Literatura académica sobre el tema "Ventricular function"

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Artículos de revistas sobre el tema "Ventricular function"

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Conti, C. Richard. "Ventricular Function". Cardiovascular Innovations and Applications 3, n.º 2 (1 de julio de 2018): 267–68. http://dx.doi.org/10.15212/cvia.2017.0057.

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Mills, P. "VENTRICULAR FUNCTION". Heart 71, n.º 4 Suppl (1 de abril de 1994): 33–34. http://dx.doi.org/10.1136/hrt.71.4_suppl.33.

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Vincent, Jean-Louis. "Ventricular function". Baillière's Clinical Anaesthesiology 6, n.º 2 (junio de 1992): 381–93. http://dx.doi.org/10.1016/s0950-3501(05)80234-x.

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Greenberg, S. Bruce y Satinder K. Sandhu. "VENTRICULAR FUNCTION". Radiologic Clinics of North America 37, n.º 2 (marzo de 1999): 341–59. http://dx.doi.org/10.1016/s0033-8389(05)70098-1.

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DIEBOLD, B. y M. TURIEL. "RIGHT VENTRICULAR FUNCTION." Echocardiography 15, n.º 8-2 (noviembre de 1998): S1—S2. http://dx.doi.org/10.1111/j.1540-8175.1998.tb00955.x.

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Dhainaut, Jean François y Pierre Squara. "Right ventricular function". Current Opinion in Anaesthesiology 5, n.º 2 (1992): 235–39. http://dx.doi.org/10.1097/00001503-199205020-00007.

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Amory, D., S. Goldstein, A. Spotnitz, P. Scholz y B. Wagner. "RIGHT VENTRICULAR FUNCTION". Anesthesiology 81, SUPPLEMENT (septiembre de 1994): A173. http://dx.doi.org/10.1097/00000542-199409001-00172.

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Redington, Andrew N. "Right ventricular function". Cardiology Clinics 20, n.º 3 (agosto de 2002): 341–49. http://dx.doi.org/10.1016/s0733-8651(02)00005-x.

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Sheehan, Florence H. "Left ventricular function". International Journal of Cardiac Imaging 11, S1 (marzo de 1995): 55. http://dx.doi.org/10.1007/bf01142222.

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Visser, Lance C. "Right Ventricular Function". Veterinary Clinics of North America: Small Animal Practice 47, n.º 5 (septiembre de 2017): 989–1003. http://dx.doi.org/10.1016/j.cvsm.2017.04.004.

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Tesis sobre el tema "Ventricular function"

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McCormick, Matthew. "Ventricular function under LVAD support". Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:0d49ba30-b508-4c69-9ba6-b398d4338c01.

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This thesis presents a finite element methodology for simulating fluid–solid interactions in the left ventricle (LV) under LVAD support. The developed model was utilised to study the passive and active characteristics of ventricular function in anatomically accurate LV geometries constructed from normal and patient image data. A non–conforming ALE Navier–Stokes/finite–elasticity fluid–solid coupling system formed the core of the numerical scheme, onto which several novel numerical additions were made. These included a fictitious domain (FD) Lagrange multiplier method to capture the interactions between immersed rigid bodies and encasing elastic solids (required for the LVAD cannula), as well as modifications to the Newton–Raphson/line search algorithm (which provided a 2 to 10 fold reduction in simulation time). Additional developments involved methods for extending the model to ventricular simulations. This required the creation of coupling methods, for both fluid and solid problems, to enable the integration of a lumped parameter representation of the systemic and pulmonary circulatory networks; the implementation and tuning of models of passive and active myocardial behaviour; as well as the testing of appropriate element types for coupling non–conforming fluid– solid finite element models under high interface tractions (finding that curvilinear spatial interpolations of the fluid geometry perform best). The behaviour of the resulting numerical scheme was investigated in a series of canonical test problems and found to be convergent and stable. The FD convergence studies also found that discontinuous pressure elements were better at capturing pressure gradients across FD boundaries. The ventricular simulations focused firstly on studying the passive diastolic behaviour of the LV both with and without LVAD support. Substantially different vortical flow features were observed when LVAD outflow was included. Additionally, a study of LVAD cannula lengths, using a particle tracking algorithm to determine recirculation rates of blood within the LV, found that shorter cannulas improved the recirculation of blood from the LV apex. Incorporating myocardial contraction, the model was extended to simulate the full cardiac cycle, converging on a repeating pressure–volume loop over 2 heart beats. Studies on the normal LV geometry found that LVAD implementation restricts the recirculation of early diastolic inflow, and that fluid–solid coupled models introduce greater heterogeneity of myocardial work than was observed in equivalent solid only models. A patient study was undertaken using a myocardial geometry constructed using image data from an LVAD implant recipient. A series of different LVAD flow regimes were tested. It was found that the opening of the aortic valve had a homogenising effect on the spatial variation of work, indicating that the synchronisation of LVAD outflow with the cardiac cycle is more important if the valve remains shut. Additionally, increasing LVAD outflow during systole and decreasing it during diastole led to improved mixing of blood in the ventricular cavity – compared with either the inverse, or holding outflow constant. Validation of these findings has the potential to impact the treatment protocols of LVAD patients.
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Clark, Simon John. "Right ventricular function in respiratory distress syndrome". Thesis, University of Liverpool, 2001. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250478.

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Hedman, Anders. "Noninvasive evaluation of the effects of coronary artery bypass grafting on myocardial function /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-761-8/.

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Danton, Mark Henry Dunn. "Studies in right ventricular function : employing the conductance catheter method for ventricular volume determination". Thesis, Queen's University Belfast, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326470.

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Small, Alexander. "Assessing ventricular function in patients with atrial fibrillation". Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3564/.

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The Frank-Starling law states that the stroke volume of a regular cardiac beat increases in response to an increase in the volume of blood filling the heart. If this law applies in atrial fibrillation (AF) as well as in sinus rhythm (SR) then cardiac function will depend on the duration of diastole in the preceding beat as well as the duration of the indexed beat. Aim: The aim of this thesis was to develop a series of tools which would allow an assessment of the changes in cardiac function from one beat to the next in AF and SR. A secondary aim was to find a means of describing rhythm in a way that reflected possible functional change. Methods: List-mode radionuclide ventriculography, RNVG, acquisitions of 373 patients in AF and a comparative group of 385 patients in SR were made. Software was written which allowed tightly defined preceding and indexed beat selection criteria to be established. Left ventricular ejection fraction (LVEF) and other functional parameters (pre-systolic volume, systolic time, the ratio of pre-systolic to end-diastolic volume, peak filling rate and first third filling fraction) were calculated for images created using different beat selection criteria based on the quartiles of beat length. Assessment used both variable and fixed time formatting and included a comparison of results achieved in the first and second half of the scan. Traditional linear measures of heart rate variability together with descriptors of the Poincar´e plot and cycle length entropy were used to describe rhythm in both AF and SR patients. Results: Substantial variation with indexed and preceding beat length was seen in both SR and AF in all the systolic parameters measured and in particular in LVEF where the standard deviation of LVEF for any one patient was found to be 8.2% in SR and 14.1% in AF. A combination of descriptors of rhythm was found to have good correlation with the range of LVEF measured. Examination of the results for LVEF in several clinical sub-groups suggests that the range of LVEF may have clinical interest. The techniques were applied in a small clinical study which considered the value of radio-frequency ablation in patients with AF and heart failure. In this study, measures of Sample entropy and the range of LVEF appeared to have prognostic value. Conclusion: A tool which allows the investigation of beat-to-beat functional variation in RNVG has been produced. It has been shown that the functional variation depending on beat selection criteria is substantial and may have clinical significance both in patients with underlying pathology and prognostically in patients undergoing radiofrequency ablation (RFA).
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Macnee, W. "Right ventricular function in chronic bronchitis and emphysema". Thesis, University of Glasgow, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383973.

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Maret, Eva. "Noninvasive Evaluation of Myocardial Ischemia and Left Ventricular Function". Doctoral thesis, Linköpings universitet, Klinisk fysiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-18315.

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The general aim of this thesis was, following the path of the ischemic cascade, to evaluate the feasibility of some new non-invasive techniques for the detection of myocardial ischemia, the extent of infarcted myocardium, and for the quantification of systolic left ventricular function. Reduced longitudinal myocardial velocity and displacement may be early signs of ischemia. We evaluated the diagnostic sensitivity and specificity of pulsed tissue Doppler for the detection of ischemia and scar during dobutamine stress testing and compared it with myocardial perfusion scintigraphy (SPECT) in patients with a history of unstable angina. Pulsed tissue Doppler was useful for objective quantification of left ventricular longitudinal shortening and for differentiation between patients with a normal, ischemic or necrotic myocardium. The coronary flow velocity reserve (CFVR) of the left anterior descending artery (LAD) was studied with transthoracic Doppler echocardiography (TTDE) during adenosine stress. Patients with a clinical suspicion of stress induced myocardial ischemia were investigated, and the results were compared with the findings from SPECT. A CFVR >2 in the LAD could exclude significant coronary artery disease in a clinical setting, however, in cases with low CFVR, multiple cardiovascular and metabolic risk factors as well as epicardial coronary artery disease or microvascular dysfunction might be responsible. TTDE is a promising tool, e.g. for follow-up after coronary interventions or for evaluating endothelial function over time. A third study focused on the importance of accurate and reproducible measurements of left ventricular volumes and ejection fraction (LVEF). Patients with known or suspected coronary artery disease with different levels of LVEF were enrolled. We compared the LVEF determined with an automatic echocardiographic method with manual planimetry, visual assessment of LVEF and with quantitative myocardial gated SPECT. The software using learned pattern recognition and artificial intelligence (AutoEF) applied on biplane apical echocardiographic views reduced the variation in measurements without increasing the time required. The method seems to be able to reduce variation in the assessment of LVEF in clinical patients, especially for less experienced readers. We evaluated a new feature tracking software for its ability to detect infarcted myocardium on cine-MR images. Patients were selected based on the presence or absence of myocardial scar in the perfusion area of the LAD. The software tracked myocardial wall motion and allowed the calculation of velocity, displacement and strain in radial and longitudinal directions. Feature tracking of cine-MR images detected scar segments with transmurality >50% within the distribution of the LAD with 80% sensitivity and 86% specificity (radial strain), without the need for the administration of gadolinium-based contrast. In summary, we have evaluated some of the noninvasive techniques in the wide array of diagnostic tools available for the diagnosis of ischemic heart disease. Their availability, low costs, freedom from radiation and repeatability are essential as well as their diagnostic ability.
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Müller-Brunotte, Richard. "Diastolic heart function in hypertension-induced left ventricular hypertrophy /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-898-3/.

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Flannery, Daniel John. "Noninvasive assessment of ventricular function after acute myocardial infarction". Thesis, Queen's University Belfast, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335937.

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Clarkson, Peter Bruce Mark. "Studies of left ventricular diastolic function inhealth and disease". Thesis, University of Dundee, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337397.

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Libros sobre el tema "Ventricular function"

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Raineri, Angelo, Robert D. Leachman y Jan J. Kellermann, eds. Assessment of Ventricular Function. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-8003-0.

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A, Raineri, Kellermann Jan J y Leachman Robert D, eds. Assessment of ventricular function. New York: Plenum, 1985.

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Ježek, V., M. Morpurgo y R. Tramarin, eds. Right Ventricular Hypertrophy and Function in Chronic Lung Disease. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-3853-2.

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Fogel, Mark A., ed. Ventricular Function and Blood Flow in Congenital Heart Disease. Malden, Massachusetts, USA: Blackwell Publishing, 2005. http://dx.doi.org/10.1002/9780470994849.

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Wilson, Mathew G. Left ventricular morphology and function in elite British athletes. Wolverhampton: University of Wolverhampton, 2002.

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A, Fogel Mark, ed. Ventricular function and blood flow in congenital heart disease. Malden, Mass: Blackwell Futura, 2005.

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Biomekhanika neodnorodnosteĭ serdechnoĭ mysht͡s︡y. Moskva: "Nauka", 1993.

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N, Burns Peter, ed. Handbook of contrast echocardiography: Left ventricular function an myocardial perfusion. Berlin: Springer, 2000.

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H, Gaasch William y LeWinter Martin M, eds. Left ventricular diastolic dysfunction and heart failure. Philadelphia: Lea & Febinger, 1994.

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Dries, David J. Right ventricle: The neglected neighbor of the left. Austin, Tx: R.G. Landes, 1994.

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Capítulos de libros sobre el tema "Ventricular function"

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Opie, Lionel H. "Ventricular Function". En Essential Cardiology, 31–44. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6705-2_3.

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Warltier, David C. "Ventricular Function". En Anesthesiology and the Cardiovascular Patient, 39–50. Dordrecht: Springer Netherlands, 1996. http://dx.doi.org/10.1007/978-94-009-1622-7_4.

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Graham, T. P. "Ventricular function". En Congenital Heart Disease, 145–61. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4872-3_6.

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Kühl, Harald P. "Left ventricular function". En Three-dimensional Echocardiography, 55–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-11179-2_4.

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Ayres, S. M. "Abnormal Ventricular Function". En Update in Intensive Care and Emergency Medicine, 1–11. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-83453-0_1.

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Westerhof, Nicolaas, Nikolaos Stergiopulos y Mark I. M. Noble. "Assessing Ventricular Function". En Snapshots of Hemodynamics, 129–35. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-6363-5_19.

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Westerhof, Nicolaas, Nikolaos Stergiopulos, Mark I. M. Noble y Berend E. Westerhof. "Assessing Ventricular Function". En Snapshots of Hemodynamics, 155–62. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91932-4_20.

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Vieillard-Baron, Antoine. "Right Ventricular Function". En Echocardiography in ICU, 139–44. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-32219-9_12.

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Dohi, Kaoru, Norikazu Yamada y Masaaki Ito. "Right Ventricular Function". En Diagnosis and Treatment of Pulmonary Hypertension, 217–36. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-287-840-3_17.

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Mertens, Luc y Mark K. Friedberg. "Systolic Ventricular Function". En Echocardiography in Pediatric and Congenital Heart Disease, 96–131. Oxford, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118742440.ch7.

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Actas de conferencias sobre el tema "Ventricular function"

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Hartley, C. J., R. S. Rabinovitz, B. Patel, L. J. Suignard, H. Litowitz, J. E. Chelly, M. O. Jeroudi et al. "Ultrasonic Sensors For Measuring Regional Ventricular Function". En 1988 Los Angeles Symposium--O-E/LASE '88, editado por Alan I. West. SPIE, 1988. http://dx.doi.org/10.1117/12.945236.

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Jhun, Choon-Sik, Mark B. Ratcliffe y Julius M. Guccione. "Ventricular Wall Stress and Pump Function of Ventricular Septal Defect of Congenital Heart Defects". En ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206320.

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About 36,000 infants are born each year with a congenital heart defect (CHD) and charges for treatment surpass $2.2 billion for inpatient surgery alone. Of many different types of CHDs, ventricular septal defect (VSD) is the most common class (∼1/3 of CHDs) of heart deformity present at birth. Though many close spontaneously and rarely require treatment, VSD still accounts for ∼15% of defects requiring an invasive procedure within the first year of life [1]. Generally, the ventricular performance is indexed by geometry, shape, diastolic and systolic function, and myocardial contractility [2]. Ejection fraction (EF) and end-systolic (ES) wall stress also used to assess the ventricular function [3–5]. Ratcliffe and Guy suggested that the assessment of LV function focusing on indices of systolic function, such as EF and contractility (EES), is misleading because the shift of end-systolic pressure-volume relationship (ESPVR) and increase/decrease in EES and coincident shift of end-diastolic pressure-volume relationship (EDPVR) may result in pseudo increase/decrease of EF even though there may not be any significant change in true LV function (i.e., Starling relationship) [6]. Though Sagawa and associates proposed the ESPVR as a reliable index of intrinsic systolic function [7], it requires derivation of the pressure-volume relationship at different loading conditions by using a noninotropic vasoconstrictor or vasodilator. This may consequently enforce a significant burden on infants with a failing heart. Moreover, irreversible complication of muscle structure can be generated [8–10]. Thus, rigorous quantification of the pump function associated with mechanics has been hindered especially for infants.
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Bhattacharya, Shamik y Zhaoming He. "Tricuspid Valve Annulus Tension". En ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53088.

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Functional tricuspid regurgitation is a direct outcome of right ventricular dilatation and tricuspid annulus dilatation. The mechanism underlying functional tricuspid regurgitation is believed to be multifactorial and related to abnormalities in right ventricular volume, function and shape. Changes in the right ventricle geometry may lead to alterations in the positions of the papillary muscles (PM) of the tricuspid valve (TV). PM displacement happens in right ventricular dilatation but its correlation with tricuspid annulus dilatation is still unknown. The unique structure and orientation of tricuspid PM has role to play in TV annulus mechanics and right ventricular mechanics (Fig.1). It has been already shown that annulus tension (AT) is a parameter to evaluate left ventricular function that, previously, was evaluated via the left ventricular geometry and pressure [1–3].
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Ferreira, Antonio, Yajuan Wang, John Gorcsan y James F. Antaki. "Assessment of Cardiac Function During Mechanical Circulatory Support: A Simulation Study". En ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53735.

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Ventricular Assist Devices (VADs) have emerged as the standard of care for advanced heart failure (HF) patients, requiring mechanical circulatory support [1]. Regardless of the intended use, a small — yet significant — portion of implanted patients exhibits signs of recovery of the left ventricular function [2].
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Nemchyna, O., N. Solowjowa, M. Dandel, Y. Hrytsyna, J. Stein, J. Knierim, F. Schoenrath, V. Falk y C. Knosalla. "Left Ventricular Diastolic Function Assessed by Speckle Tracking Echocardiography in Patients with Left Ventricular Aneurysm". En 51st Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery (DGTHG). Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1742937.

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Chyou, Anthony, Barbara E. Klein, Ronald Klein, R. G. Barr, Mary F. Cotch, Amy Praestgaard, Tien Y. Wong, David A. Bluemke, Joao Lima y Steven M. Kawut. "Retinal Vascular Changes And MRI-Defined Right Ventricular Structure And Function: The Multi-Ethnic Study Of Atherosclerosis Right Ventricular Function Study". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3451.

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Lander, P. y E. J. Barbari. "Analyzing ventricular late potentials using a vector magnitude function". En Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94379.

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AIHARA, KOICHIRO, YUJI NAKAZATO, YASUNOBU KAWANO, KAORU NAKAZATO, MASAYUKI YASUDA, TAKASHI TOKANO y HIROYUKI DAIDA. "SCREENIG OF LEFT VENTRICULAR FUNCTION BY SIGNAL-AVERAGED ELECTROCARDIOGRAM". En Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0160.

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Chesler, N. C., A. Roldan, R. R. Vanderpool y R. Naeije. "How to measure pulmonary vascular and right ventricular function". En 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5333835.

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Kachenoura, Nadjia, Emilie Bollache, Alban Redheuil, Stephanie Clement-Guinaudeau, Ludivine Perdrix, Benoit Diebold, Magalie Ladouceur y Elie Mousseaux. "Right ventricular diastolic function evaluation in magnetic resonance imaging". En 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7408593.

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Informes sobre el tema "Ventricular function"

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Velikov, Toni, Elena Kinova, Bozhidar Krastev, Emilia Stoyanova, Tsvetelina Velikova y Assen Goudev. Left Ventricular Function Assessed by Myocardial Deformation and Torsion in Chronic Hemodialysis Patients. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, octubre de 2020. http://dx.doi.org/10.7546/crabs.2020.10.16.

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Liu, Gejing, Man Ren, Yingshi Du, Xinni Xu, Ruoyu Zhao, Yu Wu, Yongming Liu y Liang Qi. A meta-analysis of Effect of thyroid hormone replacement therapy on the Cardiac diastolic function in Patients with Subclinical Hypothyroidism. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, febrero de 2023. http://dx.doi.org/10.37766/inplasy2023.2.0083.

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Review question / Objective: P:Subclinical Hypothyroidism(Age over 18); I:thyroid hormone replacement therapy; C:baseline(before-after study in the same patient); O:Cardiac diastolic function measurement by echocardiography. Condition being studied: Subclinical hypothyroidism is associated with anomalies left ventricular diastolic functions, however, there are still disputes about whether to use levothyroxine for treatment. This meta-analysis aimed to determine whether levothyroxine (LT4), commonly used to treat hypothyroidism, affects cardiovascular indices in SCH patients as measured by echocardiography.
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Yang, Ming, Fan Guo y Zhi Cheng Jing. Prognostic value of preoperative assessment of left ventricular function in patients undergoing percutaneous coronary intervention. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, febrero de 2022. http://dx.doi.org/10.37766/inplasy2022.2.0031.

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Adhyapak, Srilakshmi y Prahlad Menon. Improvements In Regional Left Ventricular Function After Late Percutaneous Coronary Intervention After Anterior Myocardial Infarction. Peeref, octubre de 2022. http://dx.doi.org/10.54985/peeref.2210p4745262.

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