Littérature scientifique sur le sujet « APS, Complement System, Coagulation Cascade »
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Articles de revues sur le sujet "APS, Complement System, Coagulation Cascade"
Panebianco, Lauren M., et Teresa Gentile. « The Role of Monitoring Complement Levels in Catastrophic Antiphospholipid Syndrome : A Case Series of 4 Patients ». Blood 134, Supplement_1 (13 novembre 2019) : 4872. http://dx.doi.org/10.1182/blood-2019-128297.
Texte intégralLiang, Yan, Shang-Bo Xie, Chang-Hao Wu, Yuan Hu, Qin Zhang, Si Li, Yin-Guang Fan et al. « Coagulation cascade and complement system in systemic lupus erythematosus ». Oncotarget 9, no 19 (11 décembre 2017) : 14862–81. http://dx.doi.org/10.18632/oncotarget.23206.
Texte intégralFletcher-Sandersjöö, Alexander, Marc Maegele et Bo-Michael Bellander. « Does Complement-Mediated Hemostatic Disturbance Occur in Traumatic Brain Injury ? A Literature Review and Observational Study Protocol ». International Journal of Molecular Sciences 21, no 5 (26 février 2020) : 1596. http://dx.doi.org/10.3390/ijms21051596.
Texte intégralBerkowitz, Shani, Joab Chapman, Amir Dori, Shany Guly Gofrit, Nicola Maggio et Efrat Shavit-Stein. « Complement and Coagulation System Crosstalk in Synaptic and Neural Conduction in the Central and Peripheral Nervous Systems ». Biomedicines 9, no 12 (20 décembre 2021) : 1950. http://dx.doi.org/10.3390/biomedicines9121950.
Texte intégralGavriilaki, Eleni, Akrivi Chrysanthopoulou, Ioanna Sakellari, Ioannis Batsis, Despina Mallouri, Tasoula Touloumenidou, Apostolia Papalexandri et al. « Linking Complement Activation, Coagulation, and Neutrophils in Transplant-Associated Thrombotic Microangiopathy ». Thrombosis and Haemostasis 119, no 09 (2 juillet 2019) : 1433–40. http://dx.doi.org/10.1055/s-0039-1692721.
Texte intégralArnout, J. « Mechanism of action of Lupus anticoagulants ». Hämostaseologie 21, no 02 (2001) : 44–49. http://dx.doi.org/10.1055/s-0037-1619504.
Texte intégralHuber, Silke, Mariam Massri, Marco Grasse, Verena Fleischer, Sára Kellnerová, Verena Harpf, Ludwig Knabl et al. « Systemic Inflammation and Complement Activation Parameters Predict Clinical Outcome of Severe SARS-CoV-2 Infections ». Viruses 13, no 12 (26 novembre 2021) : 2376. http://dx.doi.org/10.3390/v13122376.
Texte intégralMutch, Nicola J. « Polyphosphate as a haemostatic modulator ». Biochemical Society Transactions 44, no 1 (9 février 2016) : 18–24. http://dx.doi.org/10.1042/bst20150207.
Texte intégralTiwari, Ritudhwaj, Anurag R. Mishra, Flora Mikaeloff, Soham Gupta, Ali Mirazimi, Siddappa N. Byrareddy, Ujjwal Neogi et Debasis Nayak. « In silico and in vitro studies reveal complement system drives coagulation cascade in SARS-CoV-2 pathogenesis ». Computational and Structural Biotechnology Journal 18 (2020) : 3734–44. http://dx.doi.org/10.1016/j.csbj.2020.11.005.
Texte intégralBazargani, Farhan, Russell P. Rother et Magnus Braide. « The Roles of Complement Factor C5a and CINC-1 in Glucose Transport, Ultrafiltration, and Neutrophil Recruitment during Peritoneal Dialysis ». Peritoneal Dialysis International : Journal of the International Society for Peritoneal Dialysis 26, no 6 (novembre 2006) : 688–96. http://dx.doi.org/10.1177/089686080602600614.
Texte intégralThèses sur le sujet "APS, Complement System, Coagulation Cascade"
UGOLINI, Sara. « PROTEOMIC ANALYSIS OF BIOLOGICAL MATERIAL FROM WOMEN WITH ANTIPHOSPHOLIPID SYNDROME. A RETROSPECTIVE STUDY ». Doctoral thesis, 2016. http://hdl.handle.net/11562/938687.
Texte intégralObjective. During the last decade, the role of complement system activation in the pathogenesis of antiphospholipid syndrome (APS) has been debated but an evident correlation with the proinflammatory and procoagulant phenotype in this disease has never been found. We carried out a retrospective study on APS women to evaluate the expression of some soluble and local complement and coagulation molecules supposed to be involved in promoting thrombotic events and pregnancy-related clinical complications.Methods. Complement C5a and C5b-9 complex, Tissue Factor (TF) and Tissue Factor Pathway Inhibitor (TFPI) plasma levels of APS patients and matched healthy women were analyzed by ELISA assays. Placenta samples of APS patients and controls were subjected to immunoblotting analysis with specific antibodies for complement component 5a Receptor (C5aR), CD46 (MCP, Membrane Cofactor Protein), CD55 (DAF, Decay Accelerating Factor), CD59, p-selectin, TF and TFPI. Mutations and polymorphisms analysis in the complement regulator genes were also performed. Results. In APS patients, both in pregnant and non-pregnant conditions, we found higher plasma levels of C5a, C5b-9 and TF compared to healthy subjects. In APS placenta, C5aR, C5b-9 and p-selectin levels were higher than in controls, conversely, CD46, CD55 and CD59 levels were lower compared to healthy subjects. Since clinical management of pregnancy in APS requires anticoagulant therapy (heparin), plasma and placenta content of TF and TFPI were affected by this treatment. In APS placenta donors, we did not find mutations or allele variances in the CD46, CD55 and CD59 genes.Conclusions. Our data suggest complement activation both in pregnant and non-pregnant APS patients as supported by the increase in key molecules from complement system. In this study, higher levels of activated complement molecules were associated to a proinflammatory and procoagulant state in APS subjects. The lower placental CD46, CD55 and CD59 expression may impair local protection, favoring local complement hyperactivation and leading to inflammation, thrombosis, tissue damage and possible fetal death, but further studies are necessary to better understand the mechanisms involved in complement dysregulation in APS syndrome.