Literatura académica sobre el tema "Pancreatiti"
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Artículos de revistas sobre el tema "Pancreatiti"
Zakharova, M. A., D. S. Gorin, L. A. Marinova y A. G. Kriger. "X-ray-negative pancreatic dust stones in patients with chronic pancreatiti". Khirurgiya. Zhurnal im. N.I. Pirogova, n.º 12 (2019): 137. http://dx.doi.org/10.17116/hirurgia2019121137.
Texto completoRisse, O., C. Arvieux, J. Abba y C. Létoublon. "Chirurgia delle complicanze delle pancreatiti acute". EMC - Tecniche Chirurgiche Addominale 19, n.º 1 (marzo de 2013): 1–14. http://dx.doi.org/10.1016/s1283-0798(13)63954-6.
Texto completoHadjiangelis, Nicos P. y Doreen J. Addrizzo-Harris. "Cryptic Miliary Tuberculosis With a Prodrome Resembling Pancreatiti". Chest 124, n.º 4 (enero de 2003): 330S. http://dx.doi.org/10.1378/chest.124.4_meetingabstracts.330s.
Texto completoShaikh, Danial H., Ahmed Alemam, Jennifer von Ende, Haider Ghazanfar, Anil Dev y Bhavna Balar. "Ansa Pancreatica, an Uncommon Cause of Acute, Recurrent Pancreatitis". Case Reports in Gastroenterology 15, n.º 2 (1 de julio de 2021): 587–93. http://dx.doi.org/10.1159/000516686.
Texto completoBhattacharyya, B. K., S. Chowdhury, S. Das, S. Mukherjee y D. Bhattacharjee. "Treatment of Pancreatic Exocrine Insufficiency with Enteric Coated Pancreatin Formulations: An Overview". International Journal of Pharmaceutical Sciences and Nanotechnology 6, n.º 3 (30 de noviembre de 2013): 2125–30. http://dx.doi.org/10.37285/ijpsn.2013.6.3.3.
Texto completoAdibelli, Zehra Hilal, Mustafa Adatepe, Cetin Imamoglu, Ozgur Sipahi Esen, Nazif Erkan y Mehmet Yildirim. "Anatomic variations of the pancreatic duct and their relevance with the Cambridge classification system: MRCP findings of 1158 consecutive patients". Radiology and Oncology 50, n.º 4 (1 de diciembre de 2016): 370–77. http://dx.doi.org/10.1515/raon-2016-0041.
Texto completoBragado, M. J., J. I. San Roman, A. González, L. J. García, M. A. López y J. J. Calvo. "Impairment of Intracellular Calcium Homoeostasis in the Exocrine Pancreas after Caerulein-Induced Acute Pancreatitis in the Rat". Clinical Science 91, n.º 3 (1 de septiembre de 1996): 365–69. http://dx.doi.org/10.1042/cs0910365.
Texto completoSommer, Camille Anne y C. Mel Wilcox. "Pancreatico-pericardial fistula as a complication of chronic pancreatitis". F1000Research 3 (29 de enero de 2014): 31. http://dx.doi.org/10.12688/f1000research.3-31.v1.
Texto completoMihai, Catalina, Mariana Floria, Radu Vulpoi, Loredana Nichita, Cristina Cijevschi Prelipcean, Vasile Drug y Viorel Scripcariu. "Pancreatico-Pleural Fistula – from Diagnosis to Management. A Case Report". Journal of Gastrointestinal and Liver Diseases 27, n.º 4 (31 de diciembre de 2018): 465–69. http://dx.doi.org/10.15403/jgld.2014.1121.274.ple.
Texto completoSajika Dighe, Raju Shinde, Sangita Shinde y Mohit Gupte. "A rare case of pancreaticopleural fistula patient presented in surgery OPD". International Journal of Research in Pharmaceutical Sciences 11, SPL4 (21 de diciembre de 2020): 1329–32. http://dx.doi.org/10.26452/ijrps.v11ispl4.4301.
Texto completoTesis sobre el tema "Pancreatiti"
Fernandes, Cátia Conceição da Encarnação. "Clínica médica e cirúrgica em animais de companhia: pancreatite em animais de companhia". Master's thesis, Universidade de Évora, 2015. http://hdl.handle.net/10174/18224.
Texto completoFitzsimmons, Deborah. "Quality of life in pancreatic cancer and chronic pancreatitis". Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326844.
Texto completoMalluta, Éverson Fernando. "Avaliação do pâncreas através da ecoendoscopia em pacientes portadores de Doença de Crohn". Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5147/tde-28012009-151207/.
Texto completoBACKGROUND: Pancreas injury usually is not mentioned as an extra-intestinal manifestation of Crohns disease, but the few available series suggest pancreatic injury in a significant proportion of these patients, ranging from 1.2% to 58% in this series. Endoscopic ultrasound (EUS) presents the advantage of having a much higher sensitivity than the abdominal ultrasound or computadorized tomography in evaluating pancreas abnormalities. Compared to the endoscopic retrograde cholangiopancreatography, EUS has at least an equal sensitivity, with a lower complication rate. AIMS: To evaluate the incidence of pancreatic alterations by means of the EUS in Crohns disease, correlating them with clinical, endoscopic and biochemical data. METHODS: Fifty one patients with Crohns disease, age between 18 and 60 years-old (mean = 38), without previous history of pancreatic disease, diabetes mellitus or alcoholism, were submitted to EUS. The control-group was formed by 20 patients submitted to EUS with no previous history of pancreatic or Crohns disease. Clinical, endoscopic and biochemical data were collected in order to determine possible predictive factors. Eleven variables were analyzed, both in pancreatic parenchyma and ducts. The patients with 3 or more alterations were submitted to magnetic resonance. Pancreatic function was determined using fecal elastase assay in 39 patients. RESULTS: Of the 51 analyzed patients, 56% were female, with mean diagnosis time of seven years (1-25) and Crohns disease Activity Index (CDAI) of 102 (20-419). Two patients (3.9%) presented 4 alterations in the EUS exam, 3 (5.9%) presented 3 alterations, 11 (21.5%) had 2 alterations and 13 (25.5%) had 1 alteration in the EUS, which were statistically significant when compared to the control-group, in whom only 16% presented 1 exam alteration (p<0.001). The parenchymal abnormalities were more common, totalizing 39 findings when compared to 11 ductal abnormalities. The patients with 3 or more alterations in the exam were submitted to magnetic resonance; however, pancreatic lesions were not detected. Four patients (10%) had low fecal elastase measurement, suggestive of exocrine pancreatic insufficiency. None of these patients had significant pancreatic alterations in EUS. The only predictive factor that correlated with the number of alterations in EUS was isolated ileal disease. CONCLUSIONS: Patients with Crohns disease had a higher incidence of pancreatic abnormalities (10% with 3 or more alterations in endoscopic ultrasound) in comparison to the control-group. These alterations were found most frequently in the pancreatic parenchyma, which might explain the lack of correlation with the magnetic resonance, which is more prone to detect duct abnormalities. The only predictive factor to these alterations on EUS was ileal disease
Sampaio, Cristina Cardoso. "Pancreatite alcoólica". Master's thesis, [s.n.], 2014. http://hdl.handle.net/10284/4842.
Texto completoA pancreatite alcoólica é uma das principais complicações do consumo excessivo de álcool. O risco de desenvolver a doença aumenta com a ingestão de doses crescentes de álcool, sugerindo que os efeitos tóxicos estão relacionados com a quantidade de álcool ingerida. No entanto, apenas uma minoria dos alcoólicos desenvolve pancreatite, o que indica a necessidade de um fator desencadeador adicional para iniciar a lesão pancreática clinicamente evidente. A pancreatite alcoólica começa como um processo agudo necroinflamatório/ autodigestivo, progredindo com repetidos episódios de necroinflamação. O seu espectro clínico inclui pancreatite aguda (dor abdominal aguda e elevados níveis séricos das enzimas pancreáticas) e pancreatite crónica (dor abdominal, má digestão e diabetes). Têm sido feitos avanços significativos que fornecem uma visão sobre os mecanismos moleculares da lesão pancreática provocada pelo álcool, principalmente no que diz respeito aos seus efeitos tóxicos sobre as células acinares pancreáticas e recentemente, nas células estreladas pancreáticas (PSCs), que desempenham um papel fundamental na fibrose, caraterística da pancreatite crónica alcoólica. Alcoholic pancreatitis is a major complication of alcohol abuse. The risk of developing pancreatitis increases with increasing doses of alcohol, suggesting that alcohol exerts dose-related toxic effects on the pancreas. However, it is also clear that only a minority of alcoholics develop the disease, indicating that an additional trigger may be required to initiate clinically evident pancreatic injury. Alcoholic pancreatitis is thought to begin as an acute necroinflammatory/autodigestive process in a susceptible individual and to progress with repeated episodes. The clinical spectrum of the disease includes acute pancreatitis (acute abdominal pain and raised sérum levels of pancreatic enzymes) and chronic pancreatitis (abdominal pain, maldigestion, diabetes). Significant advances have been made in recent years that provide an insight into the molecular mechanisms of alcohol-related pancreatic injury, particularly with respect to the direct toxic effects of alcohol on pancreatic acinar cells and on the recently characterized PSCs, with may play a key role in the fibrosis of alcoholic chronic pancreatitis.
Шевченко, Володимир Порфирович, Владимир Порфирьевич Шевченко, Volodymyr Porfyrovych Shevchenko, V. O. Bratushka, Y. I. Sobolev, I. A. Myslovsky, O. V. Kravez et al. "The results of surgical treatment of pancreatic pseudocysts following acute pancreatitis". Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27527.
Texto completoIkuta, Kozo. "Nardilysin inhibits pancreatitis and suppresses pancreatic ductal adenocarcinoma initiation in mice". Kyoto University, 2019. http://hdl.handle.net/2433/242408.
Texto completoWilson, Benjamin Gregg. "Feline pancreatic lipase: purification and validation of a clinically significant radioimmunoassay for the diagnosis of feline pancreatitis". Texas A&M University, 2003. http://hdl.handle.net/1969.1/1581.
Texto completoAbou, Khalil Jad. "Pancreatic fistulas after pancreatico-duodenectomies: are pancreatico-gastrostomies safer than pancreatico-jejunostomies? a quasi-experiment and propensity-score adjusted analysis". Thesis, McGill University, 2014. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=122998.
Texto completoCONTEXTE les fistules pacreatiques (PF) constituent une cause significative de la morbiditée et mortalité subie par les patients qui recoivent une pancreaticoduodenectomie (PD). La technique ideale pour retablir la continuité pancreatico-enterique est inconnue. Il n'est pas donné que les Pancreatico-Gastrostomies (PG) donne moi de PF et de complications post-operatives que les Pancreatico-Jejunostomies(PJ). BUT: Nous avons comparé le profile de complications post-operatoire chez les patients ayant subi une PG or PJ apres PD au Centre Universitaire de Sante McGill. METHODOLOGIE: Nous avons collecté des données pre-operatoires ainsi que les complications post- operatoires pour les patients ayant subi une PD dans notre base de données entre 1999 et 2011 et ayant subi une reconstruction par PG ou PJ. Nous avons performé une regression logistique ajustée pour un" propensity-score" pour identifier l'effet de la technique chirurgicale sur les PF, les delais de la motilitée gastrique (DGE), et les complications totales. nous avons utilisé les classifications ISGPF et Strasberg et Linehan pour PF et la definition ISGPS pour DGE. La morbidité totale a été evaluée par la classification Clavien-Dindo et l'Index Comprehensif de Morbidité (CCI). RESULTATS 23/103 et 20/103 (p=0.49) des patients ont developpé une PF et 74/103 et 55/103 patients ont eu DGE en periode post operatoire dans les groupes PG et PJ respectivement (p=0.02). Le grade Clavien-Dindo des complications n'etait pas different entre les groupes (p=0.29) mais le CCI l'etait (38.4 vs. 31.4 for PG vs. PG respectivement, p=0.02.) l'analyse multivariable ajustée pour le "Propensity-score" n'a pas montré d'effet de la technique chirurgical sur PF (p=0.89), DGE grades B/C (p=0.9) ou CCI (p=0.41) mais il restait un effet sur le DGE de toutes les grades de severité (p=0.012.) CONCLUSION Les patients ayant recu une PG n'avaient pas moins de PF que ceux ayant recu une PJ aprés PD a notre institution; Les deux groupes ont souffert du meme profile de complications, mais le groupe PG avait plus de DGE de toutes grades.
Camargo, Enilton Aparecido. "Caracterização da pancreatite aguda induzida por fosfolipases 'A IND. 2' secretorias". [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308940.
Texto completoTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
Made available in DSpace on 2018-08-10T04:13:39Z (GMT). No. of bitstreams: 1 Camargo_EniltonAparecido_D.pdf: 1739135 bytes, checksum: faa68531f796834af8491c48ada0ccc0 (MD5) Previous issue date: 2007
Resumo: A pancreatite aguda e uma doença inflamatória do pâncreas caracterizada por intensa necrose pancreática e efeitos sistêmicos secundários como lesão pulmonar, os quais são a principal causa da mortalidade observada nessa doença. Ha evidencia de que as fosfolipases A2 (PLA2) tem um importante papel na fisiopatologia da pancreatite aguda. O objetivo deste trabalho foi investigar a capacidade de PLA2s de induzir pancreatite em ratos e os mecanismos envolvidos nesse fenômeno. As seguintes PLA2s foram utilizadas: piratoxina-I (homologo Lys-49 de PLA2 desprovido de atividade catalitica), bothropstoxina-II (homologo Asp-49 de PLA2 com baixa atividade catalítica) e a PLA2 proveniente do veneno de Naja moçambique moçambique (que possui alta atividade catalítica). Ratos Wistar machos (200-250 g) provenientes do CEMIB/UNICAMP foram utilizados. As PLA2s foram injetadas no ducto biliopancreatico de animais anestesiados e apos diferentes tempos experimentais foram avaliados o extravasamento de proteínas plasmáticas no pâncreas, infiltrado de neutrofilos no pâncreas e pulmão e amilase serica. A analise histológica do pâncreas e pulmão também foi realizada em alguns grupos experimentais. Piratoxina-I foi capaz de causar extravasamento de proteínas plasmáticas no pâncreas, infiltrado de neutrofilos no pulmão e os níveis sericos de amilase. Alem disso, a piratoxina-I causou alterações histológicas nos tecidos pancreático (infiltrado de neutrofilos, necrose de células acinares e edema intersticial) e pulmonar (edema intersticial e diminuição do espaço alveolar), que foram mais evidentes nos tempos iniciais da pancreatite (4-12h). Bothropstoxina-II e a PLA2 do veneno de Naja moçambique moçambique, a semelhança da Piratoxina-I, também foram capazes de aumentar o extravasamento de proteínas plasmáticas e o influxo de neutrofilos no tecido pancreático por mecanismos nao relacionados a sua atividade catalítica. Entretanto, o influxo de neutrofilos para o pulmão e o aumento dos níveis sericos de amilase causados por essas PLA2s foi dependente de sua atividade catalítica. As PLA2s também causaram secreção deamilase de acinos pancreáticos isolados, que foi dependente da atividade catalítica dessas enzimas. Adicionalmente, com o objetivo de entender o mecanismo envolvido na pancreatite induzida pela PLA2 de Naja mocambique mocambique animais foram tratados com os seguintes agentes farmacológicos: pentoxifilina (inibidor da sintese de TNF-á), SR140333 (antagonista de receptor NK1), icatibant (antagonista de receptor B2), L-NAME (inibidor não seletivo das NOS), aminoguanidina (inibidor preferencial da NOS induzivel), indometacina (inibidor não seletivo de COX), celecoxib (inibidor seletivo de COX-2), PCA4248 (antagonista dos receptores de PAF) e AA861 (inibidor da 5-lipoxigenase). Em conjunto, nossos dados mostraram que os efeitos locais e secundários são multimediados, envolvendo a participação de bradicinina, substancia P, NO, TNF-á, PAF e metabolitos das COXs. Em conclusão, demonstramos que as PLA2s secretorias são capazes de induzir pancreatite aguda em ratos quando injetadas no ducto biliopancreatico, um quadro caracterizado por efeitos inflamatórios locais e secundários cuja mediação farmacológica envolve vários fatores. Alem disso, a pancreatite aguda induzida pelas PLA2s reproduz algumas alterações observadas na pancreatite aguda em humanos, representando uma nova estratégia de estudo da fisiopatologia da pancreatite aguda
Abstract: Acute pancreatitis is an inflammatory disease of the pancreas that is characterized by intense pancreatic necrosis and remote systemic effects such as the lung injury that is the main cause of death during acute pancreatitis. There are evidences that phospholipases A2 (PLA2) have an important role in the acute pancreatitis pathophysiology. The aim of this work was to investigate the ability of PLA2 to induce acute pancreatitis in rats, and the mechanisms underlying this phenomenon. The following PLA2s were used: piratoxin-I (a Lys-49 PLA2 homologue devoid of catalytic activity), bothropstoxin-II (an Asp-49 PLA2 homologue with low catalytic activity) and PLA2 from Naja mocambique mocambique venom (high catalytic activity). Male Wistar rats (200-250g) provided by CEMIBUNICAMP have been used. The PLA2s were injected into the common bile duct of anesthetized rats, after which pancreatic plasma protein extravasation, pancreatic and lung neutrophil infiltration and serum levels of amylase were measured. Histology of the pancreatic and lung tissue has also been carried out in some experimental group. Piratoxin-I was able to increase the pancreatic plasma protein extravasation, lung neutrophil infiltration and serum amylase levels. In addition, Piratoxin-I caused histological changes in the pancreatic (neutrophil infiltration, areas of acinar cell necrosis and interstitial edema) and lung (interstitial edema and diminuition of alveolar space) tissues, which were more evident in the early periods (4-12h) after the injection. Bothropstoxin-II and PLA2 from Naja mocambique mocambique venom were also able to increase the plasma protein extravasation and neutrophil influx in the pancreatic tissue by mechanisms unrelated to their catalytic activity. However, the remote lung neutrophil influx caused by these PLA2s was shown to depend on their catalytic activity. The enhancement of serum amylase levels was also dependent on the catalytic activity of these enzymes. The PLA2s also caused amylase secretion from isolated pancreatic acini, which was dependent on their catalytic activity. Next, in order to further understand the mechanisms involved in pancreatitis induced by PLA2 Naja mocambique mocambique, animals were treated with the following pharmacological agents: pentoxifylline (TNF-á synthesis inhibitior), SR140333 (NK1receptor antagonist), icatibant (B2 receptor antagonist), L-NAME (non-selective NOS inhibitor), aminoguanidine (preferential inducible NOS inhibitor), indomethacin (nonselective COX inhibitor), celecoxib (selective COX-2 inhibitor), PCA4248 (PAF receptor antagonist) and AA861 (5-lipoxygenase inhibitor). Taken together our data showed that local and remote effects are multimediated, involving the participation of bradykinin, substance P, NO, TNF-á, PAF and COXs metabolites. In conclusion, we have shown that secretory PLA2s are able to induce acute pancreatitis in rats when injected into the common bile duct. Therefore, PLA2-induce acute pancreatitis reproduces some aspects of the human disorder representing a new strategy to study the pathophysiology of acute pancreatitis
Doutorado
Farmacologia
Doutor em Farmacologia
Karjula, H. (Heikki). "Diagnosis, treatment and prophylaxis of pancreatic fistulas in severe necrotizing pancreatitis and the long-term outcome of acute pancreatitis". Doctoral thesis, Oulun yliopisto, 2019. http://urn.fi/urn:isbn:9789526224312.
Texto completoTiivistelmä Akuutti nekrotisoiva haimatulehdus ja erityisesti siihen liittyvä bakteeri-infektio on sairaus, johon liittyy korkea komplikaatio- ja kuolleisuusriski. Tautia usein komplisoi infektion lisäksi nekroosiin liittyvä haimafisteli, joka tekee hoidosta entistä haasteellisemman. Viime aikaisissa tutkimuksissa on käsitelty runsaasti mini-invasiivista nekrosektomiaa, mutta suhteellisen vähän on tutkimuksia nekrotisoivaan haimatulehdukseen liittyvästä fisteliongelmasta. Haimatulehdus-potilaiden pitkäaikaisennuste on myös epäselvä. Tämän väitöskirjatutkimuksen tavoitteena oli selvittää nekrotisoivaan haimatulehdukseen liittyvän haimafistelin yleisyyttä, diagnostiikkaa, ehkäisyä ja hoitoa. Lisäksi tarkasteltiin akuuttiin haimatulehdukseen sairastuneiden potilaiden pitkäaikaisennustetta. Ensimmäisessä osatyössä ilmeni, että kaikille potilaille, joille suoritettiin haiman nekrosektomia kehittyi fisteli ja endoskooppinen transpapillaarinen haimateiden stenttaus (ETPS) osoittautui hyväksi ja turvalliseksi hoidoksi fistelin hoidossa. Toisessa prospektiivisessa randomoidussa kontrolloidussa osatyössä tutkittiin profylaktista haimateiden stenttausta nekrotisoivassa haimatulehduksessa. Tutkimus osoitti, etteivät potilaat hyötyneet stenttauksesta: toimenpiteestä oli enemmän haittaa kuin hyötyä. Tämän tutkimuksen mukaan protetisointia ei suositella tehtäväksi taudin alkuvaiheessa. Kolmannessa osatyössä selvitettiin haiman nekrosektomian jälkeisen haimafistelin diagnosointia. Tutkimustuloksen mukaan haimafistelin osoittamiseksi dreenieritteen amylaasitasoa mittaamalla tarvitaan useita mittauskertoja, koska yksittäisen mittauksen sensitiivisyys on matala. Neljännessä osatyössä analysoitiin Oulun yliopistollisessa sairaalassa 1995–2012 akuutin haimatulehduksen sairastaneiden työikäisten potilaiden pitkäaikaisennustetta ja kuolinsyitä. Noin kymmenen vuoden seurannassa tutkimusryhmän (n = 1 644) kuolleisuus oli yli nelinkertainen verrattuna ikä- ja sukupuolivakioituihin verrokeihin (n = 8 220). Merkittävin kuolleisuutta lisäävä tekijä oli alkoholi. Tutkimuksemme osoitti, että infektoituneen haimanekroosiin liittyvä haimafisteli on huomioitava hoidossa. Varhaisesta profylaktisesta haimateiden protetisoinnista ei tutkimuksessa osoitettu olevan hyötyä. Alkoholin aiheuttaman haimatulehduksen pitkäaikaisennusteen mortaliteetti on korkea johtuen alkoholin käytöstä ja siihen liittyvistä sairauksista
Libros sobre el tema "Pancreatiti"
V, Balakrishnan, Kumar Harish, S. Sudhindran y A. G. Unnikrishnan. Chronic pancreatitis and pancreatic diabetes in India. Editado por Indian Pancreatitis Study Group. Cochin, India]: Indian Pancreatitis Study Group, 2006.
Buscar texto completoG, Beger H., Büchler Markus 1955- y Malfertheiner P. 1950-, eds. Standards in pancreatic surgery. Berlin: Springer-Verlag, 1993.
Buscar texto completoKanojia, Ravi P. Laparoscopic lateral pancreatico-jejunostomy for chronic pancreatitis in children. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7849-4.
Texto completoC, Langley William, ed. Pancreatitis research advances. New York: Nova Biomedical Books, 2007.
Buscar texto completo1952-, Johnson C. D. y Imrie C. W, eds. Pancreatic disease: Towards the year 2000. 2a ed. London: Springer, 1999.
Buscar texto completo1932-, Burns Gerard P. y Bank S, eds. Disorders of the pancreas: Current issues in diagnosis and management. New York: McGraw-Hill, Health Professions Division, 1992.
Buscar texto completoLankisch, P. G. y Peter A. Banks. Pancreatitis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-80320-8.
Texto completoAdams, David B., Peter B. Cotton, Nicholas J. Zyromski y John Windsor, eds. Pancreatitis. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118924907.
Texto completoA, Banks Peter, ed. Pancreatitis. Berlin: Springer, 1998.
Buscar texto completoC, Carter David y Warshaw Andrew L. 1939-, eds. Pancreatitis. Edinburgh: Churchill Livingstone, 1989.
Buscar texto completoCapítulos de libros sobre el tema "Pancreatiti"
Tuan, Le Quan Anh y Pham Minh Hai. "Laparoscopic Internal Drainage of Pancreatic Pseudocysts". En Mastering Endo-Laparoscopic and Thoracoscopic Surgery, 345–48. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3755-2_50.
Texto completoForsmark, Chris E. "Pancreatitis-Related Pancreatic Masses: Chronic Pancreatitis". En Pancreatic Masses, 75–84. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19677-0_6.
Texto completoAl-Salem, Ahmed H. "Pancreatitis and Pancreatic Pseudocyst". En Atlas of Pediatric Surgery, 221–27. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29211-9_25.
Texto completoArshad, Ali y Ashley Dennison. "Acute Pancreatitis, Chronic Pancreatitis and Pancreatic Neoplasms". En Gastroenterology For General Surgeons, 103–17. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-92768-8_10.
Texto completoCarter, D. C. "Pancreatico-duodenectomy for Chronic Pancreatitis". En Aktuelle Pankreaschirurgie, 149–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75265-0_21.
Texto completoBadalov, Nison y Scott Tenner. "Chronic Pancreatitis and Pancreatic Pseudocysts". En Practical Gastroenterology and Hepatology: Small and Large Intestine and Pancreas, 420–26. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444328417.ch57.
Texto completoGuarise, Alessandro, Niccolò Faccioli, Giovanni Morana y Alec J. Megibow. "Chronic Pancreatitis vs Pancreatic Tumors". En Imaging of the Pancreas, 329–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-68251-6_18.
Texto completoSunderland, G. T. y C. W. Imrie. "Pancreatic Fistulas in Acute Pancreatitis". En Pancreatic Fistulas, 61–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77418-8_4.
Texto completoVesentini, S. "Pancreatic Fistulas in Chronic Pancreatitis". En Pancreatic Fistulas, 70–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77418-8_5.
Texto completoHindy, Pierre y Scott Tenner. "Chronic Pancreatitis and Pancreatic Pseudocysts". En Practical Gastroenterology and Hepatology Board Review Toolkit, 378–82. Oxford, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781119127437.ch63.
Texto completoActas de conferencias sobre el tema "Pancreatiti"
Hasanova, S. Y. k. "To the question of the treatment of pancreatic necrosis". En General question of world science. Наука России, 2021. http://dx.doi.org/10.18411/gq-31-07-2021-06.
Texto completoThomas, David, Emma Bermingham, Mark Roberts y Wayne Young. "An investigation into the effect of high fat and carbohydrate diets on a range of biomarkers associated with pancreatitis in dogs". En 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/uvdt4784.
Texto completoClark, K. y K. E. Gross. "Pancreatico-Pleural Fistula and Pancreatico-Pericardial Fistula: Unusual Complications of Pancreatitis". En American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6439.
Texto completoMARINGHINI, A., R. PATTI, A. TERMINI, M. CIAMBRA y P. BIFFARELLA. "PANCREATIC FUNCTION TESTS IN DIAGNOSIS OF CHRONIC PANCREATITIS". En Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0012.
Texto completoBudzinskiy, S., S. Shapovalianz, E. Fedorov, M. Zakharova y E. Platonova. "ENDOSCOPIC PANCREATIC DUCT STENTING IN MANAGEMENT OF CHRONIC PANCREATITIS". En ESGE Days. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1704663.
Texto completoRuud, T. E., W. Müller-Esterl, H. Fritz, J. O. Stadaas y A. O. Aasen. "RELATIONS BETWEEN APROTININ CONCENTRATIONS AND HEMODYNAMICS IN EXPERIMENTAL ACUTE PANCREATITIS". En XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644333.
Texto completoMalinka, T., B. Globke, L. Timmermann, F. Klein, J. Pratschke y M. Bahra. "Necrotising pancreatitis of the pancreatic remnant (NECROREMNANTITIS): A new definition of severe pancreatitis after pancreatectomy". En Viszeralmedizin 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1695503.
Texto completoBOCKMAN, DALE E. "PATHOLOGY OF CHRONIC PANCREATITIS: RELATIONSHIP TO PANCREATIC ANATOMY AND NERVES". En Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0001.
Texto completoLAYER, PETER y JUTTA KELLER. "HUMAN EXOCRINE PANCREATIC SECRETION IN NORMAL PANCREAS AND CHRONIC PANCREATITIS". En Proceedings of the 92nd Course of the International School of Medical Sciences. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814447249_0010.
Texto completoLi, Lisha, Heping Wang y Yulin Li. "Network Analysis Reveals the Connection Between Chronic Pancreatitis and Pancreatic Cancer". En International Conference on Biomedical and Biological Engineering. Paris, France: Atlantis Press, 2016. http://dx.doi.org/10.2991/bbe-16.2016.59.
Texto completoInformes sobre el tema "Pancreatiti"
Research, Gratis. Gallstone Pancreatitis. Gratis Research, enero de 2020. http://dx.doi.org/10.47496/gr.blog.08.
Texto completoAlseidi, Adnan. Pancreatic Anatomy. Touch Surgery Simulations, 2018. http://dx.doi.org/10.18556/touchsurgery/2018.s0111.
Texto completoJayasinghe, Ravindri, Sonali Ranasinghe, Chandrani Kuruppu, Umesh Jayarajah y Sanjeewa Seneviratne. Clinical characteristics and outcomes of acute pancreatitis following spinal surgery: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, julio de 2022. http://dx.doi.org/10.37766/inplasy2022.7.0017.
Texto completoSahin-Toth, Miklos. Mouse Model of Human Hereditary Pancreatitis. Fort Belvoir, VA: Defense Technical Information Center, septiembre de 2015. http://dx.doi.org/10.21236/ada624309.
Texto completoMiller, George. Divergent Effects of Dendritic Cells on Pancreatitis. Fort Belvoir, VA: Defense Technical Information Center, septiembre de 2015. http://dx.doi.org/10.21236/ada624310.
Texto completoBeatty, Gregory, Patrick Guirnalda y Santiago L. Luque. Listeria Vaccines for Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, octubre de 2013. http://dx.doi.org/10.21236/ada600605.
Texto completoLu, Lei, Hangbin Jin, Jing Yang, Jianfeng Yang y Xiaofeng Zhang. Early versus delayed cholecystectomy for mild gallstone pancreatitis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, abril de 2022. http://dx.doi.org/10.37766/inplasy2022.4.0033.
Texto completoWang, Lei. Molecular Probes for Pancreatic Cancer Imaging. Portland State University Library, enero de 2000. http://dx.doi.org/10.15760/etd.3105.
Texto completoYu, David. The Replication Stress Response in Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, octubre de 2013. http://dx.doi.org/10.21236/ada599228.
Texto completoYu, Davis S. The Replication Stress Response in Pancreatic Cancer. Fort Belvoir, VA: Defense Technical Information Center, diciembre de 2014. http://dx.doi.org/10.21236/ada621841.
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