Littérature scientifique sur le sujet « B-acute lymphoblastic leukemia »
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Articles de revues sur le sujet "B-acute lymphoblastic leukemia"
Juárez-Avendaño, Gerardo, Nuria Citlalli Luna-Silva, Euler Chargoy-Vivaldo, Laura Alicia Juárez-Martínez, Mayra Noemí Martínez-Rangel, Noemí Zárate-Ortiz, Edith Martínez-Valencia, Briceida López-Martínez, Rosana Pelayo et Juan Carlos Balandrán. « Poor Prognosis Biomolecular Factors Are Highly Frequent in Childhood Acute Leukemias From Oaxaca, Mexico ». Technology in Cancer Research & ; Treatment 19 (1 janvier 2020) : 153303382092843. http://dx.doi.org/10.1177/1533033820928436.
Texte intégralSugisaki, Manato, Kenji Imamura, Yukie Terasaki, Hiromasa Iino, Takumi Hoshino, Nahoko Hatsumi, Hiroshi Handa et Satoru Takada. « Slowly progressing acute lymphoblastic leukemia with prolonged leukopenia ». SAGE Open Medical Case Reports 11 (janvier 2023) : 2050313X2311777. http://dx.doi.org/10.1177/2050313x231177758.
Texte intégralLoghavi, Sanam, Jeffery L. Kutok et Jeffrey L. Jorgensen. « B-Acute Lymphoblastic Leukemia/Lymphoblastic Lymphoma ». American Journal of Clinical Pathology 144, no 3 (1 septembre 2015) : 393–410. http://dx.doi.org/10.1309/ajcpan7bh5dnywzb.
Texte intégralHurwitz, CA, MR Loken, ML Graham, JE Karp, MJ Borowitz, DJ Pullen et CI Civin. « Asynchronous antigen expression in B lineage acute lymphoblastic leukemia ». Blood 72, no 1 (1 juillet 1988) : 299–307. http://dx.doi.org/10.1182/blood.v72.1.299.299.
Texte intégralHurwitz, CA, MR Loken, ML Graham, JE Karp, MJ Borowitz, DJ Pullen et CI Civin. « Asynchronous antigen expression in B lineage acute lymphoblastic leukemia ». Blood 72, no 1 (1 juillet 1988) : 299–307. http://dx.doi.org/10.1182/blood.v72.1.299.bloodjournal721299.
Texte intégralAdamaki, Maria, Spiros Vlahopoulos, George I. Lambrou, Athanasios G. Papavassiliou et Maria Moschovi. « Aberrant AML1 gene expression in the diagnosis of childhood leukemias not characterized by AML1-involved cytogenetic abnormalities ». Tumor Biology 39, no 3 (mars 2017) : 101042831769430. http://dx.doi.org/10.1177/1010428317694308.
Texte intégralLi, Shiyong, et Glen Lew. « Is B-Lineage Acute Lymphoblastic Leukemia With a Mature Phenotype and L1 Morphology a Precursor B-Lymphoblastic Leukemia/Lymphoma or Burkitt Leukemia/Lymphoma ? » Archives of Pathology & ; Laboratory Medicine 127, no 10 (1 octobre 2003) : 1340–44. http://dx.doi.org/10.5858/2003-127-1340-iballw.
Texte intégralSilva, Alessandra Suelen Jardim, Gustavo Henrique de Medeiros Oliveira, Lenilton Silva DA Silva Júnior, Hugo Henrique de Freitas Ferreira, Maria das Graças Pereira Araujo, Victor lima Soares, Rodrigo Villar Freitas et al. « Clinical Utility of Flow Cytometry Immunophenotyping in Acute Lymphoblastic Leukemia ». Blood 136, Supplement 1 (5 novembre 2020) : 8. http://dx.doi.org/10.1182/blood-2020-143281.
Texte intégralBuhring, HJ, I. Sures, B. Jallal, FU Weiss, FW Busch, WD Ludwig, R. Handgretinger, HD Waller et A. Ullrich. « The receptor tyrosine kinase p185HER2 is expressed on a subset of B- lymphoid blasts from patients with acute lymphoblastic leukemia and chronic myelogenous leukemia ». Blood 86, no 5 (1 septembre 1995) : 1916–23. http://dx.doi.org/10.1182/blood.v86.5.1916.bloodjournal8651916.
Texte intégralKhabirova, Eleonora, Laura Jardine, Tim H. H. Coorens, Simone Webb, Taryn D. Treger, Justin Engelbert, Tarryn Porter et al. « Single-cell transcriptomics reveals a distinct developmental state of KMT2A-rearranged infant B-cell acute lymphoblastic leukemia ». Nature Medicine 28, no 4 (14 mars 2022) : 743–51. http://dx.doi.org/10.1038/s41591-022-01720-7.
Texte intégralThèses sur le sujet "B-acute lymphoblastic leukemia"
Oliveira, Tiago M. « The importance of glycosylation in Acute Lymphoblastic Leukemia ». Thesis, Griffith University, 2021. http://hdl.handle.net/10072/410463.
Texte intégralThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
Institute for Glycomics
Griffith Health
Full Text
Besada, Rana Hany. « BiTEs and CAR-Ts : immunotherapy in childhood B-cell acute lymphoblastic leukemia ». Thesis, Massachusetts Institute of Technology, 2018. http://hdl.handle.net/1721.1/115699.
Texte intégralCataloged from PDF version of thesis.
Includes bibliographical references (pages 18-21).
B-cell acute lymphoblastic leukemia is the most common pediatric cancer, responsible for the most cancer-related deaths in children. Advances in chemotherapy over the past half-century have steadily increased the remission and survival of children with B-cell acute lymphoblastic leukemia to nearly 90%. However, the problems of minimal residual disease and relapsed and refractory disease persist. Personalized, targeted therapies have improved outcomes among the minority of patients for whom chemotherapy is ineffective. Immunotherapy, specifically bispecific T-cell engaging antibody therapy and chimeric antigen receptor T-cell therapy, has proven an effective treatment for relapsed and refractory B-cell acute lymphoblastic leukemia in children. These new modalities, however, have also introduced new adverse side effects to the treatment regimen. Though immunotherapy has increased remission and survival, more work must be done to reduce adverse effects and eliminate relapsed and refractory disease.
by Rana Hany Besada.
S.M.
Morisot, Sebastien. « Détermination of the frequency of leukemia stem cells in childhood precursor B cell acute lymphoblastic leukemias ». Paris 11, 2009. http://www.theses.fr/2009PA11T022.
Texte intégralSoto-Feliciano, Yadira M. (Yadira Marie). « PHF6 is a novel regulator of B-cell identity in acute lymphoblastic leukemia ». Thesis, Massachusetts Institute of Technology, 2016. http://hdl.handle.net/1721.1/103165.
Texte intégralThis electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.
Cataloged from student-submitted PDF version of thesis. Vita.
Includes bibliographical references.
Mutations in the zinc finger gene PHF6 are seen in approximately 20% of adult T-cell acute lymphoblastic leukemias and 3% of adult acute myeloid leukemias. The notable absence of PHF6 mutations in B-cell lineage malignancies has led to the hypothesis that PHF6 may act as a lineage-specific tumor suppressor gene. Recent work from our group described a role for PHF6 as a positive regulator of growth in B-cell acute lymphoblastic leukemia (B-ALL). To identify the mechanisms by which PHF6 acts to promote B-ALL growth in vivo, we utilized CRISPR-Cas9 to delete Phf6 in murine B-ALL cells. Transplantation of Phf6 knockout cells (Phf6KO) into immunocompetent recipients significantly extended disease latency and survival. Strikingly, these mice developed lymphomas, characterized by significantly enlarged lymph nodes, decreased disease burden in the spleen and increased expression of the canonical T-cell marker CD4, suggesting that Phf6KO B-ALL cells adopt alternate lineage programs in vivo. To dissect the molecular mechanisms by which Phf6 regulates this lineage decision, we carried out a combination of RNA and chromatin immunoprecipitation (ChIP-Seq) sequencing analyses in Phf6WT and Phf6KO cells. RNA sequencing analysis revealed many differentially expressed genes in Phf6KO B-ALL cells. Notably, genes and gene sets that were significantly down-regulated in Phf6KO cells included those involved in pathways important for B-cell development and function. ChIP-Seq analysis of PHF6 and several histone marks revealed that PHF6 and H3K27ac signals co-localize close to the transcription start site and enhancer regions of a significant proportion of differentially expressed genes. Transcription factor binding motif analysis revealed significant enrichment for several well-described transcriptional regulators of B-cell development. Importantly, we demonstrated that the transcription factors TCF12 and NF-kB co-immunoprecipitated with PHF6 in Phf6WT B-ALL cells. These findings discovered a novel role for PHF6 in the maintenance of B-cell identity in B-ALL, by activating genes that are crucial for B-cell lineage maintenance. Collectively, these results indicate that loss-of-function of Phf6 in B-ALL leads to an unstable cell identity state, in which cells need to acquire alternate developmental programs in order to survive. These findings could potentially explain the absence of PHF6 mutations in human B-cell lineage malignancies.
by Yadira M. Soto-Feliciano.
Ph. D.
James, Alva Rani [Verfasser]. « LncRNAs signature defining major subtypes of B-cell acute lymphoblastic leukemia / Alva Rani James ». Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2019. http://d-nb.info/1189140330/34.
Texte intégralNicoletti, Simon. « Natural Killer Cells and Pre-B Acute Lymphoblastic Leukemia : Evidence for an Unconventional Cytotoxicity Pathway ». Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS383.
Texte intégralNatural Killer (NK) cells are innate lymphoid cells with anti-infectious and anti-tumoral activities. Among neoplasia, pre-B acute lymphoblastic leukemias (pre-B ALL) represent the most common form of cancer in childhood and were shown to be resistant to NK cell mediated cytotoxicity although the mechanisms explaining this phenomenon are incompletely understood.In the present work, we investigated the relative immune resistance of pediatric pre-B ALL targets to activated NK cells. We developed a flow cytometry based cytotoxicity assay to assess the NK activity and the involvement of long term cytotoxic pathways. Although pre-B ALL blasts were strongly resistant at 4h, we found a considerable delayed NK killing at 25h.Further investigations revealed that cell contact was mandatory for efficient killing but also that neither the granule exocytosis nor the death receptor pathway were involved. Target cell death was caspase independent but mitochondria signaling amplified it. We then showed that NK cells from patients with X-linked chronic granulomatous disease could not kill efficiently ALL blasts and that NK cells expressed key components of a NADPH oxidase complex that was distinct from the phagocyte type. Our work reveals an uncharacterized effector pathway among cytotoxic lymphocytes and establishes key molecular requirements for this unconventional pathway
Ueno, Hiroo. « Landscape of driver mutations and their clinical impacts in pediatric B-cell precursor acute lymphoblastic leukemia ». Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263562.
Texte intégralSartor, Chiara <1988>. « Research of predictive biomarkers to anti-CD22 antibody-drug conjugate treatment in B-cell acute lymphoblastic leukemia ». Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2022. http://amsdottorato.unibo.it/10252/1/PhD%20thesis_Chiara%20Sartor_XXXIV%20ciclo.pdf.
Texte intégralAuer, Franziska [Verfasser], Arndt [Gutachter] Borkhardt et Hermann [Gutachter] Aberle. « Paired Box 5 (PAX5) in B cell precursor acute lymphoblastic leukemia / Franziska Auer ; Gutachter : Arndt Borkhardt, Hermann Aberle ». Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2017. http://d-nb.info/1123197628/34.
Texte intégralGroeneveld-Krentz, Stefanie [Verfasser]. « The clinical relevance of aneuploidy in relapses of pediatric B-cell precursor acute lymphoblastic leukemia / Stefanie Groeneveld-Krentz ». Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2020. http://d-nb.info/1223927180/34.
Texte intégralLivres sur le sujet "B-acute lymphoblastic leukemia"
St-Denis, Emily Jean. The progression of precursor B cell acute lymphoblastic leukemia in murine models. 2005.
Trouver le texte intégralChapitres de livres sur le sujet "B-acute lymphoblastic leukemia"
Fukano, Reiji. « Mature B-Cell Acute Lymphoblastic Leukemia ». Dans Pediatric Acute Lymphoblastic Leukemia, 73–80. Singapore : Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-15-0548-5_8.
Texte intégralGastier-Foster, Julie M. « Precursor B-Cell Acute Lymphoblastic Leukemia ». Dans Molecular Pathology Library, 287–307. Boston, MA : Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-5698-9_24.
Texte intégralGorczyca, Wojciech. « B-Cell Acute Lymphoblastic Leukemia/Lymphoma ». Dans Flow Cytometry in Neoplastic Hematology, 495–516. 4e éd. Boca Raton : CRC Press, 2022. http://dx.doi.org/10.1201/9781003197935-17.
Texte intégralGhanem, Hady, Hagop Kantarjian, Nitin Jain et Elias Jabbour. « Management of B-Cell Acute Lymphoblastic Leukemia ». Dans Cancer Consult : Expertise for Clinical Practice, 22–28. Oxford, UK : John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118589199.ch3.
Texte intégralHogan, Laura E., Luke D. Maese, Keith J. August et Jennifer L. McNeer. « Treatment of Pediatric B- and T-Cell Acute Lymphoblastic Leukemia ». Dans Clinical Management of Acute Lymphoblastic Leukemia, 75–104. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-85147-7_4.
Texte intégralRoberts, Kathryn G., et Charles G. Mullighan. « Molecular Pathways and Targets in B-Cell Progenitor Acute Lymphoblastic Leukemia ». Dans Clinical Management of Acute Lymphoblastic Leukemia, 3–32. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-85147-7_1.
Texte intégralGorczyca, Wojciech. « B-Cell Acute Lymphoblastic Leukemia/Lymphoma (B-ALL/LBL) ». Dans Atlas of Differential Diagnosis in Neoplastic Hematopathology, 663–78. 4e éd. Boca Raton : CRC Press, 2021. http://dx.doi.org/10.1201/9781003120445-40.
Texte intégralMolina, John C., et Nirali N. Shah. « Monoclonal Antibody-Based Treatment and Other New Agents for B-Lineage Acute Lymphoblastic Leukemia ». Dans Clinical Management of Acute Lymphoblastic Leukemia, 295–328. Cham : Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-85147-7_13.
Texte intégralLarson, R. A., C. D. Bloomfield et C. A. Schiffer. « Cancer and Leukemia Group B Studies in Acute Lymphoblastic Leukemia ». Dans Haematology and Blood Transfusion / Hämatologie und Bluttransfusion, 420–26. Berlin, Heidelberg : Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78350-0_75.
Texte intégralKulhalli, Rahul, Chinmay Savadikar et Bhushan Garware. « Toward Automated Classification of B-Acute Lymphoblastic Leukemia ». Dans Lecture Notes in Bioengineering, 63–72. Singapore : Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-15-0798-4_7.
Texte intégralActes de conférences sur le sujet "B-acute lymphoblastic leukemia"
Ain, N. U., A. Kainat, A. Hurera et J. Bierenbaum. « Acute Adult B-lymphoblastic Leukemia Presenting as Hypercalcemia ». Dans American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3389.
Texte intégralDobson, Stephanie M., Robert Vanner, Esmé Waanders, Jessica McLeod, Olga I. Gan, Zhaohui Gu, Debbie Payne-Turner et al. « Abstract A25 : Evolving functional heterogeneity in B-acute lymphoblastic leukemia ». Dans Abstracts : Fourth AACR International Conference on Frontiers in Basic Cancer Research ; October 23-26, 2015 ; Philadelphia, PA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.fbcr15-a25.
Texte intégralDobson, Stephanie M., Robert Vanner, Esmé Waanders, Olga I. Gan, Jessica McLeod, Ildiko Grandal, Debbie Payne-Turner et al. « Abstract LB-341 : Evolving functional heterogeneity in B-acute lymphoblastic leukemia ». Dans Proceedings : AACR 107th Annual Meeting 2016 ; April 16-20, 2016 ; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-lb-341.
Texte intégralKane, Shriya, Yali Ding, Chandrika Gowda, Jonathon Lee Payne, Soumya Iyer, Pavan K. Dhanyamraju, Chunhua Song et al. « Abstract 2927 : Targeted combination treatment for B-cell acute lymphoblastic leukemia ». Dans Proceedings : AACR Annual Meeting 2020 ; April 27-28, 2020 and June 22-24, 2020 ; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-2927.
Texte intégralSpinella, Jean-François, Virginie Saillour, Chantal Richer, Manon Ouimet, Pauline Cassart, Jasmine Healy, Eric Bareke et al. « Abstract 4335 : The genomic landscape of childhood pre-B acute lymphoblastic leukemia ». Dans Proceedings : AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012 ; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4335.
Texte intégralZanetti, SR, T. Velazco-Hernandez, F. Gutierrez-Agüera, H. Roca-Ho, D. Sánchez-Martínez, P. Petazzi, R. Torres et al. « CD19 and CD22-directed biespecific CAR for B-cell Acute Lymphoblastic Leukemia ». Dans 32. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1687121.
Texte intégralSchröder, C. F., Ž. Antić, U. zur Stadt, M. Tang, M. Zimmermann, C. Eckert, B. Fedders, M. Stanulla, G. Cario et A. K. Bergmann. « Comprehensive molecular and clinical characterization of DUX4-rearranged B-acute lymphoblastic leukemia ». Dans 34. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1768545.
Texte intégralLiu, S., Q. Sun, K. M. Debatin et L. H. Meyer. « Effective targeting of Wnt Signaling in B Cell Precursor Acute Lymphoblastic Leukemia ». Dans 34. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1768528.
Texte intégralLenk, L., M. Carlet, A. Cousins, G. Cario, C. Halsey, I. Jeremias, E. Hobeika, H. Jumaa, A. Alsadeq et DM Schewe. « CD79a/CD79b Promote CNS-Involvement and Leukemic Engraftment in Pediatric B-cell Precursor Acute Lymphoblastic Leukemia ». Dans 33. Jahrestagung der Kind-Philipp-Stiftung für pädiatr. onkolog. Forschung. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1709765.
Texte intégralChen, Zhengshan, Seyedmehdi Shojaee, Maike Buchner, Huimin Geng, Jae Woong Lee, Lars Klemm, Eugene Park et al. « Abstract 2075 : Signaling thresholds and negative B cell selection in acute lymphoblastic leukemia ». Dans Proceedings : AACR 106th Annual Meeting 2015 ; April 18-22, 2015 ; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2075.
Texte intégral