Literatura académica sobre el tema "NMSC"
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Artículos de revistas sobre el tema "NMSC"
Weston, Andrew, Marie Hughes, Gillian Corbett, Neil Graham y Franz Strydom. "Incidence of Non-Melanoma Skin Cancers in Patients with Chronic Lymphocytic Leukaemia: A Retrospective Study in a Bay of Plenty, New Zealand Population". Blood 138, Supplement 1 (5 de noviembre de 2021): 4692. http://dx.doi.org/10.1182/blood-2021-147766.
Texto completoBislimi Berisha, Reihane, Djordje Dzokic y Shkendije Dobruna. "Evaluation of Pre-operative Dermoscopy in Early Diagnosis of Non-melanocytic Skin Cancer". Open Access Macedonian Journal of Medical Sciences 9, B (2 de diciembre de 2021): 1660–63. http://dx.doi.org/10.3889/oamjms.2021.7539.
Texto completoAntonijevic, Aleksandar, Natasa Rancic, Mirko Ilic, Biljana Kocic, Jasmina Stevanovic y Marija Milic. "Trends in incidence of non-melanoma and melanoma skin cancers in central Serbia". Srpski arhiv za celokupno lekarstvo 146, n.º 7-8 (2018): 391–95. http://dx.doi.org/10.2298/sarh161121002a.
Texto completoHu, Dailun, Philip K. Nicholls, Changfu Yin, Khama Kelman, Qionglan Yuan, Wayne K. Greene, Zhongli Shi y Bin Ma. "Immunofluorescent Localization of Non-myelinating Schwann Cells and Their Interactions With Immune Cells in Mouse Thymus". Journal of Histochemistry & Cytochemistry 66, n.º 11 (18 de mayo de 2018): 775–85. http://dx.doi.org/10.1369/0022155418778543.
Texto completoGhahartars, Mehdi, Shiva Najafzadeh, Shabnam Abtahi, Mohammad Javad Fattahi y Abbas Ghaderi. "Investigation of Interleukin-27 in the Sera of Nonmelanoma Skin Cancer Patients". Dermatology Research and Practice 2018 (19 de noviembre de 2018): 1–5. http://dx.doi.org/10.1155/2018/8321302.
Texto completoMøllersen, Kajsa, Herbert Kirchesch, Maciel Zortea, Thomas R. Schopf, Kristian Hindberg y Fred Godtliebsen. "Computer-Aided Decision Support for Melanoma Detection Applied on Melanocytic and Nonmelanocytic Skin Lesions: A Comparison of Two Systems Based on Automatic Analysis of Dermoscopic Images". BioMed Research International 2015 (2015): 1–8. http://dx.doi.org/10.1155/2015/579282.
Texto completoMistry, Nisha, Zenaida Abanto, Chris Bajdik y Jason K. Rivers. "Demographic and Tumor Characteristics of Patients Diagnosed with Nonmelanoma Skin Cancer: 13-Year Retrospective Study". Journal of Cutaneous Medicine and Surgery 16, n.º 1 (enero de 2012): 32–38. http://dx.doi.org/10.1177/120347541201600107.
Texto completoDacosta Byfield, Stacey, Diana Chen, Yeun Mi Yim y Carolina Reyes. "Age distribution of patients with advanced non-melanoma skin cancer in the United States". Archives of Dermatological Research 305, n.º 9 (21 de abril de 2013): 845–50. http://dx.doi.org/10.1007/s00403-013-1357-2.
Texto completoPerkins, Joanna L., Yan Liu, Pauline A. Mitby, Joseph P. Neglia, Sue Hammond, Marilyn Stovall, Anna T. Meadows et al. "Nonmelanoma Skin Cancer in Survivors of Childhood and Adolescent Cancer: A Report From the Childhood Cancer Survivor Study". Journal of Clinical Oncology 23, n.º 16 (1 de junio de 2005): 3733–41. http://dx.doi.org/10.1200/jco.2005.06.237.
Texto completoBorik-Heil, Liliane, Georg Endler, Walther Parson, Andreas Zuckermann, Lisa Schnaller, Keziban Uyanik-Ünal, Peter Jaksch et al. "Cumulative UV Exposure or a Modified SCINEXA™-Skin Aging Score Do Not Play a Substantial Role in Predicting the Risk of Developing Keratinocyte Cancers after Solid Organ Transplantation—A Case Control Study". Cancers 15, n.º 3 (30 de enero de 2023): 864. http://dx.doi.org/10.3390/cancers15030864.
Texto completoTesis sobre el tema "NMSC"
Fraser, Tyler Malcolm. "Molecular Dynamics of p21 and Fluorescent Sphingomyelin in Keratinocytes Exposed to UVB". DigitalCommons@CalPoly, 2018. https://digitalcommons.calpoly.edu/theses/1985.
Texto completoHill, Natasha Tremayne. "Vitamin D receptor and 1alpha, 25-dihydroxyvitamin D3 mediated regulation of DeltaNp63alpha". Wright State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=wright1450456950.
Texto completoOnder, Zeynep. "Characterization of the Nucleocytoplasmic Transport of the Cutaneous HPV8 E7 Protein". Thesis, Boston College, 2014. http://hdl.handle.net/2345/3828.
Texto completoSome non melanoma skin cancers (NMSC) have been associated with human papillomavirus (HPV) pathogenesis, like epidermodysplasia verruciformis (EV) and squamous cell carcinoma (SCC). EV is a genetically inherited skin disease that develops when the individuals are infected with cutaneous HPV types belonging to the β-genus, especially types 5 and 8. Transgenic mouse lineages expressing all early genes of cutaneous HPV8 develop papillomas, dysplasias and SCC after UV irradiation and this correlates with enhanced HPV8 oncogenes expression. We have previously discovered that the nuclear localization of mucosal HPV16 E7 and HPV11 E7 proteins is mediated by their zinc-binding domain via a Ran-dependent pathway and independent of nuclear import receptors and that a patch of hydrophobic residues within the zinc-binding domain of HPV16 E7 and HPV11 E7 proteins is responsible for their nuclear import via hydrophobic interactions with FG nucleoporins. Here we investigated the nucleocytoplasmic traffic of cutaneous HPV8 E7 protein using confocal microscopy to analyze the intracellular localization of EGFP-8E7, its subdomains and its mutants after transient transfections. We also investigated the nuclear import ability of GST-8E7, its subdomains and mutants using in vitro nuclear import assays in digitonin-permeabilized HeLa cells. In addition, we performed isolation assays to study the direct interaction between HPV8 E7 and two FG nucleoporins, Nup62 and Nup153 or the nuclear export receptor, CRM1. We found that the nuclear import of cutaneous HPV8 E7 is mediated by a nuclear localization signal (NLS) located within its zinc-binding domain. Furthermore, we determined that the hydrophobic residues within the 65LRLFV69 patch are responsible for the nuclear import and nuclear localization of HPV8 E7 via direct hydrophobic interactions with FG nucleoporins, Nup62 and Nup153, whereas the positively charged arginine 66 plays no significant role in the function of the NLS. In addition, we examined the nuclear export mechanism of cutaneous HPV8 E7 protein and showed that it has a leucine-rich nuclear export signal (NES) in its C-terminal domain that is recognized by the CRM1 nuclear export receptor. These studies are essential for understanding the nucleocytoplasmic traffic of cutaneous HPV8 E7 protein
Thesis (PhD) — Boston College, 2014
Submitted to: Boston College. Graduate School of Arts and Sciences
Discipline: Biology
Kandell, Rebecca Marie. "Assessing the Photoprotective Effects of Fluorescent Sphingomyelin Against UVB Induced DNA Damage in Human Keratinocytes". DigitalCommons@CalPoly, 2018. https://digitalcommons.calpoly.edu/theses/1872.
Texto completoAlshammari, Eid Salem. "∆Np63α Positively Regulates ERK3 Expression in Non-Melanoma Skin Cancer". Wright State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1556229754222487.
Texto completoCERAOLO, MARIA GRAZIA. "DEREGULATION OF UV-ACTIVATED INFLAMMASOME BY HPV 38 IN HUMAN KERATINOCYTES". Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/543118.
Texto completoNon-melanoma skin cancers (NMSC) are the most common human malignancies occurring in fair-skinned adult population, representing approximately 30% of total cancer. The etiology of this cancer is multifactorial, since genotypic, phenotypic and/or environmental factors can be involved in its development. Several studies have reported that the exposure to ultraviolet irradiation (UVB) represents a key risk factor in the development of skin cancer. Furthermore, infectious agents could be an additional risk factor and this hypothesis is supported by the evidence that immunocompromised people, e.g. organ transplant recipients (OTRs), show a higher risk of developing NMSC compared with immunocompetent individuals. Several infectious agents are able to colonize the skin and especially a subgroup of beta cutaneous human papillomavirus (HPV) is considered the most likely additional etiological factors of NMSC. In particular, the presence of β-HPV has been reported in patients with a rare cell-mediated immunity disorder, called epidermodysplasia verruciformis (EV), which causes high rates of susceptibility to develop skin squamous cell carcinoma (SCC) on sun-exposed areas. Thus, it provided the initial clues about infectious agents, such as β HPV, as potential additional risk factor for NMSC. Several studies have demonstrated the transforming properties of the viral oncoproteins, E6 and E7, in in vitro and in vivo experimental models. Both proteins, altering the functions of tumour suppressor gene products, e.g. p53 and retinoblastoma (pRb), are able to deregulate key cellular events, such as cell cycle, DNA repair, apoptosis and senescence. Based on these in vitro results, our group has generated a transgenic (Tg) mouse model that express E6 and E7 oncogenes from beta HPV38 in the basal level of the skin and mucosal epithelia. Those mice were highly susceptible to UV-radiation; in fact, the exposure of HPV38 E6/E7 Tg mice to UVB caused the development of actinic keratosis-like lesions that are considered as precursors of SCC in humans and a few week later SCC. Conversely, the wild-type mice did not develop any type of skin lesions when exposed to the same dose of UV radiations. However, the mechanism of the UV/HPV38 cooperation has not been elucidated yet. The aim of this work thesis was to dissect the molecular mechanisms of such cooperation. We have first determined whether the expression of the viral oncogenes in the Tg animals alters the pattern of gene expression induced by UV radiation. The analysis of the entire transcriptome pointed out that more than 300 genes are deregulated in the Tg mice after UVB exposure, compared to the wild type animals. In particular, the HPV38 E6 and E7 oncoproteins were able to down regulate the expression of proteins involved in the inflammasome complexes or the downstream effectors, such as IL-18. Interestingly, many studies in keratinocyte cell lines provided evidence that UV radiation activate the inflammasome pathway, leading the secretion of specific pro-inflammatory cytokines, like IL-1β and IL-18. RT-qPCR experiments confirmed the transcriptome findings, showing that IL-18 expression is up-regulated in wild-type mice after UV exposure, while it is strongly inhibited in HPV38 E6/E7 Tg mice. Using primary human keratinocytes (HPKs) as experimental models, we dissected the mechanisms involved in the UV-induced IL-18 expression as well as the role of HPV38 in the deregulation of these events. RT-qPCR analysis showed an UV-induced upregulation of IL-18 mRNA expression as well as some key inflammasome components, like AIM2 and ASC, in HPKs cells; conversely, the presence of E6 and E7 oncoproteins strongly down regulated IL-18 both at the expression and secretion levels. Using another in vitro experimental model, such as keratinocytes expressing the human telomerase reverse transcriptase gene (hTERT), in order to prolong the life span of the cells, we found that this cell line presents a higher basal level of IL-18 compared to HPKs and the presence of E6 and E7 plays an inhibitory role. Transient transfection experiments in hTERT HPKs using a vector containing the isolated IL-18 promoter cloned in front the luciferase reporter gene confirmed the ability of HPV38 E6 and E7 to repress the IL-18 promoter activity. The analysis of IL-18 promoter by TFBIND bioinformatics tool revealed the presence of 15 putative binding sites for p53, which is well known to be activated by UV radiation. Performing chromatin immunoprecipitation assays (CHIP), we showed that p53 is recruited to two distinct regions of the IL-18 promoters that includes four p53 responsive elements. Importantly, HPV38 E6 and E7 efficiently prevent p53 recruitment to these IL-18 promoter regions. Together these results corroborated our hypothesis that beta HPV38 play an important role in the inhibition UV-induce inflammasome response, by altering the gene expression and the production of proteins involved in the inflammasome pathways as well as of specific cytokines (e.g. IL-18). Most likely, the down regulation of IL-18 may impair the ability of the keratinocytes to recruit the immune cell population at the UV-exposed area, causing a defect in the elimination of cells harboring DNA damage. In conclusion, all these alterations may lead to the accumulation of UV-induced DNA damage and development of non-melanoma skin cancer.
Norburn, Jill. "NATIONAL MERIT FINALISTS AT THE UNIVERSITY OF CENTRAL FLORIDA-TRENDS, ATTRITION, AND RETENTION1997-2005". Doctoral diss., University of Central Florida, 2006. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/4069.
Texto completoEd.D.
Department of Educational Research, Technology and Leadership
Education
Educational Leadership
Westphal, Kathi. "Molekulare Mechanismen kutaner humaner Papillomviren (HPV) während der Hautkarzinogenese". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2009. http://dx.doi.org/10.18452/15998.
Texto completoIn the last years epidemiologic and molecular biological studies accumulated increasing evidence that cutaneous human papillomaviruses are etiologically involved in the formation of non-melanoma skin cancer (NMSC). The presented work aims to identify the underlying molecular mechanisms of the viral proteins E6 and E7 of cutaneous HPV types. The E6 and E7 genes of the different HPV types 1, 4, 5, 8, 20, and RTRX7, which are in vivo associated with cutaneous benign or malignant lesions, were studied. Natural host cells of these viruses, human primary keratinocyts (HPK) of the skin, were infected with recombinant E6 and E7 encoding retroviruses. The following analyses were performed in monolayer culture (non-differentiated keratinocytes) or in organotypic skin culture (induction of keratinocyte differentiation). The expression of E6 and E7 elongated the life span of monolayer HPK and significantly increased the doubling rate. An activation of the telomerase, characteristic for immortalized cells, was only detected in HPV 8 E6 positive cells. In organotypic skin cultures E7 of HPV 1, 4 and 38 induced drastic changes in differentiation and proliferation. Additionally an impairment of the normal cell cycle control in suprabasale HPV 5 E7 and 8 E7 cultures was seen. Hints for a strong invasive potential of E7 infected HPK were proven for HPV 8 E7 and expanded to HPV 4 E7, HPV 38 E7 and RTRX E7. The viral E6 and E7 genes of cutaneous and mucosal HPV types exhibit different molecular mechanisms. The multistep model of carcinogenesis includes a series of fundamental cell transformations necessary for tumorigenesis. Mechanisms for the modulation of cell differentiation and proliferation by cutaneous HPV types 4, 5, 8 and 38 described in this work could potentially contribute to the induction and progression of early stages of squamous cell carcinoma.
Barra, Tiago Bruno Areal. "O papel formativo do Movimento Nacional dos Meninos e Meninas de Rua (MNMMR) na comunidade do Lagamar atravÃs da perspectiva dos participantes: uma experiÃncia de construÃÃo da resiliÃncia e empoderamento". Universidade Federal do CearÃ, 2015. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14035.
Texto completoA sociedade tem passado por um conjunto de transformaÃÃes em sua estrutura. Um dos aspectos mais interessantes dessa transformaÃÃo à observar como a juventude passa por tais mudanÃas de maneira rÃpida, mas nem sempre guiada pelo aspecto qualitativo desse processo. A juventude, principalmente da periferia, està sendo vÃtima de uma ordem social que se estrutura na Ãtica da desumanizaÃÃo. Tendo como base essa premissa, a presente pesquisa tem como objetivo compreender o papel formativo do Movimento Nacional dos Meninos e Meninas de Rua (MNMMR) na Comunidade do Lagamar atravÃs da perspectiva dos participantes, observando esse processo dentro de uma experiÃncia de construÃÃo da resiliÃncia e do empoderamento. Para compreender esse processo formativo (MACEDO, 2010), utilizei da construÃÃo das histÃrias de vida (DELORY-MOMBERGER, 2008; FERRAROTTI, 1996; NÃVOA & FINGER, 2010; PINEAU, 2006, 2012), de acordo com os procedimentos da entrevista narrativa (JOVCHELOVITCH & BAUER, 2002) buscando compor traÃos identitÃrios importantes do MNMMR do Cearà que acaba por completar 30 anos de histÃria em 2015. A categoria da resiliÃncia (ANTUNES, 2011; ASSIS, 2006; CYRULNIK, 2004, 2012; MELILLO & OJEDA, 2005; POLETTI, 2013; YUNES, 2003) e do empoderamento forjado no processo de lutas e conquistas sociais (FREIRE, 1983, 1986, 2006; LOBO, 2011; SAWAIA, 2008) ajudam a compreender esse processo de exclusÃo social e de tomada de consciÃncia crÃtica frente Ãs adversidades sociais. Nas histÃrias de vida, fica evidenciado a importÃncia social do MNMMR, tornando-se um movimento social importante para a Comunidade do Lagamar por atuar com os sujeitos em situaÃÃo de rua. A relevÃncia mostra que as dimensÃes de resiliÃncia e do empoderamento, podem servir de significados para a melhoria de uma formaÃÃo humana propiciada pelo prÃprio MNMMR. O presente trabalho investigativo traz à tona um diÃlogo formativo, que contempla tambÃm um processo educativo, uma aÃÃo educativa construÃda no seio da rua, tendo como protagonista a juventude da periferia da cidade de Fortaleza.
Dang-Heine, Chantip. "Genexpressionsprofil und Aktivität humaner Papillomviren in nicht-melanozytären Hauttumoren". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2010. http://dx.doi.org/10.18452/16152.
Texto completoDuring development of non-melanoma skin cancer, several risk factors are involved: UV-exposition, pigmentation, age, and potentially human papilloma virus (HPV). The molecular mechanisms underlying tumourgenesis in squamous cell carcinoma (SCC) and its pre-cancerosis actinic keratosis (AK) are not fully understood. In this study, the gene expression profile and HPV-infection status were analysed in SCC from immunocompetent and organ transplanted, immunocompromised patients.By global transcriptome analysis from cutaneous SCC, AK and healthy skin, 118 genes were identified differentially expressed in a cDNA-microarray. The expression of 11 out of 13 selected genes (85%) was investigated by real-time RT-PCR (qPCR) and the expression of three genes remarkably induced in SCC correlated with the progression to AK until SCC. These genes encoded for Metalloproteinase-1, which is involved in the remodelling of extracellular matrix, and the protooncogene RAB31 and Tenascin-C (Tn-C). Tn-C protein is expressed in SCC-tissue at the invasion front in basal cells and in keratinocytes in the Stratum papillare and retikulare, but not in healthy skin. This study, the 2243 bp Tn-C-specific splice-variant has for the first time detected in SCC, but not in normal skin. Thus it might serve as diagnostic marker of SCC progression. The data of the transcriptome analysis indicates that a simultaneous dysregulation of oncogene expression and DNA-repair, cell-cycle and proliferation, proteolysis and adhesion molecules exists in SCC. Additionally, the expression of HPV in SCC and thus the causal relationship between HPV-infection and tumourgenesis of SCC in immunocompromised patients was investigated. The HPV-infection pattern in SCC-tissue and normal skin was assessed by detection of DNA from cutaneous HPV-types. Viral E6/E7-mRNA-transcripts of the cutaneous HPV-types 8, 9, 15 were expressed selectively in AK and SCC. In contrast, no HPV-specific mRNA was present in HPV-DNA positive normal skin. The analysis of the open reading frame from the respective E6-protein genes unravelled one single pointmutation, which is not been characterized so far in terms of e.g. its impact on protein structure. The viral activity of the oncogenes E6 and E7 of cutaneous HPV-types indicates a potential function of HPV in the tumourgenesis of SCC in immunocompromised individuals.
Libros sobre el tema "NMSC"
Tibbles, April. NMSA curriculum guidelines. Columbus, Ohio (4807 Evanswood Dr., Columbus 43229-6292): National Middle School Association, 1991.
Buscar texto completoNMS medicine. 7a ed. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2012.
Buscar texto completoD, Wolfsthal Susan, ed. NMS medicine. 6a ed. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins, 2008.
Buscar texto completoNMS medicine casebook. Philadelphia, PA: Lippincott Williams & Wilkins, 2009.
Buscar texto completoPfeifer, Samantha M. NMS obstetrics and gynecology. 7a ed. Philadelphia: Wolters Kluwer/Lippincott Williams & Wilkins Health, 2011.
Buscar texto completoFilomena, Moscatelli, ed. James Turrell. Milano: Charta, 2009.
Buscar texto completoJames, Turrell. James Turrell. Editado por Moscatelli Filomena. Milano: Charta, 2009.
Buscar texto completoCouncil, Nursing and Midwifery. Professional advice from the NMC. London: Nursing and Midwifery Council, 2002.
Buscar texto completoConference, New Media Consortium Summer. 2007 NMC summer conference proceedings. Austin, Tex: New Media Consortium, 2007.
Buscar texto completoSteve, Merrett y Rignall Julian, eds. NMS: The complete games guide. (London): EMAP Images, 1993.
Buscar texto completoCapítulos de libros sobre el tema "NMSC"
Knapp, F. F. y Ashutosh Dash. "Locoregional Radionuclide Therapy for Nonmelanoma Skin Cancer (NMSC)". En Radiopharmaceuticals for Therapy, 253–64. New Delhi: Springer India, 2016. http://dx.doi.org/10.1007/978-81-322-2607-9_13.
Texto completoGrüne, Lars y Jürgen Pannek. "Economic NMPC". En Nonlinear Model Predictive Control, 221–58. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46024-6_8.
Texto completoGrüne, Lars y Jürgen Pannek. "Distributed NMPC". En Nonlinear Model Predictive Control, 259–95. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46024-6_9.
Texto completoAndrews, Anne M., Greg A. Gerhardt, Lynette C. Daws, Mohammed Shoaib, Barbara J. Mason, Charles J. Heyser, Luis De Lecea et al. "NMS". En Encyclopedia of Psychopharmacology, 901. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_4419.
Texto completoFroehlich, Stephan J., Carlo A. Lackerbauer, Guenter Rudolph, Jan Rémi, Soheyl Noachtar, Werner J. Heppt, Annette Cryer et al. "NMS". En Encyclopedia of Molecular Mechanisms of Disease, 1485. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7515.
Texto completoGrancharova, Alexandra y Tor Arne Johansen. "Explicit Stochastic NMPC". En Explicit Nonlinear Model Predictive Control, 157–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28780-0_7.
Texto completoGrancharova, Alexandra y Tor Arne Johansen. "Semi-explicit Distributed NMPC". En Explicit Nonlinear Model Predictive Control, 209–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-28780-0_9.
Texto completoGuo, Jian, Yu Sasaki, Lei Wang y Shuang Wu. "Cryptanalysis of HMAC/NMAC-Whirlpool". En Advances in Cryptology - ASIACRYPT 2013, 21–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-42045-0_2.
Texto completoChen, Tinghuai. "NMRC: A Technique for Redundancy". En Fault Diagnosis and Fault Tolerance, 119–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77179-8_4.
Texto completoVon Unold, P. "NMS, Nitrate Monitoring System". En Field Screening Europe 2001, 363–66. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-010-0564-7_62.
Texto completoActas de conferencias sobre el tema "NMSC"
Lamont, Sarah M., Kyungmann Kim, Thomas Havighurst, Eneida Mendonça, Gary S. Wood, Stephen Snow y Howard H. Bailey. "Abstract A52: A phase III skin cancer chemoprevention study of DFMO in subjects with a history of non-melanoma skin cancer (NMSC): Follow-up of NMSC events greater than 5 years post-study participation". En Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a52.
Texto completoGeweid, Gamal G. N., Fartash Vasefi y Kouhyar Tavakolian. "A Novel Nonparametric Technique for Segmenting Multimode Hyperspectral Images Obtained From Non-Melanoma Skin Cancer Lesions". En 2020 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/dmd2020-9045.
Texto completoListik, Clarice, Rubens Gisbert Cury, Sara Carvalho Barbosa Casagrande, Eduardo Listik, Debora Arnaut, Natally Santiago, Júlia Machado et al. "Improvement of non-motor symptoms and quality of life after DBS stimulation for dystonia: one-year follow-up". En XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.253.
Texto completoSaeidpourazar, Reza y Nader Jalili. "Forced-Controlled Nanomanipulation Utilizing Nano-Robotic Manipulator and Nanomechanical Cantilever". En ASME 2008 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. ASMEDC, 2008. http://dx.doi.org/10.1115/smasis2008-629.
Texto completoCowie, Jim. "CRL/NMSU". En the 6th conference. Morristown, NJ, USA: Association for Computational Linguistics, 1995. http://dx.doi.org/10.3115/1072399.1072414.
Texto completoTomsic, Alejandro Z., Tyler Crain y Marc Shapiro. "PhysiCS-NMSI". En EuroSys '16: Eleventh EuroSys Conference 2016. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2911151.2911166.
Texto completoYebi, Adamu, Bin Xu, Xiaobing Liu, John Shutty, Paul Anschel, Simona Onori, Zoran Filipi y Mark Hoffman. "Nonlinear Model Predictive Control Strategies for a Parallel Evaporator Diesel Engine Waste Heat Recovery System". En ASME 2016 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/dscc2016-9801.
Texto completoYu, Shuangcheng, Yichi Zhang, Chen Wang, Won-kyu Lee, Biqin Dong, Teri W. Odom, Cheng Sun y Wei Chen. "Characterization and Design of Functional Quasi-Random Nanostructured Materials Using Spectral Density Function". En ASME 2016 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. American Society of Mechanical Engineers, 2016. http://dx.doi.org/10.1115/detc2016-60118.
Texto completo"NMDC 2019 Program". En 2019 IEEE 14th Nanotechnology Materials and Devices Conference (NMDC). IEEE, 2019. http://dx.doi.org/10.1109/nmdc47361.2019.9084022.
Texto completo"NMDC 2019 Committee". En 2019 IEEE 14th Nanotechnology Materials and Devices Conference (NMDC). IEEE, 2019. http://dx.doi.org/10.1109/nmdc47361.2019.9084001.
Texto completoInformes sobre el tema "NMSC"
Pope, Noah Gale y Christopher Hobbs. NMAC case studies: NMAC discovers leak of radioactive liquid at THORP, Sellafield Plant, UK. Office of Scientific and Technical Information (OSTI), septiembre de 2015. http://dx.doi.org/10.2172/1215824.
Texto completoKarpius, Peter Joseph. NDA & NMAC at LANL Overview. Office of Scientific and Technical Information (OSTI), agosto de 2019. http://dx.doi.org/10.2172/1558957.
Texto completoBoring, Ronald y David Gertman. Human reliability analysis for NMSS P-406. Office of Scientific and Technical Information (OSTI), mayo de 2011. http://dx.doi.org/10.2172/1467674.
Texto completoBecker, Robert C. NMMC Picture Archiving and Communication Systems (PACS). Fort Belvoir, VA: Defense Technical Information Center, abril de 2007. http://dx.doi.org/10.21236/ada503753.
Texto completoWest, Rebecca Lynn. Nuclear Security Objectives of an NMAC System. Office of Scientific and Technical Information (OSTI), enero de 2018. http://dx.doi.org/10.2172/1416266.
Texto completoOrlando, D. A., R. C. Hogg y K. M. Ramsey. NMSS handbook for decommissioning fuel cycle and materials licensees. Office of Scientific and Technical Information (OSTI), marzo de 1997. http://dx.doi.org/10.2172/459359.
Texto completoSteckle, Jr., Warren y D. Veirs. Technical review of NMC gasket filter degradation. Office of Scientific and Technical Information (OSTI), julio de 2008. http://dx.doi.org/10.2172/1159585.
Texto completoThomas, Robert L. The Submarine Force: Utility in the Future NMS? Fort Belvoir, VA: Defense Technical Information Center, enero de 1997. http://dx.doi.org/10.21236/ada442607.
Texto completoHodge, C. Qualification of SRS's KAMS NMC and GIS Systems. Office of Scientific and Technical Information (OSTI), julio de 2003. http://dx.doi.org/10.2172/812411.
Texto completoKing, M., E. Renedo y D. Croucher. Review of the evaluation activities for the NMS. National Physical Laboratory, abril de 2022. http://dx.doi.org/10.47120/npl.iea9.
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