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Bibliographies thématiques / Cardiovascular fluid mechanic / Livres

Livres sur le sujet « Cardiovascular fluid mechanic »

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Auteur : Grafiati

Publié le 14 mars 2023

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1

Pedrizzetti, Gianni, et Karl Perktold, dir. Cardiovascular Fluid Mechanics. Vienna : Springer Vienna, 2003. http://dx.doi.org/10.1007/978-3-7091-2542-7.

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2

Waite, Lee. Biofluid mechanics in cardiovascular systems. New York : McGraw-Hill, 2006.

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3

Pedrizzetti, Gianni. Fluid Mechanics for Cardiovascular Engineering. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-85943-5.

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4

P, Verdonck, et Perktold K, dir. Intra and extracorporeal cardiovascular fluid dynamics. Southampton : Computational Mechanics Publications, 1998.

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5

Yoganathan, A. P. (Ajit Prithiviraj), 1951- et Rittgers Stanley E. 1947-, dir. Biofluid mechanics : The human circulation. 2^e éd. Boca Raton : Taylor & Francis, 2012.

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6

1947-, Rittgers Stanley E., et Yoganathan A. P. 1951-, dir. Biofluid mechanics : The human circulation. Boca Raton : CRC/Taylor & Francis, 2007.

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7

B, Lumsden Alan, Kline William E. 1948-, Kakadiaris Ioannis A et SpringerLink (Online service), dir. Pumps and Pipes : Proceedings of the Annual Conference. Boston, MA : Springer Science+Business Media, LLC, 2011.

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8

Hayashi, K., Hiroyuki Abe et Sato M. Data book on mechanical properties of living cells, tissues, and organs. Tokyo : Springer, 1996.

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9

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. New York, NY : Springer New York, 2013.

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10

Pedrizzetti, Gianni, et Karl Perktold. Cardiovascular Fluid Mechanics. Springer London, Limited, 2014.

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11

(Editor), Gianni Pedrizzetti, et Karl Perktold (Editor), dir. Cardiovascular Fluid Mechanics (CISM International Centre for Mechanical Sciences). Springer, 2004.

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12

Pedrizzetti, Gianni. Fluid Mechanics for Cardiovascular Engineering : A Primer. Springer International Publishing AG, 2022.

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13

Pedrizzetti, Gianni. Fluid Mechanics for Cardiovascular Engineering : A Primer. Springer International Publishing AG, 2021.

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14

(Editor), K. Perktold, et P. Verdonck (Editor), dir. Intra and Extracorporeal Cardiovascular Fluid Dynamics (Advances in Fluid Mechanics). 2^e éd. WIT Press (UK), 2000.

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15

(Editor), K. Perktold, et P. Verdonck (Editor), dir. Intra and Extracorporeal Cardiovascular Fluid Dynamics : Vol. 2 - Fluid Structure Interaction (Advances in Fluid Mechanics). WIT Press (UK), 2000.

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16

Waite, Lee, et Jerry Fine. Applied Biofluid Mechanics. McGraw-Hill Professional, 2007.

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17

Vascular Grafts : Experiment and Modelling (Advances in Fluid Mechanics, Vol. 34). Computational Mechanics, Inc., 2003.

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18

Verdonck, P. Intra and Extracorporeal Cardiovascular Fluid Dynamics : Volume 1, General Principles in Application (Advances in Fluid Mechanics Vol 22). WIT Press (UK), 1998.

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19

Applied Biofluid Mechanics. McGraw-Hill Professional, 2007.

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20

Applied Biofluid Mechanics. McGraw-Hill Education, 2017.

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21

Colebourn, Claire, et Jim Newton. Field guide to critical care echocardiography. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198757160.003.0008.

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This chapter is intended as a summary or reference for clinician echocardiographers at the bedside. It gives step-by-step algorithms which address both commonly asked questions and clinical situations which require rapid decision-making using echocardiography in the critically ill. These algorithms cover fluid status and fluid responsiveness, cardiovascular parameters, assessment of the shocked and breathless patient including trauma, assessment of the patient with a clinical diagnosis of pulmonary embolism, and assessment of the unwell obstetric patient and patients who are weaning from mechanical ventilatory support.

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22

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics. Taylor & Francis Group, 2006.

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23

Farmakis, Dimitrios, John Parissis et Gerasimos Filippatos. Acute heart failure : epidemiology, classification, and pathophysiology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0051.

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Acute heart failure is defined as the rapid development or change of symptoms and signs of heart failure that requires urgent medical attention and usually hospitalization. Acute heart failure is the first reason for hospital admission in individuals aged 65 or more and accounts for nearly 70% of the total health care expenditure for heart failure. It is characterized by an adverse prognosis, with an in-hospital mortality rate of 4-7%, a 2-3-month post-discharge mortality of 7-11%, and a 2-3-month readmission rate of 25-30%. The majority of patients have a previous history of heart failure and present with normal or increased blood pressure, while about half of them have a preserved left ventricular ejection fraction. A high prevalence of cardiovascular or non-cardiovascular comordid conditions is further observed, including coronary artery disease, arterial hypertension, atrial fibrillation, diabetes mellitus, renal dysfunction, chronic lung disease, and anaemia. Different classification systems have been proposed for acute heart failure, reflecting the clinical heterogeneity of the syndrome; the categorization to acutely decompensated chronic heart failure vs de novo acute heart failure and to hypertensive, normotensive, and hypotensive acute heart failure are among the most widely used and clinically relevant classifications. The pathophysiology of acute heart failure involves several pathogenetic mechanisms, including volume overload, pressure overload, myocardial loss, and restrictive filling, while several cardiovascular and non-cardiovascular causes or precipitating factors lead to acute heart failure through a single of these mechanisms or a combination of them. Regardless of the underlying mechanism, peripheral and/or pulmonary congestion is the hallmark of acute heart failure, resulting from fluid retention and/or fluid redistribution. Myocardial injury and renal dysfunction are also involved in the precipitation and progression of the syndrome.

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24

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics : The Human Circulation. CRC, 2006.

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25

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics : The Human Circulation, Second Edition. Taylor & Francis Group, 2012.

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26

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics : The Human Circulation, Second Edition. Taylor & Francis Group, 2012.

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27

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics : The Human Circulation, Second Edition. Taylor & Francis Group, 2012.

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28

Chandran, Krishnan B., Ajit P. Yoganathan et Stanley E. Rittgers. Biofluid Mechanics : The Human Circulation, Second Edition. Taylor & Francis Group, 2012.

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29

Kline, William E., Mark G. Davies, Alan B. Lumsden et Ioannis Kakadiaris. Pumps and Pipes : Proceedings of the Annual Conference. Springer, 2014.

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30

Kline, William E., Mark G. Davies et Alan B. Lumsden. Pumps and Pipes : Proceedings of the Annual Conference. Springer, 2011.

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31

Wilton, Niall, Brian J. Anderson et Bruno Marciniak. Anatomy, physiology, and pharmacology in paediatric anaesthesia. Sous la direction de Jonathan G. Hardman et Neil S. Morton. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0069.

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Anaesthesia for children is tempered by changes that occur during both growth and development. Drug dose is affected by size and clearance maturation processes as well as the changing body composition that occurs with age. All organ systems undergo these maturation changes and most are complete within the first few years of life. Normal physiological variables in infancy and childhood are quite different from adults. The central nervous, cardiovascular, and respiratory systems are particularly important. Cerebral immaturity and plasticity impacts sensitivity to drugs, pain responses, and behaviour and increases potential harm from apoptosis with anaesthesia. The heart undergoes a transition from fetal to adult circulation during the first few weeks of life. Undiagnosed congenital defects are not uncommon. The neonate is very susceptible to conditions that trigger an increase in pulmonary vascular resistance, with reversion to fetal circulatory patterns. Respiratory anatomy and mechanics affect the propensity to apnoea, airway maintenance, artificial ventilation modalities, uptake of inhalational agents, and tracheal tube sizes. Metabolic rate and oxygen requirements increase with decreasing age. This physiology influences diverse aspects that include the rate of desaturation during apnoea, hypoglycaemia during starvation, cardiac output, drug metabolism, fluid requirements, and heat production or loss.

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32

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2013.

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33

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2013.

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34

Thiriet, Marc. Tissue Functioning and Remodeling in the Circulatory and Ventilatory Systems. Springer, 2016.

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