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Libros sobre el tema "Tumour metastasis"

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Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 20 de febrero de 2023

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1

Károly, Lapis, Eckhardt S y International Union Against Cancer, eds. Carcinogenesis and tumour progression. Budapest: Akadémiai Kiadó, 1987.

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2

Windle, J. M. The role of transglutaminase in tumour growth and metastasis. Birmingham: University of Birmingham, 1985.

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3

M, Chadwick C., ed. Receptors in tumour biology. Cambridge: Cambridge University Press, 1986.

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4

Banfalvi, Gaspar. Homeostasis - Tumor - Metastasis. Dordrecht: Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-007-7335-6.

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5

T, Galeotti, ed. Cell membranes and cancer: Proceedings of the Second International Workshop on Membranes in Tumour Growth, Rome, Italy, June 17-20, 1985. Amsterdam: Elsevier Science, 1985.

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6

Dickson, Robert B. y Marc E. Lippman, eds. Mammary Tumor Cell Cycle, Differentiation, and Metastasis. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-1259-8.

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7

Motomichi, Torisu, Yoshida Takeshi y International Symposium on Basic Mechanisms and Clinical Treatments of Tumor Metastasis (1982 : Fukuoka-shi, Japan), eds. Basic mechanisms and clinical treatment of tumor metastasis. New York: Academic Press, 1985.

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8

Akira, Watanabe, ed. Cancer metastases research. New York: Nova Science Publishers, 2008.

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9

Cardona, Kenneth y Shishir K. Maithel, eds. Primary and Metastatic Liver Tumors. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91977-5.

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10

E, Abrey Lauren, Chamberlain Marc C y Engelhard Herbert H, eds. Leptomeningeal metastases. New York: Springer, 2005.

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11

Oral cancer metastasis. New York: Springer, 2010.

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12

Stacker, Steven A. y Marc G. Achen. Lymphangiogenesis in cancer metastasis. [Dordrecht?]: Springer, 2009.

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13

Sawaya, Raymond. Intracranial metastases: Current management strategies. Malden, Mass., USA: Blackwell Futura, 2004.

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14

1921-, Franks L. M. y Hart I, eds. Tumour progression and metastasis. Oxford: Oxford University Press for the Imperial CancerResearch Fund, 1988.

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15

Hoskin, Peter. Radiotherapy planning for metastatic disease. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199696567.003.0021.

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Chapter 16 discusses radiotherapy planning for metastatic disease, predominantly for patients with bone metastasis, spinal cord compression, and brain metastasis. The techniques for such treatments are specific to this indication rather than the primary tumour site.
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16

Goepel, John. Pathology of testicular tumours. Editado por James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0091.

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Tumours of the testis are uncommon and are usually germ cell tumours. They present most often as a scrotal mass in a young man, and are the most frequent malignant tumour in this age group. The incidence has risen over recent decades and is higher in Western Europe. A history of testicular maldescent is a significant risk factor. About 50% are pure seminoma; the remainder non-seminomas may have a single but more usually a mixed histology. Non-seminomas are all called teratoma in the British system. Metastasis readily occurs to paraaortic lymph nodes or the lungs, and some patients present with advanced metastatic disease. Radical orchidectomy is the usual treatment of the primary tumour, with chemotherapy for metastatic disease. There are other tumours that arise in the testis, which will require a different management strategy. Finally, there are tumours that arise in paratesticular tissue or the spermatic cord.
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17

Joniau, Steve, S. Van Bruwaene, J. Karnes, G. De Meerleer, P. Gontero, M. Spahn y A. Briganti. High-risk prostate cancer. Editado por James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0066.

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In this chapter, patients with adverse tumour characteristics and a high risk of tumour progression are discussed. In the current era of PSA testing, the proportion of patients presenting with high-risk prostate cancer (PCa) is estimated between 10% and 20% with a 10-year cancer specific survival approaching 40% for those not receiving active local treatment. The prevalence of high-risk disease varies with community PSA use, and is higher in countries (e.g. 30% in the United Kingdom) with little PSA testing. Adequate staging with magnetic resonance imaging for tumour extension, computer tomography for visceral metastases, and bone scan for skeletal metastasis is advocated in this group. The treatment of locally advanced or high-risk prostate cancer is a contemporary challenge.
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18

Jamie, Goode, Chadwick Derek, Novartis Foundation y Symposium on the Tumour Microenvironment: Causes and Consequences of Hypoxia and Acidity (2000 : London, England), eds. The tumour microenvironment: Causes and consequences of hypoxia and acidity. Chichester: Wiley, 2001.

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19

Intermolecular cross-talk in tumor metastasis. Amsterdam: IOS Press, 1999.

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20

The Tumour Microenvironment - No. 240: Causes and Consequences of Hypoxia and Acidity (Novartis Foundation Symposia). Wiley, 2001.

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21

Rawat, S., L. Horgan y C. M. S. Royston. Laparoscopic surgery. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198510567.003.0009.

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Laparoscopic staging for abdominal malignancies 324Laparoscopic splenectomy 326Laparoscopic inguinal hernia repair 328Laparoscopic Nissen fundoplication 332Laparoscopic cholecystectomy 336Laparoscopic appendicectomy 342Obesity surgery 346Laparoscopy is an effective and useful tool for the diagnosis and staging of abdominal malignancies. Staging is of paramount importance in planning treatment for localized and advanced disease. It is imperative to accurately identify those patients with a potentially resectable, localized tumour and those patients with advanced disease or distant metastasis. Despite improvements in preoperative staging with dynamic computed tomography (CT) and endoscopic ultrasonography, unexpected liver or peritoneal metastases are found in 10–20% of patients with oesophageal, gastric and pancreatic cancer. The need for laparotomy can therefore be obviated in these patients....
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22

Xu, Ke, ed. Tumor Metastasis. InTech, 2016. http://dx.doi.org/10.5772/61798.

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23

Hodgkiss, Andrew. Psychiatric consequences of particular cancers. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0004.

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Certain tumour types can cause psychopathology through direct biological mechanisms such as metastatic spread to the brain, release of onconeuronal antibodies, ectopic hormone secretion, or release of pro-inflammatory cytokines. Lung cancers, adenocarcinoma of the pancreas, brain tumours, and ovarian tumours are considered in detail. Confusional states due to brain metastases, syndrome of inappropriate ADH secretion, hypercalcaemia of malignancy, and anti-Hu encephalitis are found in lung cancers. Severe depression, due to interleukin-6 release and its actions on the HPA axis and tryptophan metabolism, is common in adenocarcinoma of the pancreas. Anti-NMDA-receptor limbic encephalitis, clinically indistinguishable from acute schizophrenia, can complicate teratomas. Gliomas, pituitary tumours, and thyroid, adrenal, and testicular tumours can also disrupt mental health through various biological mechanisms described here.
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24

Barzilai, Ori, Mark H. Bilsky y Ilya Laufer. Spine Metastases. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0028.

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A decision-making framework called NOMS (neurologic, oncologic, mechanical, and systemic) facilitates and guides therapeutic decisions for patients with spinal metastases. Patients should be evaluated for signs of myelopathy or cauda equina. The Epidural Spinal Cord Compression scale facilitates reporting of the degree of radiographic spinal cord compression. A determination of the expected histology-specific tumor response to conventionally fractionated external beam radiation and systemic therapy should be made. Radiation therapy effectively treats biologic pain and radiosensitive tumors such as multiple myeloma. Patients should undergo a careful evaluation of movement-associated pain as tumor-induced spinal instability is an independent indication for surgery. Determination of tumor-associated mechanical instability can be facilitated by the Spinal Instability Neoplastic Score. Herein, the authors present a case of spinal multiple myeloma managed using the NOMS framework and in consideration of current evidence and treatment paradigms.
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25

Banfalvi, Gaspar. Homeostasis - Tumor - Metastasis. Springer, 2016.

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26

Banfalvi, Gaspar. Homeostasis - Tumor - Metastasis. Springer, 2013.

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27

Banfalvi, Gaspar. Homeostasis - Tumor - Metastasis. Springer London, Limited, 2013.

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28

(Editor), Curtis C. Harris y Lance A. Liotta (Editor), eds. Genetic Mechanisms in Carcinogenesis and Tumour Progression (UCLA Symposia on Molecular and Cellular Biology, New Series). John Wiley & Sons Inc, 1990.

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29

Hodgkiss, Andrew. Introduction to cancer biology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0001.

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A brief introduction to cancer biology, aimed at psychiatrists, is offered. Selective DNA transcription, the cell cycle, receptor tyrosine kinases, and cell signalling pathways are introduced, using the EGFR/RAS/MAPK pathway as an exemplar. The molecular pathology of oncogenesis is summarized, including discussion of oncogenes, tumour suppressor genes, and examples of driver mutations. The exploitation of such mutations in stratified medicine, using molecularly targeted agents, is mentioned. Finally, Hanahan and Weinberg’s six hallmarks of cancer are listed, adding angiogenesis and metastasis to the picture of oncogenesis.
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30

Scott-Brown, Martin. Symptom control in cancer. Editado por Patrick Davey y David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0329.

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Treatment in cancer is aimed at improving survival (curing where possible) and/or improving symptoms. Symptoms may be caused by the cancer itself (primary tumour, metastases, or paraneoplastic phenomenon) or by the treatments patients undergo to treat the cancer (surgery, radiotherapy, chemotherapy, hormone therapy, and biological therapy). Therefore, symptom control is one of the key roles of oncologists as they treat cancer patients. The most important part of symptom control in cancer patients is to elucidate the underlying cause of the symptom. Symptom control is most effective when the underlying cause is targeted; for example, shoulder pain may be treated most effectively by local radiotherapy if it is due to a bone metastasis in the humeral head, by dexamethasone if it is referred pain due to diaphragmatic irritation from hepatomegaly, and by amitriptyline or gabapentin if it is neuropathic pain due to cervical nerve root irritation. Covering all symptom control in cancer patients is beyond the remit of this chapter; however, it will cover the control of pain and nausea and vomiting, as these are very common symptoms in cancer patients.
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31

Zehnder, Pascal y George N. Thalmann. Muscle-invasive bladder cancer. Editado por James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0078.

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In the United Kingdom, >4,000 people die of bladder cancer every year. This reflects around one-third of affected patients and occurs in those with primary metastatic disease, with invasion at presentation, and in persons whose tumour progresses to invasion from non-invasive disease. The outcome from invasive cancers has not dramatically altered over the last 30 years, due to a lack of screening programmes, a lack of advances in treatment, and the fact that many patients present with tumours at an advanced stage. Around 50% of patients with invasive disease die from bladder cancer despite radical treatment, suggesting the disease is metastatic at presentation. Cure is rarely possible in patients with locally advanced tumours and lymph node metastases. Therapeutic options include systemic chemotherapy and salvage radical treatment for responders or palliation. Following radical cystectomy for cancer, patients require lifelong follow-up for both oncologic and functional reasons.
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32

Liotta, L. A. y I. R. Hart. Tumor Invasion and Metastasis. Springer Netherlands, 2012.

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33

Lasfar, Ahmed y Karine Cohen-Solal, eds. Tumor Progression and Metastasis. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.77832.

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34

Liotta, L. A. y I. R. Hart. Tumor Invasion and Metastasis. Springer, 2012.

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35

Preusser, Matthias, Gabriele Schackert y Brigitta G. Baumert. Metastatic brain tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0019.

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Brain metastasis is a common clinical challenge in cancer patients, particularly those with lung cancer, breast cancer, and melanoma. The prognosis is poor, with median overall survival times measured in months for most patient populations. Established treatments include neurosurgical resection, radiotherapy (including stereotactic radiosurgery and stereotactic radiotherapy, whole-brain radiotherapy, and new radiation techniques), and supportive care measures. Recently, more and more targeted therapies such as EGFR inhibitors, HER2 antagonists, BRAF inhibitors, ALK inhibitors, and immune checkpoint inhibitors are demonstrating some efficacy in brain metastasis patients and should be considered in the clinical setting.
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36

Schiff, David, Jonathan Sherman y Paul D. Brown. Metastatic tumours: spinal cord, plexus, and peripheral nerve. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0020.

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Systemic cancers produce substantial neurological morbidity when they spread to the spinal epidural space, producing epidural spinal cord compression—a neurological emergency. Less often, metastases spread directly to spinal cord parenchyma to manifest as intramedullary spinal cord metastasis or result in peripheral nerve dysfunction via compression of the brachial, lumbosacral, or, rarely, the cervical plexus. This chapter reviews the clinical manifestations and risk factors for development of these entities, the diagnostic approach, management options including the role of surgery, radiation (including stereotactic body radiation therapy), and chemotherapy, as well as the neurological prognosis.
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37

Metastatic Progression and Tumour Heterogeneity. MDPI, 2020. http://dx.doi.org/10.3390/books978-3-03928-854-0.

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38

Jordan, Nerissa. Non-metastatic neurological manifestations of malignancy. Editado por Patrick Davey y David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0238.

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Neurological complications of systemic malignancy are frequent. They may reflect direct local effects of the tumour; CNS infection; side effects of chemotherapy or radiotherapy; nutritional or metabolic derangements; or a paraneoplastic syndrome. The paraneoplastic neurological syndromes are a group of disorders associated with a malignancy outside the nervous system. The pathophysiology is immune-mediated, with the tumour’s expression of neuronal proteins invoking antibody formation, which in turn results in neurological symptoms. This chapter will mainly focus on these syndromes.
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39

(Editor), U. Reinhold y W. Tilgen (Editor), eds. Minimal Residual Disease in Melanoma: Biology, Detection and Clinical Relevance (Recent Results in Cancer Research). Springer, 2000.

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40

Metastatic Spinal Tumors. Thieme Medical Publishers, Incorporated, 2014.

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41

Felbaum, Daniel R., Jonathan H. Sherman y Walter C. Jean. Pineal Tumors. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0003.

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Pineal region tumors can include a variety of histologies including pineal parenchymal tumor, germ cell tumor, glial tumor, metastasis and meningioma. The workup for pineal region tumors includes standard magnetic resonance imaging for anatomic imaging, as well as cerebrospinal fluid markers to assess for certain germ cell tumors. Cerebrospinal fluid diversion may be necessary if patients present with hydrocephalus. If surgical resection is indicated based on the suspected diagnosis, magnetic resonance venogram is an important study that influences the surgical trajectory. This chapter reviews common pineal region tumors in the setting of a case presentation. Management strategies and surgical approaches are also discussed in this chapter. Pearls for how to select the surgical approach and complication avoidance are also presented.
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42

Bower, Mark, Louise Robinson y Sarah Cox. Endocrine and metabolic complications of advanced cancer. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0142.

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Cancer produces endocrine and metabolic complications in two ways. Firstly, the primary tumour or its metastases may interfere with the function of endocrine glands, kidneys, or liver by invasion or obstruction. Secondly, tumours may give rise to remote effects without local spread and these actions are termed paraneoplastic manifestations of malignancy. Generally, these paraneoplastic syndromes arise from secretion by tumours of hormones, cytokines, and growth factors, but also occur when normal cells secrete products in response to the presence of tumour. This chapter reviews the pathogenesis, epidemiology, and management of the commonest paraneoplastic endocrinopathies including hypercalcaemia, Cushing’s syndrome, the syndrome of inappropriate antidiuresis, non-islet cell tumour hypoglycaemia, enteropancreatic hormone syndromes, Carcinoid syndrome, phaeochromocytoma, gonadotrophin secretion syndromes, prolactin and oxytocin secretion, and paraneoplastic pyrexia. The chapter concludes with a brief discussion of the management of metabolic disease in the context of advanced malignancy including hyperglycaemia, thyroid dysfunction, metabolic bone disease, renal failure, liver failure, and lactic acidosis.
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43

Abou-Al-Shaar, Hussam y Mark A. Mahan. Dumbbell Nerve Sheath Tumors. Editado por Meghan E. Lark, Nasa Fujihara y Kevin C. Chung. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190617127.003.0019.

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A dumbbell tumor is a nerve sheath tumor that arises from a spinal nerve in the neural foramen and grows as a dumbbell-shaped mass. The differential diagnosis for a dumbbell tumor includes schwannoma, neurofibroma, malignant peripheral nerve sheath tumor, and metastases, among others. MR imaging is considered the gold-standard imaging modality for diagnosis of dumbbell tumors. Surgical approaches that are tailored to the individual patient’s case can be utilized. The chapter reviews dumbbell tumors, including a case example and covers the incidence, clinical presentation, imaging features, decision-making strategy, surgical approaches, outcomes, and potential complications associated with their management.
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44

Torisu, Motomichi y Takeshi Yoshida. Basic Mechanisms and Clinical Treatment of Tumor Metastasis. Elsevier Science & Technology Books, 2013.

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45

Ben-Baruch, Adit, ed. The Inflammatory Milieu of Tumors: Cytokines and Chemokines that Affect Tumor Growth and Metastasis. BENTHAM SCIENCE PUBLISHERS, 2012. http://dx.doi.org/10.2174/97816080525611120101.

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46

Stanford, David Richard. Biochemical characterization of metastatic tumor cells. 1985.

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47

Abrey, Lauren E. y Jeffrey J. Raizer. Brain Metastases. Springer, 2014.

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48

Brain Metastases from Primary Tumors. Elsevier, 2014. http://dx.doi.org/10.1016/c2013-0-18945-3.

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49

Brain Metastases from Primary Tumors. Elsevier, 2015. http://dx.doi.org/10.1016/c2013-0-23234-7.

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50

New Developments in Metastasis Suppressor Research. Nova Science Pub Inc, 2007.

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