Littérature scientifique sur le sujet « Cholesterol Physiological effect »
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Articles de revues sur le sujet "Cholesterol Physiological effect"
Khudiar, Khalisa Kadim. « Effect of Magnetic Water on Some Physiological Aspects of Adult Male Rabbits ». Iraqi Journal of Veterinary Medicine 36, no 0E (4 avril 2012) : 120–26. http://dx.doi.org/10.30539/iraqijvm.v36i0e.405.
Texte intégralNgestiningsih, Dwi, Rejeki Andayani Rahayu et Lusiana Batubara. « Effect of Superoxide Dismutase (SOD) Supplementation on Plasma Levels of Malondialdehyde (MDA), Total Cholesterol and LDL Cholesterol in the Elderly ». Journal of Biomedicine and Translational Research 5, no 2 (10 octobre 2019) : 29–33. http://dx.doi.org/10.14710/jbtr.v5i2.4679.
Texte intégralFreeman, Dilys J., Christopher J. Packard, James Shepherd et Dairena Gaffney. « Polymorphisms in the Gene Coding for Cholesteryl Ester Transfer Protein are Related to Plasma High-Density Lipoprotein Cholesterol and Transfer Protein Activity ». Clinical Science 79, no 6 (1 décembre 1990) : 575–81. http://dx.doi.org/10.1042/cs0790575.
Texte intégralIkram, Naila, Shehzada A. A. Babar, Tahir Aslam, Hira Malik, Natasha Zahid et Anas Bin Tariq. « Physiological effect of cholecystectomy on lipid profile of patients with cholelithiasis ». International Journal of Scientific Reports 6, no 7 (23 juin 2020) : 243. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20202638.
Texte intégralDomingues, Marco M., Bárbara Gomes, Axel Hollmann et Nuno C. Santos. « 25-Hydroxycholesterol Effect on Membrane Structure and Mechanical Properties ». International Journal of Molecular Sciences 22, no 5 (4 mars 2021) : 2574. http://dx.doi.org/10.3390/ijms22052574.
Texte intégralReis, S. A., L. L. Conceição, D. D. Rosa, N. P. Siqueira et M. C. G. Peluzio. « Mechanisms responsible for the hypocholesterolaemic effect of regular consumption of probiotics ». Nutrition Research Reviews 30, no 1 (20 décembre 2016) : 36–49. http://dx.doi.org/10.1017/s0954422416000226.
Texte intégralPOST, Sabine M., Jaap TWISK, L. V. D. FITS, Elly C. M. DE WIT, Marco F. M. HOEKMAN, Willem H. MAGER et Hans M. G. PRINCEN. « Lipoprotein cholesterol uptake mediates up-regulation of bile-acid synthesis by increasing cholesterol 7α-hydroxylase but not sterol 27-hydroxylase gene expression in cultured rat hepatocytes ». Biochemical Journal 341, no 2 (8 juillet 1999) : 339–46. http://dx.doi.org/10.1042/bj3410339.
Texte intégralSeltman, H., W. Diven, M. Rizk, B. J. Noland, R. Chanderbhan, T. J. Scallen, G. Vahouny et A. Sanghvi. « Regulation of bile-acid synthesis. Role of sterol carrier protein2 in the biosynthesis of 7α-hydroxycholesterol ». Biochemical Journal 230, no 1 (15 août 1985) : 19–24. http://dx.doi.org/10.1042/bj2300019.
Texte intégralLI, Feng, et Y. David HUI. « Synthesis and secretion of the pancreatic-type carboxyl ester lipase by human endothelial cells ». Biochemical Journal 329, no 3 (1 février 1998) : 675–79. http://dx.doi.org/10.1042/bj3290675.
Texte intégralStoll, Peter, Andreas Gutzwiller, Martin Jost, Heiner Schneeberger, Robert Sieber, Hannes B. Staehelin, Christian Steffen et Guenther Ritzel. « Short-term effect of whole milk and milk fermented by Pseudomonas fluorescens on plasma lipids in adult boars ». British Journal of Nutrition 66, no 1 (juillet 1991) : 129–38. http://dx.doi.org/10.1079/bjn19910016.
Texte intégralThèses sur le sujet "Cholesterol Physiological effect"
Volk, Catherine B. « Role of inhibition of protein prenylation in the cholesterol-dependent and cholesterol-independent effects of simvastatin ». Virtual Press, 2006. http://liblink.bsu.edu/uhtbin/catkey/1339597.
Texte intégralDepartment of Biology
Jain, Deepak M. « Effect of corn fibre oil and its constituents on cholesterol metabolism and intestinal sterol transporter gene expression in hamsters ». Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98732.
Texte intégralBarake, Roula. « Effects of plant sterols and glucomannan on parameters of cholesterol kinetics in hyperlipidemic individuals with and without type 2 diabetes ». Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=83964.
Texte intégralVanstone, Catherine A. « Influence of phytosterols versus phytostanols on plasma lipid levels and cholesterol metabolism in hypercholesterolemic humans ». Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=33854.
Texte intégralVarady, Kristina A. « Effect of plant sterol supplementation and endurance training on cardiovascular disease risk parameters and cholesterol kinetics in previously sedentary hypercholesterolemic adults ». Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111831.
Texte intégralObjective. The aim of this study was to examine the independent and combined effects of plant sterols and exercise on blood lipid levels, and LDL particle size in previously sedentary, hypercholesterolemic adults. An additional objective of this trial was to assess the underlying mechanism by which this combination therapy modulates whole body cholesterol metabolism, to in turn improve lipid profiles.
Methods. In an 8-week, parallel-arm trial, 84 subjects were randomized to 1 of 4 interventions: (1) plant sterols and exercise,(2) plant sterols alone, (3) exercise alone, or (4) control. Blood lipid concentrations were measured using enzymatic kits, and LDL particle size was assessed using polyacrylamide gel electrophoresis. Cholesterol absorption and synthesis were determined using the single isotope single tracer technique and the deuterium incorporation approach, respectively.
Results. Plant sterol supplementation decreased (P < 0.01) total cholesterol concentrations by 8.2% when compared to baseline. Exercise increased (P < 0.01) HDL cholesterol levels by 7.5% while decreasing (P < 0.01) triglyceride concentrations by 13.3% when compared to baseline. Exercise reduced (P < 0.05) post-treatment LDL peak particle size from 255 to 253 A, and decreased (P < 0.05) the proportion of large LDL particles by 13.1%. Plant sterols had no effect on particle size distribution. Plant sterol supplementation decreased (P < 0.01) intestinal cholesterol absorption by 18%, while exercise had no effect on cholesterol absorption. Non-significant increases in cholesterol synthesis rates of 63%, 59%, and 57%, were observed in the combination, exercise, and plant sterol groups, respectively, relative to control.
Conclusion. These findings suggest that this combination therapy yields the most favourable alterations in lipid profiles when compared to each intervention alone. This combined intervention exerts its beneficial effects on lipid profiles by suppressing intestinal cholesterol absorption. Therefore, this lifestyle therapy may be an effective means of decreasing the risk of cardiovascular disease in hypercholesterolemic adults.
Journoud, Mélanie. « The effect of plant sterols on lipid profiles and cholesterol kinetics of hypercholesterolemic individuals with type 2 diabetes compared with non-diabetic controls / ». Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=80296.
Texte intégralPlasma total cholesterol (TC) decreased with placebo and PS (10.9% and 9.7% in non-diabetic versus 11.6% and 13.6% in diabetic participants, p < 0.05). Plasma low-density lipoprotein cholesterol (LDL) significantly decreased with PS in both groups. The reduction in LDL with PS was greater in diabetic compared to non-diabetic individuals (29.8% versus 14.9%, p < 0.05). Cholesterol absorption decreased on average (p = 0.06) by 26.5% with PS compared with placebo in the diabetic group only. Therefore, a controlled heart healthy diet reduced TC and LDL concentrations in non-diabetic and diabetic individuals. Adding PS as adjuncts to a hypocholesterolemic dietary treatment was associated with lower LDL concentrations and cholesterol absorption in hypercholesterolemic participants with type 2 diabetes.
Anema, Richard Flagg. « A single case study of the acute effects of strenuous exercise on blood lipids ». Scholarly Commons, 1991. https://scholarlycommons.pacific.edu/uop_etds/2210.
Texte intégralBone, Emily C. « A determination of the effects of nutrition and physical activity education on cholesterol levels over time in the worksite ». Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1246465.
Texte intégralDepartment of Family and Consumer Sciences
Mazier, Marie Jeanne Patricia. « Influence of diet fat saturation on rates of cholesterol synthesis and esterification in healthy young men ». Thesis, 1994. http://hdl.handle.net/2429/8877.
Texte intégral« Effects of octadecaenoic acids and apple polyphenols on blood cholesterol ». 2007. http://library.cuhk.edu.hk/record=b5893326.
Texte intégralThesis (M.Phil.)--Chinese University of Hong Kong, 2007.
Includes bibliographical references (leaves 148-173).
Abstracts in English and Chinese.
ACKNOWLEDGEMENTS --- p.i
ABSTRACT --- p.ii
LIST OF ABBREVIATIONS --- p.vi
TABLE OF CONTENTS --- p.x
Chapter CHAPTER 1 --- GENERAL INTRODUCTION
Chapter 1.1 --- Introduction to Cholesterol and Its Related Diseases --- p.1
Chapter 1.1.1 --- Chemistry of cholesterol --- p.1
Chapter 1.1.2 --- Physiological importance of cholesterol --- p.1
Chapter 1.1.3 --- Pathological effects of cholesterol --- p.3
Chapter 1.1.3.1 --- Mechanism of atherosclerosis --- p.3
Chapter 1.2 --- Cholesterol Homeostasis --- p.6
Chapter 1.2.1 --- Liver as the main organ for cholesterol metabolism --- p.6
Chapter 1.2.2 --- Regulatory sites of cholesterol metabolism --- p.6
Chapter 1.2.2.1 --- Regulation of cholesterol absorption by acyl coenzyme A: cholesterol acyltransferase (ACAT) --- p.6
Chapter 1.2.2.2 --- Sterol regulatory element-binding protein 2 (SREBP-2) as a transcription factor for 3 -hydroxy-3 -methylglutaryl coenzyme A reductase (HMGR) and low-density lipoprotein receptor (LDLR) --- p.10
Chapter 1.2.2.3 --- Roles ofLDLR --- p.11
Chapter 1.2.2.4 --- Rate limiting role of HMGR in cholesterol de novo synthesis --- p.14
Chapter 1.2.2.5 --- Roles of liver-X-receptor-a (LXR-a) in cholesterol catabolism --- p.16
Chapter 1.2.2.6 --- Roles of CYP7A1 in catabolism of cholesterol into bile acids --- p.19
Chapter 1.2.2.7 --- Roles of cholesterol ester transfer protein (CETP) in maintaining cholesterol distribution in blood --- p.22
Chapter CHAPTER 2 --- EFFECT OF OCTADECAENOIC ACIDS ON BLOOD CHOLESTEROL IN HAMSTERS
Chapter 2.1 --- Introduction --- p.25
Chapter 2.1.1 --- Effects of polyunsaturated fatty acids (PUFAs) on blood cholesterol --- p.25
Chapter 2.1.2 --- Differential effects of 18-C PUFAs on lowering blood cholesterol in vivo --- p.25
Chapter 2.1.3 --- "Structures, metabolism and conjugation of octadecaenoic acids (ODA)" --- p.26
Chapter 2.1.4 --- Objectives --- p.26
Chapter 2.2 --- Experiment 1 --- p.28
Chapter 2.2.1 --- Materials and methods --- p.28
Chapter 2.2.1.1 --- Experimental fatty acids --- p.28
Chapter 2.2.1.1.1 --- Isolation of LN from flaxseed --- p.28
Chapter 2.2.1.1.2 --- Isolation of CLN from tung seed --- p.28
Chapter 2.2.1.2 --- Animals --- p.29
Chapter 2.2.1.3 --- Diets --- p.30
Chapter 2.2.1.4 --- Plasma lipid measurements --- p.30
Chapter 2.2.1.5 --- Plasma CETP activity measurement --- p.30
Chapter 2.2.1.6 --- "Measurement of liver SREBP-2, LDLR, HMGR and CYP7A1 protein abundance by Western blotting" --- p.34
Chapter 2.2.1.7 --- "Measurement of hepatic SREBP-2, LDLR, HMGR, LXR, CYP7A1, CETP, SR-B1 and LCAT mRNA by real time PCR" --- p.35
Chapter 2.2.1.7.1 --- Extraction of mRNA --- p.35
Chapter 2.2.1.1.2 --- Complementary DNA synthesis --- p.36
Chapter 2.2.1.7.3 --- Real-time polymerase chain reaction (PCR) anaylsis --- p.36
Chapter 2.2.1.8 --- Determination of cholesterol in liver --- p.37
Chapter 2.2.1.9 --- Determination of fecal neutral and acidic sterols --- p.38
Chapter 2.2.1.9.1 --- Determination of fecal neutral sterols --- p.39
Chapter 2.2.1.9.2 --- Determination of fecal acidic sterols --- p.41
Chapter 2.2.1.10 --- Statistics --- p.43
Chapter 2.2.2 --- Results --- p.44
Chapter 2.2.2.1 --- Growth and food intake --- p.44
Chapter 2.2.2.2 --- Organ weights --- p.44
Chapter 2.2.2.3 --- "Effects of ODA on serum TC, TG and HDL-C" --- p.44
Chapter 2.2.2.4 --- Effect of ODA on liver cholesterol --- p.48
Chapter 2.2.2.5 --- Effect of ODA on fecal neutral sterol output --- p.48
Chapter 2.2.2.6 --- Effect of ODA on fecal acidic sterol output --- p.48
Chapter 2.2.2.7 --- Effect of ODA on cholesterol balance in hamsters --- p.52
Chapter 2.2.2.8 --- Effect of ODA on plasma CETP activity --- p.52
Chapter 2.2.2.9 --- Correlation between blood TC and liver cholesterol --- p.52
Chapter 2.2.2.10 --- Correlation between blood HDL-C and liver cholesterol --- p.52
Chapter 2.2.2.11 --- Correlation between blood nHDL/HDL ratio and liver cholesterol --- p.52
Chapter 2.2.2.12 --- Effect ofODA on liver SREBP-2 immunoreactive mass --- p.58
Chapter 2.2.2.13 --- Effect of ODA on liver LDLR immunoreactive mass --- p.58
Chapter 2.2.2.14 --- Effect of ODA on liver HMGR immunoreactive mass --- p.58
Chapter 2.2.2.15 --- Effect of ODA on liver LXR immunoreactive mass --- p.58
Chapter 2.2.2.16 --- Effect of ODA on liver CYP7A1 immunoreactive mass --- p.63
Chapter 2.2.2.17 --- Effects ofODA on hepatic CETP mRNA --- p.65
Chapter 2.2.2.18 --- Effects of ODA on hepatic LDLR mRNA --- p.65
Chapter 2.2.2.19 --- Effects of ODA on hepatic LXR mRNA --- p.65
Chapter 2.2.2.20 --- Effects of ODA on hepatic CYP7A1 mRNA --- p.65
Chapter 2.3 --- Experiment 2 --- p.70
Chapter 2.3.1 --- Materials and Methods --- p.70
Chapter 2.3.1.1 --- Experimental diets --- p.70
Chapter 2.3.1.2 --- Animals --- p.70
Chapter 2.3.1.3 --- Intestinal acyl coenzyme A: cholesterol acyltransferase (ACAT) activity measurement --- p.70
Chapter 2.3.1.3.1 --- Preparation of intestinal microsome --- p.71
Chapter 2.3.1.3.2 --- ACAT activity assay --- p.71
Chapter 2.3.2 --- Results --- p.73
Chapter 2.3.2.1 --- Growth and food intake --- p.73
Chapter 2.3.2.2 --- Organ weights --- p.73
Chapter 2.3.2.3 --- "Effect of ODA on serum TC, TG and HDL-C" --- p.73
Chapter 2.3.2.4 --- Effect of ODA feeding on fecal neutral sterol content --- p.77
Chapter 2.3.2.5 --- Effect of ODA feeding on fecal acidic sterol content --- p.77
Chapter 2.3.2.6 --- Effect of ODA feeding on intestinal acyl coenzyme A: acyl cholesterol transferase (ACAT) activity --- p.77
Chapter 2.4 --- Discussion --- p.81
Chapter CHAPTER 3 --- EFFECT OF OCTADECAENOIC ACIDS ON CHOLESTEROL-REGULATING GENES IN HepG2
Chapter 3.1 --- Introduction --- p.86
Chapter 3.1.1 --- HepG2 as a model of cholesterol regulation --- p.86
Chapter 3.1.2 --- Effect of polyunsaturated fatty acids (PUFAs) on cholesterol regulating genes in cultured cells --- p.87
Chapter 3.1.3 --- Objectives --- p.89
Chapter 3.2 --- Materials and Methods --- p.90
Chapter 3.2.1 --- Cell culture --- p.90
Chapter 3.2.2 --- "Measurement of SREBP-2, LDLR, HMGR and CYP7A1 protein abundance by Western blotting" --- p.92
Chapter 3.2.3 --- "Measurement of cellular SREBP-2, LDLR, HMGR, LXR, CYP7A1 and CETP mRNA by real time PCR" --- p.93
Chapter 3.2.4 --- Statistics --- p.93
Chapter 3.3 --- Results --- p.95
Chapter 3.3.1 --- Effect of ODA on HepG2 SREBP-2 immunoreactive mass --- p.95
Chapter 3.3.2 --- Effect of ODA on HepG2 HMGR immunoreactive mass --- p.95
Chapter 3.3.3 --- Effect of ODA on HepG2 LDLR immunoreactive mass --- p.95
Chapter 3.3.4 --- Effect of ODA on HepG2 LXR immunoreactive mass --- p.95
Chapter 3.3.5 --- Effect of ODA on HepG2 CYP7A1 immunoreactive mass --- p.96
Chapter 3.3.6 --- Effect of ODA supplementation on HepG2 SREBP-2 mRNA expression --- p.102
Chapter 3.3.7 --- Effect of ODA supplementation on HepG2 SREBP-2 mRNA expression --- p.102
Chapter 3.3.8 --- Effect of ODA supplementation on HepG2 LDLR mRNA expression --- p.102
Chapter 3.3.9 --- Effect of ODA supplementation on HepG2 LXR mRNA expression --- p.106
Chapter 3.3.10 --- Effect of ODA supplementation on HepG2 CYP7A1 mRNA expression --- p.106
Chapter 3.3.11 --- Effect of ODA supplementation on HepG2 CETP mRNA expression --- p.106
Chapter 3.4 --- Discussion --- p.110
Chapter CHAPTER 4 --- EFFECT OF APPLE POLYPHENOLS ON BLOOD CHOLESTEROL IN HAMSTERS
Chapter 4.1 --- Introduction --- p.114
Chapter 4.1.1 --- Apple is a commonly consumed fruit worldwide --- p.114
Chapter 4.1.2 --- Potential health effects of apples --- p.114
Chapter 4.1.3 --- Abundance of polyphenols in apple --- p.115
Chapter 4.1.4 --- Fuji variety of apple --- p.116
Chapter 4.1.5 --- Objectives --- p.116
Chapter 4.2 --- Materials and Methods --- p.118
Chapter 4.2.1 --- Isolation of AP --- p.118
Chapter 4.2.2 --- Characterization of AP extract --- p.118
Chapter 4.2.3 --- Effect of AP on CETP activity in vitro --- p.118
Chapter 4.2.4 --- Effect of AP on blood cholesterol in hamsters --- p.119
Chapter 4.2.4.1 --- Animals --- p.119
Chapter 4.2.4.2 --- Diets --- p.120
Chapter 4.2.4.3 --- Plasma lipids measurement --- p.121
Chapter 4.2.4.4 --- Plasma CETP activity measurement and immunoreactive mass by Western blotting --- p.123
Chapter 4.2.4.5 --- "Measurement of liver SREBP-2, LDL-R, HMG-R and CYP7A1 protein abundance by Western blotting" --- p.124
Chapter 4.2.4.6 --- Statistics --- p.124
Chapter 4.3 --- Results --- p.125
Chapter 4.3.1 --- Polyphenol content in AP --- p.125
Chapter 4.3.2 --- Effect of AP on CETP activity in vitro --- p.125
Chapter 4.3.3 --- Growth and food intake --- p.128
Chapter 4.3.4 --- Organ weights --- p.128
Chapter 4.3.5 --- Effect of AP supplementation on the plasma lipid profile of hamsters --- p.131
Chapter 4.3.6 --- Effect of AP feeding on plasma CETP activity of the hamsters --- p.131
Chapter 4.3.7 --- Effect of AP on plasma CETP immunoreactive mass --- p.134
Chapter 4.3.8 --- Effect of AP on liver SREBP-2 immunoreactive mass --- p.134
Chapter 4.3.9 --- Effect of AP on liver LDLR immunoreactive mass --- p.134
Chapter 4.3.10 --- Effect of AP on liver HMGR immunoreactive mass --- p.134
Chapter 4.3.11 --- Effect of AP on liver CYP7A1 immunoreactive mass --- p.134
Chapter 4.3.12 --- Effect of AP on liver cholesterol level --- p.140
Chapter 4.4 --- Discussion --- p.142
Chapter CHAPTER 5 --- CONCLUSION --- p.145
REFERENCES --- p.148
Livres sur le sujet "Cholesterol Physiological effect"
G, Williams David. Cholesterol. Ingram, TX : Mountain Home Pub., 1988.
Trouver le texte intégralMattjus, Peter. Interaction of cholesterol with sphingomyelins and phosphatidylcholines in model membranes. Åbo : Åbo Akademis Förlag, 1996.
Trouver le texte intégralThe cholesterol hoax : 101+ lies. Carson City, NV : Bridger House Publishers, 1997.
Trouver le texte intégralRogers, Sherry A. The cholesterol hoax. Sarasota, Fla : Sand Key Co., 2008.
Trouver le texte intégralSymes, David. Cholesterol : Reducing your risk. London : Optima, 1994.
Trouver le texte intégralAssociation, Family Heart, dir. Cholesterol : Reducing your risk. London : Macdonald Optima, 1990.
Trouver le texte intégralLeppamäki, Petra. Interrelationship between sterol and phospholipid homeostasis in cultured fibroblasts. Åbo : Åbo Akademis Förlag, 2002.
Trouver le texte intégralFischer, William L. Secrets to a healthy heart and low cholesterol : Proven guidelines and documented facts for the natural self-treatment and prevention of heart disease, high cholesterol, and other related ailments in conjunction with the world-famous breakthrough formula by Prof. Flemming Norgaard, M.D., D.D.S. Canfield, Ohio : Fischer Pub. Corp., 1993.
Trouver le texte intégralAbastado, Philippe. Cholestérol : Maladie réelle et malade imaginaire. Le Plessis-Robinson : Institut Synthélabo pour le progrès de la connaissance, 1998.
Trouver le texte intégralservice), ScienceDirect (Online, dir. The HDL handbook : Biological functions and clinical implications. London : Academic, 2010.
Trouver le texte intégralChapitres de livres sur le sujet "Cholesterol Physiological effect"
Drexel, Heinz. « Dyslipidaemia ». Dans The ESC Handbook on Cardiovascular Pharmacotherapy, sous la direction de Heinz Drexel et Massimo Francesco Piepoli, 33–48. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198759935.003.0002.
Texte intégralDrexel, Heinz. « Dyslipidaemia ». Dans The ESC Handbook on Cardiovascular Pharmacotherapy, sous la direction de Heinz Drexel et Massimo Francesco Piepoli, 33–48. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198759935.003.0002_update_001.
Texte intégralCegla, Jaimini, et James Scott. « Lipid disorders ». Dans Oxford Textbook of Medicine, sous la direction de Timothy M. Cox, 2055–97. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0232.
Texte intégralMorales-Villegas, Enrique C., et Kausik K. Ray. « PCSK9 Inhibition with Evolocumab Reaching Physiologic LDL-C Levels for Reducing Atherosclerotic Burden and Cardiovascular Disease-The Full Landscape ». Dans Frontiers in Cardiovascular Drug Discovery : Volume 4, 148–85. BENTHAM SCIENCE PUBLISHERS, 2019. http://dx.doi.org/10.2174/9781681083995118040007.
Texte intégralActes de conférences sur le sujet "Cholesterol Physiological effect"
Sun, Hyung Jin, Yunjie Wang et Katherine Yanhang Zhang. « Changes in the Mechanical Properties of Arterial Elastin With Cholesterol Effect ». Dans ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14591.
Texte intégralSudirman, Muhamad Seto. « Effectiveness of Ficus Elastica Roxb. Ex Hornem Leaf Extract in Reducing Total Cholesterol Level in High Fat Induced Diet Wistar Male Rats ». Dans The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.05.10.
Texte intégralMeade, T. W. « THE EPIDEMIOLOGY OF HAEMOSTATIC AND OTHER VARIABLES IN CORONARY ARTERY DISEASE ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643833.
Texte intégral