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Littérature scientifique sur le sujet « AD non amnesico »

Auteur : Grafiati

Publié le 9 mars 2023

Mis à jour le 31 juillet 2025

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Articles de revues sur le sujet "AD non amnesico"

Souza, Leonardo Cruz de, Maxime Bertoux, Aurélie Funkiewiez, et al. "Frontal presentation of Alzheimer's disease: A series of patients with biological evidence by CSF biomarkers." Dementia & Neuropsychologia 7, no. 1 (2013): 66–74. http://dx.doi.org/10.1590/s1980-57642013dn70100011.

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ABSTRACT Besides its typical amnesic presentation, focal atypical presentations of Alzheimer's disease (AD) have been described in neuropathological studies. These phenotypical variants of AD (so-called "atypical AD") do not follow the typical amnestic pattern and include non-amnestic focal cortical syndromes, such as posterior cortical atrophy and frontal variant AD. These variants exhibit characteristic histological lesions of Alzheimer pathology at post-mortem exam. By using physiopathological markers, such as cerebrospinal fluid markers, it is now possible to establish in vivo a biological

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Aguilar-Navarro, Sara G., Itzel I. Gonzalez-Aparicio, José Alberto Avila-Funes, Teresa Juárez-Cedillo, Teresa Tusié-Luna та Alberto Jose Mimenza-Alvarado. "Association between ApoE ε4 Carrier Status and Cardiovascular Risk Factors on Mild Cognitive Impairment among Mexican Older Adults". Brain Sciences 11, № 1 (2021): 68. http://dx.doi.org/10.3390/brainsci11010068.

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Mild cognitive impairment (MCI) (amnestic or non-amnestic) has different clinical and neuropsychological characteristics, and its evolution is heterogeneous. Cardiovascular risk factors (CVRF), such as hypertension, diabetes, or dyslipidemia, and the presence of the Apolipoprotein E ε4 (ApoE ε4) polymorphism have been associated with an increased risk of developing Alzheimer’s disease (AD) and other dementias but the relationship is inconsistent worldwide. We aimed to establish the association between the ApoE ε4 carrier status and CVRF on MCI subtypes (amnestic and non-amnestic) in Mexican ol

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Nevzorova, K. V., Y. A. Shpilyukova, E. Yu Fedotova, A. G. Burmak, A. A. Shabalina, and S. N. Illarioshkin. "The experience of diagnosing Alzheimer’s disease based on the study of cerebrospinal fluid biomarkers." S.S. Korsakov Journal of Neurology and Psychiatry 125, no. 1 (2025): 91. https://doi.org/10.17116/jnevro202512501191.

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Objective. To evaluate the frequency of Alzheimer’s disease (AD) confirmed by cerebrospinal fluid (CSF) biomarkers in a cohort of patients with classical (amnesic) and atypical phenotypes of this disease. Material and methods. The study included 63 patients (24 men and 39 women; median age 65 years [60; 71]). All patients were divided into 3 groups according to the phenotype: a classic amnesic phenotype (n=32), frequent non-amnesic phenotypes (n=21) and rare non-amnesic phenotypes (n=10). All patients underwent lumbar puncture followed by the CSF biomarkers evaluation (beta-amyloid 1—42 (Aβ1—4

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Putcha, Deepti, Nicole Carvalho, Sheena Dev, Scott M. McGinnis, Bradford C. Dickerson, and Bonnie Wong. "Verbal Encoding Deficits Impact Recognition Memory in Atypical “Non-Amnestic” Alzheimer’s Disease." Brain Sciences 12, no. 7 (2022): 843. http://dx.doi.org/10.3390/brainsci12070843.

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Memory encoding and retrieval deficits have been identified in atypical Alzheimer’s disease (AD), including posterior cortical atrophy (PCA) and logopenic variant primary progressive aphasia (lvPPA), despite these groups being referred to as “non-amnestic”. There is a critical need to better understand recognition memory in atypical AD. We investigated performance on the California Verbal Learning Test (CVLT-II-SF) in 23 amyloid-positive, tau-positive, and neurodegeneration-positive participants with atypical “non-amnestic” variants of AD (14 PCA, 9 lvPPA) and 14 amnestic AD participants. Reco

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Bergeron, David, Jean-Mathieu Beauregard, Jean-Guimond, et al. "Posterior Cingulate Cortex Hypometabolism in Non-Amnestic Variants of Alzheimer’s Disease." Journal of Alzheimer's Disease 77, no. 4 (2020): 1569–77. http://dx.doi.org/10.3233/jad-200567.

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Background: Hypometabolism of the posterior cingulate cortex (PCC) is an important diagnostic feature of late-onset, amnestic Alzheimer’s disease (AD) measured with 18F-fluorodeoxyglucose positron emission tomography (FDG-PET). However, it is unclear whether PCC hypometabolism has diagnostic value in young-onset, non-amnestic variants of AD, which exhibit less pathology in the hippocampus and default mode network. Objective: Evaluate the prevalence and diagnostic value of PCC hypometabolism in non-amnestic variants of AD. Methods: We retrospectively identified 60 patients with young-onset, aty

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Polden, Megan, and Trevor J. Crawford. "Eye Movement Latency Coefficient of Variation as a Predictor of Cognitive Impairment: An Eye Tracking Study of Cognitive Impairment." Vision 7, no. 2 (2023): 38. http://dx.doi.org/10.3390/vision7020038.

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Studies demonstrated impairment in the control of saccadic eye movements in Alzheimer’s disease (AD) and people with mild cognitive impairment (MCI) when conducting the pro-saccade and antisaccade tasks. Research showed that changes in the pro and antisaccade latencies may be particularly sensitive to dementia and general executive functioning. These tasks show potential for diagnostic use, as they provide a rich set of potential eye tracking markers. One such marker, the coefficient of variation (CV), is so far overlooked. For biological markers to be reliable, they must be able to detect abn

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Becker, James T., Olurotimi Bajulaiye, and Christine Smith. "Longitudinal analysis of a two-component model of the memory deficit in Alzheimer's disease." Psychological Medicine 22, no. 2 (1992): 437–45. http://dx.doi.org/10.1017/s0033291700030385.

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SYNOPSISThe memory deficit in Alzheimer's disease (AD) has been characterized as consisting of multiple components. The purpose of this study was to confirm the utility of a two-process model, and to examine changes in the nature and extent of the neuropsychological deficits after a one-year interval. The results replicate the initial observation that the memory loss in AD can be described as consisting of a focal amnesic syndrome and a dysexecutive syndrome characterized by failure of rapid information processing and search of both episodic and semantic memory. One year after the initial obse

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Xue, Xiaofan, Shanshan Mei, Anqi Huang, et al. "Alzheimer’s Disease Related Biomarkers Were Associated with Amnestic Cognitive Impairment in Parkinson’s Disease: A Cross-Sectional Cohort Study." Brain Sciences 14, no. 8 (2024): 787. http://dx.doi.org/10.3390/brainsci14080787.

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Background: Cognitive impairment is common in patients with Parkinson’s disease (PD) and occurs through multiple mechanisms, including Alzheimer’s disease (AD) pathology and the involvement of α-synucleinopathies. We aimed to investigate the pathological biomarkers of both PD and AD in plasma and neuronal extracellular vesicles (EVs) and their association with different types of cognitive impairment in PD patients. Methods: A total of 122 patients with PD and 30 healthy controls were included in this cross-sectional cohort study between March 2021 and July 2023. Non-dementia PD patients were d

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Andrejeva, Nadeshda, Maren Knebel, Vasco Dos Santos, et al. "Neurocognitive Deficits and Effects of Cognitive Reserve in Mild Cognitive Impairment." Dementia and Geriatric Cognitive Disorders 41, no. 3-4 (2016): 199–209. http://dx.doi.org/10.1159/000443791.

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Background/Aims: Mild cognitive impairment (MCI) is a frequent syndrome in the older population, which involves an increased risk to develop Alzheimer's disease (AD). The latter can be modified by the cognitive reserve, which can be operationalized by the length of school education. MCI can be differentiated into four subtypes according to the cognitive domains involved: amnestic MCI, multiple-domain amnestic MCI, non-amnestic MCI and multiple-domain non-amnestic MCI. While neurocognitive deficits are a constituent of the diagnosis of these subtypes, the question of how they refer to the cogni

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Bairami, Styliani, Vasiliki Folia, Ioannis Liampas, et al. "Exploring Verbal Fluency Strategies among Individuals with Normal Cognition, Amnestic and Non-Amnestic Mild Cognitive Impairment, and Alzheimer’s Disease." Medicina 59, no. 10 (2023): 1860. http://dx.doi.org/10.3390/medicina59101860.

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Background and Objectives: The present study explored the utilization of verbal fluency (VF) cognitive strategies, including clustering, switching, intrusions, and perseverations, within both semantic (SVF) and phonemic (PVF) conditions, across a continuum of neurocognitive decline, spanning from normal cognitive ageing (NC) to mild cognitive impairment (MCI) and its subtypes, amnestic (aMCI) and non-amnestic (naMCI), as well as AD. Materials and Methods: The study sample was derived from the Hellenic Longitudinal Investigation of Aging and Diet (HELIAD) cohort. The sample included 1607 NC ind

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Thèses sur le sujet "AD non amnesico"

DA, RE FULVIO. "An MRI-based analysis of the longitudinal progression of atrophy in amnestic and non-amnestic phenotypes of Alzheimer’s disease." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2020. http://hdl.handle.net/10281/261941.

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Recenti studi sulla progressione della malattia d’Alzheimer(AD)suggeriscono che la patologia possa essere trasmessa da un’area all’altra del cervello tramite diffusione locale o lungo le fibre assonali.Tuttavia,quest’ipotesi necessita di maggiori conferme ed è ancora meno chiaro se questi modelli possano essere applicabili alle varianti non amnesiche di AD(naAD),un gruppo di fenotipi AD caratterizzati da relativo risparmio all’esordio della memoria episodica e deficit cognitivi dominio-specifici.Pochi studi ad oggi hanno infatti analizzato la progressione longitudinale della malattia in naAD,

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Chapitres de livres sur le sujet "AD non amnesico"

Santana Isabel, Baldeiras Inês, Santiago Beatriz, et al. "Underlying Biological Processes in Mild Cognitive Impairment: Amyloidosis Versus Neurodegeneration." In Advances in Alzheimer’s Disease. IOS Press, 2018. https://doi.org/10.3233/978-1-61499-876-1-645.

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The amyloid cascade hypothesis proposes amyloid-&beta; (A&beta;) as the earliest and key pathological hallmark of Alzheimer's disease (AD), but this mandatory &ldquo;amyloid-first pathway&rdquo; has been contested. Longitudinal studies of mild cognitive impairment (MCI) patients represent an opportunity to investigate the intensity of underlying biological processes (amyloidosis versus neurodegeneration) and their relevance for progression to AD. We re-examined our cohort of amnestic MCI, grouped according to cerebrospinal fluid (CSF) biomarkers, aiming at establishing their pr

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Förster Stefan, Buschert Verena C., Teipel Stefan J., et al. "Effects of a 6-Month Cognitive Intervention on Brain Metabolism in Patients with Amnestic MCI and Mild Alzheimer's Disease." In Advances in Alzheimer’s Disease. IOS Press, 2011. https://doi.org/10.3233/978-1-60750-793-2-605.

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The effect of cognitive intervention on brain metabolism in AD is largely unexplored. Therefore, we aimed to investigate cognitive parameters and18FDG PET to test for effects of a cognitive intervention in patients with aMCI or mild AD. Patients with aMCI (N = 24) or mild AD (N = 15) were randomly assigned either to cognitive intervention groups (IGs), receiving weekly sessions of group-based multicomponent cognitive intervention, or active control groups (CGs), receiving pencil-paper exercises for self-study. We obtained resting-state FDG-PET scans and neuropsychological testing at baseline a

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Actes de conférences sur le sujet "AD non amnesico"

Gonçalves, Brenda, Isadora Ribeiro, Thamires Magalhães, et al. "NEUROPSYCHOLOGICAL TESTS AS PREDICTORS OF CONVERSION TO ALZHEIMER’S DISEASE IN BETA-AMYLOID POSITIVE INDIVIDUALS." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda007.

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Background: amnestic Mild Cognitive Impairment (aMCI) refers to a possibletransitional stage between healthy aging and dementia and has an increased chance of converting to Alzheimer’s disease (AD). Objectives: to assess whether neuropsychological tests can predict the conversion to AD in patients with aMCI and altered CSF amyloid peptide (βA+). Methods: 48 individuals underwent neuropsychological assessment (time 0 and time 1), being 18 healthy controls and 30 aMCI βA+, who performed a single CSF collection (time 0). All subjects with aMCI scored 0.5 in the Memory category of the Clinical Dem

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Almeida, Eliane Borca, Geise Silva, Isabella Avolio, et al. "INVESTIGATION OF EPISODIC MEMORY DEFCITS IN PATIENTS WITH MILD COGNITIVE IMPAIRMENT (MCI)." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda043.

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Background: MCI can be classified as amnestic (aMCI) or non-amnestic (naMCI). Patients with aMCI are at increased risk of developing Alzheimer’s disease (AD). The clinical diagnosis encompasses episodic memory decline with preservation of activities of daily living, in addition to possible changes in other cognitive domains. Nevertheless, there is a lack of studies in the Brazilian population comparing the performance of aMCI on different episodic memory tests. Objectives: This study investigated episodic memory alterations in patients with aMCI and healthy controls (HC) through population-val

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