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Bibliographies thématiques / CD1 proteins / Livres

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Livres sur le sujet « CD1 proteins »

Auteur : Grafiati

Publié le 25 janvier 2025

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1

Pontus, Aspenstrøm, dir. The pombe Cdc 15 homology proteins. Austin, Tex : Landes Bioscience, 2009.

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2

Anderluh, Gregor, et Robert Gilbert, dir. MACPF/CDC Proteins - Agents of Defence, Attack and Invasion. Dordrecht : Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-8881-6.

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3

Metzner, Karin. Zur CD8+-T-Lymphozyten : Interleukin-16 und Identifikation und Charakterisierung anderer sezernierter Proteine. [s.l.] : [s.n.], 1997.

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4

Pumphrey, Nicholas Jonathan. Delineating the signalling potential of the cytoplasmic tail of CD31, and related protein CD66a. Birmingham : University of Birmingham, 2000.

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5

B, Kastan M., et Imperial Cancer Research Fund (Great Britain), dir. Checkpoint controls and cancer. Plainview, NY : Cold Spring Harbor Laboratory Press, 1997.

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6

A, Schreiber, dir. Primate phylogeny from a human perspective : A study based on the immunological technique of comparative determinant analysis (CDA). Stuttgart : G. Fischer, 1996.

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7

Workshop on Mechanisms and Specificity of HIV Entry into Host Cells (1989 San Francisco, Calif.). Mechanisms and specificity of HIV entry into host cells. New York : Plenum Press, 1991.

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8

Gilbert, Robert, et Gregor Anderluh. MACPF/CDC Proteins - Agents of Defence, Attack and Invasion. Ingramcontent, 2014.

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9

Gilbert, Robert, et Gregor Anderluh. MACPF/CDC Proteins - Agents of Defence, Attack and Invasion. Springer, 2016.

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10

Gilbert, Robert, et Gregor Anderluh. MACPF/CDC Proteins - Agents of Defence, Attack and Invasion. Springer London, Limited, 2014.

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11

Patterson, Marc C. Congenital Disorders of Glycosylation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0066.

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Congenital disorders of glycosylation (CDG) comprise a family of multisystem diseases in which N- and O-linked glycosylation and glypiation of a variety of proteins and lipids is deficient. The hypoglycosylation of multiple glycoconjugates impairs normal development of the brain (and other organs), and is associated with both episodic and chronic organ dysfunction. Developmental disorders; seizures; strokelike episodes (and stroke); hearing and visual loss; peripheral neuropathy; coagulopathy; and immune, liver, endocrine, cardiac, and cutaneous manifestations may occur in varying combinations. Specific therapy is available for MPI-CDG and SLC35C2-CDG. Most forms of O-linked CDG affect muscle; these include congenital muscular dystrophies and limb girdle dystrophies.

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12

Livingston, Schuyler, Benjamin Young, Martin Markowitz, Poonam Mathur et Bruce L. Gilliam. HIV Virology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190493097.003.0017.

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HIV is a member of the lentivirus subfamily of retroviruses. Two distinct groups of viruses are pathogenic in humans: HIV-1 and HIV-2. Both are transmitted sexually and known to cause immunodeficiency disease. HIV enters the cell through use of the CD4 receptor and chemokine co-receptors, primarily CCR5 and CXCR4. The viral genome is transcribed from RNA to DNA by reverse transcriptase and integrated into the host genome by integrase. The HIV genome encodes 15 proteins, comprising three categories: structural, regulatory, and accessory. After budding from the host cell, the virus matures into its infectious form through cleavage of viral precursor proteins by protease.

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13

Schreiber, Arnd, et Klausdieter Bauer. Primate Phylogeny from a Human Perspective : A Study Based on the Immunological Technique of Comparative Determinant Analysis(Cda). Fischer Gustav Verlag GmbH & Co. KG, 1995.

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14

Schreiber, Arnd, et Klausdieter Bauer. Primate Phylogeny from a Human Perspective : A Study Based on the Immunological Technique of Comparative Determinant Analysis (Cda). VCH Publishers, 1995.

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15

Tausig, Benjamin. Bangkok is Ringing. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780190847524.001.0001.

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Bangkok Is Ringing is an on-the-ground sound studies analysis of the political protests that transformed Thailand in 2010–11. Bringing the reader through sixteen distinct “sonic niches” where dissidents used media to broadcast to both local and diffuse audiences, the book catalogues these mass protests in a way that few movements have ever been catalogued. The Red Shirt and Yellow Shirt protests that shook Thailand took place just before other international political movements, including the Arab Spring and Occupy Wall Street. Bangkok Is Ringing analyzes the Thai protests in comparison with these, seeking to understand the logic not only of political change in Thailand, but across the globe. The book is attuned to sound in a great variety of forms. The author traces the history and use in protest of specific media forms, including community radio, megaphones, CDs, and live concerts. The research took place over the course of sixteen months, and the author worked closely with musicians, concert promoters, activists, and rank-and-file protesters. The result is a detailed and sensitive ethnography that argues for an understanding of sound and political movements in tandem. In particular, it emphasizes the necessity of thinking through constraint as a fundamental condition of both political movements and the sound that these movements produce. In order to produce political transformations, the book argues, dissidents must be sensitive to the ways that their sounding is constrained and channeled.

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16

Colbert, Robert A., et Paul Bowness. Immune mechanisms : HLA-B27. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0006.

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HLA-B27 is present in the majority of patients with ankylosing spondylitis (AS). Although we have learned a considerable amount about the natural immunologic function of HLA class I proteins, this has not provided a definitive mechanism of AS pathogenesis. While HLA-B27 is adept at presenting antigenic peptides to CD8+ T cells, ‘arthritogenic’ peptides targeted by a cross-reactive T or natural killer cell response have not been described, nor have autoreactive T cells been found. Newer concepts have evolved based on the propensity of HLA-B27 to ‘misbehave’, both inside cells and on the cell surface. Misfolded HLA-B27 molecules may stimulate an endoplasmic reticulum stress response, promoting production of IL-23 and then IL-17 and related cytokines. Aberrant cell-surface HLA-B27 molecules are ligands for natural killer and related immunoreceptors, and recognition can lead to IL-17 proinflammatory responses. There is growing evidence to suggest that these aberrant behaviours contribute to AS pathogenesis.

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17

Querschnitts-Leitlinien zur Therapie mit Blutkomponenten und Plasmaderivaten. Deutscher Ärzteverlag, 2021. http://dx.doi.org/10.47420/9783769137309.

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Empfehlungen zur strengen Indikationsstellung von Blutprodukten Die Querschnitts-Leitlinien stehen in engem Zusammenhang mit den ebenfalls von der Bundesärztekammer herausgegebenen Richtlinien zur Therapie mit Blutkomponenten und Plasmaderivaten (Hämotherapie) nach §§ 12a und 18 Transfusionsgesetz. Beide Werke werden jeweils aufeinander abgestimmt. Die Kenntnis der Querschnitts-Leitlinien ist für jeden Arzt, der Blutprodukte anwendet und Hämostasestörungen behandelt, unerlässlich. Die in den Leitlinien formulierten Handlungsempfehlungen berücksichtigen den aktuellen Stand der Wissenschaft. Weitere Charakteristika: konkrete Handlungsempfehlungen Hervorhebung der wissenschaftlichen Eviden kritische Bewertung von hämotherapeutischen Behandlungen Berücksichtigung der Problematik des „Off-Label-Use“ in allgemeinen Ausführungen und konkreten Handlungsempfehlungen Indikationsstellung: der Transfusion von Erythrozyten und Thrombozyten für Plasma zur therapeutischen Anwendung und Humanalbumin zur Bestrahlung von Blutprodukten zur Transfusion CMV- bzw. Parvovirus B19-getesteter sowie bestrahlter Blutprodukte für Prokoagulatoren (Fibrinogen, PPSB, Faktor VII, rekombinanter Faktor VIIa, Faktor XIII, Fibrinkleber) für Inhibitoren (Antithrombin, Protein C, rekombinantes aktiviertes Protein C, C1-Esterase-Inhibitor) für humane Immunglobuline Empfehlungen zur: Therapie mit Humanalbumin Hämophiliebehandlung Auswahl von Thrombozytenkonzentraten

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18

Düzgünes, Nejat. Mechanisms and Specificity of HIV Entry into Host Cells. Springer London, Limited, 2012.

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19

Düzgünes, Nejat. Mechanisms and Specificity of HIV Entry into Host Cells. Springer, 2012.

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20

Fas Signaling. Springer, 2006.

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21

Bosworth, Brian P., Brian R. Landzberg et Elisa McEachern. Neurological Manifestations of Gastrointestinal and Hepatic Diseases. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0190.

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Digestive diseases may have protean neurological manifestations, and should be considered during neurological evaluation of patients. Neurological manifestations of celiac disease (CD) may involve both central and peripheral nervous system, including syndromes of cerebellar and myoclonic ataxia, encephalopathy and dementia, seizures, CNS vasculitis and progressive multifocal leukoencephalopathy, peripheral neuropathy, and myopathy. Gluten sensitivity has been frequently implicated as a cause of neuropathy, with up to 49% of celiac disease patients experiencing some form of neuropathy and almost 40% meeting the criteria for peripheral neuropathy. Gluten ataxia is one of the most common neurological manifestations of celiac disease. Neurologic manifestations of inflammatory bowel disease (IBD) have long been recognized, with an incidence ranging from less than 1% to 35%. These manifestations may precede or, more commonly, follow a diagnosis of IBD. Hepatic encephalopathy, Wilson’s disease, and acute intermittent porphyria are examples of liver diseases associated with hepatic disorders.

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22

Corbeil, Denis. Prominin-1 : New Insights on Stem & Cancer Stem Cell Biology. Springer, 2012.

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23

Corbeil, Denis. Prominin-1 : New Insights on Stem and Cancer Stem Cell Biology. Springer London, Limited, 2012.

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24

Corbeil, Denis. Prominin-1 : New Insights on Stem & Cancer Stem Cell Biology. Springer, 2014.

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25

Corbeil, Denis. Prominin-1 : New Insights on Stem & Cancer Stem Cell Biology. Springer, 2012.

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26

Bhatnagar, Subhash C. Neuroscience for the Study of Communicative Disorders. 2^e éd. Lippincott Williams & Wilkins, 2001.

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27

Antonucci, Lauren A. High-Performance Nutrition for Masters Athletes. Human Kinetics, 2022. http://dx.doi.org/10.5040/9781718225275.

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“I wish I’d had this book 50 years ago … how much more effectively could we have prepared, how much better could we have performed, and how much healthier could we have been?” —Kathrine Switzer, Author of Marathon Woman, First Woman to Officially Register for and Run the Boston Marathon, and Cofounder and Board Chair of 261 Fearless Inc. “Nutrition becomes even more important as we age. High-Performance Nutrition for Masters Athletes gives you expert advice on how to fuel smarter, recover quicker, and enjoy moving at any age.” —Meb Keflezighi, Winner of the Boston Marathon, the New York City Marathon, and an Olympic Medal “It’s about time we have a fact-based nutrition bible for Masters athletes. Lauren Antonucci has delivered the guide that every Masters athlete needs to do what they love with more energy and less injury.” —Heidi Skolnik, MS, CDN, FACSM, President of Nutrition Conditioning LLC and Author of Nutrient Timing for Peak Performance “Lauren Antonucci’s advice is grounded in both science and real-world experience. High-Performance Nutrition for Masters Athletes will be a valuable resource for all athletes who are looking to get the best from ourselves as we get older.” —Gordon Bakoulis, Five-Time U.S. Olympic Marathon Trials Qualifier, Running Coach, and Editorial Director of New York Road Runners It’s an undisputed fact that the body’s physiological needs change as you age. But that doesn’t mean you can’t continue to compete and perform at your best. In High-Performance Nutrition for Masters Athletes, you’ll find practical advice for fueling your active lifestyle for decades to come. In this essential guide, you will learn the following: - Easy-to-follow fueling strategies for precompetition, competition, and recovery- Ways to energize with carbohydrate, build muscle with protein, and meet your hydration needs for optimal athletic performance- The role dietary supplements can play in your nutrition plan and the devastating effect of underfueling- The impact chronic conditions such as high blood pressure and cardiovascular disease have on nutritional needs- Sample meal plans to reset eating habits and meet the needs of the Masters competitor You’ll also find profiles of amazing athletes, from Olympians to everyday athletes, who have adapted their nutrition and training regimens to stay at the top of their sports, year in and year out. Comprehensive yet practical, High-Performance Nutrition for Masters Athletes provides the latest research, guidance, and strategies that you need to train harder, perform stronger, and recover faster. Fuel smart, and never let age slow you down.

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