Littérature scientifique sur le sujet « Autoantibody reactivity »
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Articles de revues sur le sujet "Autoantibody reactivity"
Biggs, Catherine M., Svetlana Kostjukovits, Kerry Dobbs, Saila Laakso, Paula Klemetti, Helena Valta, Mervi Taskinen, Outi Mäkitie et Luigi D. Notarangelo. « Diverse Autoantibody Reactivity in Cartilage-Hair Hypoplasia ». Journal of Clinical Immunology 37, no 6 (19 juin 2017) : 508–10. http://dx.doi.org/10.1007/s10875-017-0408-4.
Texte intégralYu, Xuechen, Melanie Uhde, Peter Green et Armin Alaedini. « Autoantibodies in the Extraintestinal Manifestations of Celiac Disease ». Nutrients 10, no 8 (20 août 2018) : 1123. http://dx.doi.org/10.3390/nu10081123.
Texte intégralTakeuchi, K., S. J. Turley, E. M. Tan et K. M. Pollard. « Analysis of the autoantibody response to fibrillarin in human disease and murine models of autoimmunity. » Journal of Immunology 154, no 2 (15 janvier 1995) : 961–71. http://dx.doi.org/10.4049/jimmunol.154.2.961.
Texte intégralSibanda, Elopy N., Margo Chase-Topping, Lorraine T. Pfavayi, Mark E. J. Woolhouse et Francisca Mutapi. « Evidence of a distinct group of Black African patients with systemic lupus erythematosus ». BMJ Global Health 3, no 5 (septembre 2018) : e000697. http://dx.doi.org/10.1136/bmjgh-2017-000697.
Texte intégralDellavance, Alessandra, Danielle C. Baldo, Bing Zheng, Rodrigo A. Mora, Marvin J. Fritzler, Falk Hiepe, Johan Rönnelid et al. « Establishment of an international autoantibody reference standard for human anti-DFS70 antibodies : proof-of-concept study for a novel Megapool strategy by pooling individual specific sera ». Clinical Chemistry and Laboratory Medicine (CCLM) 57, no 11 (25 octobre 2019) : 1754–63. http://dx.doi.org/10.1515/cclm-2019-0087.
Texte intégralVan Haren, Keith, Beren H. Tomooka, Brian A. Kidd, Brenda Banwell, Amit Bar-Or, Tanuja Chitnis, Silvia N. Tenembaum et al. « Serum autoantibodies to myelin peptides distinguish acute disseminated encephalomyelitis from relapsing– remitting multiple sclerosis ». Multiple Sclerosis Journal 19, no 13 (23 avril 2013) : 1726–33. http://dx.doi.org/10.1177/1352458513485653.
Texte intégralPilyugin, Maxim, Magdalena Ratajska, Maciej Stukan, Nicole Concin, Robert Zeillinger et Irmgard Irminger-Finger. « BARD1 Autoantibody Blood Test for Early Detection of Ovarian Cancer ». Genes 12, no 7 (25 juin 2021) : 969. http://dx.doi.org/10.3390/genes12070969.
Texte intégralPutterman, Chaim, Irene Blanco, Nicole Jordan, Vered Daniel-Carmi, Liron Belanis-Meirovich, Rachel Sorek, Ornit Cohen-Gindi et al. « Autoantibody profiling for Systemic Lupus Erythematosus diagnosis using the Immunarray CHIP™ (P4018) ». Journal of Immunology 190, no 1_Supplement (1 mai 2013) : 42.10. http://dx.doi.org/10.4049/jimmunol.190.supp.42.10.
Texte intégralGabusi, Loi, Gissi, Spinelli, Bernardi et Buzzi. « Effectiveness of Topical Application of Heterologous Platelet Rich Plasma (PRP) in Oral Mucous Membrane Pemphigoid. A Report of a Case ». Proceedings 35, no 1 (12 décembre 2019) : 57. http://dx.doi.org/10.3390/proceedings2019035057.
Texte intégralCaudie, Christiane. « Monoclonal IgM Autoantibody Reactivity in M-IgM Peripheral Neuropathy ». Clinical Reviews in Allergy & ; Immunology 19, no 1 (2000) : 7–18. http://dx.doi.org/10.1385/criai:19:1:7.
Texte intégralThèses sur le sujet "Autoantibody reactivity"
LOVATO, Laura. « Proteomic analysis of autoantibody reactivity to central nervous system antigens in sera and cerebrospinal fluid of multiple sclerosis patients ». Doctoral thesis, 2009. http://hdl.handle.net/11562/337547.
Texte intégralMultiple Sclerosis (MS) belongs to a large group of inflammatory demyelinating diseases of the central nervous system (CNS), in which the autoimmune response is directed to myelin and myelin-producing cells, the oligodendrocytes, and eventually leads to demyelination and oligodendrocyte loss. Heterogeneity in morphological alterations of the brain is detectable by magnetic resonance imaging (MRI), histopathological evaluation, as well as in clinica1 presentation. Three are the major forms of MS. It is not clear which factors are responsible for the different courses. Relapsing-remitting (M)-MS is the most frequent form (85%-90% of patients) characterized by unpredictable attacks (relapses) followed by periods of months to years of relative quiet (remission) with no new signs of disease activity. Most RR-MS patients later develop secondary progressive (SP)-MS, in which they begin to have neurologic decline between their acute attacks without any definite periods of remission. About 10%-15% of patients present with insidious disease onset and steady progression, termed primary progressive (PP)-MS. The autoimmune reaction has been shown to involve the activation of both T and B lymphocytes. The activation of CD4+ autoreactive T cells and their differentiation into a Thl phenotype are crucial events in the initial steps, and these cells are probably also important players in the long-term evolution of the disease. Damage of the target tissue, the central nervous system, is, however, most likely mediated by other components of the immune system, such as antibodies (Igs), complement, CD8+ T cells, and factors produced by innate immune cells. The observation that Igs are elevated in the cerebrospinal fluid (CSF) of MS patients has been the most important and earliest evidence suggesting a role for B cells and antibodies in the pathology of MS. In most of the studies the search for autoantigens has focused on myelin proteins and other CNS components. Although some studies emphasize the relevance of myelin-specific antibodies, others fail to confirm these data. We have overcome such restrictive approaches using a large pane1 of antigens derived from target tissue extracts. In the present study, we compared by bidimensional polyacrylamide gel electrophoresis (2D-PAGE) and immunoblotting the IgG repertoires from serum and CSF of contro1 and MS patients against antigens derived from CNS normal white matter. The reactive spots were then identified by mass spectrometry (MaS) andlor immunoblotting. This immunomic approach enabled the identification of a restricted number of neural protein isoforms specifically recognized by MS sera and CSF, which were mostly localized on oligodendrocytes andlor cytoskeleton. Almost al1 MS patients had CSF IgG directed to isoforms of one of the oligodendroglial molecules transketolase (TK), cyclic nucleotide phosphodiesterase type I (CNPase I), collapsin response mediator protein 2 and tubulin P4. Interestingly, 50% of MS CSF IgG recognized TK, which was mostly localized on oligodendrocytes in human white matter from normal and MS samples. IgG autoreactivity to cytoskeletal proteins (radixin, sirtuin 2 and actin interacting protein 1) was prevalent in secondary progressive MS patients. Among the proteins recognized by serum IgG, almost al1 MS patients specifically recognized a restricted number of neuronal/cytoskeletal proteins, while CNPase I was the oligodendroglial antigen most frequently recognized (44%) by MS seric IgG. Our immunomic approach shed new light on the autoimmune repertoire present in MS patients revealing nove1 oligodendroglial andlor neuronal putative autoantigens, which may have potential important pathogenic implications and also serve as biomarkers of disease as well as useful diagnostic tools.
Chapitres de livres sur le sujet "Autoantibody reactivity"
Klinman, Dennis M., Akira Shirai et Yoshiaki Ishigatsubo. « Polyclonal B Cell Activation and B Cell Cross-Reactivity During Autoantibody Production in Systemic Lupus Erythematosus ». Dans Advances in Experimental Medicine and Biology, 115–23. Boston, MA : Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2427-4_12.
Texte intégralActes de conférences sur le sujet "Autoantibody reactivity"
Zucht, H.-D., P. Budde, L. Steeg, M. Tuschen, S. Bhandari et P. Schulz-Knappe. « PS1:20 Mining for common reactivity patterns of human autoantibodies against endogenous protein targets using clustered autoantibody reactivities ». Dans 11th European Lupus Meeting, Düsseldorf, Germany, 21–24 March 2018, Abstract presentations. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-abstract.68.
Texte intégralFakhroo, Aisha, Fatma Ali, Gheyath K. Nasrallah, Nico Marr et Hadi Mohamad Yassine. « Detection of antinuclear antibodies targeting intracellular signal transduction, metabolism, apoptotic processes and cell death in critical COVID-19 patients ». Dans Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0095.
Texte intégralNomura, S., H. Nagata, N. Sone, K. Oda, T. Kokawa et K. Yasunaga. « ANALYSIS OF PLATELET ANTIGENS FOR ANTI-PLATELET ANTIBODIES IN ITP USING FLOW CYTOMETRY ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644582.
Texte intégralNugent, Diane. « IDENTIFICATION OF ANTIPLATELET ANTIBODY IDIOTYPlSS ASSOCIATED WITH GLYCOPROTEIN Ib SPECIFICITY, PRESENT IN ITP PLASMA AND PRODUCED BY HUMAN HYBRIDOMAS FROM ITP SPLEEN CELL FUSIONS ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644758.
Texte intégralYoshioka, A., M. Shima, I. Tanaka, T. Fujiwara, H. Nakai et H. Fukui. « ANALYSIS OF HUMAN FACTOR VIII INHIBITOR EPITOPES TO FACTOR VIII POLYPEPTIDES BY IMMUNOBLOTTING ». Dans XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644029.
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