CASO: Un hombre blanco de 92 años con historial de demencia senil del tipo Alzheimer, glaucoma, y constipación fue tratado con indometacina para un episodio de pseudogota. Después de seis dosis de indometacina de 25 mg, el paciente se puso bien agitado, confuso, y física y verbalmente agresivo con las enfermeras. Indometacina fue descontinuada y el paciente se recuperó con una dosis de haloperidol 0.5 mg oral diario por un período de 10 días. DISCUSIÓN: Solo algunos casos de psicosis han sido reportados en la literatura con el uso de indometacina en pacientes sobre 60 años. El mecanismo por el cual indometacina produce la psicosis no es bien comprendido pero se cree que la psicosis aguda puede estar relacionada a la similaridad de la estructura molecular de indometacina y serotonina. Tanto la indometacina y serotonina tienen un componente indólico que podría explicar el desarrollo de psicosis aguda en éste paciente. CONCLUSIONES: Los proveedores de cuidado de la salud deben estar concientes que los pacientes con historial de demencia que están recibiendo indometacina podrían desarrollar severos problemas de conducta asi como efectos secundarios gastrointestinal y renal. OBJECTIF: Décrire le cas d'une personne âgée présentant avec des troubles de comportement suite à la prise d'indométhacine. RÉSUMÉ DU CAS: Un homme blanc de 92 ans présentant avec une histoire de démence de type Alzheimer, de glaucome, et de constipation a été traité avec de l'indométhacine 25 mg trois fois par jour pour un épisode de pseudogoutte. Après 2 jours de traitement avec l'indométhacine, à raison de 25 mg trois fois par jour, le patient a présenté avec de l'agitation et de la confusion. Il est devenu agressif physiquement et verbalement envers le personnel infirmier. L'indométhacine a été discontinué et le patient a reçu de haloperidol 0.5 mg chaque jour pour une période de 10 jours. DISCUSSION: Seulement quelques cas de psychose induite par l'indométhacine ont été rapportés dans la littérature chez des patients de plus de 60 ans. Le mécanisme d'action par lequel la psychose se manifeste n'est pas encore bien élucidé. Il a été postulé que la psychose serait associée à la similarité dans les structures chimiques de lindométhacine et la sérotonine. La molécule de l'indométhacine et celle de la sérotonine possèdent un groupe indolique, ce qui pourrait expliquer le développement d'une psychose aiguë. CONCLUSIONS: Il est important de surveiller les changements de comportement chez les patients souffrant de démence lorsque l'indométhacine est prescrit.
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Katzman, Robert. « A neurologist's view of Alzheimer's disease and dementia ». International Psychogeriatrics 16, no 3 (septembre 2004) : 259–73. http://dx.doi.org/10.1017/s1041610204000456.

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Senile dementia was the third most common admission diagnosis for New York psychiatric hospitals at the start of the twentieth century and the distinction between vascular and senile dementia was understood by psychiatrists even then. The term Alzheimer's disease (AD) was originally introduced to distinguish a pre-senile dementia from the common general paresis, but Alzheimer raised the possibility that pre-senile AD might not be distinguishable in clinical or histological terms from senile dementia. By the late 1970s it had become clear that the most common disorder producing dementia in elderly people was clinically and pathologically identical to pre-senile AD. AD is malignant, reducing remaining life expectancy by almost half and raising the risk of death over five years threefold (cancer raises it fourfold). Synapse loss associated with beta amyloid oligomers is a strong determinant of cognitive decline in patients with AD. Tau-containing neurofibrillary tangles usefully track disease severity. Unmodifiable risk factors include mutations in three genes which affect the production or metabolism of beta amyloid, the risk factor gene for Apolipoprotein $\epsilon$4 and female gender. The overriding risk factor is age, the prevalence of AD doubling with every five years of age until 90. Low education, head injury and low folate levels are examples of potentially modifiable risk factors. Since a delay of onset of five years would halve the number of patients with the disease, clinical trials for such putative protective factors as estrogens, folic acid, vitamin E, statins, and NSAIDs have begun. Cognitive and leisure activity may be protective against the development of AD but any protective function can only be confirmed by clinical trials.
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Probst, Botez et Tolnay. « Neuropathology of Alzheimer’s disease ». Therapeutische Umschau 56, no 2 (1 février 1999) : 88–93. http://dx.doi.org/10.1024/0040-5930.56.2.88.

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Ziel dieser Übersichtsarbeit ist eine Schilderung der Morphologie und eine Darstellung einzelner pathogenetischer Faktoren der Alzheimer Krankheit (AK). Die Übersicht beinhaltet weiter eine Beschreibung der Silberkornkrankheit, einer häufigen, mit der AK verwandten, jedoch noch weitgehend verkannten Demenzursache des hohen Alters. Makroskopisch zeigt das Gehirn eines an AK Verstorbenen vor allem eine Atrophie des medialen Temporallappens, wobei die Hippocampi, die Entorhinalrinde und die Mandelkerne besonders stark betroffen sind. Histologische Merkmale der AK sind: ein regionalspezifischer Neuronenverlust, eine Verminderung der kortikalen Synapsendichte sowie intra- und extrazelluläre Ablagerungen abnormer Proteine. Die intraneuronale Ansammlung abnormer fibrillärer Strukturen, die vor allem auf die Hyperphosphorylierung des Zytoskelett-assoziierten Proteins Tau zurückzuführen ist, beeinträchtigt die Funktion der betroffenen Nervenzellen und ist für deren Untergang mitverantwortlich. Die Ausbreitung dieser Veränderungen über den Hippocampus hinaus auf den gesamten Neokortex führt zur Alzheimer-Demenz. Die extrazelluläre Ablagerung von Abeta-Amyloid im Neuropil (senile Plaques) ist bei der AK besonders stark ausgeprägt. Zwischen der Gesamtzahl kortikaler Plaques und klinischen Parametern der Demenz besteht offenbar keine klare Korrelation. Eine solche findet sich jedoch bezüglich des Anteils neuritischer Plaques, d.h. des Ausmaßes an neuritischer Degeneration innerhalb seniler Plaques. Die Übersicht beinhaltet weiter eine kurze Schilderung der kongophilen Angiopathie und der Alzheimer-assoziierten Veränderungen der cholinergischen Innervation des Gehirns. Schließlich werden die genetischen Risikofaktoren der AK diskutiert.
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Jorm, A. F. « Controlled and automatic information processing in senile dementia : a review ». Psychological Medicine 16, no 1 (février 1986) : 77–88. http://dx.doi.org/10.1017/s0033291700002567.

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SynopsisThe evidence on cognitive decline in senile dementia is reviewed with respect to the distinction between controlled and automatic processing. It is argued that controlled processing, which requires the attentional resources of the individual, declines in early senile dementia. However, automatic processing, which does not require attentional resources, is well maintained until late in the disorder. Implications of this view are drawn for the development of effective screening instruments which can be used for the early detection of dementia in the general population.
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SASANUMA, SUMIKO. « Differentiation between aphasia and dementia (senile dementia). » Japan Journal of Logopedics and Phoniatrics 27, no 4 (1986) : 311–13. http://dx.doi.org/10.5112/jjlp.27.311.

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Gorzoni, Milton Luiz, Renato Moraes Alves Fabbri et Sueli Luciano Pires. « Senile dementia and pharmacological drugs ». Dementia & ; Neuropsychologia 7, no 2 (juin 2013) : 176–80. http://dx.doi.org/10.1590/s1980-57642013dn70200007.

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ABSTRACT Side effects and drug interactions are common in the elderly and highly relevant in the demented, being routinely confused with symptoms of cognitive impairment. Which drugs are most consumed by this patient group? Do prescription patterns differ between the demented and non-demented? Objective: To define drug consumption quantitatively and qualitatively in demented (D) and non-demented (ND) elderly. Methods: Patients were divided into men and women, by age group (<80 and ≥80 years), non-demented and demented status, and consumers of ≤3 or >3 drugs. As a criterion comparing groups, the Chi-square (Fisher's exact) test was employed. This study is part of Project No. 405/10 approved by the Ethics Committee of the institution. Results: The sample had a mean age of 81.5±8.8 years, 29 D (21 women and 8 men) and 21 ND (16 women and 5 men), 12 consumers of up to three drugs (7 D and 5 ND) and 38 consumers of 3 medications or more (22 D and 16 ND). The most used drugs among dementia patients were aspirin, angiotensin-converting enzyme inhibitors, statins, selective serotonin reuptake inhibitors, and vitamins. Drugs most consumed by non-demented included vitamins, aspirin, calcium carbonate, proton pump inhibitors, statins and alendronate sodium. There was no statistical significance on any of the comparisons, although the number of elderly consumers of vitamins in the ND had a p-value of 0.06 (Yates). Conclusion: The elderly in this series, regardless of dementia status, gender or age group, had similar drug consumption patterns and used multiple drugs simultaneously.
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Ambani, Bandish. « A Case of Senile Dementia ». Homoeopathic Links 19, no 1 (2006) : 42–44. http://dx.doi.org/10.1055/s-2006-923818.

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KRINGLEN, E., et M. BERGEM. « TWIN STUDY OF SENILE DEMENTIA ». Alzheimer Disease & ; Associated Disorders 2, no 4 (1988) : 398. http://dx.doi.org/10.1097/00002093-198802040-00028.

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Rosin, A. J., L. Abramowitz, J. Diamond et P. Jesselson. « Environmental Management of Senile Dementia ». Social Work in Health Care 11, no 1 (12 décembre 1985) : 33–43. http://dx.doi.org/10.1300/j010v11n01_03.

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McConathy, Walter, Andras Lacko et Janos Kalman. « Senile Dementia and Apolipoprotein E4 ». Dementia and Geriatric Cognitive Disorders 8, no 4 (1997) : 258. http://dx.doi.org/10.1159/000106641.

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Howell, PaulR. « MENINGIOMA MISDIAGNOSED AS SENILE DEMENTIA ». Lancet 331, no 8586 (mars 1988) : 657. http://dx.doi.org/10.1016/s0140-6736(88)91468-7.

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Ferrari, E., L. Cravello et F. Magri. « Eating behaviour in senile dementia ». Experimental Gerontology 42, no 1-2 (janvier 2007) : 141. http://dx.doi.org/10.1016/j.exger.2006.06.014.

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Biringer, Francois, James R. Anderson et Denise Strubel. « Self-recognition in senile dementia ». Experimental Aging Research 14, no 4 (décembre 1988) : 177–80. http://dx.doi.org/10.1080/03610738808259745.

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Orrell, Martin, et Paul Bebbington. « Life Events and Senile Dementia Affective Symptoms ». British Journal of Psychiatry 166, no 5 (mai 1995) : 613–20. http://dx.doi.org/10.1192/bjp.166.5.613.

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BackgroundPrevious research in the elderly has linked threatening life events with depression. Dementia sufferers are known to be sensitive to stressful changes in their daily life such as relocation. This study investigates whether threatening life events are associated with depressive symptoms in dementia sufferers.MethodUsing the Life Events and Difficulties Schedule, this study examined life events before admission in a group of 70 dementia patients compared with two control groups: dementia sufferers in the community and mentally fit elderly people matched for age and sex.ResultsLife events with severe threat were not significantly more frequent in the dementia patients than in two control groups. However, depressive symptoms in the dementia sufferers were significantly associated with independent severe life events. This strong association was maintained when a multivariate analysis was used to control for the effects of other social factors and severity of cognitive impairment. This association appears to be specific to the threat aspect of life events since there was no association between depressive symptoms and events relating to change in the social environment.ConclusionIn dementia sufferers, threatening life events are associated with depressive symptoms. This means that dementia sufferers are responsive to stress in the same way as cognitively intact individuals, and clinicians need to be more aware of the social influences on them.
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Wiegersma, Sjoerd, Emmy Van Dongen et Hans Lomans. « Errors of Free Recall in Senile Dementia ». Perceptual and Motor Skills 63, no 2 (octobre 1986) : 617–18. http://dx.doi.org/10.2466/pms.1986.63.2.617.

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Errors made in the free recall of nouns by patients with senile dementia were classified as to their paradigmatic, phonological, or syntagmatic relationships with presented words. Dementia patients at four levels of dementia severity made relatively more paradigmatic errors than normals. Syntagmatic errors only occurred for greater severity. Such errors show the relative preservation in dementia of semantic knowledge about words as well as the decreased paradigmatic and syntagmatic discriminability of words.
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Zhang, Jianrong, Yu’e Wu, Yanfang Huang, Shuqing Zhang, Liuqin Xu, Xiaoyun Huang, Xingshui Wang et Qingping Huang. « Effect of the Mendelsohn maneuver and swallowing training in patients with senile vascular dementia complicated with dysphagia ». Journal of International Medical Research 49, no 5 (mai 2021) : 030006052110131. http://dx.doi.org/10.1177/03000605211013198.

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Objective We investigated the effect of the Mendelsohn maneuver and swallowing training in patients with senile vascular dementia complicated with dysphagia. Methods We randomly classified 214 patients with senile vascular dementia and swallowing dysfunction into a control group (CG, n = 106) and observation group (OG, n = 108). Both groups underwent health education, psychological intervention, and training of the oral muscle group. The OG additionally underwent the Mendelsohn maneuver and swallowing training. The Hasegawa Dementia Scale (HDS), China Stroke Scale (CSS), and Neurobehavioral Cognitive Status Examination (NCSE) were used to evaluate dementia, neurological impairment, and cognitive dysfunction, respectively. Results The OG had a higher rate of effective therapy than the CG. After intervention, the OG showed better swallowing function than the CG. At 15 days and 1 month after intervention, the OG had higher video fluoroscopic swallowing exam scores than the CG. The OG had lower serum interleukin (IL)-1, IL-6, and tumor necrosis factor-α levels than the CG. After intervention, the OG had higher HDS and NCSE scores and lower CSS scores than the CG. Conclusions The Mendelsohn maneuver and swallowing training can improve swallowing function in patients with senile vascular dementia complicated with dysphagia and help to ameliorate the inflammatory response.
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Sun, Wei, Jinxia Liu, Lihua Liu et Xiuzhi Wang. « Management Strategy of Alzheimer’s Patients under the Medical-Care Integration Model Based on Big Data Evaluation ». BioMed Research International 2022 (11 octobre 2022) : 1–11. http://dx.doi.org/10.1155/2022/9896935.

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In the context of the era of big data, the management of Alzheimer’s patients has aroused widespread concern in the society. What should the elderly and people with dementia do and how the society should accommodate these special groups have aroused heated discussions in the society. The goal of the medical-nursing integration model is to realize the integrated model of medical care-nursing-rehabilitation-old care, and to better satisfy the needs by providing medical services, rehabilitation care, and health management. The medical needs of the elderly can help the elderly maintain a healthy state; the elderly care needs can be better met through elderly care services, and the burden on families and society can be reduced. With the advancement of the medical-care integration model, new solutions have been provided for the management of senile dementia patients. Therefore, under the medical-care integration model, this paper managed Alzheimer’s patients based on the big data algorithm based on association rules and compared it with the management of Alzheimer’s patients under the traditional model, and drew the following conclusions: compared with the management satisfaction of Alzheimer’s patients under the traditional model, family members and patients’ total satisfaction with nursing management under the medical-care integration model was greatly improved; compared with the management of Alzheimer’s patients under the traditional model, the incidence of adverse events in the management of Alzheimer’s patients under the medical-care integration model was greatly reduced, which was reduced to 17.6%; under the medical-care integration model, the anxiety and depression scores of the elderly patients with dementia decreased rapidly; under the medical-care integration model, the living ability of senile dementia patients has been greatly improved; the use of the medical-care integration model to manage senile dementia patients has greatly improved the sleep quality, mental quality, and quality of life of senile dementia patients.
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Engelhardt, Eliasz, et Lea T. Grinberg. « Alzheimer and vascular brain disease : Senile dementia ». Dementia & ; Neuropsychologia 9, no 2 (juin 2015) : 184–88. http://dx.doi.org/10.1590/1980-57642015dn92000013.

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Alois Alzheimer is best known for his description of a novel disease, subsequently named after him. However, his wide range of interests also included vascular brain diseases. He described Senile dementia, a highly heterogeneous condition, and was able not only to distinguish it from syphilitic brain disease, but also to discriminate two clinicopathological subtypes, that may be labeled a "arteriosclerotic subtype", comparable to the present clinicopathological continuum of "Vascular cognitive impairment", and another as a "neurodegenerative subtype", characterized by primary [cortical] ganglion cell [nerve cells] degeneration, possibly foreshadowing a peculiar presenile disease that he was to describe some years later and would carry his name. He also considered the possibility of a senile presentation of this disease subtype, which was described by Oskar Fischer a short time later. Considering the clinicopathological overlapping features of the "arteriosclerotic subtype" of Senile dementia with Arteriosclerotic atrophy of the brain, it might be possible to consider that both represent a single condition.
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Zheng, Wenxin, Xinyao Sun et Jianghua Liu. « Efficacy of Donepezil Hydrochloride plus Olanzapine for Senile Dementia and Its Effect on the Recovery of Cognitive Function ». Evidence-Based Complementary and Alternative Medicine 2022 (11 août 2022) : 1–5. http://dx.doi.org/10.1155/2022/4156312.

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Objective. To investigate the efficacy of donepezil hydrochloride plus olanzapine for senile dementia and its effect on the recovery of cognitive function. Methods. A total of 60 patients with senile dementia admitted to our hospital from April 2020 to July 2021 were recruited and assigned to receive either olanzapine alone (observation group) or donepezil hydrochloride plus olanzapine (experimental group) via the random number table method, with 30 patients in each group. Results. The combined therapy resulted in significantly higher clinical efficacy versus monotherapy of olanzapine ( P < 0.05 ). Before treatment, the difference in the scores of cognitive function between the two groups did not come up to the statistical standard ( P > 0.05 ). Donepezil hydrochloride plus olanzapine was associated with significantly higher scores of cognitive function in patients versus olanzapine alone ( P < 0.05 ). The two groups had a similar incidence of adverse reactions ( P > 0.05 ). Conclusion. Donepezil hydrochloride plus olanzapine substantially enhances the recovery of cognitive function of patients with senile dementia and features a manageable safety. Further trials are, however, required prior to clinical promotion.
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Engelhardt, Eliasz, et Marleide da Mota Gomes. « Alzheimer's 100th anniversary of death and his contribution to a better understanding of Senile dementia ». Arquivos de Neuro-Psiquiatria 73, no 2 (février 2015) : 159–62. http://dx.doi.org/10.1590/0004-282x20140207.

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Initially the trajectory of the historical forerunners and conceptions of senile dementia are briefly presented, being highlighted the name of Alois Alzheimer who provided clinical and neuropathological indicators to differentiate a group of patients with Senile dementia. Alzheimer's examination of Auguste D’s case, studied by him with Bielschowsky’s silver impregnation technique, permitted to identify a pathological marker, the intraneuronal neurofibrillary tangles, characterizing a new disease later named after him by Kraepelin – Alzheimer’s disease. Over the time this disorder became one of the most important degenerative dementing disease, reaching nowadays a status that may be considered as epidemic.
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Arai, Hiroyuki. « Genetic Risk Factors in Senile Dementia. » Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 36, no 4 (1999) : 251–55. http://dx.doi.org/10.3143/geriatrics.36.251.

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Poggenpoel, Marie. « A new publication on Senile Demetia ». Health SA Gesondheid 1, no 2 (1 décembre 1996) : 32. http://dx.doi.org/10.4102/hsag.v1i2.310.

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A basic guide for nurses and caregivers on the care and management of Alzheimer's Disease by Janice Mymin. *Please note: This is a reduced version of the abstract. Please refer to PDF for full text.
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KANATA, Suzue, Tetsuji KADOTANI, Yoko WATANABE, Noriko KUROSAKI et Hidetoshi KODAMA. « A chromosomal aberrations on senile dementia. » Proceedings of the Japan Academy. Ser. B : Physical and Biological Sciences 65, no 5 (1989) : 91–94. http://dx.doi.org/10.2183/pjab.65.91.

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TSUJI, Shoji. « Diagnosis and Management of Senile Dementia. » JOURNAL OF THE JAPANESE ASSOCIATION OF RURAL MEDICINE 44, no 6 (1996) : 790–94. http://dx.doi.org/10.2185/jjrm.44.790.

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KANEKO, Mitsuo. « Senile Dementia is Preventable and Treatable. » JOURNAL OF THE JAPANESE ASSOCIATION OF RURAL MEDICINE 47, no 6 (1999) : 813–18. http://dx.doi.org/10.2185/jjrm.47.813.

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KENNEDY, JOHN S. « Senile Dementia of the Alzheimer Type ». Journal of Clinical Psychopharmacology 6, no 3 (juin 1986) : 198. http://dx.doi.org/10.1097/00004714-198606000-00026.

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