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Livres sur le sujet « Antigeni tumore associati »

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Auteur : Grafiati

Publié le 10 mars 2023

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1

C, Ghosh Bimal, et Ghosh Luna, dir. Tumor markers and tumor-associated antigens. New York : McGraw-Hill, 1987.

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2

Gires, Olivier, et Barbara Seliger. Tumor-associated antigens : Identification, characterization, and clinical applications. Weinheim : Wiley-VCH, 2009.

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3

Chuang, Hong-Yang. Synthesis and Vaccine Evaluation of the Tumor Associated Carbohydrate Antigen RM2 from Prostate Cancer. Berlin, Heidelberg : Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46848-7.

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4

Hamburger Symposium über Tumormarker. (6th 1991 Hamburg, Germany). Tumor associated antigens, oncogenes, receptors, cytokines in tumor diagnosis and therapy at the beginning of the Nineties : 6th Symposium on Tumor Markers, Hamburg 1991. München : W. Zuckschwerdt Verlag, 1992.

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5

Gires, Olivier, et Barbara Seliger, dir. Tumor‐Associated Antigens. Wiley, 2009. http://dx.doi.org/10.1002/9783527625970.

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6

Tumor-Associated Antigens : Identification, Characterization, and Clinical Applications. Wiley-VCH Verlag GmbH, 2009.

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7

Gires, Olivier, et Barbara Seliger. Tumor-Associated Antigens : Identification, Characterization, and Clinical Applications. Wiley & Sons, Incorporated, John, 2009.

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8

(Editor), Dirk Nagorsen, et F. M. Marincola (Editor), dir. Analyzing T Cell Responses : How to analyze cellular immune responses against tumor associated antigens. Springer, 2005.

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9

Marincola, Francesco M., et Dirk Nagorsen. Analyzing T Cell Responses : How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer London, Limited, 2006.

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10

Marincola, Francesco M., et Dirk Nagorsen. Analyzing T Cell Responses : How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer, 2010.

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11

Guo, Xiangqian, Jianying Zhang, Bilian Jin et Qing Zhu, dir. Tumor-Associated Antigens and Their Autoantibodies : From Discovering to Clinical Utilization. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-797-7.

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12

Alharbi, Yousef, Manish S. Patankar et Rebecca J. Whelan. Antibody-Based Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0006.

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With their role in connecting disease-associated antigens to the cellular immune response, antibodies hold considerable promise as therapeutic agents. This chapter discusses three classes of therapeutic antibodies that have been developed for use in ovarian cancer therapy. The first includes antibodies selected against tumor-associated antigens such as MUC16/CA125, mesothelin, epithelial cell adhesion molecule, and folate receptor α‎. Antibodies in the second class target proteins such as CTLA-4 and PD1 that act as immune response checkpoint receptors. The third class of antibodies target secreted factors that promote tumor growth: targets in this class include vascular endothelial growth factor, cytokines, and chemokines. The development of each of these is described. The chapter also discusses the complications presented by soluble antigens, which serve to limit the applicability of antigens (such as MUC16/CA125) that are both cell-surface associated and circulating and the prospects for the combination of antibody-based immunotherapy and chemotherapy.

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13

Greten, H., et R. Klapdor. New Tumour Associated Antigens : Two Years Clinical Experience with Monoclonal Antibodies. Thieme Publishing Group, 1986.

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14

Chuang, Hong-Yang. Synthesis and Vaccine Evaluation of the Tumor Associated Carbohydrate Antigen RM2 from Prostate Cancer. Springer, 2015.

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15

Chuang, Hong-Yang. Synthesis and Vaccine Evaluation of the Tumor Associated Carbohydrate Antigen RM2 from Prostate Cancer. Springer Berlin / Heidelberg, 2015.

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16

Chuang, Hong-Yang. Synthesis and Vaccine Evaluation of the Tumor Associated Carbohydrate Antigen RM2 from Prostate Cancer. Springer, 2016.

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17

EBNA1 and EpsteinBarr Virus Associated Tumours Springerbriefs in Cancer Research. Springer-Verlag New York Inc., 2013.

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18

Hepatitis B Research Advances. Nova Science Pub Inc, 2007.

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19

Abe, Hiroyuki, Amane Sasada, Shigeki Tabata et Minako Abe. Heat Shock Protein Vaccine Therapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0009.

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Despite advances in chemotherapeutic regimens, ovarian cancer has a poor prognosis. Therefore important effective treatments are urgently needed. Many studies have reported that the immune system plays a critical role in disease progression and overall survival. One known effective immunotherapy is the dendritic cell (DC)-based vaccine pulsed with tumor-associated antigens. This chapter reports on a method of production of a novel DC-based vaccine. The key technologies are (a) monocyte collection without leukapheresis, (b) monocyte expansion, (c) production of dendritic cells, (d) multiple overlapping long peptides with heat shock protein 70, and (e) combination immunotherapy approach. The next generation of immunotherapy for ovarian cancer will be focused on combination approaches that simultaneously augment immunity while preventing local immune suppression. Possible combinations which might be useful to help patients with ovarian cancer are summarized in this chapter.

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20

Cooperberg, Matthew, et Peter Carroll. Prostate cancer. Sous la direction de James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0064.

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Management of prostate cancer remains controversial, in large part because of its wide heterogeneity in terms of aggressiveness and prognosis. Early detection efforts based on prostate specific antigen (PSA) and aggressive treatment of high-risk cancers have yielded major improvements in mortality rates, but overtreatment of low-risk cancers—those unlikely to cause symptoms or threaten life if they were never detected—is associated with high rates of avoidable toxicity and cost. Prostate cancer can be effectively risk-stratified based on tools (e.g. nomograms, CAPRA score) integrating the PSA level, Gleason grade, clinical stage, and extent of biopsy tissue involvement. Most men with low-risk tumours are eligible for active surveillance, a programme of careful monitoring based on PSA and follow-up biopsies. Men with higher-risk cancers are best served with radical prostatectomy or radiation therapy.

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