Littérature scientifique sur le sujet « Amoeboid motility »
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Articles de revues sur le sujet "Amoeboid motility"
Leo, Angela, Erica Pranzini, Laura Pietrovito, Elisa Pardella, Matteo Parri, Paolo Cirri, Gennaro Bruno et al. « Claisened Hexafluoro Inhibits Metastatic Spreading of Amoeboid Melanoma Cells ». Cancers 13, no 14 (15 juillet 2021) : 3551. http://dx.doi.org/10.3390/cancers13143551.
Texte intégralPietrovito, Laura, Giuseppina Comito, Matteo Parri, Elisa Giannoni, Paola Chiarugi et Maria Letizia Taddei. « Zoledronic Acid Inhibits the RhoA-mediated Amoeboid Motility of Prostate Cancer Cells ». Current Cancer Drug Targets 19, no 10 (23 décembre 2019) : 807–16. http://dx.doi.org/10.2174/1568009619666190115142858.
Texte intégralKlemm, Lucas C., Ryan A. Denu, Laurel E. Hind, Briana L. Rocha-Gregg, Mark E. Burkard et Anna Huttenlocher. « Centriole and Golgi microtubule nucleation are dispensable for the migration of human neutrophil-like cells ». Molecular Biology of the Cell 32, no 17 (15 août 2021) : 1545–56. http://dx.doi.org/10.1091/mbc.e21-02-0060.
Texte intégralCallan-Jones, A. C., et R. Voituriez. « Active gel model of amoeboid cell motility ». New Journal of Physics 15, no 2 (18 février 2013) : 025022. http://dx.doi.org/10.1088/1367-2630/15/2/025022.
Texte intégralPeretz-Soroka, Hagit, Reuven Tirosh, Jolly Hipolito, Erwin Huebner, Murray Alexander, Jason Fiege et Francis Lin. « A bioenergetic mechanism for amoeboid-like cell motility profiles tested in a microfluidic electrotaxis assay ». Integrative Biology 9, no 11 (2017) : 844–56. http://dx.doi.org/10.1039/c7ib00086c.
Texte intégralDalal, Swapnil, Alexander Farutin et Chaouqi Misbah. « Amoeboid swimming in a compliant channel ». Soft Matter 16, no 6 (2020) : 1599–613. http://dx.doi.org/10.1039/c9sm01689a.
Texte intégralSaito, Koji, Yuta Ozawa, Keisuke Hibino et Yasutaka Ohta. « FilGAP, a Rho/Rho-associated protein kinase–regulated GTPase-activating protein for Rac, controls tumor cell migration ». Molecular Biology of the Cell 23, no 24 (15 décembre 2012) : 4739–50. http://dx.doi.org/10.1091/mbc.e12-04-0310.
Texte intégralCopos, Calina A., Robert D. Guy, Sam Walcott, Juan Carlos del Alamo et Alex Mogilner. « Mechanosensitive Adhesion Explains Stepping Motility in Amoeboid Cells ». Biophysical Journal 112, no 3 (février 2017) : 433a. http://dx.doi.org/10.1016/j.bpj.2016.11.2315.
Texte intégralBullock, Timothy L., Airlie J. McCoy, Helen M. Kent, Thomas M. Roberts et Murray Stewart. « Structural basis for amoeboid motility in nematode sperm ». Nature Structural Biology 5, no 3 (mars 1998) : 184–89. http://dx.doi.org/10.1038/nsb0398-184.
Texte intégralCopos, Calina A., Sam Walcott, Juan C. del Álamo, Effie Bastounis, Alex Mogilner et Robert D. Guy. « Mechanosensitive Adhesion Explains Stepping Motility in Amoeboid Cells ». Biophysical Journal 112, no 12 (juin 2017) : 2672–82. http://dx.doi.org/10.1016/j.bpj.2017.04.033.
Texte intégralThèses sur le sujet "Amoeboid motility"
Zanchi, Roberto. « The involvement of the endocytic cycle in amoeboid cell motility ». Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608458.
Texte intégralLeo, Angela. « The study of cell motility and plasticity in cancer : the role of the crosstalk between BM-MSCs and tumor in osteosarcoma progression and Claisened Hexafluoro as potential inhibitor of amoeboid motility in metastatic melanoma ». Doctoral thesis, Università di Siena, 2021. http://hdl.handle.net/11365/1128636.
Texte intégralLewis, Owen Leslie. « Mathematical Investigation of Hydrodynamic Contributions to Amoeboid Cell Motility in Physarum polycephalum ». Thesis, University of California, Davis, 2015. http://pqdtopen.proquest.com/#viewpdf?dispub=3685252.
Texte intégralIn this work, we investigate the role of intracellular fluid flow in the migration of Physarum polycephalum. We develop two distinct models. Initially, we model the intracellular space of a physarum plasmodium as a peristaltic chamber. We derive a PDE relating the deformation of the chamber boundary and the flux of fluid along the chamber center line. We then solve this PDE for two distinct boundary deformations and evaluate the characteristic stress associated with the peristaltic flow. We compare the derived stress, as well as the relative phase of the deformation and flow waves, with values seen in experiments. Second, we develop a poro-elastic model of the interior of physarum that accounts for cytoskeletal structure, as well as adhesive interactions with the substrate. We develop this model within a framework similar to the Immersed Boundary method, which readily allows for computer simulation. We then use this model to simulate cell crawling across a range of parameters that characterize the coordination of adhesion to the substrate. We identify a spatio-temporal form of adhesion coordination that is consistent with experiments. We also show that this form is both efficient and robust, when compared to similar forms of adhesion coordination.
Ingram, Mark Edward. « Whole Skin Locomotion Inspired by Amoeboid Motility Mechanisms : Mechanics of the Concentric Solid Tube Model ». Thesis, Virginia Tech, 2006. http://hdl.handle.net/10919/35100.
Texte intégralMaster of Science
D'Alessandro, Joseph. « Collective regulation of the amoeboid motility : the role of short and long-range interactions in vegetative Dictyostelium discoideum ». Thesis, Lyon, 2016. http://www.theses.fr/2016LYSE1039/document.
Texte intégralCell motility is fundamental in many physiological, either normal or pathological, phenomena. Yet, although these most often involve several cells moving at the same time, how the interactions between cells affect both individual and collective dynamics remains a poorly understood question. In this thesis, I used vegetative Dictyostelium discoideum cells as a model to study this collective regulation of the motility. I relied mainly on the thorough analysis of numerous cell trajectories in various situations to (i) characterise a secreted factor used to down-regulate the cells’ motility (biochemical nature, response pathway, secretion and response dynamics) and (ii) quantitatively analyse and model the dynamics of spreading cell colonies of controlled initial shape, size and density. Last, I describe a dynamic aggregation phenomenon that occurs when the cells are seeded at high density in a nutrient-rich medium
Wyse, Meghan M. « CXCL12 Mediated Regulation of the Cytoskeletal Regulator mDia2 Formin Induces Amoeboid Conversions and Cellular Plasticity in Migrating Human Breast Carcinoma Cells ». University of Toledo / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1404042854.
Texte intégralKulawiak, Dirk Alexander [Verfasser], Harald [Akademischer Betreuer] Engel, Harald [Gutachter] Engel, Markus [Gutachter] Bär et Carsten [Gutachter] Beta. « Physical minimal models of amoeboid cell motility / Dirk Alexander Kulawiak ; Gutachter : Harald Engel, Markus Bär, Carsten Beta ; Betreuer : Harald Engel ». Berlin : Technische Universität Berlin, 2017. http://d-nb.info/1156010403/34.
Texte intégralNègre, Paulin. « Mécanismes de motilité et guidage sous flux des leucocytes humains ». Thesis, Aix-Marseille, 2018. http://www.theses.fr/2018AIXM0747/document.
Texte intégralA fast and efficient immunity response needs leukocytes’ability to migrate within the entire organism. Their migration, called amoeboid, is characterized by a high speed (10-20 μm.min-1) and a great adaptability to move through various environment, either two-dimensional as luminal endothelial surface or tri-dimensional (3D) environment as tissue. Since the observation of leukocytes migrating without adhesion through solid 3D medium, amoeboid migration is described as requiring either adhesion or friction with solid support to permit motility. We showed here that effector T lymphocytes are able to swim without any interaction with solid substrate. Propulsion is based on actin retrograde flow coupled with transmembrane proteins linked to cytoskeleton (like integrins) which drag a brush of polymeric molecules in interaction with the medium. Furthermore, cell guidance is required for many crucial functions as organism growth or immune system. However, when crawling on luminal endothelial surfaces, cells are exposed to blood flow and they robustly orient either with or against the flow with unknown mechanisms. We showed that lymphocytes and neutrophils flow orientation can be explain without any molecular flow sensor of shear stress. Lamellipodium for neutrophils and uropod for lymphocytes is non-adherent and orients in the direction of flow like a wind vane. Front-rear cell polarization aligns the axis of the whole cell with the non-adherent pole oriented by flow. Flow mechanotaxis of leukocytes relies on passive mechanisms without mechanotransduction
Buttery, Shawnna Marie Roberts Thomas M. « Characterization of the cytosolic proteins involved in the amoeboid motility of ascaris sperm ». 2003. http://etd.lib.fsu.edu/theses/available/etd-08192004-100416.
Texte intégralAdvisor: Dr. Thomas M. Roberts, Florida State University, College of Arts and Sciences, Department of Biological Science. Title and description from dissertation home page (viewed Aug. 23, 2004). Includes bibliographical references.
Chapitres de livres sur le sujet "Amoeboid motility"
Shimmen, T. « Mechanisms of Cytoplasmic Streaming and Amoeboid Movement ». Dans Muscle Contraction and Cell Motility, 172–205. Berlin, Heidelberg : Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76927-6_6.
Texte intégralItaliano, Joseph E., Murray Stewart et Thomas M. Roberts. « How the assembly dynamics of the nematode major sperm protein generate amoeboid cell motility ». Dans International Review of Cytology, 1–34. Elsevier, 2001. http://dx.doi.org/10.1016/s0074-7696(01)02002-2.
Texte intégralFukui, Yoshio. « Toward a New Concept of Cell Motility : Cytoskeletal Dynamics in Amoeboid Movement and Cell Division ». Dans International Review of Cytology, 85–127. Elsevier, 1993. http://dx.doi.org/10.1016/s0074-7696(08)61514-4.
Texte intégralMaynard Smith, John, et Eors Szathmary. « The origin of eukaryotes ». Dans The Major Transitions in Evolution. Oxford University Press, 1997. http://dx.doi.org/10.1093/oso/9780198502944.003.0012.
Texte intégralActes de conférences sur le sujet "Amoeboid motility"
Xiong, Yuan, et Pablo A. Iglesias. « Automated characterization of amoeboid motility ». Dans 2009 43rd Annual Conference on Information Sciences and Systems (CISS). IEEE, 2009. http://dx.doi.org/10.1109/ciss.2009.5054747.
Texte intégralIngram, Mark, et Dennis Hong. « Whole Skin Locomotion Inspired by Amoeboid Motility Mechanisms ». Dans ASME 2005 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/detc2005-85419.
Texte intégralWyse, Meghan M., Andrea L. Nestor-Kalinoski et Kathryn M. Eisenmann. « Abstract C50 : CXCL12-triggered amoeboid cell motility is mediated through a RhoA-directed signaling hub ». Dans Abstracts : AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013 ; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c50.
Texte intégralEisenmann, Kathryn. « Abstract LB-270 : Regulation of the cortical actin cytoskeleton and amoeboid motility through the mDia2 formin;DIP complex ». Dans Proceedings : AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010 ; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-270.
Texte intégralMohamed, Islam, Ahmed Moahmed, Mennatallah Abdelkader, Alaaeldin Saleh et Ala-Eddin Al-Moustafa. « Elaeagnus Angustifolia : a Promising Medicinal Plant for Cancer Theraby ». Dans Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0124.
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