Littérature scientifique sur le sujet « Bifidobacterium pseudolongum »
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Articles de revues sur le sujet "Bifidobacterium pseudolongum"
Mao, Bingyong, Jiayu Gu, Dongyao Li, Shumao Cui, Jianxin Zhao, Hao Zhang et Wei Chen. « Effects of Different Doses of Fructooligosaccharides (FOS) on the Composition of Mice Fecal Microbiota, Especially the Bifidobacterium Composition ». Nutrients 10, no 8 (16 août 2018) : 1105. http://dx.doi.org/10.3390/nu10081105.
Texte intégralVasquez, Nadia, Antonia Suau, Fabien Magne, Philippe Pochart et Marie-Agnès Pélissier. « Differential Effects of Bifidobacterium pseudolongum Strain Patronus and Metronidazole in the Rat Gut ». Applied and Environmental Microbiology 75, no 2 (21 novembre 2008) : 381–86. http://dx.doi.org/10.1128/aem.01731-08.
Texte intégralXiao, Yue, Jianxin Zhao, Hao Zhang, Qixiao Zhai et Wei Chen. « Colonized Niche, Evolution and Function Signatures of Bifidobacterium pseudolongum within Bifidobacterial Genus ». Foods 10, no 10 (27 septembre 2021) : 2284. http://dx.doi.org/10.3390/foods10102284.
Texte intégralGAVINI, F., V. DELCENSERIE, K. KOPEINIG, S. POLLINGER, H. BEERENS, C. BONAPARTE et M. UPMANN. « Bifidobacterium Species Isolated from Animal Feces and from Beef and Pork Meat ». Journal of Food Protection 69, no 4 (1 avril 2006) : 871–77. http://dx.doi.org/10.4315/0362-028x-69.4.871.
Texte intégralYaeshima, Tomoko, Tomohiko Fujisawa et Tomotari Mitsuoka. « Bifidobacterium globosum, Subjective Synonym of Bifidobacterium pseudolongum, and Description of Bifidobacterium pseudolongum subsp. pseudolongum comb. nov. and Bifidobacterium pseudolongum subsp. globosum comb. nov. » Systematic and Applied Microbiology 15, no 3 (août 1992) : 380–85. http://dx.doi.org/10.1016/s0723-2020(11)80211-0.
Texte intégralTurroni, Francesca, Elena Foroni, Paola Pizzetti, Vanessa Giubellini, Angela Ribbera, Paolo Merusi, Patrizio Cagnasso et al. « Exploring the Diversity of the Bifidobacterial Population in the Human Intestinal Tract ». Applied and Environmental Microbiology 75, no 6 (23 janvier 2009) : 1534–45. http://dx.doi.org/10.1128/aem.02216-08.
Texte intégralNeuzil-Bunesova, Vera, Gabriele Andrea Lugli, Nikol Modrackova, Marie Makovska, Jakub Mrazek, Chahrazed Mekadim, Sarka Musilova et al. « Bifidobacterium canis sp. nov., a novel member of the Bifidobacterium pseudolongum phylogenetic group isolated from faeces of a dog (Canis lupus f. familiaris) ». International Journal of Systematic and Evolutionary Microbiology 70, no 9 (1 septembre 2020) : 5040–47. http://dx.doi.org/10.1099/ijsem.0.004378.
Texte intégralGu, Jiayu, Bingyong Mao, Shumao Cui, Xuemei Liu, Hao Zhang, Jianxin Zhao et Wei Chen. « Metagenomic Insights into the Effects of Fructooligosaccharides (FOS) on the Composition of Luminal and Mucosal Microbiota in C57BL/6J Mice, Especially the Bifidobacterium Composition ». Nutrients 11, no 10 (12 octobre 2019) : 2431. http://dx.doi.org/10.3390/nu11102431.
Texte intégralKim, Byoung Jun, Hee-Youn Kim, Yeo-Jun Yun, Bum-Joon Kim et Yoon-Hoh Kook. « Differentiation of Bifidobacterium species using partial RNA polymerase β-subunit (rpoB) gene sequences ». International Journal of Systematic and Evolutionary Microbiology 60, no 12 (1 décembre 2010) : 2697–704. http://dx.doi.org/10.1099/ijs.0.020339-0.
Texte intégralSimpson, P. J., C. Stanton, G. F. Fitzgerald et R. P. Ross. « Genomic Diversity and Relatedness of Bifidobacteria Isolated from a Porcine Cecum ». Journal of Bacteriology 185, no 8 (15 avril 2003) : 2571–81. http://dx.doi.org/10.1128/jb.185.8.2571-2581.2003.
Texte intégralThèses sur le sujet "Bifidobacterium pseudolongum"
Hoffmann, Mareike Jasmin [Verfasser]. « Taxonomische Einordnung und phänotypische Charakterisierung der Bifidobacterium pseudolongum-Gruppe aus Rinderfäzes und anderen Herkünften / Mareike Jasmin Hoffmann ». Hannover : Bibliothek der Tierärztlichen Hochschule Hannover, 2012. http://d-nb.info/1024440745/34.
Texte intégralPinchaud, Katleen. « Impact d'un apport alimentaire en acide arachidonique sur le microbiote intestinal et l'axe intestin-cerveau. Conséquences pour une prévention de la maladie d'Alzheimer par les probiotiques ». Electronic Thesis or Diss., Université de Lorraine, 2021. http://www.theses.fr/2021LORR0190.
Texte intégralAlzheimer's disease (AD) is a neurodegenerative disease associated with aging, consisting of a major public health problem worldwide. There is currently no effective treatment or established preventions for this disease, highlighting the importance of the design of preventive strategies in the fight against this disease. Western diets are particularly rich in ω-6 fatty acids, especially arachidonic acid (ARA), which are converted into many inflammation mediators in the organism. Previous laboratory results have shown that dietary intake of ARA sensitizes mice to the neurotoxicity of Aβ peptide oligomers, the main agent of AD. In this thesis work, we studied the impact of this dietary intake of ARA on the fecal microbiota and the microbiota-gut-brain axis. For 9 to 12 weeks, three groups of 15 mice have consumed one of these 3 diets : a conventional murine diet (“Sdt-ARA” of 5% lipids), a moderately high-fat diet of 15% lipids including 31.9% of linoleic acid ("HL-ARA"), and a diet of 15% lipids including 25.3% linoleic acid and 6.6% ARA ("HL+ARA").Analysis of the fecal microbiota revealed that the "HL-ARA" diet promotes proliferation of the Bifidobacterium pseudolongum strain, which has been show to possess anti-inflammatory activities. Furthermore, the supply of ARA suppresses this proliferation by favouring Escherichia-Shigella multiplication (with potentiating effects on inflammation). A 3-to-11-fold increase in mRNA levels of the pro-inflammatory cytokines IL-1β and TNFα has been observed in the colon of mice fed with the "HL+ARA" diet. Enrichment of ARA led to an increase of the expression of other mediators of colon inflammation such as CD40, adiponectin, and ICAM-1, as well as the claudin 1 and occludin markers which are components of cellular tight junctions. Therefore, these changes in expression suggest that the integrity of the intestinal barrier has been compromised. Moreover, dietary intake of ARA led to a 10 fold increase in TNFα mRNA levels in mesenteric adipose tissue, while the impact on the liver was found to be much more moderate. At the cerebral level, a 9 weeks food intake of ARA led to a 1.5-1.8-fold increase in the protein level of GFAP in the hippocampus and the cortex, while no changes of the microglial marker Iba1 has not been reported. We also found decreased levels of IL-6 and TNFα mRNA in the global brain tissue.These results indicate that dietary intake of ARA induces low-grade systemic inflammation which may promote the expansion of an Alzheimer's-like process in the brain if an inducing signal occurs. As such, we also evaluated the impact of an innovative oral administration model of D-galactose, which has been described as an aging accelerator compound , leading to impairment of cognitive capacities, and the appearance of amyloid plaques in the mouse brains through elevated oxidative stress. We also studied the survival in the digestive tract of two strains of Streptococcus thermophilus with anti-inflammatory activity in vitro as reported by previous laboratory work, and of a strain of Lactobacillus plantarum capable of metabolizing ω-6 polyunsaturated fatty acids in conjugated fatty acids. This thesis work aims to assess the ability of these probiotic strains to fight against low-grade inflammation induced by dietary intake of ARAs and oxidative stress induced by D-galactose. New work is underway in the laboratory to investigate in more details all the biological processes at work in the different models and to determine the impact of probiotics on a combined administration of D-galactose and an ARA-enriched diet
Actes de conférences sur le sujet "Bifidobacterium pseudolongum"
Song, Qian, Xiang Zhang, Weixin Liu, Yunfei Zhou, Fenfen Ji, Qing Li, Yanqiang Ding, Yating Zhang, Yasi Pan et Jun Yu. « IDDF2022-ABS-0075 Bifidobacterium pseudolongum and its generated acetate suppress non-alcoholic fatty liver disease-associated hepatocellular carcinoma ». Dans Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 2–4 September 2022. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2022. http://dx.doi.org/10.1136/gutjnl-2022-iddf.8.
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