Literatura académica sobre el tema "Stress dicarbonylé"
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Artículos de revistas sobre el tema "Stress dicarbonylé"
Csongová, Melinda, Jean L. J. M. Scheijen, Marjo P. H. van de Waarenburg, Radana Gurecká, Ivana Koborová, Tamás Tábi, Éva Szökö, Casper G. Schalkwijk y Katarína Šebeková. "Association of α-Dicarbonyls and Advanced Glycation End Products with Insulin Resistance in Non-Diabetic Young Subjects: A Case-Control Study". Nutrients 14, n.º 22 (21 de noviembre de 2022): 4929. http://dx.doi.org/10.3390/nu14224929.
Texto completoAlouffi, Sultan y Mohd Wajid Ali Khan. "Dicarbonyls Generation, Toxicities, Detoxifications and Potential Roles in Diabetes Complications". Current Protein & Peptide Science 21, n.º 9 (11 de diciembre de 2020): 890–98. http://dx.doi.org/10.2174/1389203720666191010155145.
Texto completoPark, Min, Takanori Nishimura, Carlos D. Baeza-Garza, Stuart T. Caldwell, Pamela Boon Li Pun, Hiran A. Prag, Tim Young et al. "Confirmation of the Cardioprotective Effect of MitoGamide in the Diabetic Heart". Cardiovascular Drugs and Therapy 34, n.º 6 (26 de septiembre de 2020): 823–34. http://dx.doi.org/10.1007/s10557-020-07086-7.
Texto completoPeter, Andreas, Erwin Schleicher, Elisabeth Kliemank, Julia Szendroedi, Alfred Königsrainer, Hans-Ulrich Häring, Peter P. Nawroth y Thomas Fleming. "Accumulation of Non-Pathological Liver Fat Is Associated with the Loss of Glyoxalase I Activity in Humans". Metabolites 14, n.º 4 (7 de abril de 2024): 209. http://dx.doi.org/10.3390/metabo14040209.
Texto completoRabbani, Naila, Maryam Al-Motawa y Paul J. Thornalley. "Protein Glycation in Plants—An Under-Researched Field with Much Still to Discover". International Journal of Molecular Sciences 21, n.º 11 (30 de mayo de 2020): 3942. http://dx.doi.org/10.3390/ijms21113942.
Texto completoShumaev, Konstantin B., Olga V. Kosmachevskaya, Elvira I. Nasybullina, Enno K. Ruuge y Alexey F. Topunov. "Role of Nitric Oxide-Derived Metabolites in Reactions of Methylglyoxal with Lysine and Lysine-Rich Protein Leghemoglobin". International Journal of Molecular Sciences 24, n.º 1 (22 de diciembre de 2022): 168. http://dx.doi.org/10.3390/ijms24010168.
Texto completoAhmad, Khurshid, Sibhghatulla Shaikh, Eun Ju Lee, Yong-Ho Lee y Inho Choi. "Consequences of Dicarbonyl Stress on Skeletal Muscle Proteins in Type 2 Diabetes". Current Protein & Peptide Science 21, n.º 9 (11 de diciembre de 2020): 878–89. http://dx.doi.org/10.2174/1389203720666191119100759.
Texto completoRabbani, Naila, Mingzhan Xue y Paul J. Thornalley. "Methylglyoxal-induced dicarbonyl stress in aging and disease: first steps towards glyoxalase 1-based treatments". Clinical Science 130, n.º 19 (23 de agosto de 2016): 1677–96. http://dx.doi.org/10.1042/cs20160025.
Texto completoNigro, Cecilia, Alessia Leone, Francesca Fiory, Immacolata Prevenzano, Antonella Nicolò, Paola Mirra, Francesco Beguinot y Claudia Miele. "Dicarbonyl Stress at the Crossroads of Healthy and Unhealthy Aging". Cells 8, n.º 7 (19 de julio de 2019): 749. http://dx.doi.org/10.3390/cells8070749.
Texto completoTatone, Carla, Ursula Eichenlaub-Ritter y Fernanda Amicarelli. "Dicarbonyl stress and glyoxalases in ovarian function". Biochemical Society Transactions 42, n.º 2 (20 de marzo de 2014): 433–38. http://dx.doi.org/10.1042/bst20140023.
Texto completoTesis sobre el tema "Stress dicarbonylé"
Halkoum, Rym. "Rôle du glyoxal dans la sénescence cellulaire : implications dans le vieillissement de la peau". Electronic Thesis or Diss., Sorbonne université, 2021. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2021SORUS016.pdf.
Texto completoSenescence is a well-characterized cellular state associated with specific markers such as permanent cell proliferation arrest, and the secretion of messenger molecules by cells expressing the Senescence-Associated Secretory Phenotype (SASP). The SASP can display autocrine and paracrine effects which contribute to the senescent phenotype reinforcement and propagation. The SASP composition depends on many factors such as the cell type or the nature of the stress that induces senescence. Since the skin constitutes a barrier with the external environment, it is particularly subjected to different types of stresses, and consequently prone to premature cellular aging. Glyoxal, a dicarbonyl compound produced during glucose metabolism and lipid peroxidation, is a precursor of Advanced Glycation End-products (AGEs), whose presence marks normal and pathological aging. My thesis work showed that glyoxal treatment provokes oxidative stress by increasing reactive oxygen species and AGEs levels and induce senescence in human keratinocytes. Furthermore, glyoxal-induced senescence bears a unique molecular progression profile: an “early-stage” when AKT-FOXO3a-p27KIP1 pathway mediates cell-cycle arrest, and a “late-stage” senescence maintained by the p16INK4/pRb pathway. Moreover, we characterized the resulting secretory phenotype during early senescence by mass spectrometry in order to find new targets for senomorphic ingredients. Our study provides evidence that glyoxal can affect keratinocyte functions and act as a driver of human skin aging
Ashour, Amal. "Dicarbonyl stress and dysfunction of the glyoxalase system in periodontal diseases". Thesis, University of Warwick, 2016. http://wrap.warwick.ac.uk/80026/.
Texto completoKimzey, Michael John. "Identification, Characterization, and Quantification of Dicarbonyl Adducts in the Plasma Proteome in Type-2 Diabetes". Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/145123.
Texto completoRosenstock, Philip [Verfasser], Iris [Gutachter] Thondorf, Rüdiger [Gutachter] Horstkorte y Lars-Oliver [Gutachter] Klotz. "Untersuchungen humaner natürlicher Killer-Zellen und ihrer Sialyltransferasen nach Dicarbonyl-Stress / Philip Rosenstock ; Gutachter: Iris Thondorf, Rüdiger Horstkorte, Lars-Oliver Klotz". Halle (Saale) : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2020. http://d-nb.info/1215098855/34.
Texto completoYang, Kai. "Formation and Metabolism of Sugar Metabolites, Glyoxal and Methylglyoxal, and their Molecular Cytotoxic Mechanisms in Isolated Rat Hepatocytes". Thesis, 2011. http://hdl.handle.net/1807/31650.
Texto completoBanach, Monica Sofia. "Hepatocyte Cytotoxicity Induced by Hydroperoxide (Oxidative Stress Model) or Dicarbonyls (Carbonylation Model): Prevention by Bioactive Nut Extracts or Catechins". Thesis, 2009. http://hdl.handle.net/1807/18164.
Texto completoCapítulos de libros sobre el tema "Stress dicarbonylé"
Kovacic, Peter y Ratnasamy Somanathan. "Novel Mechanism for Advanced Glycation End Product (AGE) Toxicity: α-Dicarbonyls, Electron Transfer, Radicals, Oxidative Stress, and Antioxidants". En Systems Biology of Free Radicals and Antioxidants, 3405–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-30018-9_153.
Texto completoRabbani, Naila, Mingzhan Xue y Paul J. Thornalley. "Dicarbonyl stress and the glyoxalase system". En Oxidative Stress, 759–77. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-818606-0.00036-5.
Texto completoActas de conferencias sobre el tema "Stress dicarbonylé"
Bulkescher, R., S. Herzig, J. Szendrödi, PP Nawroth y J. Zemva. "Dicarbonyl stress in endothelial cells alters mitochondrial protein homeostasis". En Late Breaking Abstracts Diabetes Kongress 2021 – 55. Jahrestagung der DDG Präzisionsmedizin – Eine Reise in die Zukunft der Diabetologie www.diabeteskongress.de. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1730861.
Texto completoBellier, J., MJ Nokin, F. Durieux, F. Journe, G. Ghanem, V. Castronovo y A. Bellahcène. "PO-219 Methylglyoxal-induced dicarbonyl stress: role in melanoma progression and response to therapy". En Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.254.
Texto completoBellahcene, A., J. Bellier, O. Peulen, G. Rademaker, B. Charloteaux, S. Van Laere, M. Herfs, C. Lambert, V. Castronovo y MJ Nokin. "PO-226 Dicarbonyl stress induces ECM remodelling and MAPK signalling activation in metastatic breast cancer cells". En Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.260.
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