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Littérature scientifique sur le sujet « Alveolar Hypoventilation »

Auteur : Grafiati
Publié le 1 février 2025

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Articles de revues sur le sujet "Alveolar Hypoventilation"

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Muzumdar, Hiren, et Raanan Arens. « Central Alveolar Hypoventilation Syndromes ». Sleep Medicine Clinics 3, no 4 (décembre 2008) : 601–15. http://dx.doi.org/10.1016/j.jsmc.2008.08.006.

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Seriff, Nathan S. « ALVEOLAR HYPOVENTILATION WITH NORMAL LUNGS : THE SYNDROME OF PRIMARY OR CENTRAL ALVEOLAR HYPOVENTILATION * ». Annals of the New York Academy of Sciences 121, no 3 (16 décembre 2006) : 691–705. http://dx.doi.org/10.1111/j.1749-6632.1965.tb14238.x.

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Milerad, J., H. Lagercrantz et O. Lofgren. « Alveolar hypoventilation treated with medroxyprogesterone. » Archives of Disease in Childhood 60, no 2 (1 février 1985) : 150–55. http://dx.doi.org/10.1136/adc.60.2.150.

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Guilleminault, Christian, Riccardo Stoohs, Hartmut Schneider, Thomas Podszus, J. Herman Peter et Peter von Wichert. « Central Alveolar Hypoventilation and Sleep ». Chest 96, no 5 (novembre 1989) : 1210–12. http://dx.doi.org/10.1378/chest.96.5.1210.

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Kim, Heakyung, et John R. Bach. « Central alveolar hypoventilation in neurosarcoidosis ». Archives of Physical Medicine and Rehabilitation 79, no 11 (novembre 1998) : 1467–68. http://dx.doi.org/10.1016/s0003-9993(98)90245-3.

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HIDA, Wataru. « Novel Aspects of Primary Alveolar Hypoventilation ». Internal Medicine 40, no 6 (2001) : 463–64. http://dx.doi.org/10.2169/internalmedicine.40.463.

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AlOtair, Hadil, Abdulaziz Alzeer, Mohammed Abdou et Shaden Qasrawi. « Unusual case of central alveolar hypoventilation ». Saudi Medical Journal 39, no 3 (12 mars 2018) : 305–10. http://dx.doi.org/10.15537/smj.2018.3.210803.

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Ji, Ki-Hwan. « The Need for Titration With Polysomnography of Noninvasive Positive Pressure Ventilation in a Patient With Hypoventilation Syndrome With Kyphoscoliosis ». Sleep Medicine Research 13, no 1 (30 juin 2022) : 59–62. http://dx.doi.org/10.17241/smr.2022.01256.

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Attended noninvasive positive airway pressure (PAP) with polysomnography (PSG) is recommended for determining the level of ventilatory support and bilevel PAP required for patients with chronic alveolar hypoventilation syndromes. We present a case of a 24-year-old male with kyphoscoliosis who presented with dyspnea. The patient was diagnosed with chronic alveolar hypoventilation and had bilevel PAP titrated by referring to tidal volume and arterial gas analysis during the day. After further titration with PSG there was stable ventilation during non-rapid eye movement sleep but unstable on entering rapid eye movement sleep.
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Rojanapairat, Oragun, Abigail Beggs, Melisa Chang et Aaron Thomas. « 1235 REM Sleep Associated Hypoventilation ». Sleep 43, Supplement_1 (avril 2020) : A471. http://dx.doi.org/10.1093/sleep/zsaa056.1229.

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Abstract Introduction Hypoventilation is a spectrum of respiratory disorders that is frequently found in patients with chronic obstructive pulmonary disease, restrictive lung disease (eg obesity, neuromuscular, severe interstitial lung disease, and chest wall disease), chronic sedative use, and hypothyroidism. Rapid eye movement (REM) sleep hypoventilation may be the first manifestation of hypoventilation prior to development of non-REM sleep hypoventilation and eventual awake alveolar hypoventilation. We present a case of hypoventilation during REM sleep with mild restriction on pulmonary function testing, prior to the development of obesity hypoventilation syndrome (OHS). Report of Case 68-year-old male with past medical history of diastolic heart failure, class Ill obesity (BMI 46), hypertension, chronic kidney disease lllb, and diabetes mellitus underwent split night polysomnography for evaluation of snoring, witnessed apneas and excessive daytime sleepiness. The study was significant for an apnea hypopnea index of 105/hour, and REM sleep sustained desaturation to a nadir of 72% without apneas or hypopneas, suspicious for hypoventilation. The derangements during REM sleep did not correct during PAP titration despite CPAP and supplemental oxygen. End tidal capnography was not available for the study. Follow up PFT demonstrated normal spirometry, mild restrictive lung volumes, ERV 27%, and severely depressed DLCO which corrected for alveolar volume. Daytime arterial blood gas did not reveal hypercapnia or hypoxemia (7.37/39/78/23). He underwent successful nocturnal titration with average volume assured pressure support with the final settings of IPAP 24-30, EPAP 20, VT 560 (8 ml/kg IBW), rate of 12 breaths per minute and no supplemental oxygen. Conclusion This patient demonstrates REM sleep hypoventilation without overt OHS during all stages of sleep, which likely would progress to OHS over time. OHS is associated with increased rates of chronic heart failure, pulmonary hypertension, hospitalizations for respiratory failure, and mortality. Early recognition and treatment are important in improving morbidity and mortality.
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Hou, Linle, Jonathan Stoll, Lauren Pioppo, Jack Xu et Wajahat Khan. « Transient Acquired Hypoventilation Syndrome Secondary to Uncal Herniation Is Successfully Treated with Bilevel Noninvasive Positive Pressure Ventilation ». Case Reports in Critical Care 2018 (17 décembre 2018) : 1–3. http://dx.doi.org/10.1155/2018/7013916.

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Background.To describe an unusual presentation of acquired hypoventilation syndrome treated successfully with noninvasive positive pressure ventilation.Case Presentation.We report a case report of a 48-year-old male who presented to the emergency room for recurrent syncope. He was found to have a ventricular colloid cyst causing uncal herniation. The patient was noted to be intermittently apneic and bradypnic. Transient hypoventilation was successfully treated with noninvasive positive pressure ventilation and the patient made a full neurological recovery following transcallosal resection of the colloid cyst. Subsequently, the hypoventilation resolved.Conclusion.With prompt surgical intervention, full neurological recovery is possible after cerebral uncal herniation. In rare circumstances, this can result in transient alveolar hypoventilation. Bilevel noninvasive positive pressure ventilation can be used to successfully manage the hypoventilation.
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Thèses sur le sujet "Alveolar Hypoventilation"

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Di, Maria Julie. « Prise en charge des troubles respiratoire du sommeil chez les patients lésés médullaires ˸ optimisation du diagnostic à la prise en charge ». Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASW012.

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Les lésions de la moelle épinière sont responsables de tableaux cliniques très variables en fonction du niveau anatomique et de la sévérité de la lésion. Parmi les conséquences de la lésion médullaire, les troubles du sommeil occupent une place importante, au premier rang desquels les troubles respiratoires qui sont 3 à 4 fois plus prévalent qu'en population générale et de nature variée (apnées obstructives et/ou centrale, hypoventilation alvéolaire).En dépit de leur excès de prévalence, les mécanismes physiopathologiques des troubles respiratoires du sommeil associés à la lésion médullaire ne sont pas parfaitement élucidés. De plus, en raison des multiples comorbidités souvent associées à la lésion médullaire, les problématiques liées au sommeil sont fréquemment reléguées au second plan et sous diagnostiquées. L'objectif de cette thèse est d'enrichir les connaissances relatives à la prise en charge spécifique des troubles respiratoires du sommeil chez les patients lésés médullaires, depuis la physiopathologie jusqu'aux modalités de traitement en passant par les stratégies de dépistages et les conséquences spécifiques.Au cours de cette thèse nous avons abordé la physiopathologie des troubles respiratoire nocturne des patients lésés médullaires et notamment le rôle des mouvements de fluides nocturnes au décubitus. Nos résultats soulignent l'importance du phénotypage et la nécessité de stratégies de dépistage adaptées des troubles respiratoires. Les conséquences spécifiques notamment vésico-sphinctérienne et cardiovasculaires ont également été abordée. Enfin, les bénéfices et limites des traitements conventionnels ont été étudiés notamment les déterminants spécifiques de l'observance au traitement
Spinal cord injuries (SCI) lead to variable clinical presentations depending on the level and severity of the lesion. Sleep disorders are a frequent complication of SCI: sleep disordered breathing (SDB) is 3-4 times more frequent than in the general population and includes obstructive and/or central apneas and alveolar hypoventilation.Despite its increased prevalence, the pathophysiological mechanisms underpinning SDB associated with SCI are poorly understood. Furthermore, the presence of other complications linked to SCI means that sleep disorders are often under investigated. The objective of this thesis is to improve knowledge both of the pathophysiology and management of SDB, focussing on screening, treatment modalities and consequences.We look at the pathophysiology of SDB in SCI patients and notably the role of rostral fluid shift when lying down. Our results underline the importance of accurate phenotyping and adapted SDB screening strategies in this population. We also examine specific consequences such as bladder and bowel function and cardiovascular morbidity. Finally, the benefits and limits of conventional treatments are explored, focussing on the factors associated with treatment to continual positive airway pressure (CPAP) and non-invasive ventilation (NIV)
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PANHALEUX, PHILIPPE. « L'hypoventilation alveolaire primitive : a propos d'un cas ». Nantes, 1989. http://www.theses.fr/1989NANT058M.

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Caron, Christophe. « Les hypoventilations alvéolaires d'origine centrale : à propos d'un cas et revue de la littérature ». Lille 2, 1991. http://www.theses.fr/1991LIL2M138.

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Petit, François-Xavier Chabot Jean-François. « HYPOVENTILATION ALVEOLAIRE ET OBESITE PRESENTATION ET ETUDE DE FAISABILITE D'UN PROTOCOLE DE RECHERCHE DIAGNOSTIQUE ET THERAPEUTIQUE MULTICENTRIQUE / ». [S.l.] : [s.n.], 2000. http://www.scd.uhp-nancy.fr/docnum/SCDMED_T_2000_PETIT_FRANCOIS_XAVIER.pdf.

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LARROQUE, BEATRICE. « Malaises graves du nourrisson par atteinte du tronc cerebral : leurs relations avec le syndrome d'ondine, la mort subite inexpliquee du nourrisson ». Aix-Marseille 2, 1988. http://www.theses.fr/1988AIX20279.

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Livres sur le sujet "Alveolar Hypoventilation"

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Junna, Mithri R., Bernardo J. Selim et Timothy I. Morgenthaler. Central sleep apnea and hypoventilation syndromes. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0018.

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Sleep disordered breathing (SDB) may occur in a variety of ways. While obstructive sleep apnea is the most common of these, this chapter reviews the most common types of SDB that occur independently of upper airway obstruction. In many cases, there is concurrent upper airway obstruction and neurological respiratory dysregulation. Thus, along with attempts to correct the underlying etiologies (when present), stabilization of the upper airway is most often combined with flow generators (noninvasive positive pressure ventilation devices) that modulate the inadequate ventilatory pattern. Among these devices, when continuous positive airway pressure (CPAP) alone does not allow correction of SDB, adaptive servo-ventilation (ASV) is increasingly used for non-hypercapnic types of central sleep apnea (CSA), while bilevel PAP in spontaneous-timed mode (BPAP-ST) is more often reserved for hypercapnic CSA/alveolar hypoventilation syndromes. Coordination of care among neurologists, cardiologists, and sleep specialists will often benefit such patients.
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Joynt, Gavin M., et Gordon Y. S. Choi. Blood gas analysis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0072.

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Arterial blood gases allow the assessment of patient oxygenation, ventilation, and acid-base status. Blood gas machines directly measure pH, and the partial pressures of carbon dioxide (PaCO2) and oxygen (PaO2) dissolved in arterial blood. Oxygenation is assessed by measuring PaO2 and arterial blood oxygen saturation (SaO2) in the context of the inspired oxygen and haemoglobin concentration, and the oxyhaemoglobin dissociation curve. Causes of arterial hypoxaemia may often be elucidated by determining the alveolar–arterial oxygen gradient. Ventilation is assessed by measuring the PaCO2 in the context of systemic acid-base balance. A rise in PaCO2 indicates alveolar hypoventilation, while a decrease indicates alveolar hyperventilation. Given the requirement to maintain a normal pH, functioning homeostatic mechanisms result in metabolic acidosis, triggering a compensatory hyperventilation, while metabolic alkalosis triggers a compensatory reduction in ventilation. Similarly, when primary alveolar hypoventilation generates a respiratory acidosis, it results in a compensatory increase in serum bicarbonate that is achieved in part by kidney bicarbonate retention. In the same way, respiratory alkalosis induces kidney bicarbonate loss. Acid-base assessment requires the integration of clinical findings and a systematic interpretation of arterial blood gas parameters. In clinical use, traditional acid-base interpretation rules based on the bicarbonate buffer system or standard base excess estimations and the interpretation of the anion gap, are substantially equivalent to the physicochemical method of Stewart, and are generally easier to use at the bedside. The Stewart method may have advantages in accurately explaining certain physiological and pathological acid base problems.
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Pevernagie, Dirk. Positive airway pressure therapy. Sous la direction de Sudhansu Chokroverty, Luigi Ferini-Strambi et Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0017.

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This chapter describes positive airway pressure (PAP) therapy for sleep disordered breathing. Continuous PAP (CPAP) acts as a mechanical splint on the upper airway and is the treatment of choice for moderate to severe obstructive sleep apnea (OSA). Autotitrating CPAP may be used when the pressure demand for stabilizing the upper airway is quite variable. In other cases, fixed CPAP is sufficient. There is robust evidence that CPAP reduces the symptomatic burden and risk of cardiovascular comorbidity in patients with moderate to severe OSA. Bilevel PAP is indicated for treatment of respiratory diseases characterized by chronic alveolar hypoventilation, which typically deteriorates during sleep. Adaptive servo-ventilation is a mode of bilevel PAP used to treat Cheyne–Stokes respiration with central sleep apnea . It is crucial that caregivers help patients get used to and be compliant with PAP therapy. Education, support, and resolution of adverse effects are mandatory for therapeutic success.
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Albright, Robert C. Acid-Base and Electrolyte Disorders. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0474.

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The most important principle in understanding disorders of water balance is that sodium balance is determined by the adequacy of the effective circulating volume, while water balance is determined by osmoregulation and the interplay between vasopressin activity, renal concentrating and diluting ability, and thirst. Disorders of sodium balance can be determined only by clinical examination. Orthostatic hypotension implies volume depletion and sodium deficiency. Edema implies volume excess and sodium excess. Potassium is predominantly an intracellular cation. The intracellular balance of potassium is regulated by endogenous factors such as acidemia, sodium, adenosine triphosphatase, insulin, catecholamines, and aldosterone. Clinically, it is absolutely critical to follow a stepwise approach to acid-base disorders. Metabolic acidosis is defined as a primary disturbance in which the retention of acid consumes endogenous alkali stores. This is reflected by a decrease in bicarbonate. Metabolic alkalosis is defined as a primary disturbance in which plasma bicarbonate is increased. The signs and symptoms of metabolic alkalosis include weakness, muscle cramps, hyperreflexia, alveolar hypoventilation, and arrhythmias.
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Latronico, Nicola, Simone Piva et Victoria McCredie. Long-Term Implications of ICU-Acquired Muscle Weakness. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0024.

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Intensive care unit-acquired weakness (ICUAW) is a significant and common complication with major implications for survivors of critical illness. ICUAW is a clinical diagnosis made in the presence of generalized muscle weakness that occurs in the setting of critical illness when other causes of muscle weakness have been excluded. Critical illness polyneuropathy and myopathy are the most common causes of ICUAW. Short-term implications of ICUAW include alveolar hypoventilation and an increased risk of pulmonary aspiration, atelectasis, and pneumonia—factors which may contribute to acute respiratory failure and ICU re-admission. In the long term, ICUAW has been associated with physical disturbances, including unsteady gait, sensory loss, foot drop, and, in more severe cases, persistent quadriparesis and ventilator dependency. ICUAW appears to heavily influence the failure of ICU patients to return to baseline health status post-discharge. There is a paucity of evidenced-based therapeutic strategies to reduce the incidence of ICUAW; however, early rehabilitative therapy might represent an effective measure in improving functional status.
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Fahner, Marcie. Coloring Book - You Will Get Better - Congenital Central Alveoloar Hypoventilation Syndrome. Independently Published, 2021.

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Chapitres de livres sur le sujet "Alveolar Hypoventilation"

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Weitzenblum, E. « Central alveolar hypoventilation syndrome ». Dans Sleep, 577–79. Boston, MA : Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0217-3_45.

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Peter, Helga, et Thomas Penzel. « Congenital Central Alveolar Hypoventilation Syndrome ». Dans Springer Reference Medizin, 1. Berlin, Heidelberg : Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-642-54672-3_408-1.

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Aboussouan, Loutfi S., et Umur Hatipoǧlu. « Alveolar Hypoventilation and Non-invasive Ventilation in COPD ». Dans Complex Sleep Breathing Disorders, 145–52. Cham : Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-57942-5_13.

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Chang, J. « Congenital Central Alveolar Hypoventilation Syndrome ». Dans Encyclopedia of Sleep, 314–18. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-378610-4.00326-0.

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Chang, Jacqueline. « Congenital central alveolar hypoventilation syndrome ». Dans Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-822963-7.00118-3.

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Brown, L. K. « Sleep-Related Idiopathic Nonobstructive Alveolar Hypoventilation ». Dans Encyclopedia of Sleep, 309–13. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-378610-4.00325-9.

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Ianus, Vlad D., et Lee K. Brown. « Sleep related idiopathic non-obstructive alveolar hypoventilation ». Dans Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-822963-7.00331-5.

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Ianus, Vlad D., et Lee K. Brown. « Sleep related idiopathic non-obstructive alveolar hypoventilation ». Dans Reference Module in Neuroscience and Biobehavioral Psychology. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-12-822963-7.00331-5.

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Shah, Jayant. « Chapter-04 Alveolar Ventilation, Hypoventilation and Sleep Studies ». Dans Pulmonary Function Tests Made Easy, 31–38. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10688_4.

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Pires, Rute. « Non-Invasive Ventilation in Alveolar Obesity-Hypoventilation Syndrome ». Dans Advances in Medical Diagnosis, Treatment, and Care, 117–35. IGI Global, 2020. http://dx.doi.org/10.4018/978-1-7998-3531-8.ch008.

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Obesity is the main risk factor for several sleep breathing disorders, including obstructive sleep apnea syndrome (OSAS), either alone or associated with chronic obstructive pulmonary disease (COPD), and alveolar obesity-hypoventilation syndrome (AOHS). In several of these conditions, the indicated treatment includes the use of non-invasive ventilation during sleep, such as the use of continuous positive airway pressure (continuous positive airway pressure or CPAP) and two-level pressure (BIPAP, bi-level positive airway pressure). In this chapter, a brief review is made of what the most recent studies say regarding the treatment of SOHA with non-invasive ventilation (NIV), comparing different ventilation modes and/or treatments.
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Actes de conférences sur le sujet "Alveolar Hypoventilation"

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Moreira, João Victor Aguiar, Isabela Maria Bernardes Goulart, Diogo Fernandes dos Santos, Isabella Sabião Borges, Pedro Otávio Rego de Aguiar, Thaciany Soares Ferreira, Leonardo Peixoto Garcia et al. « Bilateral diaphragmatic eventration and alveolar hypoventilation in congenital myotonic dystrophy ». Dans XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.533.

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Context: Congenital myotonic dystrophy (CMD) is a subtype of type 1 myotonic dystrophy presented in the neonatal period associated with a 16–40% mortality rate. CMD cause significant morbidity and mortality and often require intensive intervention at birth because of hypotonia, respiratory failure and feeding difficulties. It can cause respiratory problems including ineffective cough, recurrent pulmonary infections, orthopnea, dyspnea, poor sleep, apnea and snoring. However, there are few descriptions about diaphragmatic impairment in CMD. We present a baby who had bilateral diaphragmatic eventration associated with CMD. Case report: A term outborn female baby with normal birth weight, delivered by cesarean presenting hypotonia and breathing difficulty since birth. There was no history of meconium aspiration syndrome and aspiration pneumonia. Neurological examination showed a severe hypotonia, eyelid ptosis, oral motor weakness and suction inability, without contractures. Chest X-rays confirmed the bilateral diaphragmatic paralysis. Electroneuromyography confirmed a marked myopathic involvement with frequent myotonic discharges. The mother presented clinical and electrical myotonic phenomena. The baby started mechanical ventilation as was not maintaining saturation on head box oxygen. After surgical repair the baby started on non-invasive respiratory support with improvement of ventilatory conditions. Conclusion: Diaphragmatic eventration is a congenital condition where the muscle maintains its normal costal attachments but is significantly elevated with limited motility. Clinical manifestations vary to life-threatening respiratory distress. Bilateral congenital diaphragmatic eventration is rarer and has more guarded prognosis. Early diaphragmatic plication enhances weaning process and may prevent or minimize the morbidity. Infants with CMD should be monitored for diaphragmatic impairment.
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Bogan, Richard K., et JoAnne T. Turner. « Clinical Correlates And Prevalence Of Alveolar Hypoventilation In A Sleep Clinic Population ». Dans American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5264.

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Aguado Agudo, M., T. Martín Carpi, J. Rodríguez Sanz et L. Torralba. « Transcutaneous capnography usefulness in alveolar hypoventilation and titration in a sleep unit. » Dans ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.981.

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Bulbul, Yilmaz, Sibel Ayik, Tevfik Ozlu et Asim Orem. « Frequency And Predictors Of Obesity Hypoventilation And Other Causes Of Alveolar Hypoventilation In Hospitalized Patients At A Tertiary Health Care Institution ». Dans American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5049.

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Goswami, Umesh, Madhu Kannapiran et Conrad Iber. « Phrenic Nerve Injury After 16 Years Of Diaphragmatic Pacing In Congenital Central Alveolar Hypoventilation Syndrome ». Dans American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5835.

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