Littérature scientifique sur le sujet « Aging cardiac »
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Articles de revues sur le sujet "Aging cardiac"
Wessells, Robert J., et Rolf Bodmer. « Cardiac aging ». Seminars in Cell & ; Developmental Biology 18, no 1 (février 2007) : 111–16. http://dx.doi.org/10.1016/j.semcdb.2006.12.011.
Texte intégralLi, Zhen, et David J. Lefer. « Demystifying Cardiac Aging ». Circulation Research 128, no 4 (19 février 2021) : 508–10. http://dx.doi.org/10.1161/circresaha.121.318741.
Texte intégralLIMACHER, MARIAN C. « Aging and Cardiac Function ». Southern Medical Journal 87, no 5 (mai 1994) : S17. http://dx.doi.org/10.1097/00007611-199405000-00003.
Texte intégralLIMACHER, MARIAN C. « Aging and Cardiac Function ». Southern Medical Journal 87, Supplement (mai 1994) : S17. http://dx.doi.org/10.1097/00007611-199405001-00003.
Texte intégralMiyamoto, Shigeki. « Autophagy and cardiac aging ». Cell Death & ; Differentiation 26, no 4 (28 janvier 2019) : 653–64. http://dx.doi.org/10.1038/s41418-019-0286-9.
Texte intégralRenlund, Dale G., et Gary Gerstenblith. « Aging and cardiac function ». International Journal of Cardiology 10, no 3 (mars 1986) : 193–96. http://dx.doi.org/10.1016/0167-5273(86)90001-x.
Texte intégralCzuriga, D., Z. Papp, I. Czuriga et Á. Balogh. « Cardiac aging – a review ». European Surgery 43, no 2 (avril 2011) : 69–77. http://dx.doi.org/10.1007/s10353-011-0600-3.
Texte intégralObas, Vanessa, et Ramachandran S. Vasan. « The aging heart ». Clinical Science 132, no 13 (9 juillet 2018) : 1367–82. http://dx.doi.org/10.1042/cs20171156.
Texte intégralChen, Bijun, Shuaibo Huang et Nikolaos G. Frangogiannis. « Aging, cardiac repair and Smad3 ». Aging 10, no 9 (20 septembre 2018) : 2230–32. http://dx.doi.org/10.18632/aging.101567.
Texte intégralWestin, Oscar, Finn Gustafsson et Emil Fosbøl. « Occult cardiac amyloidosis ? » Aging 11, no 20 (19 octobre 2019) : 8739–40. http://dx.doi.org/10.18632/aging.102383.
Texte intégralThèses sur le sujet "Aging cardiac"
Inuzuka, Yasutaka. « Suppression of phosphoinositide 3-kinase prevents cardiac aging in mice ». Kyoto University, 2009. http://hdl.handle.net/2433/126462.
Texte intégralHaynes, Premi. « TRANSMURAL HETEROGENEITY OF CELLULAR LEVEL CARDIAC CONTRACTILE PROPERTIES IN AGING AND HEART FAILURE ». UKnowledge, 2014. http://uknowledge.uky.edu/physiology_etds/16.
Texte intégralYang, Bo, et 杨波. « Role of lipocalin-2 in cardiac dysfunction associated with aging and dietary obesity ». Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47869641.
Texte intégralpublished_or_final_version
Pharmacology and Pharmacy
Doctoral
Doctor of Philosophy
Ueno, Linda Massako. « Effects of aging and regular exercise upon baroreflex sensitivity and cardiac autonomic activities ». Kyoto University, 2003. http://hdl.handle.net/2433/148937.
Texte intégral0048
新制・課程博士
博士(人間・環境学)
甲第10300号
人博第187号
14||151(吉田南総合図書館)
新制||人||46(附属図書館)
UT51-2003-H721
京都大学大学院人間・環境学研究科文化・地域環境学専攻
(主査)教授 森谷 敏夫, 教授 中村 榮太郎, 教授 津田 謹輔
学位規則第4条第1項該当
Amano, Masari. « Effects of exercise training, aging, gender and mental stress upon cardiac autonomic nervous function ». Kyoto University, 2003. http://hdl.handle.net/2433/148936.
Texte intégral0048
新制・課程博士
博士(人間・環境学)
甲第10299号
人博第186号
14||150(吉田南総合図書館)
新制||人||45(附属図書館)
UT51-2003-H720
京都大学大学院人間・環境学研究科文化・地域環境学専攻
(主査)教授 森谷 敏夫, 教授 津田 謹輔, 教授 田口 貞善
学位規則第4条第1項該当
Ramsey, Michael Wiechmann. « Effects of head-up tilt on mean arterial pressure, heart rate, and regional cardiac output distribution in aging rats ». Texas A&M University, 2005. http://hdl.handle.net/1969.1/3094.
Texte intégralAnstine, Lindsey J. « Valve cell dynamics in developing, mature, and aging heart valves ». The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1478692972995079.
Texte intégralLODRINI, ALESSANDRA MARIA. « Cellular senescence and failure in human and animal cardiac myocytes ». Doctoral thesis, Università degli Studi di Milano-Bicocca, 2021. http://hdl.handle.net/10281/301783.
Texte intégralDuring my PhD I was involved mainly in two research projects aimed to study myocardial dysfunction induced by aging or chemotherapy. The first study aimed to reproduce and characterize mechanisms involved in aging using cardiomyocytes (CMs) from human induced pluripotent stem cells (hiPSCs), and to test cardioprotective therapies, like cardiac progenitor cell (CPC)-derived exosomes (Exo). Aging of the heart involves adverse remodeling in CMs which results in heart failure with incidence that increases with age. Interestingly, till now we lacked a human model of cardiac aging. We reprogrammed CPCs into hiPSCs and subsequently differentiated in hiPSC-derived CMs. A senescence-like phenotype (SenCMs) was induced by short exposure (3 hours) to doxorubicin (Dox) at sub-lethal concentration (0.2 µM). 24h following DOX treatment, SenCMs were exposed to Exo (~2·103 particles/cell) collected from culture media of CPCs by ultracentrifugation. Dox treatment induced senescence, as confirmed by activation of p21 and increased SA-β-gal positivity compared to control CMs (cCMs). Biochemical analysis revealed presence of oxidative stress and a depolarized mitochondrial membrane potential due to the treatment, which resulted in decreased ATP production by mitochondria. SenCMs also showed impaired calcium handling and prolonged QTc vs. cCMs due to upregulation of INaL. These effects were mitigated by exposure to Exo. Overall, SenCMs recapitulate the phenotype of aged CMs in terms of senescence markers and electrical and metabolic properties. Additionally, exposure to CPC-derived Exo limited age-related cardiac anomalies. The second study aimed to study the cardiac dysfunction dependent on the combined administration of Dox and trastuzumab (Trz) through evaluation of cardiac performance, T-tubule organization, and electrophysiological changes in cardiac myocytes from an in-vivo rat model. Combined treatment with Dox and Trz in patients with HER2-positive cancer is limited by cardiotoxicity, as manifested by contractile dysfunction and arrhythmia. The respective roles of the two agents in the cardiotoxicity of the combined therapy are incompletely understood. Adult rats received 6 doses of either Dox or Trz, or the two agents sequentially. Dox-mediated left ventricular (LV) dysfunction was aggravated by Trz administration. Dox treatment, but not Trz, induced T-tubule disarray. Moreover, Dox, but not Trz monotherapy, induced prolonged action potential duration (APD), increased incidence of delayed afterdepolarizations (DADs) and beat-to-beat variability of repolarization (BVR), and slower Ca2+ transient decay. Although APD, DADs, BVR and Ca2+ transient decay recovered over time after the cessation of Dox treatment, subsequent Trz administration exacerbated these abnormalities. Trz, but not Dox, reduced Ca2+ transient amplitude and SR Ca2+ content. Both agents increased Ca2+ waves and downregulated SERCA. Finally, Dox increased resting Ca2+ waves, Ca2+ spark frequency, spark-mediated sarcoplasmic reticulum (SR) leak, and long-lasting Ca2+ release events (so-called Ca2+ “embers”). These results suggest that Dox, but not Trz, may cause T-tubule disarray in cardiac myocytes in vivo while also inducing overall larger changes in electrical parameters and intracellular Ca2+ handling. While Dox-induced changes in electrical parameters are reversible, subsequent Trz administration prevents their recovery. These findings illustrate the specific roles of Dox and Trz, and their interactions in cardiotoxicity and arrhythmogenicity.
ZENI, FILIPPO. « Circulating levels of soluble Receptor for Advanced Glycation End-products (sRAGE) decrease with aging and may predict age-related cardiac remodeling ». Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/170797.
Texte intégralBackground: Aging is an unavoidable risk factor in later life that can influence the onset and progression of many diseases. In fact, the high incidence of cardiovascular diseases in the elderly is mainly attributable to cardiac remodelling associated to physiological intrinsic aging. RAGE is a multi-ligand receptor involved in many age-related disorders. Its soluble isoform (sRAGE) acts as a decoy receptor being able to block the activation of the membrane-bound receptor, and its circulation levels have been found altered in several chronic and acute pathologies. The role of RAGE isoforms in aging and, in particular, cardiac senescence has never been investigated. Moreover, the finding of reliable biomarkers able to assess individual health status of subjects has important applications in prevention, diagnosis, and disease management. In this context, the aim of this study was to ascertain whether sRAGE is a biomarker of aging and age-related cardiac remodelling, and evaluate the contribution of RAGE isoforms to cardiac aging. Results: Serum of male and female from 20 to 92 years old healthy subjects was collected and sRAGE levels were evaluated by ELISA. We found a significant decrease of circulating sRAGE in males while only a trend in females. Accordingly, we observed a strong correlation of sRAGE with chronological age in male but not in female subjects. Male and female mice at different age (2.5-12-22-months, Young, Middle Age (MA) and Old, respectively) undergone 2D-echocardiography to determine the left ventricle (LV) dimensions and function during aging. Serum sRAGE similarly declines from the Young to the MA group in both sexes, and inversely correlate with LV dimensions and function, preferentially in males. No detectable amount of RAGE protein was found in LV at all ages. Rage-/- mice displayed a significant increase of LV volumes and diameters in diastole and systole, and a concomitant decrease in ejection fraction (EF) and fractional shortening (FS), compared to age-matched wt animals during aging with the strongest differences present between the MA groups. Moreover, MA Rage-/- mice exhibited higher deposition of collagen and expression of heart failure marker genes (BNP and Ankrd1) in respect to the wt counterpart. Conversely, no differences in cardiomyocytes size were observed at any age between the two genotypes. Finally, microarray functional annotation analysis based on the interaction between age-genotype revealed that the chronic lack of RAGE affected the expression of genes associated to contractile fibre function, antigen presenting process and adaptive immunity, insulin pathway, cell death and apoptosis. We also found a correlation between LV volumes and diameters in diastole and systole and differentially expressed genes involved in several processes like muscle contraction, fibrosis, wound healing and regulation of apoptosis. Conclusions: Our results indicate that sRAGE is a serum biomarker of healthy aging and age-related cardiac remodeling, preferentially in males. The absence of RAGE in mice exacerbates adverse cardiac remodeling with age. We propose that, among RAGE isoforms, sRAGE may play a pivotal role in cardiac senescence.
Sheikh, Hajer Nisar. « Tropomyosin Phosphorylation in Cardiac Health and Disease ». University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1242913472.
Texte intégralLivres sur le sujet "Aging cardiac"
United States. Congress. Senate. Special Committee on Aging. Pacemakers revisited : A saga of benign neglect : hearing before the Special Committee on Aging, United States Senate, Ninety-ninth Congres, first session, Washington, DC, May 10, 1985. Washington : U.S. G.P.O., 1986.
Trouver le texte intégralOffice, General Accounting. Medicare's policies and prospective payment rates for cardiac pacemaker surgeries need review and revision : Report to the Chairman, Special Committee on Aging, United States Senate. Washington, D.C : U.S. General Accounting Office, 1985.
Trouver le texte intégralFried, Robert, et Richard Carlton. Omega-Factor : Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Trouver le texte intégralFried, Robert, et Richard Carlton. Omega-Factor : Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Trouver le texte intégralFried, Robert, et Richard Carlton. Omega-Factor : Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Trouver le texte intégralFried, Robert, et Richard Carlton. Omega-Factor : Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Trouver le texte intégralFried, Robert, et Richard Carlton. Omega-Factor : Promoting Health, Preventing Premature Aging and Reducing the Risk of Sudden Cardiac Death. Taylor & Francis Group, 2023.
Trouver le texte intégralCardiothoracic surgery in the elderly. New York : Springer, 2011.
Trouver le texte intégralCardiothoracic surgery in the elderly. New York : Springer, 2011.
Trouver le texte intégralKatlic, Mark R. Cardiothoracic Surgery in the Elderly. Springer New York, 2014.
Trouver le texte intégralChapitres de livres sur le sujet "Aging cardiac"
Dai, Dao-Fu, Robert J. Wessells, Rolf Bodmer et Peter S. Rabinovitch. « Cardiac Aging ». Dans The Comparative Biology of Aging, 259–86. Dordrecht : Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3465-6_12.
Texte intégralChantler, Paul D., et Edward G. Lakatta. « Heart and Arterial Aging ». Dans Cardiac Adaptations, 111–44. New York, NY : Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5203-4_7.
Texte intégralFenton, Richard A., Mojca Lorbar et James G. Dobson. « Adenosine and Cardiac Aging ». Dans Developments in Cardiovascular Medicine, 143–58. Boston, MA : Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5603-9_9.
Texte intégralCesselli, Daniela, Federica D’Aurizio, Patrizia Marcon, Natascha Bergamin, Carlo Alberto Beltrami et Antonio Paolo Beltrami. « Cardiac Stem Cell Senescence ». Dans Stem Cells and Aging, 81–97. Totowa, NJ : Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-317-6_7.
Texte intégralJugdutt, Bodh I., et Anwar Jelani. « Aging and Markers of Adverse Remodeling After Myocardial Infarction ». Dans Cardiac Remodeling, 487–512. New York, NY : Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-5930-9_27.
Texte intégralKnowlton, Anne A. « Estrogen, Cardiac Protection and Aging ». Dans Integrative Biology of Women’s Health, 157–74. New York, NY : Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8630-5_9.
Texte intégralBolli, Roberto, et Piero Anversa. « Stem Cells and Cardiac Aging ». Dans Cardiovascular Regeneration and Stem Cell Therapy, 171–81. Oxford, UK : Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470988909.ch18.
Texte intégralSeara, Fernando A. C., Leonardo Maciel, Tais Hanae Kasai-Brunswick, Jose H. M. Nascimento et Antonio C. Campos-de-Carvalho. « Extracellular Vesicles and Cardiac Aging ». Dans Advances in Experimental Medicine and Biology, 33–56. Singapore : Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-1443-2_3.
Texte intégralAdams, Kent J. « Cardiac Rehabilitation and Exercise ». Dans Handbook of Clinical Nutrition and Aging, 419–36. Totowa, NJ : Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-391-0_17.
Texte intégralZhao, Cuimei, Guoping Li et Jin Li. « Non-coding RNAs and Cardiac Aging ». Dans Advances in Experimental Medicine and Biology, 247–58. Singapore : Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-1671-9_14.
Texte intégralActes de conférences sur le sujet "Aging cardiac"
Lai, Yi-Homg, Yi Yuan, Yu-Xian Liang et Jia-Han Yu. « Comparison of aging effect between cardiac complexity and baroreceptor sensitivity ». Dans 2021 International Conference on Information Technology and Biomedical Engineering (ICITBE). IEEE, 2021. http://dx.doi.org/10.1109/icitbe54178.2021.00068.
Texte intégralDimitrakopoulos, Georgios N., Konstantina Dimitrakopoulou, Ioannis A. Maraziotis, Kyriakos Sgarbas et Anastasios Bezerianos. « Supervised method for construction of microRNA-mRNA networks : Application in cardiac tissue aging dataset ». Dans 2014 36th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2014. http://dx.doi.org/10.1109/embc.2014.6943593.
Texte intégralStarc, V., et T. T. Schlegel. « The effect of aging and cardiac disease on that portion of QT interval variability that is independent of Heart Rate Variability ». Dans 2008 35th Annual Computers in Cardiology Conference. IEEE, 2008. http://dx.doi.org/10.1109/cic.2008.4749041.
Texte intégralHeyne, E., R. Musleh, L. G. Koch, S. L. Britton, T. Doenst et M. Schwarzer. « Aging Is Not Related to Increased Cardiac Mitochondrial ROS Production in a Rat Model of Genetically Determined High or Low Exercise Capacity ». Dans 51st Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1742786.
Texte intégralZhingre Sanchez, Jorge D., Emma A. Schinstock, Michael G. Bateman et Paul A. Iaizzo. « The Development and Testing of a Fixation Apparatus for Inducing the Coaptation of the Cardiac Atrioventricular Valves ». Dans 2019 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/dmd2019-3298.
Texte intégralMen, Jing, Airong Li, Zhiwen Yang, Jixu Yu, Rudolph Tanzi et Chao Zhou. « Heartbeat recovery after cardiac arrest and association of maximum pace-able heart rate with aging via optogenetic pacing and OCM imaging of Drosophila (Conference Presentation) ». Dans Optical Coherence Tomography and Coherence Domain Optical Methods in Biomedicine XXIV, sous la direction de Joseph A. Izatt et James G. Fujimoto. SPIE, 2020. http://dx.doi.org/10.1117/12.2549151.
Texte intégralCoelho, Patricia, Ana Rodrigues, Maria Vieira, Joana Liberal et Francisco Rodrigues. « The influence of Monfortinho thermal waters on human health ». Dans III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-241.
Texte intégralDuvall, Julia, Rachael Granberry, Lucy E. Dunne, Brad Holschuh, Christopher Johnson, Kevin Kelly, Bruce Johnson et Michael Joyner. « The Design and Development of Active Compression Garments for Orthostatic Intolerance ». Dans 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3480.
Texte intégralFira, Monica, Liviu Goras, Victor-Andrei Maiorescu et Mihaela Catalina Luca. « Compressed Sensing and Classification of Cardiac Beats using Patient Specific Dictionaries ». Dans 2nd International Conference on Information and Communication Technologies for Ageing Well and e-Health. SCITEPRESS - Science and and Technology Publications, 2016. http://dx.doi.org/10.5220/0005793401730179.
Texte intégralSeong, Jaehoon, Ajay K. Wakhloo et Baruch B. Lieber. « Morphological Age-Depended Development of the Human Carotid Bifurcation ». Dans ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2545.
Texte intégralRapports d'organisations sur le sujet "Aging cardiac"
Slaughter-Acey, Jaime, Kathryn Behrens, Amy M. Claussen, Timothy Usset, Carrie Neerland, Sameerah Bilal-Roby, Huda Bashir et al. Social and Structural Determinants of Maternal Morbidity and Mortality : An Evidence Map. Agency for Healthcare Research and Quality (AHRQ), décembre 2023. http://dx.doi.org/10.23970/ahrqepccer264.
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