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Literatura académica sobre el tema "Streptococcus agalactiae – Analyse cladistique"
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Artículos de revistas sobre el tema "Streptococcus agalactiae – Analyse cladistique"
Perovic, Olga, Ali A. Yahaya, Crystal Viljoen, Jean-Bosco Ndihokubwayo, Marshagne Smith, Sheick O. Coulibaly, Linda De Gouveia et al. "External Quality Assessment of Bacterial Identification and Antimicrobial Susceptibility Testing in African National Public Health Laboratories, 2011–2016". Tropical Medicine and Infectious Disease 4, n.º 4 (13 de diciembre de 2019): 144. http://dx.doi.org/10.3390/tropicalmed4040144.
Texto completoLiu, Zeliang, Xueqi Jiang, Jie Li, Wenjing Ji, Haijian Zhou, Xinyi Gong, Beibei Miao et al. "Molecular characteristics and antibiotic resistance mechanisms of clindamycin-resistant Streptococcus agalactiae isolates in China". Frontiers in Microbiology 14 (1 de marzo de 2023). http://dx.doi.org/10.3389/fmicb.2023.1138039.
Texto completoPetzer, I. M., J. Karzis, J. C. Watermeyer, T. J. Van der Schans y R. Van Reenen. "Trends in udder health and emerging mastitogenic pathogens in South African dairy herds". Journal of the South African Veterinary Association 80, n.º 1 (21 de mayo de 2009). http://dx.doi.org/10.4102/jsava.v80i1.163.
Texto completoPino-Otín, María Rosa, Cristina Gan, Eva Terrado, María Angeles Sanz, Diego Ballestero y Elisa Langa. "Antibiotic properties of Satureja montana L. hydrolate in bacteria and fungus of clinical interest and its impact in non-target environmental microorganisms". Scientific Reports 12, n.º 1 (2 de noviembre de 2022). http://dx.doi.org/10.1038/s41598-022-22419-2.
Texto completoTesis sobre el tema "Streptococcus agalactiae – Analyse cladistique"
Beauruelle, Clémence. "Locus CRISPR de Streptococcus agalactiae : marqueur génétique de la phylogénie de l'espèce et de l'évolution récente des isolats". Electronic Thesis or Diss., Tours, 2019. http://www.theses.fr/2019TOUR3806.
Texto completoWe studied the relevance of the CRISPR1 array (associated to a CRISPR-Cas II-A type) as an epidemiological marker for genotyping and phylogenetic analyses of Streptococcus agalactiae (or Group B Streptococcus (GBS)) isolates. We demonstrate that i) spacer acquisition events occurred in vivo which strongly suggest that the CRISPR1-Cas system is functionally active for adaptation ii) ancestral markers (TDR and ancestral spacers) are highly conserved and reflect the phylogenetic structure of the GBS population (in congruence with MLST) iii) CRISPR1 array shared a high degree of polymorphism (especially for leader end spacers) offering a highly discriminatory typing method (allow to separate isolates within a same ST defined by MLST). Leader end analysis also provides specific evidence on isolates recent evolution, especially encounters with MGEs. CRISPR1 array appears as a useful genetic feature to follow vaginal carriage of GBS in women and for evaluate the diversity of GBS vaginal carriage population. On the basis of these data, we assume that this method could pretend to be a reference method for phylogenetic GBS typing
Joubert, Laetitia. "Caractérisation de l'homéostasie et de l'impact de l'hème sur les capacités de virulence et de colonisation de bactéries à GRAM positif". Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLA031/document.
Texto completoHeme is a redox-reactive molecule with essential function in bacterial metabolism. However, this molecule generates reactive oxygen species responsible for its toxicity. We characterized the mechanism of heme homeostasis involving the efflux transporter HrtBA. In L. lactis, we demonstrated that HrtBA prevents from membrane and intracellular accumulation of internalized exogenous heme thanks to a menaquinone dependent mechanism. HrtBA is also present in several pathogens. In S. agalactiae, the transcription of HrtBA is regulated by a two-component system HssRS. The HssS sensor recognizes internalized exogenous heme. To clarify the role of heme of the host in the virulence of S. agalactiae, a systemic infection model in mice using luminescence (lux) and in vivo imaging (IVIS) has been set up. The monitoring of luminescence generated by heme hypersensitive (ΔhrtBA) bacteria shows that heme of host is toxic and that the capability of S. agalactiae to control heme homeostasis is crucial for infection. In the same way, by demonstrating that respiratory metabolism is crucial for infection (ΔcydA), we demonstrated that S. agalactiae depends on its capacity to acquire the heme of the host to become infectious. By using the HrtBA promoter coupled with lux operon, we studied the capacity of S. agalactiae to detect and to acquire heme in vivo during the infection. Our results show that host heme is especially biodisponible in the liver. On the contrary, heme is not detected by bacteria in the brain. Our results prove that heme of the host is an important parameter for the adaptation of S. agalactiae to its host during infection. Blocking HrtBA or heme sensor HssS could so be a target for antibiotic research against S. agalactiae and other pathogens. Finally, we show in E. faecalis that HrtBA expression also depends on a two-component system. We used the same strategy as in S. agalactiae to create a specific heme sensor that allowed us to demonstrate for the first time that E. faecalis meets and uses heme in the digestive tract