Literatura académica sobre el tema "Trigeminal complex"
Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros
Consulte las listas temáticas de artículos, libros, tesis, actas de conferencias y otras fuentes académicas sobre el tema "Trigeminal complex".
Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.
También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.
Artículos de revistas sobre el tema "Trigeminal complex"
Brown, Jeffrey A. "The Trigeminal Complex". Neurosurgery Clinics of North America 8, n.º 1 (enero de 1997): 1–10. http://dx.doi.org/10.1016/s1042-3680(18)30333-4.
Texto completoMehnert, Jan, Hauke Basedau, Lisa-Marie Sturm, Trine Nielsen, Rigmor Højland Jensen y Arne May. "Functional brainstem representations of the human trigeminal cervical complex". Cephalalgia 43, n.º 5 (mayo de 2023): 033310242311748. http://dx.doi.org/10.1177/03331024231174862.
Texto completoBALYAZINA, E. V., T. A. ISAKHANOVA y N. A. ALEKSEEVA. "CLASSICAL TRIGEMINAL NEURALGIA COMPLEX THERAPY". Kubanskij nauchnyj medicinskij vestnik 1, n.º 2 (1 de enero de 2017): 21–24. http://dx.doi.org/10.25207/1608-6228-2017-2-21-24.
Texto completoClement, M. E. y R. B. McCall. "Characterization of midline medulla role in the trigeminal depressor response". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 256, n.º 5 (1 de mayo de 1989): R1111—R1120. http://dx.doi.org/10.1152/ajpregu.1989.256.5.r1111.
Texto completoChávez, Gabriela-del-Rocío Chávez, Antonio A. F. De Salles, Timothy D. Solberg, Alessandra Pedroso, Dulce Espinoza y Pablo Villablanca. "Three-dimensional Fast Imaging Employing Steady-state Acquisition Magnetic Resonance Imaging for Stereotactic Radiosurgery of Trigeminal Neuralgia". Neurosurgery 56, n.º 3 (1 de marzo de 2005): E628. http://dx.doi.org/10.1227/01.neu.0000154709.44776.50.
Texto completoVisconti, Ciro, Leone Leone, Michele Mario Zarrelli y Alfredo Del Gaudio. "Cervical Spinal Dorsal Root Stimulation in Trigeminal Neuralgia". Pain Medicine Case Reports 5, n.º 8 (30 de noviembre de 2021): 379–83. http://dx.doi.org/10.36076/pmcr.2021.5.8.
Texto completoGaydhanker, Anuradha Prasanna y Prasanna Shravan Gaydhanker. "A study on trigeminal nerve: Does superior cerebellar artery causes trigeminal neuralgia". Indian Journal of Clinical Anatomy and Physiology 9, n.º 3 (15 de octubre de 2022): 174–78. http://dx.doi.org/10.18231/j.ijcap.2022.037.
Texto completoIgawa, Kaori, Hideki Funahashi, Yu Miyahara, Rumi Naono-Nakayama, Hisae Matsuo, Yoshihiro Yamashita, Sumio Sakoda, Toshikazu Nishimori y Yasushi Ishida. "Distribution of hemokinin-1 in the rat trigeminal ganglion and trigeminal sensory nuclear complex". Archives of Oral Biology 79 (julio de 2017): 62–69. http://dx.doi.org/10.1016/j.archoralbio.2017.03.004.
Texto completoJones, Lauren M., SooHyun Lee, Jason C. Trageser, Daniel J. Simons y Asaf Keller. "Precise Temporal Responses in Whisker Trigeminal Neurons". Journal of Neurophysiology 92, n.º 1 (julio de 2004): 665–68. http://dx.doi.org/10.1152/jn.00031.2004.
Texto completoRigoard, Philippe, Maxime Billot, Maarten Moens, Lisa Goudman, Hassan El-Hajj, Pierre Ingrand, Amine Ounajim et al. "Evaluation of External Trigeminal Nerve Stimulation to Prevent Cerebral Vasospasm after Subarachnoid Hemorrhage Due to Aneurysmal Rupture: A Randomized, Double-Blind Proof-of-Concept Pilot Trial (TRIVASOSTIM Study)". International Journal of Environmental Research and Public Health 20, n.º 10 (16 de mayo de 2023): 5836. http://dx.doi.org/10.3390/ijerph20105836.
Texto completoTesis sobre el tema "Trigeminal complex"
Barnet, Maxime. "Etude comparative chez les rats mâles et femelles de l'implication du complexe trigémino-cervical et du locus coeruleus dans l'intégration d'une douleur méningée". Electronic Thesis or Diss., Université Clermont Auvergne (2021-...), 2021. http://www.theses.fr/2021UCFAC120.
Texto completoMigraine is a chronic condition characterised by the occurrence of intense headaches associated with other symptoms such as photo/phonophobia, nausea/vomiting or allodynia: pain triggered by non-painful stimulation. These symptoms appear in attacks interspersed with intervals of calm. Migraines affect 15 to 18% of the population worldwide, and are found preferentially in women, sometimes more intensely during menstruation. Although some of the mechanisms of migraine are beginning to be understood, there are still many questions to be answered, in particular how migraine pain is integrated and whether this integration differs according to the sex of the individual.In our project, we explored in male and female rats the involvement of the trigemino-cervical complex (TCC) and the Locus Coeruleus (LC) in the development of skin allodynia in a model of meningeal pain induced by repeated injection of inflammatory soup (IS). The first brain region, the TCC, is the first central relay from the orofacial region including the meninges. The LC is a noradrenergic nucleus whose descending pathways will, among other things, modulate pain and project to the TCC. By combining behavioural analyses and immunohistological techniques, we have shown in both males and females that a dural injection of IS induces allodynia, which becomes persistent with repeated IS injections. However, the evolution of allodynia is more rapid in females. By dissociating females according to their hormonal cycle: proestrus/estrus (P/E) and Metestrus/Diestrus (M/D), we showed that P/E females had a greater cephalic allodynia than M/D females after 2 injections of IS. Furthermore, using the neuronal activation marker c-Fos, P/E females showed greater activation in the TCC following meningeal inflammation. In contrast to TCC neurons, LC neurons were not activated by IS, however, we could observe, under physiological conditions, a significant sexual dimorphism of the LC: C-Fos neurons in the LC are in greater quantity in females than in males. Finally, we examined the projections of the LC using tracing techniques and confirmed the existence of direct projections of the LC to the TCC and indirect projections using the rostro-ventral medulla as an intermediate. We also observed the existence of superficial cortical projections, which could have an influence on the meningeal vasculature, and indirectly on the TCC. However, there are no anatomical differences in these projections between males and females.Taken together, these results demonstrate that the integration of meningeal pain is dependent on the hormonal cycle, and that TCC activity reflects this integration after two injections of SI. We have also shown the existence of direct and indirect pathways that may allow the LC to modulate the TCC. Finally, our project demonstrates the need to include both sexes in scientific studies and to take into account the hormonal cycle
Dauvergne, Céline. "Etude anatomique des circuits neuronaux intervenant dans le réflexe de clignement chez le rat". Paris 6, 2004. http://www.theses.fr/2004PA066076.
Texto completoNdiaye, Awa. "Colliculus superieur et mouvement des paupières : mise en evidence de voies colliculo-faciale et colliculo-trigemino-faciale intervenant dans le réflexe de clignement chez le rat". Paris 6, 2004. http://www.theses.fr/2004PA066245.
Texto completoErvolino, Edilson. "Inervação autonômica da articulação temporomandibular em condições de normalidade e, padrão de ativação neuronal no tronco encefálico durante a vigência de artrite no complexo articular temporomandibular". Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-01102009-162540/.
Texto completoThe goals of the present study were: 1) to analyse the distribution of autonomic nerve fibers in the rat temporomandibular joint (TMJ) under normal conditions using immunofluorescence method to detect tirosyne hydroxylase (TH), neuropetide Y (NPY) and vasoactive intestinal polypeptide (VIP); 2) to verify the detailed distribution of autonomic nerve fibers in the rat temporomandibular joint by transmission electron microscopy; 3) to determine the neuronal activation pattern in the trigeminal system and in the pain modulation centers during monoarthritis induced in the rat temporomandibular joint complex (TMJC). For the first purpose, histologic sections from TMJs with intact innervation or with surgical sympatectomy and/or parasympathectomy were submitted to simultaneous detection of TH, NPY and VIP. For the second purpose, 5-hydroxidopamine treatment to detect sympathetic nerve endings was combined with surgical parasympatectomy of the otic ganglion to detect degenerated parasympathetic nerve endings in the rat TMJC, by transmission electron microscopy. For the last purpose, monoarthritis (acute, chronic and chronic-active phases) was induced in the TMJC and histologic sections from the brain stem were submitted to immunodetection of Fos protein in the trigeminal system and in the pain modulation centers (periqueductal gray matter - PAG; rostroventromedial medulla - RVM; locus coeruleus- LC; caudal ventrolateral medulla- CVLM; nucleus of the solitary tract - NTS; ventral reticular nucleus - VRN). The most important results demonstrated that the TMJC showed a discrete parasympathetic innervation (VIP-IR), while the sympathetic innervation was dense and characterized by TH-/NPY-/VIP-IR or TH-/NPY-IR nerve fibers. Autonomic nerve fibers were mainly noted associated to blood vessels and occasionally disperse in the synovial membrane. Fos-IR neurons showed significant bilateral increase in the spinal trigeminal caudal subnucleus and PAG during arthritis evolution. On the other hand, RVM, LC, CVLM and NTS only showed significant increase of Fos-IR neurons during the acute and chronic-active phases of monoarthritis. The main conclusions were: 1) the TMJC shows a dense sympathetic innervation (TH/NPY-IR or TH-/NPY-/VIP-IR) and discrete parasympathetic innervation (VIP-IR), both associated mainly to blood vessels; 2) most modulation pain centers are activated principally during acute and chronic-active arthritis, while the spinal trigeminal caudal subnucleus and PAG showed continuous activation during all phases of arthritis.
Pinto, Magali Luci [UNIFESP]. "Organização Topográfica e Quantificação das Vias Trigêmino-Rubrais em Camundongos Distróficos e Normais". Universidade Federal de São Paulo (UNIFESP), 2008. http://repositorio.unifesp.br/handle/11600/9886.
Texto completoPacientes com distrofia muscular de Duchenne apresentam alteracoes no sistema nervoso central (SNC). Mudancas no SNC tambem ocorrem nos camundongos distroficos (mdx), incluindo perda de fibras rubro-espinais. Para examinar se outras vias tambem sao reduzidas no mdx, propusemo-nos a estudar a organizacao topografica das vias trigemino-rubrais e quantificar os neuronios do Complexo Trigeminal que se projetam para o nucleo Rubro em camundongos C57BL10 (normais) e distroficos (mdx) de diferentes idades. Para tanto, os animais foram submetidos a cirurgia estereotaxica para as injecoes dos tracadores de transporte retrogrado Fluorogold, Rodamina e Fluoresceina, bilateralmente, no nucleo Rubro. Sete dias depois, os animais foram sacrificados sob anestesia atraves de perfusao transcardiaca e os encefalos foram congelados em meio de embebicao proprio no uso do aparelho criostato e, destes encefalos foram realizados cortes seriados na espessura de 35 ƒÊm. A analise foi realizada em microscopico de epifluorescencia. Foram contados os neuronios do subnucleo oral do nucleo espinal do nervo trigemeo em camundongos normais e distroficos de 3, 6 e 12 meses de idade. No Sistema Intersticial, foram contados todos os neuronios marcados ao longo de sua extensao. Nossos resultados mostraram que existe uma organizacao topografica na projecao dos neuronios do Complexo Trigeminal e do Sistema Intersticial para o nucleo Rubro, em camundongos. Todos os nucleos sensoriais do Complexo Trigeminal apresentaram intensa marcacao bilateral, com predominio contralateral quando o sitio de injecao foi na regiao magnocelular do nucleo Rubro; os neuronios apresentaram pouca ou nenhuma marcacao quando o sitio atingiu a região parvocelular e, quando o sítio de injeção atingiu a região intermediária do núcleo a qual abrange suas partes magnocelular e parvocelular, a marcação retrógrada foi mais intensa só quando o foco do sítio atingiu mais a região magnocelular. O núcleo motor do Complexo Trigeminal não foi marcado em nenhuma das situações. No Sistema Intersticial, foram marcados os neurônios apenas quando o sítio de injeção abrangeu a região magnocelular do núcleo Rubro. Também foi verificado que no Complexo Trigeminal essa organização é semelhante em ambos os grupos normais e distróficos. Os resultados mostraram uma redução de 50% no número de neurônios do Complexo Trigeminal no mdx com a idade de 3 meses. Essa redução neuronal se manteve nos camundongos mdx nos grupos de 6 e 12 meses, não ocorrendo progressão desta perda com a idade. Além disso, o grupo de camundongos C57BL10 (normais) também não apresentou perda neuronal com a idade. Concluímos que a diferença observada no complexo trigeminal no grupo de 3 meses já está estabelecida e que não é progressiva com o avanço da idade; portanto, é bem provável que os camundongos mdx já nasçam com essa redução ou que a mesma ocorra logo nas primeiras semanas após o nascimento.
TEDE
BV UNIFESP: Teses e dissertações
Melin, Céline. "Rôle de l'inhibition segmentaire dans le traitement de l'information nociceptive cutanée et méningée dans le complexe trigéminal". Thesis, Clermont-Ferrand 1, 2011. http://www.theses.fr/2011CLF1DD01/document.
Texto completoPathological disruption of segmental inhibition is thought to contribute to persistent pain hypersensitivity – including hyperalgesia, allodynia and spontaneous pain – that occurs during chronic pain states. That allodynia is also often associated with migraine – an episodic headache – suggests that a loss of synaptic inhibition is also involved in the manifestation of headache pain. However, the very high prevalence of migraine – more than 10% of the general population – raises the question as to whether processing of meningeous inputs by local neuronal network – consisting of excitatory and inhibitory interneurons – within the trigeminal nucleus, the first relay station for incoming nociceptive signals of the face and meninges, is the same as that of others, for instance cutaneous. We sought to characterize how pharmacological blockade of glycine and GABAA receptors modifies synaptic transmission between either cutaneous or meningeous primary afferent fibers and second order neurons by recording field potentials in the rat superficial medullary dorsal horn (MDH). Transcutaneous electrical stimulation evokes three negative field potentials elicited by, from the earliest to the latest, Aβ-, Aδ- and C-fiber primary afferents. Blocking segmental glycine and/or GABAA receptors strongly facilitates A-fiber-activated polysynaptic excitatory field potentials but, conversely, inhibits, or even abolishes, C-fiber field potentials. Blocking segmental GABAB receptors reverses such suppression. Interestingly, it also potentiates C-fiber field potentials under control conditions. Meningeous electrical stimulation evokes two negative field potentials elicited by, from the earliest to the latest, Aδ- and C-fiber primary afferents. Unlike cutaneous C-fiber field potentials, meningeous ones are facilitated by blocking segmental glycine and/or GABAA receptors. These results demonstrate that MDH processing of cutaneous and meningeous inputs are different. Only activation of cutaneous A-fiber primary afferents inhibits cutaneous C-fiber inputs to the MDH by the way of polysynaptic excitatory pathways, last-order GABAergic interneurons and presynaptic GABAB receptors. In view of the gate control theory postulating that afferent volleys in non-nociceptive afferents close the gate to central transmission of nociceptive inputs, our results suggest that the state of the gate depends on firing activities of both A-fiber primary afferents and polysynaptic excitatory circuits, i.e. the inhibitory tone, within the dorsal horn
Libros sobre el tema "Trigeminal complex"
Shaw, Pamela y David Hilton-Jones. The lower cranial nerves and dysphagia. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0429.
Texto completoEl-Kadri, Moutaz y George Hart. Extrasystoles. Editado por Patrick Davey y David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0110.
Texto completoCapítulos de libros sobre el tema "Trigeminal complex"
Ehland, Elise L., Roger A. Meyer y Shahrokh C. Bagheri. "Trigeminal Nerve Injuries". En Complex Dental Implant Complications, 217–37. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-47012-8_9.
Texto completoShigenaga, Yoshio y Atsushi Yoshida. "Trigeminal Brainstem Nuclear Complex, Anatomy". En Encyclopedia of Pain, 4054–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4602.
Texto completoHu, James W. y Alain Woda. "Trigeminal Brainstem Nuclear Complex, Physiology". En Encyclopedia of Pain, 4060–65. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4604.
Texto completoBereiter, David A. "Trigeminal Brain Stem Nuclear Complex, Immunohistochemistry, and Neurochemistry". En Encyclopedia of Pain, 4050–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4603.
Texto completo"34 Large Trigeminal Neurinoma". En Neurosurgery of Complex Vascular Lesions and Tumors, editado por Shigeaki Kobayashi. Stuttgart: Georg Thieme Verlag, 2005. http://dx.doi.org/10.1055/b-0034-55524.
Texto completoSrivastava, Devjit y Manohar Sharma. "Trigeminal interventions for head and neck cancer pain". En Practical Management of Complex Cancer Pain, 239–50. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199661626.003.0019.
Texto completoGozalov, Aydin, Messoud Ashina y Joanna M. Zakrzewska. "Cranial neuralgias and persistent idiopathic facial pain". En Oxford Textbook of Headache Syndromes, editado por Michel Ferrari, Joost Haan, Andrew Charles, David W. Dodick, Fumihiko Sakai y Christopher Kennard, 237–47. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198724322.003.0027.
Texto completoTheofanis, Thana N. y Patrick Greaney. "Management of Complex Scalp Injuries". En Neurotrauma, 177–86. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190936259.003.0020.
Texto completoWu, Jiang y Jianguo Cheng. "Interventional Treatment of Neuropathic Pain". En Neuropathic Pain, editado por Jianguo Cheng, 53–62. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190298357.003.0007.
Texto completoAbbott, Jillie, Claire Patterson y Elena Semino. "Patient support groups". En Trigeminal Neuralgia and Other Cranial Neuralgias, 207–16. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198871606.003.0017.
Texto completoActas de conferencias sobre el tema "Trigeminal complex"
Pavlov, Alexey N., Anatoly N. Tupitsyn, Valery A. Makarov, Fivos Panetsos, Angel Moreno, Victor Garcia-Gonzalez y Abel Sanchez-Jimenez. "Tactile information processing in the trigeminal complex of the rat". En Biomedical Optics (BiOS) 2007, editado por Valery V. Tuchin. SPIE, 2007. http://dx.doi.org/10.1117/12.709155.
Texto completoAlbuquerque, Júlia Elizabeth Nagrad de Farias, Byanka Eduarda Silva de Arruda, Fernando de Paiva Melo Neto y Francisco Nêuton de Oliveira Magalhães. "Analysis of care in neurosurgery outpatient clinic in Paraíba". En XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.277.
Texto completoMaia, Matheus Goncalves, Vivian Dias Baptista Gagliardi, Francisco Tomaz Meneses Oliveira, Eduardo dos Santos Sousa, Marina Trombin Marques, Leonardo de Sousa Bernardes y Edson Júnior Gonçalves Bechara. "Trigeminal Neuralgia associated with Wallenberg Syndrome, a case report". En XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.580.
Texto completoManzato, Luciano Bambini, José Ricardo Vanzin, Octávio Ruschel Karam, Artur Eduardo Martio, Victor Emanuel Angeliero, Luiza Rech Köhler, Paulo Moacir Mesquita Filho, Saulo de Azeredo, Yasmynni Escher y Emanuel Kerber. "Successful Endovascular Treatment of Trigeminal Neuralgia Caused by a Carotid-Cavernous Fistula: Case Report". En XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.062.
Texto completoBarile, João Paulo, Amanda Freitas Alves, Fernanda Maria Gonçalves de Sousa Moura, Sonia Maria Cesar de Azevedo Silva y Roberta Arb Saba Rodrigues Pinto. "Tolosa Hunt syndrome, case report". En XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.317.
Texto completo