Literatura académica sobre el tema "Transition to asexuality"
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Artículos de revistas sobre el tema "Transition to asexuality":
Dedukh, Dmitrij, Anatolie Marta y Karel Janko. "Challenges and Costs of Asexuality: Variation in Premeiotic Genome Duplication in Gynogenetic Hybrids from Cobitis taenia Complex". International Journal of Molecular Sciences 22, n.º 22 (9 de noviembre de 2021): 12117. http://dx.doi.org/10.3390/ijms222212117.
Beck, James B., Patrick J. Alexander, Loreen Allphin, Ihsan A. Al-Shehbaz, Catherine Rushworth, C. Donovan Bailey y Michael D. Windham. "DOES HYBRIDIZATION DRIVE THE TRANSITION TO ASEXUALITY IN DIPLOID BOECHERA?" Evolution 66, n.º 4 (8 de diciembre de 2011): 985–95. http://dx.doi.org/10.1111/j.1558-5646.2011.01507.x.
Huylmans, Ann Kathrin, Ariana Macon, Francisco Hontoria y Beatriz Vicoso. "Transitions to asexuality and evolution of gene expression in Artemia brine shrimp". Proceedings of the Royal Society B: Biological Sciences 288, n.º 1959 (22 de septiembre de 2021): 20211720. http://dx.doi.org/10.1098/rspb.2021.1720.
Kampfraath, Andries Augustus, Tjeerd Pieter Dudink, Ken Kraaijeveld, Jacintha Ellers y Zaira Valentina Zizzari. "Male Sexual Trait Decay in Two Asexual Springtail Populations Follows Neutral Mutation Accumulation Theory". Evolutionary Biology 47, n.º 4 (20 de agosto de 2020): 285–92. http://dx.doi.org/10.1007/s11692-020-09511-z.
Paland, S. "Transitions to Asexuality Result in Excess Amino Acid Substitutions". Science 311, n.º 5763 (17 de febrero de 2006): 990–92. http://dx.doi.org/10.1126/science.1118152.
Larose, Chloé, Darren J. Parker y Tanja Schwander. "Fundamental and realized feeding niche breadths of sexual and asexual stick insects". Proceedings of the Royal Society B: Biological Sciences 285, n.º 1892 (28 de noviembre de 2018): 20181805. http://dx.doi.org/10.1098/rspb.2018.1805.
Butlin, R. "Comment on "Transitions to Asexuality Result in Excess Amino Acid Substitutions"". Science 313, n.º 5792 (8 de septiembre de 2006): 1389. http://dx.doi.org/10.1126/science.1128655.
Tilquin, Anaïs y Hanna Kokko. "What does the geography of parthenogenesis teach us about sex?" Philosophical Transactions of the Royal Society B: Biological Sciences 371, n.º 1706 (19 de octubre de 2016): 20150538. http://dx.doi.org/10.1098/rstb.2015.0538.
Neiman, M., T. F. Sharbel y T. Schwander. "Genetic causes of transitions from sexual reproduction to asexuality in plants and animals". Journal of Evolutionary Biology 27, n.º 7 (26 de marzo de 2014): 1346–59. http://dx.doi.org/10.1111/jeb.12357.
Paland, S. y M. Lynch. "Response to Comment on "Transitions to Asexuality Result in Excess Amino Acid Substitutions"". Science 313, n.º 5792 (8 de septiembre de 2006): 1389. http://dx.doi.org/10.1126/science.1128745.
Tesis sobre el tema "Transition to asexuality":
Boyer, Loreleï. "Causes et conséquences évolutives de l’asexualité non-clonale chez Artemia". Thesis, Université de Montpellier (2022-….), 2022. http://www.theses.fr/2022UMONG006.
The majority of parthenogenetic species are often thought to be clonal. Clonality is costly in the long term, as it can result in accumulation of deleterious mutations and lower adaptability. However, cases reporting non-clonal asexuals are accumulating. Non-clonal asexuality has very different genomic and fitness consequences compared to clonality, and may be a key intermediate step in the transition from sex to asexuality. Additionally, asexuality may be often non-obligate, with events of cryptic sex. These events may also shape the genome and evolution of asexual lineages. In this PhD, I investigated the reproductive mode of Artemia parthenogenetica and its role in the transition from sex to asexuality and the evolution of asexual lineages. Specifically, I used the capacity of asexually produced males (“rare males”) to cross with sexual females and transmit asexuality to their offspring (contagious asexuality), to experimentally generate new lineages. I showed that diploid asexual Artemia have a non-clonal reproductive mode, in which recombination results in loss of heterozygosity (LOH) in the offspring. LOH is costly as it can reveal recessive deleterious mutations. Perhaps due to selection caused by the deleterious consequences of LOH, the recombination rate in these asexuals was lower than in a closely related sexual species. I also found that sex-asex hybrids had a mixed sexual and asexual reproduction, and that asexual females from natural populations were capable of rare sex. This means that rare events of sex in asexual Artemia could occur between a rare male and an asexual female reproducing sexually. In a review of how asexual reproductive modes were identified in the literature, I found that there was a bias in the identification and general perception of asexuals toward clonality, as an important part of the asexual species reviewed were in fact non-clonal, and evidence for clonality was often missing. Furthermore, the maj ority of non-clonal asexuals had reproductive modes that resulted in low LOH. This suggests that non-clonal asexuals often evolve secondarily toward a more clonal-like reproduction, so that even clonal species may not have been clonal throughout their evolutionary history. Finally, using genomics on contagion-generated lineages, I found that in Artemia, rare males are produced asexually through recombination and thus LOH on the ZW sex chromosomes. We know that contagious asexuality, and possibly between-lineages crosses, occurred in the evolutionary history of A. parthenogenetica. Perhaps, contagious asexuality and/or within asexual sex events provide opportunities for the gene(s) controlling asexuality to escape declining lineages into new ones. In this case, contagious asexuality through rare males may be the reason why recombination persists in asexual Artemia. Whether non-clonal asexuality and sex events occur in many parthenogenetic species is still unclear, and requires thorou gh investigation. Theoretically, there is a strong need for models taking into account the genomic consequences of non-clonal and non-obligate asexuality, and their role in the transition from sex to asexuality and the maintenance of sex
Defendini, Hélène. "Bases génétiques et conséquences évolutives de la perte de sexe dans le groupe des pucerons". Electronic Thesis or Diss., Rennes, Agrocampus Ouest, 2023. http://www.theses.fr/2023NSARA094.
Sexual reproduction is considered the ancestral reproductive mode of eukaryotes, yet it has been lost several times in many taxa. Understanding the mechanisms by which asexual lineages appear and persist over time remains a major challenge of evolutionary biology. During my PhD, I investigated the genetic basis as well as the evolutionary consequences of sex loss in aphids, a group that displays reproductive polymorphism. The ancestral reproductive mode of aphids is cyclical parthenogenesis (CP, an alternation of several parthenogenetic generations and one sexual generation), but obligate parthenogenesis (OP) is frequently observed in this group. Derived OP lineages are not able to produce sexual females though they often retain the ability to produce males. First, to characterize genomic regions involved in the transition from CP to OP reproductive mode, we used genome scan approaches on different aphid taxa that are more or less genetically related and exhibit variation in reproductive mode. We showed that the genetic basis of sex loss is different between the studied taxa, with no apparent convergence in gene content norfunctions. Thus, several independent genomic regions may be responsible for sex loss in aphids, suggesting that there are many paths that lead to asexuality in this group. Second, we studied the evolutionary consequences of the loss of sex on traits and genes essential for sexual reproduction. Since the males produced by OP lineages are unlikely to pass on their genes (because CP lineages are usually separated from OP ones), we tested the prediction that male traits should degenerate. Male production was indeed reduced in OP lineages, supposedly resulting from counter-selection, but male reproductive success was only slightly lower than in CP lineages, presumably due to the slow action of relaxed selection orunderestimation of reproductive opportunities. As OP lineages produce rare males and also do not produce sexual females, the gene expression of parthenogenetic females in these OP lineages is no longer constrained by that of other morphs. We thus predicted that the disappearance of sexual conflict (which arises when there are different morph-specific optima for a trait shared by different morphs) would result in shifts of gene expression. We therefore compared gene expression patterns of CP and OP lineages for different morphs in the pea aphid. We observed that gene expression in males from OP lineages tended towards the parthenogenetic female optimum, as predicted by theory. More surprisingly, males and parthenogenetic females of OP lineages consistently over-expressed genes typically expressed in the gonads of sexual morphs. These changes in gene expression in OP lineages may arise from the relaxation of selection or the repurposing of gene networks otherwise used in sexual lineages. This thesis illustrates the relevance of using species with polymorphic reproductive systems to understand the evolutionary history of sex loss and its consequences
Capítulos de libros sobre el tema "Transition to asexuality":
Teather, Kevin. "The Road to Sexual Reproduction". En The Evolution of Sex, 33–49. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/9780191994418.003.0003.
Actas de conferencias sobre el tema "Transition to asexuality":
Schwander, Tanja. "Convergent gene expression changes across independent transitions to asexuality: Insights from stick insects". En 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94152.