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Bibliographies thématiques / Circadian systems / Livres

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Livres sur le sujet « Circadian systems »

Auteur : Grafiati

Publié le 5 avril 2025

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1

Guo, Xinfei, et Mircea R. Stan. Circadian Rhythms for Future Resilient Electronic Systems. Cham : Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-20051-0.

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2

Ripkens, Michael. Untersuchung zur Vorhersage der Resynchronisation des zirkadianen Systems nach transmeridianen Flugen. Koln : DFVLR, 1989.

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3

Aguilar-Roblero, Raúl, Mauricio Díaz-Muñoz et Mária Luisa Fanjul-Moles, dir. Mechanisms of Circadian Systems in Animals and Their Clinical Relevance. Cham : Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-08945-4.

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4

Rensing, Ludger. Temporal Disorder in Human Oscillatory Systems : Proceedings of an International Symposium University of Bremen, 8-13 September 1986. Berlin, Heidelberg : Springer Berlin Heidelberg, 1987.

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5

Ludger, Rensing, Heiden, Uwe an der, 1942- et Mackey Michael C. 1942-, dir. Temporal disorder in human oscillatory systems : Proceedings of an international symposium, University of Bremen, 8-13 September 1986. Berlin : Springer-Verlag, 1987.

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6

H, Monk Timothy, et American Nurses Association, dir. The nurse's shift work handbook. Washington, D.C : American Nurses Pub., 1993.

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7

Redfern, P. H., I. C. Campbell, J. A. Davies et K. F. Martin, dir. Circadian Rhythms in the Central Nervous System. London : Palgrave Macmillan UK, 1985. http://dx.doi.org/10.1007/978-1-349-07837-0.

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8

H, Redfern P., et IUPHAR International Congress of Pharmacology, (9th : 1984 : London), dir. Circadian rhythms in the central nervous system. Weinheim : VCH, 1985.

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9

1942-, Redfern P. H., International Union of Pharmacology et International Congress of Pharmacology (9th : 1984 : London, England), dir. Circadian rhythms in the central nervous system. Deerfield Beach, FL, USA : Distribution for USA and Canada, VCH Publishers, 1985.

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10

1942-, Redfern P. H., International Union of Pharmacology et International Congress of Pharmacology (9th : 1984 : London, England), dir. Circadian rhythms in the central nervous system. Houndmills, Basingstoke, Hampshire : Macmillan, 1985.

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11

H, Schmidt T. F., Engel Bernard T et Blümchen Gerhard, dir. Temporal variations of the cardiovascular system. Berlin : Springer-Verlag, 1992.

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12

Japan) Sapporo Symposium on Biological Rhythmn (8th 1999 Sapporo-shi. Zeitgebers, entrainment and masking of the circadian system : Proceedings of the Eighth Sapporo Symposium on Biological Rhythm, August 10-12, 1999. Sapporo, Japan : Hokkaido University Press, 2001.

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13

Fleissner, Günther. Efferent control of visual sensitivity in arthropod eyes : With emphasis on circadian rhythms. Stuttgart : G. Fischer Verlag, 1988.

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14

Stuttgart, Universität, dir. Die Wirkung nicht-photischer Stimuli und die Rolle von Serotonin im circadianen System der Ratte. [s.l.] : [s.n.], 1998.

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15

Kleinknecht, Sabine. Ausprägung und Synchronisationsverhalten des circadianen Systems nachtaktiver Marsupialia-Arten (Petaurus breviceps, Phalangeridae ; Dasyuroides byrnei, Dasyuridae ; Monodelphis domestica, Didelphidae). [s.l.] : [s.n.], 1986.

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16

M, Buijs R., et Nederlands Instituut voor Hersenonderzoek, dir. Hypothalamic integration of circadian rhythms : Proceedings of the 19th international summer school of brain research, held at the Royal Netherlands Academy of Sciences, Amsterdam, The Netherlands from 28-31 August 1995. Amsterdam : Elsevier, 1996.

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17

1953-, White William B., dir. Blood pressure monitoring in cardiovascular medicine and therapeutics. 2^e éd. Totowa, N.J : Humana Press, 2007.

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18

Takao, Kumazawa, Kruger Lawrence et Mizumura Kazue, dir. The polymodal receptor : A gateway to pathological pain. Amsterdam : Elsevier, 1996.

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19

Aschoff, J., S. Daan et G. A. Groos. Vertebrate Circadian Systems : Structure and Physiology. Springer, 2012.

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20

Aschoff, J., S. Daan et G. A. Groos. Vertebrate Circadian Systems : Structure and Physiology. Springer, 2012.

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21

Vertebrate Circadian Systems : Structure and Physiology. Springer, 2012.

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22

Aguilar-Roblero, Raúl, Mauricio Díaz-Muñoz et Mária Luisa Fanjul-Moles. Mechanisms of Circadian Systems in Animals and Their Clinical Relevance. Springer, 2014.

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23

Aguilar-Roblero, Raúl, Mauricio Díaz-Muñoz et Mária Luisa Fanjul-Moles. Mechanisms of Circadian Systems in Animals and Their Clinical Relevance. Springer, 2015.

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24

Aguilar-Roblero, Raúl, Mauricio Díaz-Muñoz et Mária Luisa Fanjul-Moles. Mechanisms of Circadian Systems in Animals and Their Clinical Relevance. Springer, 2016.

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25

Yan, Qing. Cellular Rhythms and Networks : Implications for Systems Medicine. Springer, 2015.

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26

Yan, Qing. Cellular Rhythms and Networks : Implications for Systems Medicine. Springer, 2015.

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27

Innominato, Pasquale F., et David Spiegel. Circadian rhythms, sleep, and anti-cancer treatments. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0016.

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The circadian timing system temporally regulates biological functions relevant for psycho-physical wellbeing, spanning all the systems related to health. Hence, disruption of circadian rhythms, along with sleep cycles, is associated with the development of several diseases, including cancer. Moreover, altered circadian and sleep functions negatively impact on cancer patients’ quality of life and survival, above and beyond known determinants of outcome. This alteration can occur as a consequence of cancer, but also of anti-cancer treatments. Indeed, circadian rhythms govern also the ability of detoxifying chemotherapy agents across the 24 hours. Hence, adapting chemotherapy delivery to the molecular oscillations in relevant drug pathways can decrease toxicity to healthy cells, while increasing the number of cancer cells killing. This chronomodulated chemotherapy approach, together with the maintenance of proper circadian function throughtout the whole disease challenge, would finally result in safer and more active anticancer treatments, and in patients experiencing better quality and quantity of life.

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28

Guo, Xinfei, et Mircea R. Stan. Circadian Rhythms for Future Resilient Electronic Systems : Accelerated Active Self-Healing for Integrated Circuits. Springer, 2019.

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29

Stan, Mircea R., et Xinfei Guo. Circadian Rhythms for Future Resilient Electronic Systems : Accelerated Active Self-Healing for Integrated Circuits. Springer International Publishing AG, 2020.

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30

Rensing, L., et N. I. Jaeger. Temporal Order : Proceedings of a Symposium on Oscillations in Heterogeneous Chemical and Biological Systems, University of Bremen, September 17–22, 1984. Springer, 2011.

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31

Rensing, L. Temporal Disorder in Human Oscillatory Systems : Proceedings of an International Symposium University of Bremen, 8-13 Sept 1986 (Springer Series in Synergetics). Springer, 1987.

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32

Circadian System. Elsevier Science & Technology, 2023.

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33

Potter, Gregory D. M., et Eleanor M. Scott. Targeting the circadian system. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0016.

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Circadian (approximately 24-hour) rhythms are generated by a hierarchical system responsible for coordinating behaviour and physiology throughout the 24-hour day. Increasing evidence supports roles for disruption of the circadian system in the development of type 2 diabetes (T2DM) and depression. We outline the key aspects of circadian system regulation, discuss the findings indicating that biological disruption of the circadian system produces various behavioural and metabolic abnormalities, and review human studies which show that environmental disruption of the circadian system contributes to T2DM and depression. Finally, we will summarize the therapeutic potential of restoring circadian system organization to manage these diseases.

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34

Lam, Raymond W. Pathogenesis. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199692736.003.0003.

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• There are likely multiple processes to explain the etiology and pathophysiology of depression, with involvement of biological, psychological and social factors.• Circadian rhythmicity, stressful life events and stress reactivity can modify genetic and biological processes (gene-environment interactions) to contribute to depression.• Endophenotypes, or genetic expressions of neural systems involved in depression, will be important in the study of the pathogenesis of depression and the development of novel treatments....

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35

Redfern, P. H. Circadian Rhythms in the Central Nervous System. Palgrave Macmillan, 1985.

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36

Hurd, Mark Webster. Pacemaker-pacemaker communication in the mammalian circadian system. 1996.

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37

Keane, Michael. Fatigue. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199366149.003.0006.

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Fatigue can significantly impair healthcare providers, potentially increasing the possibility of an adverse event. The study of fatigue incorporates concepts from neuroscience, dynamic systems, human factors, and risk management, and relates these elements to circumstances faced by all healthcare professionals. An understanding of the circadian rhythm can help physicians and administrators to develop shift strategies that mitigate the effects of fatigue on human performance. Strategies such as duty-hour limits, “power naps,” and judicious use of caffeine can all help providers who must work at night or during extended shifts.

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38

Jones, Christopher R. Neurobiology of Circadian Rhythms Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0175.

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Temporal organization of nervous system function includes daily rhythms driven by a molecular-genetic hypothalamic “clock” with an intrinsic period length of approximately (circa) one day (diem). The resulting circadian rhythm influences all aspects of brain function and internally synchronizes the circadian oscillations inherent in all other body tissues. Idiosyncratic circadian characteristics interact with perceived environmental stimuli to determine each individual’s entrainment pattern of external synchronization with the day-night cycle. Idiosyncratic entrainment patterns that may come to medical attention include delayed, free-running, advanced, or absent sleep rhythms. Prolonged jet travel and shift work are difficult entrainment challenges for most people.

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39

Tononi, Giulio, et Chiara Cirelli. The Neurobiology of Sleep. Sous la direction de Dennis S. Charney, Eric J. Nestler, Pamela Sklar et Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0011.

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Sleep is required for health and well-being, and consumes roughly one-third of a human’s lifetime, yet its functions remain incompletely understood. This chapter provides an overview of so-called sleep architecture—the stages and cycles that characterize sleep, including rapid eye movement (REM) and non-REM periods. Also discussed are the numerous regions of brain and neurotransmitters that control the induction of sleep, the transitions between REM and non-REM sleep cycles, and wakefulness. Key brain systems include GABAergic neurons in the pre-optic area, the neuropeptide orexin in lateral hypothalamic neurons, histaminergic neurons in the hypothalamus, monoaminergic (norepinephrine and serotonin) and acetylcholinergic nuclei in the brainstem, and the brain’s adenosine system, all of which work in integrated circuits to control sleep and wakefulness. Overlaid on sleep-wake cycles are circadian rhythms, and the crucial role played by the suprachiasmatic nucleus in entraining such rhythms to environmental light.

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40

Wever, R. A. Circadian System of Man : Results of Experiments under Temporal Isolation. Springer London, Limited, 2013.

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41

Wever, R. A. The Circadian System of Man : Results Of Experiments Under Temporal Isolation. Springer, 2011.

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42

The Circadian System of Man : Results of Experiments Under Temporal Isolation. Springer, 2011.

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43

Lockley, Steven W. Principles of sleep–wake regulation. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198778240.003.0002.

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The 24-hour sleep–wake cycle is generated by two oscillatory processes: an endogenous hypothalamic circadian pacemaker and a sleep- and wake-dependent homeostat. These processes combine to maintain a consolidated bout of sleep at night and relatively stable waking function across the day. They also combine to determine ‘diurnal preference’—whether one is a ‘lark’ or an ‘owl’—a reflection of the phase relationship between the circadian and homeostatic processes. These processes are affected directly by light, either through resetting of the circadian pacemaker or its direct alerting effects. Sleep deficiency and circadian disruption have been associated with a higher risk of chronic disease, although the methodology for assessing these exposures is not optimal. Both sleep and the circadian system also have myriad influences on other aspects of our physiology, behaviour, and metabolism; therefore, steps should be taken to reduce their potential confounding effects in epidemiological studies.

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44

Schulkin, Jay. Evolution and Diversification of Function of an Information Molecule. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780198793694.003.0002.

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Chapter 2 begins with a depiction of the evolutionary origins of CRF in living things. CRF appears to date back hundreds of millions of years. It is found in diverse invertebrates, including flies and bees. Invertebrates’ brains look nothing like those of vertebrates except for the diverse information molecules that underlie both brain systems. There is no clear anatomical organ like the HPA axis in invertebrates, yet information molecules, including CRF, are just as important to invertebrate functioning as they are to vertebrates. CRF in invertebrates is linked to basic regulatory functions such as osmotic regulation, food intake, learning, and circadian rhythmicity. There are many examples of regulatory molecules that, over time, become adapted to serve multiple functions. Once a gene for a potent regulatory molecule exists, the potential for the differentiation of function, regulation, and mode of action exist as well.

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45

Scott, Jan. Psychological interventions for early stage bipolar disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0011.

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Clinical staging and early intervention models used in psychosis and depression have only recently been applied to individuals ‘at risk’ of bipolar disorder (BD), or experiencing a first episode of BD. This chapter briefly discusses the concept of staging and then reviews ongoing research into the adaptation and use of psychological interventions in ‘at risk’ and ‘first BD episode’ populations. Evidence indicates that the current interventions may not sufficiently target specific developmentally normal changes in cognitive–emotional and sleep–circadian regulation systems that may act as triggers for mood episodes. So the chapter discusses how to tackle these ‘dysregulations’ and how to ensure any ‘early stage’ therapy is sufficiently flexible to tackle the range of problems experienced, including mood symptoms, harmful alcohol or substance use, and/or co-morbid physical ill-health and that the therapy models must take into account that not all individuals in high-risk populations actually develop BD.

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46

Reading, Paul J. Neurological diseases and their effects on the sleep–wake cycle. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0035.

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This chapter addresses current neurobiological knowledge of how wake- and sleep-promoting systems interact to produce the daily circadian rhythm of wake and sleep and how this may be adversely affected by a variety of neurological diseases. The crucial importance of sleep quality for optimal brain function is stressed and the potential hazards of prolonged wakefulness highlighted. Insomnia relating to either sleep onset or maintenance is common and increases with normal aging. Many neurodegenerative diseases such as Alzheimer disease appear to enhance the effects of aging on the sleep–wake cycle, with increased fragmentation and reduced deep sleep. Focal pathology in the thalamus or sometimes the hypothalamus may produce striking insomnia, as may several autoimmune encephalitides. Hypersomnia is most often secondary to poor-quality nocturnal sleep, but may also relate to discrete hypothalamic pathology or traumatic head injury. The effects of epilepsy and its treatment on sleep can be significant and are discussed.

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47

Reebs, Stephan G. Effects of nonphotic factorstors on the circadian system of house sparrows and Syrian hamsters. 1989.

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48

White, William B. Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics. Humana, 2015.

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49

White, William B. Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics. Humana Press, 2000.

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50

White, William B. Blood Pressure Monitoring in Cardiovascular Medicine and Therapeutics. Springer London, Limited, 2007.

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