Littérature scientifique sur le sujet « Β2m »
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Articles de revues sur le sujet "Β2m"
Chen, Zhaosan, Nianzhi Zhang, Shuangshuang Lu, Mansoor Tariq, Junya Wang et Chun Xia. « Crystallization and preliminary X-ray diffraction analysis of the two distinct types of zebrafish β2-microglobulin ». Acta Crystallographica Section F Structural Biology Communications 71, no 6 (22 mai 2015) : 794–98. http://dx.doi.org/10.1107/s2053230x15005737.
Texte intégralLonnemann, Gerhard, et Karl M. Koch. « β2-Microglobulin Amyloidosis : Effects of Ultrapure Dialysate and Type of Dialyzer Membrane ». Journal of the American Society of Nephrology 13, suppl 1 (janvier 2002) : S72—S77. http://dx.doi.org/10.1681/asn.v13suppl_1s72.
Texte intégralShields, Michael J., Nassim Assefi, Wesley Hodgson, Ellen J. Kim et Randall K. Ribaudo. « Characterization of the Interactions Between MHC Class I Subunits : A Systematic Approach for the Engineering of Higher Affinity Variants of β2-Microglobulin ». Journal of Immunology 160, no 5 (1 mars 1998) : 2297–307. http://dx.doi.org/10.4049/jimmunol.160.5.2297.
Texte intégralStefanovic', Vladisav, Svetislav Kostic', Vidojko Djordjevic', Marina Mitic' et Momcilo Bogicevic'. « β2-Microglobulin Elimination in End-Stage Renal Disease Patients on Renal Replacement Therapy ». Peritoneal Dialysis International : Journal of the International Society for Peritoneal Dialysis 13, no 2_suppl (janvier 1993) : 520–22. http://dx.doi.org/10.1177/089686089301302s127.
Texte intégralZhang, Fan, Yu-Ming Chu et Wei-Mao Qian. « Bounds for the Arithmetic Mean in Terms of the Neuman-Sándor and Other Bivariate Means ». Journal of Applied Mathematics 2013 (2013) : 1–7. http://dx.doi.org/10.1155/2013/582504.
Texte intégralCorlin, Dorthe B., Jette W. Sen, Søren Ladefoged, Grethe Bjerregaard Lund, Mogens H. Nissen et Niels HH Heegaard. « Quantification of Cleaved β2-Microglobulin in Serum from Patients Undergoing Chronic Hemodialysis ». Clinical Chemistry 51, no 7 (1 juillet 2005) : 1177–84. http://dx.doi.org/10.1373/clinchem.2005.049544.
Texte intégralZHU, Xiaoping, Junmin PENG, Raktima RAYCHOWDHURY, Atsushi NAKAJIMA, Wayne I. LENCER et Richard S. BLUMBERG. « The heavy chain of neonatal Fc receptor for IgG is sequestered in endoplasmic reticulum by forming oligomers in the absence of β2-microglobulin association ». Biochemical Journal 367, no 3 (1 novembre 2002) : 703–14. http://dx.doi.org/10.1042/bj20020200.
Texte intégralGood, Sarah C., Katherine M. Dewison, Sheena E. Radford et Patricija van Oosten-Hawle. « Global Proteotoxicity Caused by Human β2 Microglobulin Variants Impairs the Unfolded Protein Response in C. elegans ». International Journal of Molecular Sciences 22, no 19 (4 octobre 2021) : 10752. http://dx.doi.org/10.3390/ijms221910752.
Texte intégralYuichiro Higashimoto et Yoshihiro Motomiya. « Heparin and β2-microglobulin amyloidogenesis ». GSC Biological and Pharmaceutical Sciences 22, no 2 (28 février 2023) : 070–78. http://dx.doi.org/10.30574/gscbps.2023.22.2.0061.
Texte intégralMorales, Pedro J., Judith L. Pace, Jeralyn S. Platt et Joan S. Hunt. « Placental cytotrophoblast HLA-G5 is synthesized as a β2m-free heavy chain homodimer (42.13) ». Journal of Immunology 178, no 1_Supplement (1 avril 2007) : S35—S36. http://dx.doi.org/10.4049/jimmunol.178.supp.42.13.
Texte intégralThèses sur le sujet "Β2m"
Alhawsawi, Sana Mahmoud. « β2m antibody is a suitable antibody to detect major histocompatibility complex class Ι as well as α chain antibody in healthy tissues and tissues infected with mouse parvovirus 1 ». Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1432738096.
Texte intégralCarbone, Anna Lisa. « Engineering and functional characterisation of pentameric concatenated (α4)₂(β2)₃ and (α4)₃(β2)₂ nicotinic acetylcholine receptors ». Thesis, Oxford Brookes University, 2009. https://radar.brookes.ac.uk/radar/items/6b5c0ace-ecf1-488d-95e6-b112bd79e252/1/.
Texte intégralBohlin, Maria E. « Capillary electrophoresis of β2-glycoprotein I ». Licentiate thesis, Karlstads universitet, Fakulteten för teknik- och naturvetenskap, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:kau:diva-3826.
Texte intégralJones, J. M. « β2 adrenergic receptor gene therapy during cardiopulmonary bypass ». Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.605686.
Texte intégralCross, Deborah Jane. « β2-adrenoceptor gene polymorphisms and hypertension in African Trinidadians ». Thesis, University of Nottingham, 2004. http://eprints.nottingham.ac.uk/13194/.
Texte intégralDropmann, Anne [Verfasser], et Steven [Akademischer Betreuer] Dooley. « TGF-β2 abundance in mice and men : A successful anti-TGF-β2 strategy in biliary-derived liver disease / Anne Dropmann ; Betreuer : Steven Dooley ». Heidelberg : Universitätsbibliothek Heidelberg, 2018. http://d-nb.info/1177150050/34.
Texte intégralSweetman, Chlöe A. « TGF-β2 in human milk research : Exploration of a new field methodology and new findings of biosimilar TGF-β2 in non-human milk ». Scholar Commons, 2018. http://scholarcommons.usf.edu/etd/7233.
Texte intégralWagner, Ylva. « Conformational Change of β2-glycoprotein I : Evaluation of Difference in Binding Capacity of Autoantibodies to Open and Closed Forms of β2-glycoprotein I ». Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-24838.
Texte intégralCOATTI, AURORA. « Heteromeric nicotinic receptors regulate developing and mature prefrontal circuits : interaction with other neuromodulators, and implications for sleep-related hypermotor epilepsy ». Doctoral thesis, Università degli Studi di Milano-Bicocca, 2018. http://hdl.handle.net/10281/199023.
Texte intégralThe cholinergic system extensively innervates mature prefrontal cortex (PFC) and is critical to sustain cortical activation during wakefulness and REM sleep. The implication of nicotinic receptors (nAChRs) in these processes is increasingly recognized. Moreover, nAChRs regulate developing cortical circuits during the first postnatal weeks. First, we studied mice expressing β2-V287L, a mutant subunit of the nAChR linked to Autosomal Dominant Nocturnal Frontal Lobe Epilepsy (ADNFLE). β2-V287L exerts the epileptogenic action by altering the synaptic stabilization during sensitive phases of network maturation, since its expression during the first two postnatal weeks is necessary to develop the epileptic phenotype. The peak of postnatal expression of nAChRs coincides with the excitatory to inhibitory GABAergic switch, which is determined by the progressive decrease in the ratio between the expression of the Na+/K+/Cl- cotransporter-1 (NKCC1) and the K+/Cl- cotransporter-2 (KCC2). How these transporters are regulated in PFC and thalamus is largely unknown. We first studied the distribution of NKCC1 and KCC2 in developing wild-type (WT) mice. The amount of NKCC1 and KCC2 in PFC and somatosensory cortex progressively increased during the first two postnatal weeks. NKCC1 was found in neurons as well as astrocytes. KCC2 was mainly localized in neuronal somata at birth, and subsequently migrated to the somatodendritic membranes. Next, in mice expressing β2-V287L, the KCC2 amount in PFC layer V was lower than in the control littermates at postnatal day 8 (P8), but reached higher amounts by P60. Consistently, the time course of the GABAergic switch was delayed in PFC layer V of mice carrying β2-V287L, as measured by perforated patch method. Irrespective of genotype, NKCC1 and KCC2 were highly expressed in the neuropil of thalamic nuclei at birth. Their amount remained high in the adult sensory nuclei, whereas a significant decrease of KCC2 was observed in the reticular nucleus by P40. Such a decrease was more pronounced in mice expressing β2-V287L. Our results indicate that β2-containing nAChRs interact with KCC2 during synaptogenesis as well as in mature circuits, which may contribute to the pathogenesis of ADNFLE. Second, we explored some aspects of the complex interplay between the main neurotransmitters involved in cortical arousal, i. e. acetylcholine (ACh), norepinephrine (NE) and orexin A (OrxA), in order to characterize their combined effect on the PFC layer V circuit. In particular, by patch-clamp methods, we investigated how these neurotransmitters regulate glutamate release onto pyramidal neurons in PFC layer V of adult wild-type mice. Tonic administration of ACh stimulated glutamate release, mainly through α4-containing nAChRs. Moreover, OrxA increased the glutamate release onto pyramidal neurons, through Orx Receptor 1 (OrxR1). Low concentrations of NE (10 nM) were also effective in increasing glutamatergic release. We conclude that these neuromodulators cooperate in regulating glutamatergic transmission in PFC. Such interaction may be crucial in the cellular mechanisms underlying the cognitive and executive functions in this area, in normal and pathological conditions, which are often accompanied by alterations in the theta rhythms, largely determined by pyramidal neurons dynamics. These results may yield new insights into the complex regulation of the PFC microcircuit and lay the basis for further investigations on the pathogenic mechanisms responsible of altered cortical activity in sleep-related disorders such as frontal epilepsy and narcolepsy with cataplexy.
Haidar, Malak. « Rôle du facteur de croissance transformant (TGF-β2) dans la virulence des macrophages infectés par Theileria annulata ». Thesis, Sorbonne Paris Cité, 2015. http://www.theses.fr/2015PA05T044.
Texte intégralTheileria parasites (Theileria. annulata and T. parva) are intracellular protozoa and members of the phylum Apicomplexa. Theileria parasites are the only eukaryotes that possess the property of being able to transform another eukaryote, their leukocyte host cells. Transformed leukocytes show many characteristics of tumour cells such as heightened invasive capacity; however the tumour-like phenotype can be totally reversed upon drug induced parasite death and attenuated by multiple in vitro passages. Such multiple-passaged attenuated lines are used as live vaccines against tropical theileriosis. The similarities in tumour hyper-invasiveness between Theileria-transformed leukcocytes and human lymphomas imply that observations on Theileria-induced leukocyte transformation have the potential to give generally applicable insights into the mechanisms underpinning tumour virulence. My host laboratory described higher TGF-β2 levels in virulent infected macrophages and following microarray analysis of virulent compared to attenuated macrophages found that among the 1158 TGF-β-targets, 68 genes had altered transcript levels concomitant with attenuation. In this study, we investigate the signalling pathways involved in the regulation of cellular adhesion and invasiveness of Theileria-infected cells. We were especially interested in the study of TGF-β2 signalling in Theileria-transformed virulent versus attenuated macrophages. My results indicate that following Theileria infection of macrophages, the TGF-β2 signalling pathway is activated and induces an increase in adhesion of virulent transformed macrophages through two different mechanisms: either by activating a PGE2 / EP4 / cAMP / PKA / EPAC / CREB signaling pathway, or by stimulating a GRB2 / PI3-K / AP-1 pathway. As attenuated macrophages display heightened oxidative stress, which underpins their loss of adhesion and invasiveness, in collaboration with another PhD student (Mehdi Metheni) we investigated the role of TGF-β2 in the regulation of the oxidative stress status of Theileria-infected macrophages. Our data show that high levels of TGF-β2 increase the expression of catalase, an anti-oxidant enzyme that converts H2O2 into H2O and the drop in H2O2 output results in regain of the virulence trait heightened adhesion of Theileria-transformed macrophages to fibronectin. Theileria-transformed macrophages display many features of cancer cells such as their consumption of larger quantities of glucose. The BCL-2 family protein BAD has an alternative function in glucose metabolism separate from its role in apoptosis. The activity of BAD is regulated by phosphorylation in response to growth/survival factors. BAD can be phosphorylated on Ser155 by PKA. So during my thesis studies I examined the role of PKA mediated phosphorylation of BAD in the regulation of the cellular metabolism of Theileria-transformed macrophages. My results showed that ablation of BAD S155 phosphorylation dissociates the mitochondrial complex of BAD and HK2 and cytosolic HK2 becomes ubiquitinated and degraded by the proteasome. Loss of HK2 switches the metabolism of Theileria-transformed leukocytes from Warburg-like to OXPHOS-like glycolysis
Chapitres de livres sur le sujet "Β2m"
Coon, Joanna S. Thompson, et Anne E. Tattersfield. « β2-Agonists ». Dans Anti-Inflammatory Drugs in Asthma, 137–51. Basel : Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8751-9_4.
Texte intégralKikkawa, Yamato, et Hiroshi Nishimune. « Laminin β2 ». Dans Encyclopedia of Signaling Molecules, 2816–26. Cham : Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101519.
Texte intégralVillars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, V. Kuprysyuk et I. Savysyuk. « Na2ThF6 β2 ». Dans Structure Types. Part 8 : Space Groups (156) P3m1 – (148) R-3, 516. Berlin, Heidelberg : Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-70892-6_300.
Texte intégralYamamoto, Suguru, Junichiro James Kazama, Hiroki Maruyama, Ichiei Narita et Fumitake Gejyo. « β2-Microglobulin ». Dans Uremic Toxins, 249–58. Hoboken, NJ, USA : John Wiley & Sons, Inc., 2012. http://dx.doi.org/10.1002/9781118424032.ch16.
Texte intégralHoldenrieder, S., et P. Stieber. « β2-Mikroglobulin ». Dans Springer Reference Medizin, 1653–54. Berlin, Heidelberg : Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_3400.
Texte intégralHoldenrieder, S., et P. Stieber. « β2-Mikroglobulin ». Dans Lexikon der Medizinischen Laboratoriumsdiagnostik, 1–2. Berlin, Heidelberg : Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_3400-1.
Texte intégralVillars, P., K. Cenzual, J. Daams, R. Gladyshevskii, O. Shcherban, V. Dubenskyy, V. Kuprysyuk, I. Savysyuk et R. Zaremba. « Ta2H β2 ». Dans Structure Types. Part 10 : Space Groups (140) I4/mcm – (136) P42/mnm, 209. Berlin, Heidelberg : Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19662-1_153.
Texte intégralWard, Tony Milford. « β2-Microglobulin ». Dans Proteins and Tumour Markers May 1995, 1259–75. Dordrecht : Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0681-8_50.
Texte intégralKikkawa, Yamato, et Hiroshi Nishimune. « Laminin β2 ». Dans Encyclopedia of Signaling Molecules, 1–11. New York, NY : Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101519-1.
Texte intégralAppleton, Kathryn M., Ian Cushman, Yuri K. Peterson, Balachandran Manavalan, Shaherin Basith, Sangdun Choi, Akihiro Kimura et al. « IFN-β2 ». Dans Encyclopedia of Signaling Molecules, 892. New York, NY : Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100639.
Texte intégralActes de conférences sur le sujet "Β2m"
Lei, Wei-Yi, Chin-Hsuan Hsieh, Chien-Chung Chang, Shao-Hsuan Wen, Chi-Tan Hu et Shuen-Kuei Liao. « Abstract 3622 : Total loss of HLA class I expression by two sarcomatoid hepatocellular carcinoma cell lines sHCC29 and sHCC63 was caused by a ∼49-kbp deletion at chromosome 15q15 across the β2m gene locus ». Dans Proceedings : AACR Annual Meeting 2014 ; April 5-9, 2014 ; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3622.
Texte intégralFranson, J. D. « Nonlocal cancellation of dispersion ». Dans OSA Annual Meeting. Washington, D.C. : Optica Publishing Group, 1991. http://dx.doi.org/10.1364/oam.1991.thr1.
Texte intégralGamo, Hideya. « Quasi-evanescent waves of the total internal reflection at the lossy or gain medium ». Dans OSA Annual Meeting. Washington, D.C. : Optica Publishing Group, 1986. http://dx.doi.org/10.1364/oam.1986.fx3.
Texte intégralAlom, Nur, et Bikash Kumar Sarkar. « Effect of Exit Blade Angle on the Performance of Cross Flow Hydro Turbine : A Numerical Study ». Dans ASME 2021 Gas Turbine India Conference. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/gtindia2021-76037.
Texte intégralYang, C. C., A. Villeneuve, G. I. Stegeman, Cheng-Hui Lin et Hao-Hsiung Lin. « Anisotropic Two-Photon Transition in GaAs/AlGaAs Multiple Quantum Well Waveguides ». Dans Nonlinear Optics. Washington, D.C. : Optica Publishing Group, 1992. http://dx.doi.org/10.1364/nlo.1992.pd2.
Texte intégralYulin, Chen, Chen Kangmin et Zhang Dangfang. « A Variational Finite Element Method for Solving the Blade-to-Blade Flow in Centrifugal Compressor’s Cascades With Splitter Blades on an Arbitrary Streamsheet of Revolution and a Mathematical Treatment to the Region Behind Cascades ». Dans ASME 1985 Beijing International Gas Turbine Symposium and Exposition. American Society of Mechanical Engineers, 1985. http://dx.doi.org/10.1115/85-igt-148.
Texte intégralCarroll, CL, P. Stoltz, AR Zucker et CM Schramm. « Determining β2-Agonist Response in Children with Bronchiolitis. » Dans American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5816.
Texte intégralArchała, Aneta, et Anita Płazińska. « β2-adrenergic receptor polymorphism in intracellular signalling pathways ». Dans 1st International Electronic Conference on Biomedicine. Basel, Switzerland : MDPI, 2021. http://dx.doi.org/10.3390/ecb2021-10265.
Texte intégralElshaarawey, Ahmed, Aida Abdel Hafiz, Hesham Abdou et Osama Khorais. « Applying Machine Learning to Optimize Electrical Submersible Pumps Design for Handling Viscous Oil ». Dans International Petroleum Technology Conference. IPTC, 2022. http://dx.doi.org/10.2523/iptc-22657-ms.
Texte intégralVaghasiya, J. M., A. Sikarwar, A. Dalvand, S. Basu et A. J. Halayko. « Oxidized Phosphatidylcholine Impairs β2-Adrenoceptor Mediated Relaxation of Murine Airways ». Dans American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3275.
Texte intégral