Littérature scientifique sur le sujet « Cox-2 expression »
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Articles de revues sur le sujet "Cox-2 expression"
Laino, Charlene. « COX-2 Inhibitorsʼ Effect Dependent on COX-2 Tumor Expression ». Oncology Times 26, no 11 (juin 2004) : 24. http://dx.doi.org/10.1097/01.cot.0000292129.98424.b1.
Texte intégralLaino, Charlene. « COX-2 inhibitorsʼ effect dependent on COX-2 tumour expression ». Oncology Times 1, no 6 (juillet 2004) : 13. http://dx.doi.org/10.1097/01434893-200407000-00007.
Texte intégralKarim, Mohammed Mohibul, Yoshitake Hayashi, Masanori Inoue, Yukihiro Imai, Hiroshi Ito et Misao Yamamoto. « Cox-2 expression in retinoblastoma ». American Journal of Ophthalmology 129, no 3 (mars 2000) : 398–401. http://dx.doi.org/10.1016/s0002-9394(99)00355-4.
Texte intégralTelliez, Aurelie, Christophe Furman, Nicole Pommery et Jean-Pierre Henichart. « Mechanisms Leading to COX-2 Expression and COX-2 Induced Tumorigenesis : Topical Therapeutic Strategies Targeting COX-2 Expression and Activity ». Anti-Cancer Agents in Medicinal Chemistry 6, no 3 (1 mai 2006) : 187–208. http://dx.doi.org/10.2174/187152006776930891.
Texte intégralHazar, Burhan, Melek Ergin, Ertuğrul Seyrek, Şeyda Erdoğan, ılhan Tuncer et Sibel Hakverdi. « Cyclooxygenase-2 (Cox-2) Expression in Lymphomas ». Leukemia & ; Lymphoma 45, no 7 (juillet 2004) : 1395–99. http://dx.doi.org/10.1080/10428190310001654032.
Texte intégralMungo, David V., Xinping Zhang, Regis J. O'Keefe, Randy N. Rosier, J. Edward Puzas et Edward M. Schwarz. « COX-1 and COX-2 expression in osteoid osteomas ». Journal of Orthopaedic Research 20, no 1 (janvier 2002) : 159–62. http://dx.doi.org/10.1016/s0736-0266(01)00065-1.
Texte intégralMenczer, Joseph, Letizia Schreiber, Oleg Sukmanov, Vladimir Kravtsov, Esther Berger, Abraham Golan et Tally Levy. « COX-2 expression in uterine carcinosarcoma ». Acta Obstetricia et Gynecologica Scandinavica 89, no 1 (janvier 2010) : 120–25. http://dx.doi.org/10.3109/00016340903342006.
Texte intégralGatalica, Zoran, et Brian Loggie. « COX-2 expression in pseudomyxoma peritonei ». Cancer Letters 244, no 1 (novembre 2006) : 86–90. http://dx.doi.org/10.1016/j.canlet.2005.12.013.
Texte intégralMenczer, Joseph. « Cox-2 expression in ovarian malignancies ». European Journal of Obstetrics & ; Gynecology and Reproductive Biology 146, no 2 (octobre 2009) : 129–32. http://dx.doi.org/10.1016/j.ejogrb.2009.05.030.
Texte intégralKawamoto, Toru, Tohru Asano, Junichi Shoda, Mira Datta, Takeshi Todoroki, Naomi Tanaka, Takashi Fukao et Masanao Miwa. « Immunohistochemical expression of cyclooxygenase-2 (COX-2) in gallbladder carcinoma — Association of enhanced COX-2 expression with tumor progression ». Gastroenterology 118, no 4 (avril 2000) : A189. http://dx.doi.org/10.1016/s0016-5085(00)82830-9.
Texte intégralThèses sur le sujet "Cox-2 expression"
Sun, Haipeng. « Regulation of Cyclooxygenase Gene Expression by Glucocorticoids in Cardiomyocytes ». Diss., The University of Arizona, 2007. http://hdl.handle.net/10150/194896.
Texte intégralFritzsche, Julia. « Expression von EGFR, HER-2 und COX-2 beim Zervixkarzinom : Vergleich von Primärtumoren und Rezidiven ». Doctoral thesis, Universitätsbibliothek Leipzig, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-119352.
Texte intégralKim, Janet Heejung. « Cyclooxygenase-2 Expression in Post-Mastectomy Chest Wall Relapse ». Yale University, 2006. http://ymtdl.med.yale.edu/theses/available/etd-06282006-104942/.
Texte intégralKim, Youngsoo. « Molecular characterization of cyclooxygenase-2 (COX-2) expression in murine skin carcinoma cells / ». Digital version accessible at:, 1998. http://wwwlib.umi.com/cr/utexas/main.
Texte intégralRowe, Kherie Sheheda Janelle. « Cox-2 expression : interaction of Neisseria meningitidis with human cells ». Thesis, University of Nottingham, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519420.
Texte intégralAbdalla, Salem Ishtiwi. « Cyclooxygenase-2 (cox-2) expression in Barrett's oesphageal epithelium : relationship to inflammation and cancer ». Thesis, Queen Mary, University of London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418127.
Texte intégralLysitsa, Stella. « Evolution du lichen plan buccal et expression de la COX-2 / ». Genève : [s.n.], 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000253996.
Texte intégralHuppes, Rafael Ricardo [UNESP]. « Expressão gênica de MMP-2 e 9, TIMP-1 e 2, ATM, TP53, VEGF, COX-2 e CDH-1 no TVT canino ». Universidade Estadual Paulista (UNESP), 2014. http://hdl.handle.net/11449/122030.
Texte intégralA literatura cita que 1 a 5% dos casos de tumor venéreo transmissível (TVT) primário são metastáticos. Sendo assim, é importante estudar os mecanismos que colaborem para a invasão metastática assim como para sua implantação. Dentre estes mecanismos as metaloproteinases (MMP-2 e MMP-9) e seus inibidores (TIMP-1 e TIMP-2), assim como o ATM, COX-2, VEGF e CDH-1 podem explicar a implantação tumoral no sítio primário e a ocorrência da invasão metastática do TVT no cão. O objetivo do presente trabalho é avaliar a expressão gênica dos marcadores acima e correlacionar a sua expressão com o poder de implantação e invasão metastática no TVT. Para este estudo foram avaliadas 32 amostras tumorais, que foram congeladas e delas extraídos RNAm. Utilizou-se o método de qRT-PCR para todos os transcritos. Os resultados foram comparados com sangue periférico de 10 cães saudáveis (grupo controle) com o teste de Mann Whitney. A expressão gênica de MMP-2 e TIMP-1 foi significativamente maior do que o grupo controle (p < 0,001; p = 0,037; respetivamente). A expressão dos transcritos dos genes MMP-9 e TIMP-2 não apresentou diferença estatística entre o TVT e grupo controle (p = 0,535; p = 0, 906; respetivamente). A avaliação de expressão de transcritos do ATMapresentou aumento significativo (p < 0,0001) de sua expressão no tecido tumoral (TVT) quando comparado com o grupo controle, enquanto a expressão dos transcritos do gene TP53 não apresentou diferença estatística entre os grupos (p = 0,26). Na avaliação da COX-2, VEGF, CDH-1 foi verificado aumento significativo (p < 0,0001; p < 0,0001; p = 0,04, respectivamente) da expressão de transcritos dos genes no tecido tumoral (TVT) em relação ao grupo controle. A super-expressão de MMP-2 e o TIMP-1 pode explicar a capacidade de implantação das células tumorais assim como a maior expressão de VEGF e COX-2 pode explicar o crescimento rápido local do tumor e ...
The literature reports that 1-5% of cases of primary trasmissible venereal tumor (TVT) are metastatic. Thus, it is interesting to study the mechanisms that collaborate to the metastatic invasion and implantation of TVT. Among these mechanisms, the metalloproetinases (MMP-2 and MMP-9) and their inhibitors (TIMP-1 and TIMP-2), as well as ATM, COX-2, VEGF and CDH-1 may explain the tumoral implantation in the primary site and metastatic invasion of TVT in dogs. The objectives of this study are to evaluate the gene expression of these markers and to correlate their expression with the high ability of deployment and metastatic invasion of TVT. For this study, 32 tumor samples were frozen and their mRNA were extract using the qRT-PCR method for all transcripts. The results were compared with peripheral blood of 10 healthy dogs (control group) using the Mann Whitney test. The expression of MMP-2 and TIMP-1 were significantly higher than the control group (p <0.001, p = 0.037, respectively). The expression of MMP-9 and TIMP-2 showed no statistical difference between the TVT and the control group (p = 0.535, p = 0, 906, respectively). The expression of ATM was increased in tumor tissue (TVT) when compared with the control group, while the expression of TP53 had no statistical difference between groups (p = 0.26). The evaluation of COX-2, VEGF and CDH-1 were increas in tumor tissue when compared with control group. The over expression of MMP-2 and TIMP-1 may explain the implantation ability of the tumor cells, as well as the increase of VEGF and COX-2 may explain the rapid tumor growth and the rich vasculatization. While the over expression of ATM, TP53 and CDH-1 may contribute to the low metastatic capacity of the TVT tumor
Huppes, Rafael Ricardo. « Expressão gênica de MMP-2 e 9, TIMP-1 e 2, ATM, TP53, VEGF, COX-2 e CDH-1 no TVT canino / ». Jaboticabal, 2014. http://hdl.handle.net/11449/122030.
Texte intégralCoorientador: Andrigo Barboza De Nardi
Coorientador: Mirela Tinucci Costa
Banca: Rosemere de Oliveira Vasconcelos
Banca: Geórgia Mode Magalhães
Banca: Rafael Torres Neto
Banca: Bruno Watanabe Minto
Resumo: A literatura cita que 1 a 5% dos casos de tumor venéreo transmissível (TVT) primário são metastáticos. Sendo assim, é importante estudar os mecanismos que colaborem para a invasão metastática assim como para sua implantação. Dentre estes mecanismos as metaloproteinases (MMP-2 e MMP-9) e seus inibidores (TIMP-1 e TIMP-2), assim como o ATM, COX-2, VEGF e CDH-1 podem explicar a implantação tumoral no sítio primário e a ocorrência da invasão metastática do TVT no cão. O objetivo do presente trabalho é avaliar a expressão gênica dos marcadores acima e correlacionar a sua expressão com o poder de implantação e invasão metastática no TVT. Para este estudo foram avaliadas 32 amostras tumorais, que foram congeladas e delas extraídos RNAm. Utilizou-se o método de qRT-PCR para todos os transcritos. Os resultados foram comparados com sangue periférico de 10 cães saudáveis (grupo controle) com o teste de Mann Whitney. A expressão gênica de MMP-2 e TIMP-1 foi significativamente maior do que o grupo controle (p < 0,001; p = 0,037; respetivamente). A expressão dos transcritos dos genes MMP-9 e TIMP-2 não apresentou diferença estatística entre o TVT e grupo controle (p = 0,535; p = 0, 906; respetivamente). A avaliação de expressão de transcritos do ATMapresentou aumento significativo (p < 0,0001) de sua expressão no tecido tumoral (TVT) quando comparado com o grupo controle, enquanto a expressão dos transcritos do gene TP53 não apresentou diferença estatística entre os grupos (p = 0,26). Na avaliação da COX-2, VEGF, CDH-1 foi verificado aumento significativo (p < 0,0001; p < 0,0001; p = 0,04, respectivamente) da expressão de transcritos dos genes no tecido tumoral (TVT) em relação ao grupo controle. A super-expressão de MMP-2 e o TIMP-1 pode explicar a capacidade de implantação das células tumorais assim como a maior expressão de VEGF e COX-2 pode explicar o crescimento rápido local do tumor e ...
Abstract: The literature reports that 1-5% of cases of primary trasmissible venereal tumor (TVT) are metastatic. Thus, it is interesting to study the mechanisms that collaborate to the metastatic invasion and implantation of TVT. Among these mechanisms, the metalloproetinases (MMP-2 and MMP-9) and their inhibitors (TIMP-1 and TIMP-2), as well as ATM, COX-2, VEGF and CDH-1 may explain the tumoral implantation in the primary site and metastatic invasion of TVT in dogs. The objectives of this study are to evaluate the gene expression of these markers and to correlate their expression with the high ability of deployment and metastatic invasion of TVT. For this study, 32 tumor samples were frozen and their mRNA were extract using the qRT-PCR method for all transcripts. The results were compared with peripheral blood of 10 healthy dogs (control group) using the Mann Whitney test. The expression of MMP-2 and TIMP-1 were significantly higher than the control group (p <0.001, p = 0.037, respectively). The expression of MMP-9 and TIMP-2 showed no statistical difference between the TVT and the control group (p = 0.535, p = 0, 906, respectively). The expression of ATM was increased in tumor tissue (TVT) when compared with the control group, while the expression of TP53 had no statistical difference between groups (p = 0.26). The evaluation of COX-2, VEGF and CDH-1 were increas in tumor tissue when compared with control group. The over expression of MMP-2 and TIMP-1 may explain the implantation ability of the tumor cells, as well as the increase of VEGF and COX-2 may explain the rapid tumor growth and the rich vasculatization. While the over expression of ATM, TP53 and CDH-1 may contribute to the low metastatic capacity of the TVT tumor
Doutor
Laube, Markus. « Synthese von Cyclooxygenase-2-Inhibitoren als Grundlage für die funktionelle Charakterisierung der COX-2-Expression mittels PET ». Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-160091.
Texte intégralLivres sur le sujet "Cox-2 expression"
United States. Congress. House. Committee on International Relations, dir. 106-2 MARKUP : Markup Of H. CON. RES. 232, Expressing The Sense Of Congress Concerning The Safety And Well-Being Of United States Citizens Injured While Travelling In Mexico, June 14, 2000. [S.l : s.n., 2000.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations, dir. 106-2 MARKUP : Markup Of H. CON. RES. 232, Expressing The Sense Of Congress Concerning The Safety And Well-Being Of United States Citizens Injured While Travelling In Mexico, June 14, 2000. [S.l : s.n., 2000.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations., dir. 106-2 MARKUP : Markup Of H. CON. RES. 232, Expressing The Sense Of Congress Concerning The Safety And Well-Being Of United States Citizens Injured While Travelling In Mexico, June 14, 2000. [S.l : s.n., 2000.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations., dir. 106-2 MARKUP : Markup Of H. CON. RES. 232, Expressing The Sense Of Congress Concerning The Safety And Well-Being Of United States Citizens Injured While Travelling In Mexico, June 14, 2000. [S.l : s.n., 2000.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations, dir. 106-2 MARKUP : Markup Of H. CON. RES. 232, Expressing The Sense Of Congress Concerning The Safety And Well-Being Of United States Citizens Injured While Travelling In Mexico, June 14, 2000. [S.l : s.n., 2000.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations. Subcommittee on the Middle East and Central Asia. Expressing the grave concern of Congress regarding the continuing gross violations of human rights and civil liberties of the Syrian and Lebanese people by the government of the Syrian Arab republic ; and expressing the grave concern of Congress regarding the occupation of the Republic of Lebanon by the Syrian Arab Republic : Markup before the Subcommittee on the Middle East and Central Asia of the Committee on International Relations, House of Representatives, One Hundred Ninth Congress, first session, on H. Con. Res. 18 and H. Con. Res. 32, March 2, 2005. Washington : U.S. G.P.O., 2005.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations. Subcommittee on the Middle East and Central Asia. Expressing the grave concern of Congress regarding the continuing gross violations of human rights and civil liberties of the Syrian and Lebanese people by the government of the Syrian Arab republic ; and expressing the grave concern of Congress regarding the occupation of the Republic of Lebanon by the Syrian Arab Republic : Markup before the Subcommittee on the Middle East and Central Asia of the Committee on International Relations, House of Representatives, One Hundred Ninth Congress, first session, on H. Con. Res. 18 and H. Con. Res. 32, March 2, 2005. Washington : U.S. G.P.O., 2005.
Trouver le texte intégralRights, United States Congress House Committee on International Relations Subcommittee on International Operations and Human. H. Con. Res. 28, expressing the sense of Congress that the United States should introduce and make all efforts necessary to pass a resolution criticizing the People's Republic of China for its human rights abuses in China and Tibet at the annual meeting of the United Nations Commission on Human Rights : Markup before the Subcommittee on International Operations and Human Rights of the Committee on International Relations, House of Representatives, One Hundred Sixth Congress, first session, March 2, 1999. Washington : U.S. G.P.O., 1999.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations. Subcommittee on International Operations and Human Rights. H. Con. Res. 28, expressing the sense of Congress that the United States should introduce and make all efforts necessary to pass a resolution criticizing the People's Republic of China for its human rights abuses in China and Tibet at the annual meeting of the United Nations Commission on Human Rights : Markup before the Subcommittee on International Operations and Human Rights of the Committee on International Relations, House of Representatives, One Hundred Sixth Congress, first session, March 2, 1999. Washington : U.S. G.P.O., 1999.
Trouver le texte intégralUnited States. Congress. House. Committee on International Relations. Subcommittee on International Operations and Human Rights. H. Con. Res. 28, expressing the sense of Congress that the United States should introduce and make all efforts necessary to pass a resolution criticizing the People's Republic of China for its human rights abuses in China and Tibet at the annual meeting of the United Nations Commission on Human Rights : Markup before the Subcommittee on International Operations and Human Rights of the Committee on International Relations, House of Representatives, One Hundred Sixth Congress, first session, March 2, 1999. Washington : U.S. G.P.O., 1999.
Trouver le texte intégralChapitres de livres sur le sujet "Cox-2 expression"
Dixon, D. A. « Regulation of COX-2 Expression in Human Cancers ». Dans COX-2, 52–71. Basel : KARGER, 2003. http://dx.doi.org/10.1159/000071363.
Texte intégralBennett, P., et D. Slater. « COX-2 expression in labour ». Dans Improved Non-Steroid Anti-Inflammatory Drugs : COX-2 Enzyme Inhibitors, 167–88. Dordrecht : Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-010-9029-2_10.
Texte intégralPatrono, C., P. Patrignani, M. R. Panara, F. Cipollone, G. Santini, M. G. Sciulli, M. T. Rotondo, R. Padovano et M. Di Giamberardino. « COX-2 expression and inhibition in human monocytes ». Dans Improved Non-Steroid Anti-Inflammatory Drugs : COX-2 Enzyme Inhibitors, 121–31. Dordrecht : Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-010-9029-2_7.
Texte intégralDixon, Dan A., Fernando F. Blanco, Annalisa Bruno et Paola Patrignani. « Mechanistic Aspects of COX-2 Expression in Colorectal Neoplasia ». Dans Recent Results in Cancer Research, 7–37. Berlin, Heidelberg : Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30331-9_2.
Texte intégralHinz, Burkhard, Robert Ramer et Kay Brune. « Induction of COX-2 Expression by the Endocannabinoid Derivative R(+)-Methanandamide ». Dans Advances in Experimental Medicine and Biology, 145–52. Boston, MA : Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9194-2_30.
Texte intégralCrofford, L. J. « Expression and regulation of COX-2 in synovial tissues of arthritic patients ». Dans Improved Non-Steroid Anti-Inflammatory Drugs : COX-2 Enzyme Inhibitors, 133–43. Dordrecht : Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-010-9029-2_8.
Texte intégralHerschman, Harvey R., David J. Wadleigh et Srinivasa T. Reddy. « Regulation of COX-2 Expression in Fibroblasts, Osteoblasts, Mast Cells, and Macrophages ». Dans Advances in Prostaglandin and Leukotriene Research, 41–47. Dordrecht : Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9721-0_8.
Texte intégralBerg, Jörg, Thomas Christoph, Angelika Bodenteich et Robertson Towart. « Heterogeneous Distribution of COX-2 Over-Expression in Human Colon Carcinoma Cells ». Dans Advances in Experimental Medicine and Biology, 327–30. Boston, MA : Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1810-9_70.
Texte intégralLuna, Marian, Angela Ferrario, Sam Wong et Charles J. Gomer. « Identification of MAP Kinase Pathways Involved in COX-2 Expression Following Photofrin Photodynamic Therapy ». Dans Methods in Molecular Biology, 47–63. Totowa, NJ : Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-697-9_4.
Texte intégralBazan, N. G., V. M. Marcheselli, G. Allan, K. Van Meter et J. P. Moises. « Brain COX-2 in experimental models of epilepsy and stroke : signalling pathways leading to enhanced expression ». Dans New Targets in Inflammation, 47–53. Dordrecht : Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-011-5386-7_5.
Texte intégralActes de conférences sur le sujet "Cox-2 expression"
Shao, W., C. Kuhn, D. Mayr, N. Ditsch, S. Mahner, N. Harbeck, V. Cavaillès, U. Jeschke et S. Sixou. « Untersuchungen zur differenzierten Expression von PPARγ, Cox-1 und Cox-2 beim Mammakarzinom ». Dans Abstracts zum Kongress 2019 der Bayerischen Gesellschaft für Geburtshilfe und Frauenheilkunde (BGGF) und der Österreichischen Gesellschaft für Gynäkologie und Geburtshilfe (OEGGG). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1693862.
Texte intégralRussell-Puleri, Sparkle, Eno E. Ebong et John M. Tarbell. « Mechanisms of flow-dependent endothelial COX-2 and PGI2 expression ». Dans 2014 40th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2014. http://dx.doi.org/10.1109/nebec.2014.6972924.
Texte intégralPirker, T., E. Pferschy-Wenzig et R. Bauer. « Inhibition of COX-2 mRNA expression by damask rose flowers ». Dans GA – 69th Annual Meeting 2021, Virtual conference. Georg Thieme Verlag, 2021. http://dx.doi.org/10.1055/s-0041-1736970.
Texte intégralLin, Shih-Chieh, et Shaw-Jenq Tsai. « Abstract 3083 : Dual-specificity phosphatase-2 (DUSP2) negatively control cyclooxygenase-2 (COX-2) expression in cancer cells ». Dans Proceedings : AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012 ; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3083.
Texte intégralBu, Rong, Sandeep K. Parvathareddy, Abdul K. Siraj, Padmanaban Annaiyappanaidu, Kaleem Iqbal, Maha Al Rasheed, Wael Haqawi et Khawla S. Al-Kuraya. « Abstract 729 : Prognostic significance of COX-2 over-expression inBRAFmutated Middle Eastern PTC ». Dans Proceedings : AACR Annual Meeting 2021 ; April 10-15, 2021 and May 17-21, 2021 ; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-729.
Texte intégralYang, Wan-Yu, Chih-Hsin Tang et Jing-Yuan Chuang. « Abstract 1392 : CTGF inhibits cell motility in oral cancer cells through reducing COX-2 expression ». Dans Proceedings : AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011 ; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1392.
Texte intégralShim, Minsub, Julie Foley, Colleen Anna, Yuji Mishina et Thomas E. Eling. « Abstract LB-338 : Increased apoptosis and p53 expression in sclerotome of COX-2 transgenic embryo ». Dans Proceedings : AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010 ; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-338.
Texte intégralZago, Michela, Angela Rico de Souza, Simon Rousseau, David H. Eidelman, Qutayba Hamid et Carolyn J. Baglole. « The Aryl Hydrocarbon Receptor (AhR) Exerts Post-Transcriptional Control Over Cigarette Smoke-Induced Cyclooxygenase-2 (Cox-2) Protein Expression ». Dans American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2009.
Texte intégralKillian, Megan L., Barbara Zielinska et Tammy L. Haut Donahue. « Role of IL-1 on Aggrecanase and COX-2 Gene Expression of Meniscal Explants Following Dynamic Compression ». Dans ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19110.
Texte intégralSHIN, JONG WOOK, In Won Park, Jae Chul Choi, Byoung Whui Choi et Jae Yeol Kim. « Expression Of C-ErbB-2 And COX-2 In Adenocarcinoma And Squamous Cell Carcinoma In The Surgically Removed Lung Cancers ». Dans American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3500.
Texte intégralRapports d'organisations sur le sujet "Cox-2 expression"
Shapiro, Charles L., William Burak et Robert Brueggemeier. The Effect of COX-2 Inhibitors on the Aromatase Gene Expression in Human Breast Cancer. Fort Belvoir, VA : Defense Technical Information Center, juin 2003. http://dx.doi.org/10.21236/ada418325.
Texte intégralShapiro, Charles L. The Effect of COX-2 Inhibitors on the Aromatase Gene (CYP19) Expression in Human Breast Cancer. Fort Belvoir, VA : Defense Technical Information Center, décembre 2006. http://dx.doi.org/10.21236/ada468021.
Texte intégralFields, Michael J., Mordechai Shemesh et Anna-Riitta Fuchs. Significance of Oxytocin and Oxytocin Receptors in Bovine Pregnancy. United States Department of Agriculture, août 1994. http://dx.doi.org/10.32747/1994.7568790.bard.
Texte intégralSplitter, Gary, Zeev Trainin et Yacov Brenner. Lymphocyte Response to Genetically Engineered Bovine Leukemia Virus Proteins in Persistently Lymphocytic Cattle from Israel and the U.S. United States Department of Agriculture, juillet 1995. http://dx.doi.org/10.32747/1995.7570556.bard.
Texte intégralMeidan, Rina, et Joy Pate. Roles of Endothelin 1 and Tumor Necrosis Factor-A in Determining Responsiveness of the Bovine Corpus Luteum to Prostaglandin F2a. United States Department of Agriculture, janvier 2004. http://dx.doi.org/10.32747/2004.7695854.bard.
Texte intégralDudareva, Natalia, Alexander Vainstein, Eran Pichersky et David Weiss. Integrating biochemical and genomic approaches to elucidate C6-C2 volatile production : improvement of floral scent and fruit aroma. United States Department of Agriculture, septembre 2007. http://dx.doi.org/10.32747/2007.7696514.bard.
Texte intégralGrumet, Rebecca, Rafael Perl-Treves et Jack Staub. Ethylene Mediated Regulation of Cucumis Reproduction - from Sex Expression to Fruit Set. United States Department of Agriculture, février 2010. http://dx.doi.org/10.32747/2010.7696533.bard.
Texte intégralRodriguez, Russell J., et Stanley Freeman. Gene Expression Patterns in Plants Colonized with Pathogenic and Non-pathogenic Gene Disruption Mutants of Colletotrichum. United States Department of Agriculture, février 2009. http://dx.doi.org/10.32747/2009.7592112.bard.
Texte intégralXu, Jin-Rong, et Amir Sharon. Comparative studies of fungal pathogeneses in two hemibiotrophs : Magnaporthe grisea and Colletotrichum gloeosporioides. United States Department of Agriculture, mai 2008. http://dx.doi.org/10.32747/2008.7695585.bard.
Texte intégralIzhar, Shamay, Maureen Hanson et Nurit Firon. Expression of the Mitochondrial Locus Associated with Cytoplasmic Male Sterility in Petunia. United States Department of Agriculture, février 1996. http://dx.doi.org/10.32747/1996.7604933.bard.
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