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Artículos de revistas sobre el tema "SARS-CoV-2 (virus) – Physiopathologie"

Autor: Grafiati

Publicado: 30 de noviembre de 2024

Última modificación: 31 de julio de 2025

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1

Salinas, Sara, and Yannick Simonin. "Les atteintes neurologiques liées au SARS-CoV-2 et autres coronavirus humains." médecine/sciences 36, no. 8-9 (2020): 775–82. http://dx.doi.org/10.1051/medsci/2020122.

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L’émergence récente d’un nouveau coronavirus, le SARS-CoV-2, responsable de la maladie appelée COVID-19, est un nouvel avertissement du risque pour la santé publique représenté par les zoonoses virales et notamment par les coronavirus. Principalement connus pour leur capacité à infecter les voies respiratoires supérieures et inférieures, les coronavirus peuvent également affecter le système nerveux central et périphérique, comme c’est le cas pour de nombreux virus respiratoires, tels que les virus influenza ou le virus respiratoire syncytial. Les infections du système nerveux sont un problème

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2

POZZETTO, B., I. BECHRI, M. DELOLME, et al. "État des lieux du diagnostic virologique de l’infection à SARS-CoV-2." EXERCER 31, no. 163 (2020): 215–20. http://dx.doi.org/10.56746/exercer.2020.163.215.

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L’infection COVID-19 a émergé de façon soudaine en Chine, en décembre 2019 et est devenue rapidement pandémique. Le virus responsable a été identifié comme un nouveau coronavirus probablement issu d’un virus de chauve-souris, dénommé SARS-CoV-2, ce qui a permis de mettre au point des tests diagnostiques permettant l’identification de son ARN par techniques moléculaires. En plus du rappel de quelques données virologiques, l’objet de cette revue est de présenter les tests moléculaires de diagnostic direct et les tests sérologiques actuellement disponibles pour identifier cette infection. Le diag

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3

Meunier, Nicolas. "Odorat et virus respiratoires :une relation révélée par la Covid-19." médecine/sciences 39, no. 2 (2023): 119–28. http://dx.doi.org/10.1051/medsci/2023007.

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L’odorat, sens pendant longtemps sous-estimé chez l’homme, a été mis sur le devant de la scène par sa soudaine disparition, survenue pendant la pandémie de Covid-19, dont l’anosmie est un des symptômes majeurs. Pourtant, depuis longtemps, les virus respiratoires ont été associés aux troubles de l’odorat, dont 25 % seraient liés à une infection virale. L’olfaction débute dans le nez, au sein d’un épithélium olfactif qui a la particularité de contenir des neurones en contact direct avec l’environnement. Plusieurs virus respiratoires sont connus pour leur capacité réplicative au sein de cet épith

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4

Delolme, M., Bruno Pozzetto, Josselin Rigaill, et al. "Les tests de diagnostic virologique de la Covid-19." Revue de biologie médicale 359, no. 2 (2021): 17–28. https://doi.org/10.3917/rbm.359.0017.

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L’infection Covid-19 a émergé soudainement dans la ville chinoise de Wuhan, en décembre 2019, et est devenue rapidement pandémique. Mi-janvier 2021, plus de 96 millions de cas confirmés sont survenus dans 218 pays ou territoires depuis son émergence un an auparavant, dont plus de 2 millions de cas mortels. Le virus responsable a été promptement identifié comme un nouveau coronavirus probablement issu d’un virus de chauve-souris, dénommé SARS-CoV-2, ce qui a permis de mettre au point des techniques permettant de le dépister. Le diagnostic repose principalement sur la détection du génome viral (

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5

Stepanian, Alain, A. Ibos, Virginie Siguret, and Maxime Delrue. "Complications thromboemboliques lors de la première vague de COVID-19 : quels enseignements pratiques pour le biologiste médical ?" Revue de biologie médicale 363, no. 6 (2021): 7–17. https://doi.org/10.3917/rbm.363.0007.

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Le 31 décembre 2019, l’Organisation mondiale de la Santé (OMS) est informée de l’existence de cas de pneumonies virales d’étiologie inconnue détectés à Wuhan, dans la province du Hubei, en Chine. En quelques semaines, ce nouveau virus dénommé le SARS-CoV-2 ( severe acute respiratory syndrome coronavirus 2 ), responsable de la COVID-19 ( coronavirus disease 2019 ), a conduit à une pandémie d’une ampleur exceptionnelle. Si la maladie est le plus souvent bénigne, elle est responsable chez certains patients d’un syndrome de détresse respiratoire aiguё qui nécessite une prise en charge hospitalière

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6

Placinta, Gheorghe, Victor Pantea, Lilia Cojuhari, Valentin Cebotarescu, Lidia Placinta, and Dan Croitoru. "Interaction between SARS-CoV-2 and human organism." Moldovan Medical Journal 63(2) (May 30, 2020): 57–62. https://doi.org/10.5281/zenodo.3866031.

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<strong>Background: </strong>COVID-19 is a part of the betacoronaviridae group, sabercoviridae subgroup. At the moment we are confronting a pandemy, which has a completely new pathologic pattern for the whole world. Considering the highly contagious characteristics of the virus, which is the cause of death for many people, and also the fact that many people continue to be infected with this virus there is a stringing necesity of combating this pandemy. Because of that we need the knowledge that regards the physiopathologic mechanisms, particularities of the host-invader interactions, relevance

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7

Cespedes, Mateus da Silveira, and José Carlos Rosa Pires de Souza. "Sars-CoV-2: A clinical update - II." Revista da Associação Médica Brasileira 66, no. 4 (2020): 547–57. http://dx.doi.org/10.1590/1806-9282.66.4.547.

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SUMMARY INTRODUCTION A covid-19 pandemic decreed by WHO has raised greater awareness of it. EPIDEMIOLOGY The infection, reached the mark of 2,000,000 patients in 33 countries and caused the risk of the presence of comorbidities and advanced age. TRANSMISSIBILITY The transmissibility calculated so far is similar to the H1N1 epidemic, but with lower mortality rates. PHYSIOPATHOLOGY The SARS-CoV-2 virus, of the Coronaviridae family, has the capacity for cellular invasion through the angiotensin-converting enzyme 2 does not have a lower respiratory epithelium and in the cells of the small intestin

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8

Sacramento, Carolina Q., Natalia Fintelman-Rodrigues, Suelen S. G. Dias, et al. "Unlike Chloroquine, Mefloquine Inhibits SARS-CoV-2 Infection in Physiologically Relevant Cells." Viruses 14, no. 2 (2022): 374. http://dx.doi.org/10.3390/v14020374.

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Despite the development of specific therapies against severe acute respiratory coronavirus 2 (SARS-CoV-2), the continuous investigation of the mechanism of action of clinically approved drugs could provide new information on the druggable steps of virus–host interaction. For example, chloroquine (CQ)/hydroxychloroquine (HCQ) lacks in vitro activity against SARS-CoV-2 in TMPRSS2-expressing cells, such as human pneumocyte cell line Calu-3, and likewise, failed to show clinical benefit in the Solidarity and Recovery clinical trials. Another antimalarial drug, mefloquine, which is not a 4-aminoqui

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9

Singh, Preeti, and Biplob Adhikari. "Journey of SARS-CoV-19 from Mucosa Towards Angiotensin-Converting Enzyme2 Pathway: A Review." Journal of Nepalese Society of Periodontology and Oral Implantology 5, no. 2 (2021): 92–96. http://dx.doi.org/10.3126/jnspoi.v5i2.52980.

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The disease caused by severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) or simply corona virus disease-2019 (COVID-19), has rapidly spread throughout the world. World Health Organization declared a global health emergency on January 30, 2020 announcing it as a Pandemic. The outbreak of the disease has posed serious health threat. A search was conducted for review which included various articles from 2019 to 2021 in PubMed/MEDLINE with keywords ACE2, 2019 NCOV, pandemic, physiopathology, SARS, Transmission. Both humoral and acquired immunity are targeted by the virus during progressi

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10

Pacnejer, Aliteia-Maria, Anca Butuca, Carmen Maximiliana Dobrea, et al. "Neuropsychiatric Burden of SARS-CoV-2: A Review of Its Physiopathology, Underlying Mechanisms, and Management Strategies." Viruses 16, no. 12 (2024): 1811. http://dx.doi.org/10.3390/v16121811.

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The COVID-19 outbreak, caused by the SARS-CoV-2 virus, was linked to significant neurological and psychiatric manifestations. This review examines the physiopathological mechanisms underlying these neuropsychiatric outcomes and discusses current management strategies. Primarily a respiratory disease, COVID-19 frequently leads to neurological issues, including cephalalgia and migraines, loss of sensory perception, cerebrovascular accidents, and neurological impairment such as encephalopathy. Lasting neuropsychological effects have also been recorded in individuals following SARS-CoV-2 infection

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11

González-Villalva, Adriana, Aurora de la Peña-Díaz, Marcela Rojas-Lemus, et al. "Fisiología de la hemostasia y su alteración por la coagulopatía en COVID-19." Revista de la Facultad de Medicina 63, no. 5 (2020): 45–57. http://dx.doi.org/10.22201/fm.24484865e.2020.63.5.08.

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COVID-19 global pandemic caused by Sars-CoV-2 virus, has worried to health care providers due to the high mortality rate related to coagulopathy in many patients. COVID-19 coagulopathy is mainly thrombotic, first locally in lungs but later on it becomes micro and macrovascular systemic coagulopathy. It has been associated to endothelial damage, inflammation, neutrophil-extracellular traps, monocyte and macrophage activation, cytokines storm that induce a vicious cycle of thrombosis and inflammation. The increased levels of prothrombotic factors as tissue factor, Von Willebrand factor, fibrinog

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12

Evangelista, Heitor, César Amaral, Luís Cristóvão Porto, et al. "Modeling the initial phase of SARS-CoV-2 deposition in the respiratory tract mimicked by the 11C radionuclide." PLOS ONE 16, no. 1 (2021): e0245019. http://dx.doi.org/10.1371/journal.pone.0245019.

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The knowledge on the deposition and retention of the viral particle of SARS-CoV-2 in the respiratory tract during the very initial intake from the ambient air is of prime importance to understand the infectious process and COVID-19 initial symptoms. We propose to use a modified version of a widely tested lung deposition model developed by the ICRP, in the context of the ICRP Publication 66, that provides deposition patterns of microparticles in different lung compartments. In the model, we mimicked the "environmental decay" of the virus, determined by controlled experiments related to normal s

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13

Chaturvedi, Pragya, and Sudhanshu Singh. "Significance of Epigenetics in Sars-CoV-2 Infection and Proposed Epi-Drugs for Covid-19." SAMRIDDHI : A Journal of Physical Sciences, Engineering and Technology 13, SUP 1 (2021): 21–26. http://dx.doi.org/10.18090/samriddhi.v13is1.6.

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We frequently come in contact with animal viruses through the food which we eat, the pets which we have, and our connections with nature. The enormous majority of viruses which enter our bodies pass inoffensively through our physiological systems or eradicate by our immune systems. However, on rare circumstances, a human-encounters by a virus which begins to replicate itself, accomplishing its entire lifecycle within human cells and intensifying themselves into a large number. Replication of an animal virus inside the human body is the key instant in the zoonotic process. SARS CoV-2 is one of

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14

Lopes, Luiz Thiago Oliveira, Marcelus de Andrade Oliveira, Willian Guilherme Lobato Gonçalves, et al. "Séquelles de la COVID-19 : revue intégrative de la littérature." Núcleo do Conhecimento 01, no. 08 (2023): 68–87. https://doi.org/10.32749/nucleodoconhecimento.com.br/sante/sequelles-de-la-covid.

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La grande affinité entre la protéine Spike du virus SARS-CoV-2 et le récepteur de l'enzyme de conversion de l'angiotensine 2 est postulée comme l'une des principales raisons de la forte taux de transmission virale, ce qui a conduit l'OMS à déclarer la COVID-19 comme une Urgence de Santé Publique d'Intérêt International, ainsi qu'à adopter des mesures de contenances de la propagation virale. De plus, grâce au mécanisme physiopathologique du virus, on observe généralement des symptômes non spécifiques, une évolution atypique, principalement chez les personnes âgées et immunodéprimées, ainsi qu'u

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15

Barbosa, Adriana Aparecida de Oliveira, Gabriel Cunha Beato, Pietra Antônia Filiol Belin, and Larissa Ramos Araújo. "ASPECTOS CLÍNICOS DA MÁ NUTRIÇÃO NA COVID-19." Simbio-Logias Revista Eletrônica de Educação Filosofia e Nutrição 12, no. 16 (2020): 01–19. http://dx.doi.org/10.32905/19833253.2020.12.16p01.

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The pandemic caused by the new coronavirus has sparked discussions among health professionals about the role of food and nutrition in the infectious condition caused by the SARS-CoV-2 virus in different population groups. Malnutrition, including obesity, may reflect more severe outcomes in the physiopathology of infection and systemic responses caused by COVID-19. The present work aims to make considerations directed to the nutritionist about the susceptibility of COVID-19 in individuals submitted to malnutrition, highlighting possible outcomes of the disease and the importance of nutritional

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16

Moatar, Alexandra Ioana, Aimee Rodica Chis, Catalin Marian, and Ioan-Ovidiu Sirbu. "Gene Network Analysis of the Transcriptome Impact of SARS-CoV-2 Interacting MicroRNAs in COVID-19 Disease." International Journal of Molecular Sciences 23, no. 16 (2022): 9239. http://dx.doi.org/10.3390/ijms23169239.

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According to the World Health Organization (WHO), as of June 2022, over 536 million confirmed COVID-19 disease cases and over 6.3 million deaths had been globally reported. COVID-19 is a multiorgan disease involving multiple intricated pathological mechanisms translated into clinical, biochemical, and molecular changes, including microRNAs. MicroRNAs are essential post-transcriptional regulators of gene expression, being involved in the modulation of most biological processes. In this study, we characterized the biological impact of SARS-CoV-2 interacting microRNAs differentially expressed in

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17

Cespedes, Mateus da Silveira, and José Carlos Rosa Pires de Souza. "Coronavirus: a clinical update of Covid-19." Revista da Associação Médica Brasileira 66, no. 2 (2020): 116–23. http://dx.doi.org/10.1590/1806-9282.66.2.116.

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SUMMARY INTRODUCTION A covid-19 pandemic decreed by WHO has raised greater awareness of it. EPIDEMIOLOGY The infection reached the mark of 350,000 patients in 33 countries and affected as comorbidities the presence of comorbidities and advanced age. TRANSMISSIBILITY The transmissibility calculated so far is similar to the H1N1 epidemic, but with lower mortality rates. PHYSIOPATHOLOGY The SARS-CoV-2 virus, of the Coronaviridae family, has the capacity for cellular invasion through the angiotensin-converting enzyme 2 does not have a lower respiratory epithelium and in the cells of the small inte

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18

Devaux, Christian A., Jean-Christophe Lagier, and Didier Raoult. "New Insights Into the Physiopathology of COVID-19: SARS-CoV-2-Associated Gastrointestinal Illness." Frontiers in Medicine 8 (February 18, 2021). http://dx.doi.org/10.3389/fmed.2021.640073.

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Although SARS-CoV-2 is considered a lung-tropic virus that infects the respiratory tract through binding to the ACE2 cell-surface molecules present on alveolar lungs epithelial cells, gastrointestinal symptoms have been frequently reported in COVID-19 patients. What can be considered an apparent paradox is that these symptoms (e.g., diarrhea), sometimes precede the development of respiratory tract illness as if the breathing apparatus was not its first target during viral dissemination. Recently, evidence was reported that the gut is an active site of replication for SARS-CoV-2. This replicati

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19

Teixeira, Lívia, Jairo R. Temerozo, Filipe S. Pereira-Dutra, et al. "Simvastatin Downregulates the SARS-CoV-2-Induced Inflammatory Response and Impairs Viral Infection Through Disruption of Lipid Rafts." Frontiers in Immunology 13 (February 18, 2022). http://dx.doi.org/10.3389/fimmu.2022.820131.

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Coronavirus disease 2019 (COVID-19) is currently a worldwide emergency caused by Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2). In observational clinical studies, statins have been identified as beneficial to hospitalized patients with COVID-19. However, experimental evidence of underlying statins protection against SARS-CoV-2 remains elusive. Here we reported for the first-time experimental evidence of the protective effects of simvastatin treatment both in vitro and in vivo. We found that treatment with simvastatin significantly reduced the viral replication and lung damage in

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20

Monchatre-Leroy, Elodie, Sandrine Lesellier, Marine Wasniewski, et al. "Hamster and ferret experimental infection with intranasal low dose of a single strain of SARS-CoV-2." Journal of General Virology 102, no. 3 (2021). http://dx.doi.org/10.1099/jgv.0.001567.

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Understanding the pathogenesis of the SARS-CoV-2 infection is key to developing preventive and therapeutic strategies against COVID-19, in the case of severe illness but also when the disease is mild. The use of appropriate experimental animal models remains central in the in vivo exploration of the physiopathology of infection and antiviral strategies. This study describes SARS-CoV-2 intranasal infection in ferrets and hamsters with low doses of low-passage SARS-CoV-2 clinical French isolate UCN19, describing infection levels, excretion, immune responses and pathological patterns in both anim

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21

Martin-Cardona, Albert, Josep Lloreta Trull, Raquel Albero-González, et al. "SARS-CoV-2 identified by transmission electron microscopy in lymphoproliferative and ischaemic intestinal lesions of COVID-19 patients with acute abdominal pain: two case reports." BMC Gastroenterology 21, no. 1 (2021). http://dx.doi.org/10.1186/s12876-021-01905-3.

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Abstract Background SARS-CoV-2 may produce intestinal symptoms that are generally mild, with a small percentage of patients developing more severe symptoms. The involvement of SARS-CoV-2 in the physiopathology of bowel damage is poorly known. Transmission electron microscopy (TEM) is a useful tool that provides an understanding of SARS-CoV-2 invasiveness, replication and dissemination in body cells but information outside the respiratory tract is very limited. We report two cases of severe intestinal complications (intestinal lymphoma and ischaemic colitis) in which the presence of SARS-CoV-2

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22

Zazzara, Maria Beatrice, Anna Modoni, Alessandra Bizzarro, et al. "COVID-19 atypical Parsonage-Turner syndrome: a case report." BMC Neurology 22, no. 1 (2022). http://dx.doi.org/10.1186/s12883-022-02622-4.

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Abstract Background Neurological manifestations of Sars-CoV-2 infection have been described since March 2020 and include both central and peripheral nervous system manifestations. Neurological symptoms, such as headache or persistent loss of smell and taste, have also been documented in COVID-19 long-haulers. Moreover, long lasting fatigue, mild cognitive impairment and sleep disorders appear to be frequent long term neurological manifestations after hospitalization due to COVID-19. Less is known in relation to peripheral nerve injury related to Sars-CoV-2 infection. Case presentation We repor

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23

Errasfa, Mourad. "Milk Oligosaccharides and Lectins as Candidates for Clinical Trials Against Covid-19." Current Nutrition & Food Science 16 (August 19, 2020). http://dx.doi.org/10.2174/1573401316999200819125355.

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Background: Covid-19 pandemia is causing a very high death toll around the world and a serious fall in the global economy. Many clinical trials are currently underway to check the effectiveness of some known drugs. The physiopathology associated with the virus infection is currently better understood and good prophylactic drug therapies are implemented, such as antibiotics and blood thinners, though, no specific drugs against SARS-Cov-2 were developed yet. Objective: In the present research work, it is aimed to carry out a bibliographic investigation on some active molecular species that could

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24

Oliviero, Antonio, Fernando de Castro, Francesca Coperchini, Luca Chiovato, and Mario Rotondi. "COVID-19 Pulmonary and Olfactory Dysfunctions: Is the Chemokine CXCL10 the Common Denominator?" Neuroscientist, July 13, 2020, 107385842093903. http://dx.doi.org/10.1177/1073858420939033.

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COVID-19 is an ongoing viral pandemic that emerged from East Asia and quickly spread to the rest of the world. SARS-CoV-2 is the virus causing COVID-19. Acute respiratory distress syndrome (ARDS) is definitely one of the main clinically relevant consequences in patients with COVID-19. Starting from the earliest reports of the COVID-19 pandemic, two peculiar neurological manifestations (namely, hyposmia/anosmia and dysgeusia) were reported in a relevant proportion of patients infected by SARS-CoV-2. At present, the physiopathologic mechanisms accounting for the onset of these symptoms are not y

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Mendonça Filho, Valder Cavalcante Maia, Amanda Gomes de Oliveira, Isabelle de Fátima Vieira Camelo Maia, et al. "COVID-19 in the nervous system: physiopathology and neurological manifestations." Arquivos de Neuro-Psiquiatria, July 4, 2023. http://dx.doi.org/10.1055/s-0043-1769123.

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Abstract Background Coronavirus disease 2019 (COVID-19) is a viral infection caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Although respiratory manifestations have received greater visibility during the pandemic caused by this virus, numerous neurological complaints related to coronavirus 2 infection have been documented in several countries. These records suggest that this pathogen presents neurotropism, and it can cause different neurological conditions of varying intensity. Objective To investigate the ability of coronavirus 2 to invade the central nervous system (

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26

Eslami, Majid, Anna Abdolshahi, Alireza Emadi, and Bahman Yousefi. "IMPORTANCE OF PROBIOTICS IN THE PREVENTION AND TREATMENT OF COVID-19." Journal of microbiology, biotechnology and food sciences, October 20, 2022, e4594. http://dx.doi.org/10.55251/jmbfs.4594.

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The novel severe acute respiratory syndrome COVID-19 outbreak is brought on by the SARS-CoV-2 coronavirus. Taking into account the criticism that SARS-CoV-2 has received on a global scale, efficient preventive actions and treatment for COVID-19 would be an urgent need. New strategies were developed based on immune responses. The immunomodulating properties of a few bioactive substances, minerals, and micronutrients have led to recommendations. Probiotics' therapeutic uses in COVID-19 patients were looked into in this review. A decline in the amount of various probiotic species, including Bifid

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Kazezoglu, Cemal, Arzu İrvem, Yasemin Tuba Sutasir, et al. "Urine amino acid and gamma aminobutyric acid level in COVID 19 patients." Iranian Journal of Microbiology, February 10, 2025. https://doi.org/10.18502/ijm.v17i1.17809.

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Background and Objectives: SARS CoV2 has tropism for various tissues, including the respiratory tract, brain, endothe- lium, heart, kidney and liver. Neurological symptoms can also be seen in the clinical course of the disease, and anosmia is the most common. The main objective of our study was to examine the urinary amino acid profiles of moderately severe patients diagnosed with COVID-19 with a positive RT-PCR test and try to find metabolic changes associated with the infec- tion. Also, it was aimed to investigate the neuroinhibitory Gamma-Aminobutyric acid (GABA) levels in order to examine

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Zhong, Han, Yang Zhou, Shu-Ya Mei, et al. "Scars of COVID-19: A bibliometric analysis of post-COVID-19 fibrosis." Frontiers in Public Health 10 (September 20, 2022). http://dx.doi.org/10.3389/fpubh.2022.967829.

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BackgroundThe coronavirus disease 2019 (COVID-19) becomes a worldwide public health threat. Increasing evidence proves that COVID-19-induced acute injuries could be reversed by a couple of therapies. After that, post-COVID-19 fibrosis (PCF), a sequela of “Long COVID,” earns rapidly emerging concerns. PCF is associated with deteriorative lung function and worse quality of life. But the process of PCF remains speculative. Therefore, we aim to conduct a bibliometric analysis to explore the overall structure, hotspots, and trend topics of PCF.Materials and methodsA comprehensive search was perform

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Lopes, Luiz Thiago Oliveira, Marcelus de Andrade Oliveira, Willian Guilherme Lobato Gonçalves, et al. "Séquelles de la COVID-19 : revue intégrative de la littérature." Revista Científica Multidisciplinar Núcleo do Conhecimento, August 8, 2023, 68–87. http://dx.doi.org/10.32749/nucleodoconhecimento.com.br/sante/sequelles-de-la-covid.

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La grande affinité entre la protéine Spike du virus SARS-CoV-2 et le récepteur de l’enzyme de conversion de l’angiotensine 2 est postulée comme l’une des principales raisons de la forte taux de transmission virale, ce qui a conduit l’OMS à déclarer la COVID-19 comme une Urgence de Santé Publique d’Intérêt International, ainsi qu’à adopter des mesures de contenances de la propagation virale. De plus, grâce au mécanisme physiopathologique du virus, on observe généralement des symptômes non spécifiques, une évolution atypique, principalement chez les personnes âgées et immunodéprimées, ainsi qu’u

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