Bibliographies thématiques / Dementia, metabolic imaging, neuropsychology

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Littérature scientifique sur le sujet « Dementia, metabolic imaging, neuropsychology »

Auteur : Grafiati

Publié le 9 mars 2023

Mis à jour le 31 juillet 2025

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Articles de revues sur le sujet "Dementia, metabolic imaging, neuropsychology"

Pihlajamäki, Maija, and Reisa A. Sperling. "Functional MRI Assessment of Task-Induced Deactivation of the Default Mode Network in Alzheimer’s Disease and At-Risk Older Individuals." Behavioural Neurology 21, no. 1-2 (2009): 77–91. http://dx.doi.org/10.1155/2009/276384.

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Alzheimer’s disease (AD) is the most common form of dementia in old age, and is characterized by prominent impairment of episodic memory. Recent functional imaging studies in AD have demonstrated alterations in a distributed network of brain regions supporting memory function, including regions of the default mode network. Previous positron emission tomography studies of older individuals at risk for AD have revealed hypometabolism of association cortical regions similar to the metabolic abnormalities seen in AD patients. In recent functional magnetic resonance imaging (fMRI) studies of AD, co

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Kessels, Roy P. C., Stefano Cappa, Cristina Festari, et al. "64 The Biomarker-Based Etiological Diagnosis of Neurocognitive Disorders: the European Inter-Societal Delphi Consensus." Journal of the International Neuropsychological Society 29, s1 (2023): 268–69. http://dx.doi.org/10.1017/s135561772300382x.

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Objective:In the field of neurocognitive disorders, the perspective offered by new disease-modifying therapy increases the importance of etiological diagnosis. The prescription of cerebrospinal fluid analysis (CSF) and imaging biomarkers is a common practice in the clinic but is often driven more by personal expertise and local availability of diagnostic tools than by evidence of efficacy and cost-effectiveness analysis. This leads to a widely heterogeneous dementia care across Europe. Therefore, a European initiative is currently being conducted to establish a consensus for biomarker-based di

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Cummings, J. L. "Subcortical Dementia Neuropsychology, Neuropsychiatry, and Pathophysiology." British Journal of Psychiatry 149, no. 6 (1986): 682–97. http://dx.doi.org/10.1192/bjp.149.6.682.

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Subcortical dementia refers to a clinical syndrome characterised by slowing of cognition, memory disturbances, difficulty with complex intellectual tasks such as strategy generation and problem solving, visuospatial abnormalities, and disturbances of mood and affect. The syndrome was first described by Kinnier Wilson, but further progress in development of the concept has occurred only within the past ten years. Subcortical dementia occurs in degenerative extrapyramidal disorders and has also been identified in inflammatory, infectious, and vascular conditions. Histologic, metabolic, and neuro

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M�ller, Harald E., Peter Vermathen, Markus G. Lentschig, et al. "Metabolic characterization of AIDS dementia complex by spectroscopic imaging." Journal of Magnetic Resonance Imaging 9, no. 1 (1999): 10–18. http://dx.doi.org/10.1002/(sici)1522-2586(199901)9:1<10::aid-jmri2>3.0.co;2-w.

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Camicioli, Richard, and Nancy Fisher. "Progress in Clinical Neurosciences: Parkinson's Disease with Dementia and Dementia with Lewy Bodies." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 31, no. 1 (2004): 7–21. http://dx.doi.org/10.1017/s0317167100002791.

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Dementia occurs in up to 30% of people with Parkinson's disease and is a major cause of disability. Pathologically, Parkinson's dementia, where dementia follows the onset of parkinsonism by at least one year, overlaps with dementia with Lewy bodies. We review the functional impact, definitions, neuropsychology, epidemiology and pathophysiology of Parkinson's dementia, dementia with Lewy bodies and their overlap. Associated psychiatric and imaging findings are also considered. Lastly, current and emerging approaches to assessment and treatment in patients with these Lewy body associated dementi

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Powell, Artiss L., and D. Frank Benson. "Brain imaging techniques in the diagnosis of dementia." Neuropsychology Review 1, no. 1 (1990): 3–19. http://dx.doi.org/10.1007/bf01108856.

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Nazem, Amir, Chris C. Tang, Phoebe Spetsieris, et al. "A multivariate metabolic imaging marker for behavioral variant frontotemporal dementia." Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring 10, no. 1 (2018): 583–94. http://dx.doi.org/10.1016/j.dadm.2018.07.009.

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Gupta, Vanshika, Ritu Verma, Rajeev Ranjan, et al. "Metabolic imaging patterns in posterior cortical atrophy and Lewy body dementia." Nuclear Medicine Communications 40, no. 12 (2019): 1275–82. http://dx.doi.org/10.1097/mnm.0000000000001102.

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Rapoport, Stanley I. "Discriminant Analysis of Brain Imaging Data Identifies Subjects With Early Alzheimer's Disease." International Psychogeriatrics 9, S1 (1997): 229–35. http://dx.doi.org/10.1017/s1041610297004936.

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In vivo functional brain imaging provides an opportunity to quantify and localize functional deficits associated with Alzheimer's disease (AD), in relation to dementia severity and heterogeneous cognitive profiles. Such imaging also provides a basis for distinguishing AD from other causes of dementia and for making an early diagnosis of disease. One imaging modality that can elucidate AD is positron emission tomography (PET), which is used to measure regional cerebral metabolic rates for glucose (rCMRglc) and regional cerebral blood flow (rCBF). Resting-state measurements with PET, when relate

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Standley, Katherine, Charles Brock, and Michael Hoffmann. "Advances in functional neuroimaging in dementias and potential pitfalls." Neurology International 4, no. 1 (2012): 7. http://dx.doi.org/10.4081/ni.2012.e7.

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Neuroimaging is continuously advancing at a rapid rate and has progressed from excluding relatively uncommon secondary causes (stroke, tumor) to assisting with early diagnosis and subtype of dementia. Structural imaging has given way to functional, metabolic and receptor imaging.

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Thèses sur le sujet "Dementia, metabolic imaging, neuropsychology"

FERRI, FRANCESCA. "Phenotypic and metabolic imaging characterization of posterior cognitive dysfunctions." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2015. http://hdl.handle.net/10281/94449.

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In the last decade, the differential diagnosis of dementia has become very challenging. Different clinical syndromes can in fact be associated with the same underlying pathology and different pathologies can be associated with the same clinical phenotype, making it very difficult to distinguish them in vivo. In the present project we focused on the differential diagnosis of cognitive syndromes affecting, at onset, posterior cerebral networks with particular reference to PCA, CBD and LBD, respectively. We enrolled 70 consecutive patients, referred to the memory clinic of the Neurology Depar

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Fiorenzato, Eleonora. "Cognitive and Brain Imaging Changes in Parkinsonism." Doctoral thesis, Università degli studi di Padova, 2017. http://hdl.handle.net/11577/3424966.

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The present thesis comprises three main parts: one theoretical and two experimental. The first part, composed of two chapters, will introduce the clinical and neuropathological features underlying parkinsonian disorders, namely in Parkinson’s disease (PD) as well as in atypical parkinsonisms — multiple system atrophy (MSA) and progressive supranuclear palsy (PSP) (Chapter 1). In this regard, PD and MSA are defined as synucleinopathies due to the presence of synuclein aggregates; while PSP that is characterized by tau protein accumulations, is part of tauopathies. Further, Chapter 2 will provi

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Rahman, Tupur. "Understanding Cognitive Function In Older Adults With Type 2 Diabetes Mellitus via Environmental Stress in the Functional Magnetic Resonance Imaging Environment and Metabolic Stress Associated with Glucose Ingestion." Thesis, 2011. http://hdl.handle.net/1807/31401.

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The objectives were to explore cognitive function in older adults with Type 2 Diabetes Mellitus (T2DM) through two studies: 1) a pilot study involving the metabolic stress of glucose ingestion and the environmental stress of a functional magnetic resonance imaging (fMRI) environment and 2) an fMRI study exploring the neural mechanisms through hippocampal and prefrontal functional imaging during memory and executive functioning tasks. In T2DM subjects, environmental stress produced greater impairment in memory performance and metabolic stress produced overestimation of memory performance, with

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Livres sur le sujet "Dementia, metabolic imaging, neuropsychology"

W, Parks Randolph, Zec Ronald F, and Wilson Robert S. 1948-, eds. Neuropsychology of Alzheimer's disease and other dementias. Oxford University Press, 1993.

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British Psychological Society Conference on Neuropsychology (1987 Rothesay, Scotland). Developments in clinical and experimental neuropsychology. Plenum Press, 1989.

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Crawford, John R. Developments in Clinical and Experimental Neuropsychology. Springer, 2013.

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Parker, Denis M., and John R. Crawford. Developments in Clinical and Experimental Neuropsychology. Springer London, Limited, 2013.

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Chapitres de livres sur le sujet "Dementia, metabolic imaging, neuropsychology"

Hassenstab, Jason J. "Neuropsychology and Neuroimaging in Metabolic Dysfunction." In Brain Imaging in Behavioral Medicine and Clinical Neuroscience. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6373-4_14.

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Naugle, Richard I., and Erin D. Bigler. "Brain Imaging and Neuropsychological Identification of Dementia of the Alzheimer’s Type." In Critical Issues in Neuropsychology. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4899-2534-3_7.

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Haxby, J. V. "Cognitive Deficits and Local Metabolic Changes in Dementia of the Alzheimer Type." In Imaging, Cerebral Topography and Alzheimer’s Disease. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75690-0_8.

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Alavi, A., and E. Souder. "Detection of Structural and Metabolic Changes of the Brain in Normal Aging and Dementia with Modern Imaging Techniques." In Cerebral Ischemia and Dementia. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76208-6_42.

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"Biomarkers and Imaging." In The Neuropsychology of Dementia. Cambridge University Press, 2024. http://dx.doi.org/10.1017/9781009025911.014.

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"Endocrine-, Metabolic-, Toxin-, and Drug-Related Dementia." In Imaging of Neurodegenerative Disorders, edited by Sangam G. Kanekar. Georg Thieme Verlag, 2016. http://dx.doi.org/10.1055/b-0035-129384.

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Cerami, Chiara, and Stefano F. Cappa. "Integration of Imaging in Cortical Dementia Diagnosis." In The Neuropsychology of Cortical Dementias. Springer Publishing Company, 2015. http://dx.doi.org/10.1891/9780826107275.0002.

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Stevens, John M., and Nick C. Fox. "Structural Imaging." In Early-Onset Dementia: a multidisciplinary approach. Oxford University PressNew York, NY, 2001. http://dx.doi.org/10.1093/oso/9780192630346.003.0006.

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Abstract Structural neuroimaging traditionally has been used to exclude potentially treatable surgical causes of dementia, such as neoplasm, subdural haematomas, and hydrocephalus (Moseley 1995; Alexander et al. 1995). In recent years, newly recognized entities such as AIDS dementia complex (Barker et al. 1995), and venous hypertensive encephalopathy associated with arterio-venous malformations (Deveikis 1998) have entered the list of diagnosable causes, along with a legion of medical conditions which include multiple sclerosis, neurosarcoidosis, and metabolic disorders (Moseley 1995). In most

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Lopez, Oscar L., and Sandra Bell. "Neurobiological Approaches to the Treatment of Alzheimer’s Disease." In Cognitive Neuropsychology of Alzheimer’s Disease. Oxford University PressOxford, 2004. http://dx.doi.org/10.1093/oso/9780198508304.003.0022.

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Abstract Dementia syndromes in older adults can be secondary to a wide variety of causes (e.g. infections, exposure to toxic substances, medications, avitaminosis, head trauma, cerebrovascular disease, tumors, or neurodegenerative disorders). However, the most frequent cause is Alzheimer’s disease (AD), which has been reported in up to 10% of the population age 65 or older (Jorm and Jolley 1998). Therefore, there is a significant effort to develop medications that can treat or ameliorate AD symptoms. All suspected known mechanisms of the metabolic cascade of AD have been explored with specific

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Weih Markus, Degirmenci Ümüt, Kreil Sebastian, et al. "Nuclear Medicine Diagnostic Techniques in the Era of Pathophysiology-Based CSF Biomarkers for Alzheimer's Disease." In Advances in Alzheimer’s Disease. IOS Press, 2011. https://doi.org/10.3233/978-1-60750-793-2-163.

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Nuclear medicine techniques were the first functional imaging techniques used to support the clinical diagnosis of Alzheimer's Disease (AD). Perfusion-SPECT allows registration of regional cerebral blood flow (rCBF) which is altered in a characteristic temporal-parietal pattern in AD. Numerous studies have shown the diagnostic value of reduced CBF and metabolic changes using perfusion-SPECT and FDG-PET in AD diagnosis as well as in differential diagnosis against frontotemporal dementia (FTD), dementia with Lewy-Bodies (DLB), and vascular cognitive disorders. This renders perfusion-SPECT an imp

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Actes de conférences sur le sujet "Dementia, metabolic imaging, neuropsychology"

Dias, Jéssica Azevedo, Matheus de Serpa Vale, Raquel Penido Oliveira, and Thaís Helen Rezende Pio. "Alzheimer’s: what is the difference between or frontotemporal dementia." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.544.

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Introduction: In the study of neurodegenerative diseases, Alzheimer’s disease (AD) has been classically considered with a typical presentation of cognitive symptoms and neuroanatomical changes. However, there are clinical phenotypes of AD whose neurobiological bases are similar to frontotemporal dementia (FTD). In this sense, the heterogeneity of these pictures leads to inaccurate evaluation and diagnosis processes, due to the scant knowledge about their neurocognitive symptoms. The early stages of Alzheimer’s-type dementia are classically characterized by memory impairment, whereas behavioral

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