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Artículos de revistas sobre el tema "Pathogenic"

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Publicado: 4 de junio de 2021
Última modificación: 1 de agosto de 2024

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1

Gvozdyak, R. I. "«Pathogen-1» Experiment Aggression of pathogenic bacteria in microgravity". Kosmìčna nauka ì tehnologìâ 6, n.º 4 (30 de julio de 2000): 111. http://dx.doi.org/10.15407/knit2000.04.119.

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2

Ruchel, Reinhard. "Proteinasen pathogener Pilze: Proteinases of pathogenic fungi". Mycoses 42, S1 (abril de 1999): 48–52. http://dx.doi.org/10.1111/j.1439-0507.1999.tb04527.x.

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3

Luzzatto, Lucio, Caterina Nannelli y Rosario Notaro. "Potentially pathogenic and pathogenic G6PD variants". American Journal of Human Genetics 110, n.º 11 (noviembre de 2023): 1983–85. http://dx.doi.org/10.1016/j.ajhg.2023.10.003.

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4

Gharwalova, Lucia, Marketa Kulisova, Anastasiia Vasyliuk, Helena Maresova, Andrea Palyzova, Linda Nedbalova y Irena Kolouchova. "Sphingolipids of plant pathogenic fungi". Plant Protection Science 57, No. 2 (1 de marzo de 2021): 134–39. http://dx.doi.org/10.17221/131/2020-pps.

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Glycosphingolipids in filamentous fungi are significant components of the plasma membrane and are vital for different cellular processes, such as growth, morphological transition or signal transduction. Fungal growth inhibitors targeting glycosylinositolphosphoceramide (GIPCs) biosynthesis or antifungal compounds binding to GIPCs present in membranes could present a safe way of preventing fungal growth on crops since GIPCs are not present in mammalian cells. Mass spectrometry-based shotgun lipidomics was used to analyze sphingolipids of 11 fungal strains isolated from plant material. Molecular species with inositol ceramides containing zero to five carbohydrates were identified. Differences in the amount of individual molecular species were influenced by the taxonomic affiliation. All tested strains exhibited a relatively high content (more than 40 mol.%) of GIPCs with three and more saccharides attached to the polar head. It could be assumed that the sphingolipid profiles of the tested plant pathogens would be an adaptation mechanism to antifungal plant defensins.
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5

Finn, Albert F. y Peter D. Gorevic. "Pathogenic paraproteins". Current Opinion in Rheumatology 2, n.º 4 (agosto de 1990): 652–60. http://dx.doi.org/10.1097/00002281-199002040-00017.

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6

Horvat, Rebecca T. "PATHOGENIC FUNGI". Shock 30, n.º 6 (diciembre de 2008): 753. http://dx.doi.org/10.1097/01.shk.0000336210.36795.86.

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7

Gould, EA y T. Solomon. "Pathogenic flaviviruses". Lancet 371, n.º 9611 (febrero de 2008): 500–509. http://dx.doi.org/10.1016/s0140-6736(08)60238-x.

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8

DIPERRI, G. "PATHOGENIC ENTAMOEBA". Lancet 331, n.º 8595 (mayo de 1988): 1166. http://dx.doi.org/10.1016/s0140-6736(88)91980-0.

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9

Gao, Shou-Jiang. "Pathogenic procedures". Trends in Microbiology 5, n.º 3 (marzo de 1997): 125–26. http://dx.doi.org/10.1016/s0966-842x(97)87506-3.

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10

Tran Van Nhieu, Guy. "Pathogenic paradox?" Trends in Microbiology 7, n.º 3 (marzo de 1999): 102. http://dx.doi.org/10.1016/s0966-842x(99)01473-0.

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11

Casci, Tanita. "Pathogenic conversions". Nature Reviews Genetics 13, n.º 1 (16 de diciembre de 2011): 2. http://dx.doi.org/10.1038/nrg3143.

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12

Dempsey, Laurie A. "Pathogenic antibodies". Nature Immunology 20, n.º 11 (22 de octubre de 2019): 1414. http://dx.doi.org/10.1038/s41590-019-0535-6.

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13

POOLMAN, J. "Pathogenic neisseriae". Lancet 336, n.º 8722 (octubre de 1990): 1061. http://dx.doi.org/10.1016/0140-6736(90)92518-m.

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14

Prasanna, Arun N. y Sarika Mehra. "Comparative Phylogenomics of Pathogenic and Non-Pathogenic Mycobacterium". PLoS ONE 8, n.º 8 (28 de agosto de 2013): e71248. http://dx.doi.org/10.1371/journal.pone.0071248.

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15

Foley, J. F. "Detecting a Pathogenic Activity, Not a Pathogenic Molecule". Science Signaling 7, n.º 343 (16 de septiembre de 2014): ec252-ec252. http://dx.doi.org/10.1126/scisignal.2005897.

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16

Thongboonkerd, Visith, Wararat Chiangjong, Putita Saetun, Supachok Sinchaikul, Shui-Tein Chen y Uraiwan Kositanont. "Analysis of differential proteomes in pathogenic and non-pathogenic Leptospira : Potential pathogenic and virulence factors". PROTEOMICS 9, n.º 13 (julio de 2009): 3522–34. http://dx.doi.org/10.1002/pmic.200700855.

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17

Bøe, Johs. "On the Distinction Between Pathogenic and Non-Pathogenic Staphylococci". Acta Pathologica Microbiologica Scandinavica 21, n.º 5 (14 de agosto de 2009): 721–30. http://dx.doi.org/10.1111/j.1699-0463.1944.tb04972.x.

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18

Wurtzel, Omri, Nina Sesto, J. R. Mellin, Iris Karunker, Sarit Edelheit, Christophe Bécavin, Cristel Archambaud, Pascale Cossart y Rotem Sorek. "Comparative transcriptomics of pathogenic and non‐pathogenic Listeria species". Molecular Systems Biology 8, n.º 1 (enero de 2012): 583. http://dx.doi.org/10.1038/msb.2012.11.

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19

DiMiceli, Lauren. "Distinguishing Between Pathogenic and Non-Pathogenic Species of Entamoeba". Laboratory Medicine 35, n.º 10 (1 de octubre de 2004): 613–15. http://dx.doi.org/10.1309/b81npvaw8y4bgy11.

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20

Roberts, Glenn D. "Medical Mycology: The Pathogenic Fungi and the Pathogenic Actinomycetes". Mayo Clinic Proceedings 63, n.º 10 (octubre de 1988): 1061–62. http://dx.doi.org/10.1016/s0025-6196(12)64931-3.

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21

Prasad, Rajendra, Frédéric Devaux, Sanjiveeni Dhamgaye y Dibyendu Banerjee. "Response of pathogenic and non-pathogenic yeasts to steroids". Journal of Steroid Biochemistry and Molecular Biology 129, n.º 1-2 (marzo de 2012): 61–69. http://dx.doi.org/10.1016/j.jsbmb.2010.11.011.

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22

Hopsu-Havu, V. K., C. E. Sonck y Elvi Tunnela. "Production of Elastase by Pathogenic and Non-Pathogenic Fungi". Mycoses 15, n.º 3 (24 de abril de 2009): 105–10. http://dx.doi.org/10.1111/j.1439-0507.1972.tb01357.x.

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23

Ahearn, Donald G. "Medical Mycology: The Pathogenic Fungi and the Pathogenic Actinomycetes". JAMA: The Journal of the American Medical Association 260, n.º 12 (23 de septiembre de 1988): 1794. http://dx.doi.org/10.1001/jama.1988.03410120140051.

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24

Gaminda, K. A. P. "DEOXYRIBOZYMES IN DETECTION OF PATHOGENIC BACTERIA". Biotechnologia Acta 14, n.º 5 (octubre de 2021): 5–20. http://dx.doi.org/10.15407/biotech14.05.005.

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Aim. The purpose of the review was to analyze the use of DNAzyme biosensors for the detection of pathogens. In the recent years, deoxyribozymes (DNAzymes) have a significant impact as biosensors in diverse fields, from detection of metal ions in the environment to theranostic applications and detection of microorganisms. Although routinely used sophisticated instrumental methods are available to detect pathogenic bacterial contamination, they involve time-consuming, complicated sample pre-treatment and expensive instruments. As an alternative, pathogen-specific DNAzymes have demonstrated a series of advantages: a non-destructive rapid analysis technique with in situ and real-time detection of bacteria with high sensitivity and selectivity. A wide range of pathogen-specific DNAzymes has been developed using colorimetric and fluorescence-based detections for pathogenic bacterial contamination in various samples. The current review summarizes the in vitro selection of pathogen-specific DNAzymes, various strategies utilized in the sensor designs, and their potential use in theranostic applications.
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25

Orlyankin, B. G. y T. I. Aliper. "Porcine pathogenic viruses". "Veterinary Medicine" Journal 23, n.º 01 (enero de 2020): 03–08. http://dx.doi.org/10.30896/0042-4846.2020.23.1.03-08.

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26

Kaper, James B., James P. Nataro y Harry L. T. Mobley. "Pathogenic Escherichia coli". Nature Reviews Microbiology 2, n.º 2 (febrero de 2004): 123–40. http://dx.doi.org/10.1038/nrmicro818.

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27

Parkhill, Julian y Colin Berry. "Relative pathogenic values". Nature 423, n.º 6935 (mayo de 2003): 23–24. http://dx.doi.org/10.1038/423023a.

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28

Suerbaum, Sebastian y Torkel Wadström. "Bacterial pathogenic factors". Current Opinion in Gastroenterology 11 (1995): 11–15. http://dx.doi.org/10.1097/00001574-199501001-00003.

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29

Figura, Natale y Soad Tabaqchali. "Bacterial pathogenic factors". Current Opinion in Gastroenterology 12 (enero de 1996): 11–15. http://dx.doi.org/10.1097/00001574-199601001-00003.

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30

Bisel, Ryan S. y Debra J. Ford. "Diagnosing Pathogenic Eschatology". Communication Studies 59, n.º 4 (21 de noviembre de 2008): 340–54. http://dx.doi.org/10.1080/10510970802467395.

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31

Charlotte, Harrison. "Identifying pathogenic pathways". Nature Reviews Drug Discovery 12, n.º 7 (julio de 2013): 506. http://dx.doi.org/10.1038/nrd4061.

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32

Barešić, Anja y Andrew C. R. Martin. "Compensated pathogenic deviations". BioMolecular Concepts 2, n.º 4 (1 de agosto de 2011): 281–92. http://dx.doi.org/10.1515/bmc.2011.025.

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AbstractDeleterious or ‘disease-associated’ mutations are mutations that lead to disease with high phenotype penetrance: they are inherited in a simple Mendelian manner, or, in the case of cancer, accumulate in somatic cells leading directly to disease. However, in some cases, the amino acid that is substituted resulting in disease is the wild-type native residue in the functionally equivalent protein in another species. Such examples are known as ‘compensated pathogenic deviations’ (CPDs) because, somewhere in the second species, there must be compensatory mutations that allow the protein to function normally despite having a residue which would cause disease in the first species. Depending on the nature of the mutations, compensation can occur in the same protein, or in a different protein with which it interacts. In principle, compensation can be achieved by a single mutation (most probably structurally close to the CPD), or by the cumulative effect of several mutations. Although it is clear that these effects occur in proteins, compensatory mutations are also important in RNA potentially having an impact on disease. As a much simpler molecule, RNA provides an interesting model for understanding mechanisms of compensatory effects, both by looking at naturally occurring RNA molecules and as a means of computational simulation. This review surveys the rather limited literature that has explored these effects. Understanding the nature of CPDs is important in understanding traversal along fitness landscape valleys in evolution. It could also have applications in treating diseases that result from such mutations.
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33

Jauniaux, E. "I158 PATHOGENIC MECHANISMS". International Journal of Gynecology & Obstetrics 119 (octubre de 2012): S199—S200. http://dx.doi.org/10.1016/s0020-7292(12)60188-x.

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34

Angell, E. "Pathogenic waste treatment". Environment International 23, n.º 3 (1997): IX—X. http://dx.doi.org/10.1016/s0160-4120(97)88023-3.

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35

Kaper, James B. "Pathogenic Escherichia coli". International Journal of Medical Microbiology 295, n.º 6-7 (octubre de 2005): 355–56. http://dx.doi.org/10.1016/j.ijmm.2005.06.008.

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36

Fattakhov, R. "Genesis pathogenic ecosystems". Parasitology International 47 (agosto de 1998): 329. http://dx.doi.org/10.1016/s1383-5769(98)80967-7.

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37

Bahcall, Orli G. "Classifying pathogenic variation". Nature Reviews Genetics 16, n.º 3 (18 de febrero de 2015): 131. http://dx.doi.org/10.1038/nrg3915.

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38

Holmes, Edward C. "Virology: Pathogenic passengers". Nature 478, n.º 7369 (octubre de 2011): 319–20. http://dx.doi.org/10.1038/478319a.

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39

Shoenfeld, Yehuda. "Pathogenic natural autoantibodies". Clinical Immunology Newsletter 13, n.º 2-3 (febrero de 1993): 13–19. http://dx.doi.org/10.1016/0197-1859(93)90020-k.

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40

Wackett, Lawrence P. "Plant pathogenic microorganisms". Environmental Microbiology 17, n.º 10 (octubre de 2015): 4143–44. http://dx.doi.org/10.1111/1462-2920.13067.

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41

Kiberstis, P. A. "NEUROSCIENCE: Pathogenic Tangles". Science 287, n.º 5462 (31 de marzo de 2000): 2377f—2377. http://dx.doi.org/10.1126/science.287.5462.2377f.

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42

Voelkner, Nadine. "Managing pathogenic circulation". Security Dialogue 42, n.º 3 (junio de 2011): 239–59. http://dx.doi.org/10.1177/0967010611405393.

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43

Broder, Samuel. "Pathogenic Human Retroviruses". New England Journal of Medicine 318, n.º 4 (28 de enero de 1988): 243–45. http://dx.doi.org/10.1056/nejm198801283180409.

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44

H., G. C. y J. A. von Arx. "Plant Pathogenic Fungi". Mycologia 79, n.º 6 (noviembre de 1987): 919. http://dx.doi.org/10.2307/3807701.

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45

Calam, John. "4 Pathogenic mechanisms". Baillière's Clinical Gastroenterology 9, n.º 3 (septiembre de 1995): 487–506. http://dx.doi.org/10.1016/0950-3528(95)90044-6.

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46

Solbrig, Marylou V. "Human pathogenic arenaviruses". Annals of Neurology 64, n.º 3 (25 de febrero de 2008): 355–56. http://dx.doi.org/10.1002/ana.21350.

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47

van West, Pieter y Gordon W. Beakes. "Animal pathogenic Oomycetes". Fungal Biology 118, n.º 7 (julio de 2014): 525–26. http://dx.doi.org/10.1016/j.funbio.2014.05.004.

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48

Harborne, Jeffrey B. "Plant pathogenic bacteria". Phytochemistry 27, n.º 5 (enero de 1988): 1569–70. http://dx.doi.org/10.1016/0031-9422(88)80251-6.

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49

Filip, Z., D. Kaddu-Mulindwa y G. Milde. "Survival of Some Pathogenic and Facultative Pathogenic Bacteria in Groundwater". Water Science and Technology 20, n.º 3 (1 de marzo de 1988): 227–31. http://dx.doi.org/10.2166/wst.1988.0105.

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In model experiments carried out in the laboratory the survival of bacteria in groundwater kept at 10±l °C was tested. Only two of the tested bacteria species did not survive longer than 10 - 30 days. Escherichia coli, Salmonella typhimurium, Pseudomonas aeruginosa and other pathogenic or facultative pathogenic bacteria survived up to 100 days or even more in ground-water with or without the addition of sand from an aquifer. These results can be of importance for determining groundwater protection zones.
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50

Napalkova, G. M., I. I. Korsakova, N. P. Khrapova, N. N. Piven', L. V. Lomova y T. V. Bulatova. "Differentiation of Pathogenic and Non-Pathogenic Burkholderias Using Rocket Immunoelectrophoresis". Problems of Particularly Dangerous Infections, n.º 4(106) (20 de agosto de 2010): 37–38. http://dx.doi.org/10.21055/0370-1069-2010-4(106)-37-38.

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Demostrated is the possibility to differentiate virulent strains of melioidosis and glanders etiological agents from avirulent ones and closely related microorganisms according to the presence of the antigenic complex 8, using rocket immunoelectrophoresis with the serum containing antibodies to this complex.
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