Bibliografías temáticas / Tumor-inflammation / Libros

Libros sobre el tema "Tumor-inflammation"

Siga este enlace para ver otros tipos de publicaciones sobre el tema: Tumor-inflammation.
Autor: Grafiati
Publicado: 1 de abril de 2023

Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros

Elija tipo de fuente:

Consulte los 16 mejores mejores libros para su investigación sobre el tema "Tumor-inflammation".

Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.

También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.

Explore libros sobre una amplia variedad de disciplinas y organice su bibliografía correctamente.

1

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
2

S, Grewal Iqbal, ed. Therapeutic targets of the TNF superfamily. New York: Springer Science+Business Media, 2009.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
3

Marialuisa, Melli y Parente Luca, eds. Cytokines and lipocortins in inflammation and differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of IL-1, TNF, and Lipocortins in Inflammation and Differentiation, held in Siena, Italy, October 22-25, 1989. New York, NY: Wiley-Liss, 1990.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
4

Harald, Tschesche, ed. Proteinases in inflammation and tumor invasion: Review articles including those from an international conference, Bielefeld, Federal Republic of Germany, March 14-16, 1985. Berlin: De Gruyter, 1986.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
5

E, Sim, ed. Humoral factors. Oxford: IRL Press at Oxford University Press, 1993.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
6

Dougan, Michael Lawrence. Balancing oncogenic inflammation and anti-tumor immunity in the development of novel immune therapies for cancer. 2009.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
7

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily. Springer London, Limited, 2009.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
8

Grewal, Iqbal S. Therapeutic Targets of the TNF Superfamily: Volume 647. Springer, 2010.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
9

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Incl. Those from an Internat. Conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 1986.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
10

Tschesche, Harald. Proteinases in inflammation and tumor Invasion: Review articles incl. those from an internat. conference, Bielefeld, March 14 - 16 1985. De Gruyter, Inc., 2020.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
11

Tschesche, Harald. Proteinases in Inflammation and Tumor Invasion: Review Articles Including Those from an International Conference Bielefeld, Federal Republic of Germ. Walter De Gruyter Inc, 1986.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
12

(Editor), Marialuisa Melli y Luca Parente (Editor), eds. Cytokines and Lipocortins in Inflammation and Differentiation: Proceedings of the International Conference on Molecular and Cellular Biology of Il-1, (Progress in Clinical & Biological Research). Wiley-Liss, 1990.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
13

Woolf, Eric C. y Adrienne C. Scheck. Ketogenic Diet as Adjunctive Therapy for Malignant Brain Cancer. Editado por Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0013.

Texto completo
Resumen
Malignant brain tumors are devastating, and increased survival requires new therapeutic modalities. Metabolic dysregulation results in an increased need for glucose in tumor cells, suggesting that reduced tumor growth could be achieved with decreased glucose availability either through pharmacological means or use of a high-fat, low-carbohydrate ketogenic diet (KD). KD provides increased blood ketones to support energy needs of normal tissues and has been shown to reduce tumor growth, angiogenesis, inflammation, peritumoral edema, migration, and invasion. Furthermore, this diet can enhance the activity of radiation and chemotherapy in a mouse model of glioma, thus increasing survival. In vitro studies indicate that increasing ketones in the absence of glucose reduction can also inhibit cell growth and potentiate the effects of radiation. Thus, emerging data provide strong support for the use of KD in the treatment of malignant gliomas and thus far has led to a limited number of clinical trials.
Los estilos APA, Harvard, Vancouver, ISO, etc.
14

Epenetos, Agamemnon A. y Massimo Pignatelii. Cell Adhesion Molecules in Cancer and Inflammation (Cell Adhesion and Communication, Vol 1). CRC, 1995.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
15

Proteinases in Inflamation and Tumour Invasion. Walter de Gruyter & Co, 1986.

Buscar texto completo
Los estilos APA, Harvard, Vancouver, ISO, etc.
16

Keum, NaNa, Mingyang Song, Edward L. Giovannucci y A. Heather Eliassen. Obesity and Body Composition. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0020.

Texto completo
Resumen
In 2014, an estimated 1.9 billion adults worldwide were either overweight (BMI 25–29.9) or obese (BMI ≥30). The so-called obesity epidemic began in high-income, English-speaking countries in the early 1970s, but soon spread globally; more than one-third (38%) of all adults and 600,000 children under age five are overweight or obese, as are two-thirds (69%) of adults in the United States. Excessive body fat is a major cause of type 2 diabetes, hypertension, cardiovascular and liver disease, among other disorders, and has been designated a definite cause of at least fourteen cancer sites: breast (postmenopausal), colorectum, endometrium, esophagus (adenocarcinoma), gallbladder, kidney (renal cell), pancreas, gastric cardia, liver, ovary, prostate (advanced tumors), multiple myeloma, thyroid, and meningioma. Mechanisms by which adipose tissue are thought to promote tumor growth include the endocrine and metabolic effects of fat on sex hormones, growth factors, and inflammation, as well as local chemical or mechanical injury of gastrointestinal organs.
Los estilos APA, Harvard, Vancouver, ISO, etc.
También puede estar interesado en las bibliografías sobre el tema "Tumor-inflammation" para otros tipos de fuentes:
Artículos de revistas Tesis
Ofrecemos descuentos en todos los planes premium para autores cuyas obras están incluidas en selecciones literarias temáticas. ¡Contáctenos para obtener un código promocional único!

Pasar a la bibliografía