Tesis sobre el tema "Neuromuscular function"
Crea una cita precisa en los estilos APA, MLA, Chicago, Harvard y otros
Consulte los 50 mejores tesis para su investigación sobre el tema "Neuromuscular function".
Junto a cada fuente en la lista de referencias hay un botón "Agregar a la bibliografía". Pulsa este botón, y generaremos automáticamente la referencia bibliográfica para la obra elegida en el estilo de cita que necesites: APA, MLA, Harvard, Vancouver, Chicago, etc.
También puede descargar el texto completo de la publicación académica en formato pdf y leer en línea su resumen siempre que esté disponible en los metadatos.
Explore tesis sobre una amplia variedad de disciplinas y organice su bibliografía correctamente.
PELLEGRINO, GIULIA MICHELA. "LUNG FUNCTION AND DYSPNEA IN NEUROMUSCULAR DISEASES". Doctoral thesis, Università degli Studi di Milano, 2021. http://hdl.handle.net/2434/842435.
Texto completoKalmar, Jayne M. "The effects of caffeine on human neuromuscular function". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ33488.pdf.
Texto completoWeinberg, Jan. "Studies on ventilatory function in chronic neuromuscular disorders /". Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3936-5/.
Texto completoAuburn, Richard Peter. "Screening for genes involved in Drosophila neuromuscular function". Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621081.
Texto completoPenney, Jay. "Retrograde regulation of synaptic function at «Drosophila» neuromuscular junctions". Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=119497.
Texto completoLa croissance neuronale et la fonction synaptique sont des constituants clés dans le comportement du système nerveux. Il est donc essentiel de comprendre les mécanismes moléculaires régulant ces processus à l'état normal ainsi qu'à l'état pathologique. Ma thèse a pour objectif une meilleure compréhension des mécanismes contrôlant la croissance neuronale, ainsi que la fonction et la plasticité synaptiques engendrés par la transmission glutamatergique, en utilisant comme modèle la jonction neuromusculaire (JNM) de la drosophile, Drosophila melanogaster. Cet ouvrage porte spécifiquement sur l'impact de régulateurs de la synthèse protéique sur la fonction et la plasticité synaptiques dans la JNM, et a identifié des rôles importants pour de multiples régulateurs de traduction coiffe-dépendante dans ces processus. Des manipulations génétiques menant à une augmentation de la traduction coiffe-dépendante dans le muscle post-synaptique induisent une augmentation rétrograde de la fonction synaptique. Dans chaque cas, ces manipulations n'affectent ni la croissance neuronale, ni le nombre de synapses, ni le niveau de récepteurs glutamatergiques. Plutôt, une traduction coiffe-dépendante post-synaptique conduit vers une augmentation de la probabilité de relâche pré-synaptique. Cette voie signalétique joue un rôle important dans l'expression de la plasticité homéostatique des synapses neuromusculaires. Des mutants du récepteur glutamatergique IIA (GluRIIA) montrent une réduction dans la fonction post-synaptique, qui est compensée par une augmentation de la probabilité de relâche pré-synaptique. La traduction coiffe-dépendante, sous le contrôle de la voie signalétique TOR (target of rapamycin), est essentielle pour cette réponse homéostatique rétrograde. De plus, la protéine reliée à la maladie de Parkinson, LRRK2 (leucine rich repeat kinase 2), ainsi que son orthologue chez la drosophile, dLRRK, peuvent réguler la fonction synaptique de la JNM via la traduction coiffe-dépendante. Cependant, les formes mutantes de LRRK2 sont défectueuses dans leur habileté à réguler la fonction synaptique. Globalement, ces trouvailles nous aident à comprendre les mécanismes moléculaires régulant la fonction et la plasticité synaptiques glutamatergiques.
El-Kabir, Desiree Ruth. "An assessment of ventilatory function and sleep in neuromuscular disease". Thesis, King's College London (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.440537.
Texto completoBenatar, Michael G. "Presynaptic function in development and disease at the neuromuscular junction". Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.388959.
Texto completoDhesi, Jugdeep. "Vitamin D and neuromuscular function in older people who fall". Thesis, King's College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416021.
Texto completoVoegtle, Tracy. "The effect of an acute ankle joint injury on neuromuscular function". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ58098.pdf.
Texto completoBlunk, Aline D. (Aline Dorret). "Regulation of synaptic structure and function at the Drosophila neuromuscular junction". Thesis, Massachusetts Institute of Technology, 2013. http://hdl.handle.net/1721.1/84874.
Texto completoCataloged from PDF version of thesis.
Includes bibliographical references.
Neuronal communication requires a spatially organized synaptic apparatus to coordinate neurotransmitter release from synaptic vesicles and activation of postsynaptic receptors. Structural remodeling of synaptic connections can strengthen neuronal communication and synaptic efficacy during development and behavioral plasticity. Here, I describe experimental approaches that have revealed how the actin cytoskeleton participates in transynaptic signaling to control synapse assembly. I also describe my studies on how regulation of endocytic trafficking controls synaptic growth during neuronal development. To identify regulators of synapse assembly, I carried out a large-scale EMS mutagenesis screen of the second chromosome. From this screen I identified a mutation in actin 57B that disrupts synaptic morphology and presynaptic active zone organization. Actin 57B is one of six actin genes in Drosophila and is expressed in body wall muscle during larval development. The isolated allele harbors a point mutation disrupting a highly conserved amino acid present throughout the actin family. Homozygous mutant larvae show impaired alignment and spacing of presynaptic active zones. Additionally, disruption of the organization of the postsynaptic density is observed, with mislocalization of the Spectrin cytoskeleton and the PSD-homolog Disc-Large. Phallodin staining reveals a severe disruption of postsynaptic actin surrounding presynaptic boutons, with the formation of aberrant large actin swirls. Based on these results, we hypothesize that the loss of a synaptic interaction mediated by actin 57B leads to disruption of postsynaptic cytoskeletal organization and dysregulation of signals required to organize presynaptic active zones. Additionally, I present data that provide new insights into the mechanisms controlling synaptic growth signaling during transit through the endocytic pathway. Nervous Wreck (Nwk) is a presynaptic F-BAR/SH3 protein that regulates synaptic growth signaling in Drosophila. Here, I show that Nwk acts through a physical interaction with Sorting Nexin 16 (SNX16). SNX16 promotes synaptic growth signaling by activated BMP receptors, and live imaging in neurons reveals that SNX16-positive early endosomes undergo transient interactions with Nwkcontaining recycling endosomes. We identify an alternative signal termination pathway in the absence of Snx16 that is controlled by ESCRT-mediated internalization of receptors into the endosomal lumen. Our results define a presynaptic trafficking pathway mediated by SNX116, NWK and the ESCRT complex that functions to control synaptic growth signaling at the interface between endosomal compartments. Together, these experiments have expanded our understanding of the molecular mechanisms that control synaptic growth and assembly, highlighting the role of the postsynaptic actin cytoskeleton and the presynaptic endosomal trafficking pathway as key regulators.
by Aline D. Blunk.
Ph.D.in Neuroscience
Cummings, Sarah. "Investigating Oligodendrocyte Biology and Function: Insights from Neurological and Neuromuscular Diseases". Thesis, Université d'Ottawa / University of Ottawa, 2020. http://hdl.handle.net/10393/41489.
Texto completoHartigan, Erin. "Knee function after ACL rupture and reconstruction effects of neuromuscular training". Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 203 p, 2009. http://proquest.umi.com/pqdweb?did=1896910991&sid=7&Fmt=2&clientId=8331&RQT=309&VName=PQD.
Texto completoOpar, David A. "Neuromuscular hamstring function in response to intermittent running and previous injury". Thesis, Queensland University of Technology, 2013. https://eprints.qut.edu.au/64107/4/David_Opar_Thesis.pdf.
Texto completoCôté, Patrice D. "Dystroglycan function in development and neuromuscular disease : a study by gene targeting". Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36900.
Texto completoTsang, Ka Wai Karen. "A microRNA cluster negatively regulates synaptic function at «Drosophila» larval neuromuscular junction". Thesis, McGill University, 2010. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=86715.
Texto completoLa croissance et la plasticité synaptiques sont des éléments critiques pour le bon fonctionnement du cerveau, et participent aux processus de la mémoire et l'apprentissage. Le but de nos recherches est d'identifier et de caractériser les mécanismes moléculaires qui sous-tendent le règlement de la plasticité synaptique morphologique ainsi que fonctionnelle. À cette fin, nous avons élaboré un crible d'augmentation-de-fonction génétique dont le but était d'identifier de nouveaux régulateurs de croissance synaptique, en utilisant la jonction neuromusculaire (JNM) larvaire de la mouche drosophile (Drosophila melanogaster) comme synapse modèle. Grâce à ce crible, nous avons identifié un groupe de microARN (miARN) bien conservé au niveau évolutionnaire, miR-310-313, dont l'expression affecte la plasticité synaptique à la JNM de la drosophile. Nous avons produit une délétion de ce groupe de miARN par l'excision imprecise d'un élément génétique mobile (P-element). Nos résultats démontrent que la perte de miR-310-313 entraîne une augmentation significative du courant post-synaptique évoqué et du contenu quantique, une mesure de l'efficacité de la libération de neurotransmetteur présynaptique. Ce phénotype peut être rescapé en surexprimant miR-310-313 pendant le stade larvaire du système nerveux, suggérant une exigence d'expression génétique temporelle et spatiale spécifique pour ce groupe de miARN. Les miARN répriment l'expression de leurs cibles par appariement de bases avec le 3'UTR de leur mARN. Des algorithmes quantificatifs ont prédit Khc-73, un membre de la superfamille des protéines Kinesin (KIFs), comme une des cibles les plus plausibles pour miR-310-313. Nous émettons l'hypothèse que le groupe miR-310-313 régule la fonction synaptique en réprimant l'expression de Khc-73. Dans le soutien de notre hypothèse, nous démontrons que la surexpression de Khc-73 dans les neurones moteurs de la drosophile améliore la fo
Whitney, Garrett Lynn. "The Effects of Electromyographic Biofeedback and Therapeutic Exercise on Quadriceps Neuromuscular Function". University of Toledo / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1396484469.
Texto completoVaughan, Jeremiah A. "Neuromuscular Function and Fatigue and Metabolic Responses while Cycling in the Heat". Kent State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=kent1542212848069694.
Texto completoTang, Teresa. "Effects of plasmas from seronegative myasthenia gravis patients on the function of skeletal muscle nicotinic acetylcholine receptor". Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365322.
Texto completoBouhana, Antoine. "Análise cinemática do teste de Thomas modificado e sua influência na função neuromuscular". Bachelor's thesis, [s.n.], 2017. http://hdl.handle.net/10284/5884.
Texto completoIntrodução: A ação preventiva da Fisioterapia é fundamental para reduzir o risco de lesão. A maior parte de lesões resultam de um número enorme de fatores, entre eles a flexibilidade, força muscular entre outros. Existem vários testes em Fisioterapia que podem ser utilizados no sentido de avaliar a flexibilidade, entre eles temos o teste Thomas modificado. Objetivo: perceber as repercussões biomecânicas (cinemáticas e musculares) da execução do teste de Thomas modificada e sua relação com na função neuromuscular. Metodologia: Análise da cinemática tridimensional do teste de Thomas Modificado através do sistema de captura e análise de movimento 3D, Qualisys Oqus Camera Series. Resultados: Após a análise dos dados obtidos, os resultados parecem que o modo de execução do teste permite uma estabilização razoável dos mesmos, ainda que a posição final do membro em teste não tem relação com a função neuromuscular em extensão. Por outro lado, um potencial encurtamento a nível do reto femoral, não parece influenciar a força produzida nos extensores do joelho. Conclusão: A força executada pelo paciente não influenciou o lado contralateral (em teste), e que o teste de Thomas Modificado parece ser um teste fiável, uma vez que não se verificaram compensações segmentares significativas na execução do mesmo.
Introducton: The preventive action of Physiotherapy plays an essential part in reducing injury risk, especially in sports. Most of those injuries occur due to flexibility, muscular strength issues, among other factors. There are several tests used in Physiotherapy to evaluate the patients’ flexibility, and the Modified Thomas Test is one of them. Objective: Understand the biomechanical repercussions (kinematic and muscular) of the Modified Thomas test and its influence in neuromuscular function. Methodology: Tridimensional kinematic analysis of the Modified Test of Thomas with Qualisys Oqus Camera Series, 3D movement capture and analysis program. Results: After data analysis results seem that the test procedure allows a reasonable stabilization, although the final position of the limb under test has no relation with the isokinetic neuromuscular function in extension. On the other hand, a potential shortening in the rectus femoris does not seem to influence the force produced in the knee extensors. Conclusion: The force exerted by the patient did not influence the contralateral side (under test), and that the modified Thomas test seems to be a reliable test, since there were no significant segmental compensations in the execution of the test.
N/A
Grönlund, Christer. "Spatio-temporal processing of surface electromyographic signals : information on neuromuscular function and control". Doctoral thesis, Umeå universitet, Institutionen för strålningsvetenskaper, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-958.
Texto completoPatrón, Lilian Adilene y Lilian Adilene Patrón. "DCAF12 Is Required For Synaptic Function and Plasticity at the Drosophila Neuromuscular Junction". Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/624292.
Texto completoGrönlund, Christer. "Spatio-temporal processing of surface electromyographic signals : information on neuromuscular function and control /". Umeå : Umeå universitet, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-958.
Texto completoScheurer, Sarah Ann. "The Relationship between Corticomotor Excitability and Quadriceps Neuromuscular Function in ACL Reconstructed Patients". University of Toledo / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525287124424035.
Texto completoMelillo, Julie Ann. "Influence of Muscle Motor Point Identification on Quadriceps Function Following Neuromuscular Electrical Stimulation". University of Toledo / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1556547710451478.
Texto completoMcKay, Marnee. "1000 NORMS PROJECT: MEASURES OF PHYSICAL FUNCTION ACROSS THE LIFESPAN". Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/16588.
Texto completoClark, David Rodney. "Neuromuscular assessment of trunk muscle function in loaded, free barbell back squat : implications for development of trunk stability in dynamic athletic activity". Thesis, University of Stirling, 2018. http://hdl.handle.net/1893/28080.
Texto completoSundman, Eva. "Pharyngeal function, airway protection and anesthetic agents /". Stockholm : [Karolinska institutets bibl.], 2002. http://diss.kib.ki.se/2002/91-7349-161-6.
Texto completoCerantola, Silvia. "Toll-like receptor 4, enteric nervous system and gut neuromuscular function in models of functional and inflammatory bowel disorders". Doctoral thesis, Università degli studi di Padova, 2019. http://hdl.handle.net/11577/3423310.
Texto completoHeraghty, Jane Lesley. "Investigations of respiratory muscle function in children with neuromuscular disease using a novel device". Thesis, University of Bristol, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.702155.
Texto completoPanchumarthi, Sarvari. "The Drosophila Serrate is Required for Synaptic Structure and Function at Larval Neuromuscular Junctions". Diss., The University of Arizona, 2010. http://hdl.handle.net/10150/194269.
Texto completoUzel, Sébastien G. M. "Microfluidic and optogenetic technologies to model spinal cord development and neuromuscular junction formation and function". Thesis, Massachusetts Institute of Technology, 2015. http://hdl.handle.net/1721.1/103850.
Texto completo"June 2015." Cataloged from PDF version of thesis.
Includes bibliographical references (pages 106-118).
Motor neurons located in the spinal cord and innervating muscle cells throughout the body are responsible for virtually all motor functions, from locomotion to respiration or speech. They arise from differentiation of progenitor cells within the neural tube under spatiotemporally well-defined morphogen concentration profiles, and extend axons into the peripheral nervous system following a precisely orchestrated sequence of events involving secreted chemo-attractants and repellents and dynamic expression of the corresponding ligand receptors. Finally, they form neuromuscular junctions, the synapses that transmit electrical signals to the muscle effectors. Failure for these motor neurons to develop or function properly, caused by developmental or neurodegenerative genetic disorders, or as a result of traumatic injuries, lead to highly incapacitating or even lethal malformation and conditions. Microfabricated platforms and optogenetic technologies have proven to be valuable tools to control the microenvironment, biochemical cues and the stimulation applied to neuronal tissues. Precise control of the geometry of microfluidic devices together with their ability to host 3D cell culture has enhanced the physiological relevance of such neuronal tissues relative to traditional 2D culture assays. And the ability to selectively excite neuronal cells with light has opened tremendous opportunities in the field of neuroscience. In this thesis, we combine these two technologies to stimulate and subject cells to chemical and physical microenvironments that emulate their in vivo counterpart. First, we present a microfluidic platform that generates orthogonal concentration gradients and emulates the confined appearance of motor neurons within the developing spinal cord. Then, we introduce a new device capable of forming a 3D compartmentalized neuron-muscle coculture and demonstrate remote stimulation of the myofibers by the motor neurons resulting in muscle contraction. By targeting the stem cells from which the motor neurons are derived with the light sensitive ion channel Channelrhodopsin, we form, in this microfluidic device, the first in vitro light-activatable neuromuscular junction. Keywords: microfluidics, optogenetics, morphogenesis, cell migration, neuromuscular junctions.
by Sébastien G. M. Uzel.
Ph. D.
Walker, John W. "Use of Nordic Hamstring Exercise to Improve Hamstrings Function in Patients after ACL Reconstruction". University of Toledo / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1556811041471286.
Texto completoSujkowski, Alyson L. "GABAA Receptor Homeostasis at the C. elegans Neuromuscular Junction". University of Toledo / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1278964838.
Texto completoMork, Mikie Tanya Michelle. "The effects of a specific exercise intervention on Delayed Onset Muscle Soreness, DOMS, and neuromuscular function". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ62802.pdf.
Texto completoClark, Brian C. "Adaptations in human neuromuscular function following prolonged unweighting Neurological and skeletal muscle properties and countermeasure efficacy /". Related electronic resource: Current Research at SU : database of SU dissertations, recent titles available full text, 2006. http://proquest.umi.com/login?COPT=REJTPTU0NWQmSU5UPTAmVkVSPTI=&clientId=3739.
Texto completoJonsson, Malin. "Nicotinic transmission and drugs in anesthesia : neuromuscular blocking agents and propofol : consequences for carotid body function /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-660-3/.
Texto completoTalubmook, Chusri. "The influence of elevated glucose levels and the diabetic state on neuromuscular function in the gut". Thesis, University of Hertfordshire, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.247307.
Texto completoTavoian, Dallin. "Tools and Technologies for Assessing, and Exercise Strategies for Promoting, Neuromuscular Function and Mobility in Aging". Ohio University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1615816378173099.
Texto completoTakeno, Katsumi. "Neuromuscular Function of the Shoulder Girdle and Upper Extremity Muscles in Individuals with Glenohumeral Labral Repair". University of Toledo / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1596111099423871.
Texto completoCopley, LaRae Meschelle. "Investigation of the function of myotubularin through the examination of protein-protein interactions and exclusion of MTMR1 as a frequent cause of X-linked myotubular myopathy". Connect to this title online, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1080146560.
Texto completoTitle from first page of PDF file. Document formatted into pages; contains xix, 156 p.; also includes graphics (some col.). Includes bibliographical references (p. 144-153).
Overton, Amanda Julie. "Neuromuscular Fatigue and Biomechanical Alterations during High-Intensity, Constant-Load Cycling". Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2013. https://ro.ecu.edu.au/theses/612.
Texto completoCriss, Cody R. "Central Nervous System Contributions to Subjective and Objective Measures of Function after Anterior Cruciate Ligament Reconstruction". Ohio University / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1617279837916864.
Texto completoFathi, Behrooz H. "The effect of snake venom phospholipase A2 on cloned voltage-gated potassium channels and on neuromuscular function". Thesis, University of Strathclyde, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248762.
Texto completoBuhl, Lauren Kaye. "Synaptic structure and function at the Drosophila larval neuromuscular junction : a molecular analysis of complexin and radish". Thesis, Massachusetts Institute of Technology, 2011. http://hdl.handle.net/1721.1/65285.
Texto completoCataloged from PDF version of thesis.
Includes bibliographical references.
From yeast to humans, the fusion of vesicles with target membranes is driven by the formation of a parallel four-helix bundle of SNARE proteins that are present on both the vesicular (v-SNAREs) and target plasma membranes (t-SNAREs). The full zippering of this bundle is thought to provide the driving force for membrane fusion. At synapses, vesicle fusion is exquisitely regulated by Ca2+ such that neurotransmitter release can occur within 1 ms of an action potential reaching the presynaptic terminal. This feat implies the presence of both a Ca2+ sensor and a fusion clamp that prevents vesicles from fusing in the absence of Ca2+. The presynaptic Ca2+ sensor for synchronous vesicle release is widely accepted to be Synaptotagmin-1 (Syt1), and there is growing evidence that Complexin (Cpx), which binds to the SNARE complex with high affinity and 1:1 stoichiometry, can act as a vesicle fusion clamp. As suggested by its name, however, Cpx appears to play a more complex role in vesicle release, carrying out different functions in spontaneous vs. evoked fusion events. Here we show the Drosophila express at least two Cpx isoforms that differ in the C-terminus (Cpx7A and Cpx7B) and can be further regulated by RNA editing and phosphorylation. These isoforms show different effects on spontaneous vs. evoked neurotransmitter release, with Cpx7A being a better fusion clamp and Cpx7B being a better fusion promoter. In addition, these isoforms have different effects on synaptic growth, which may be linked to their effects on synaptic physiology.
by Lauren Kaye Buhl.
Ph.D.
Breuer, Marlen [Verfasser] y Annette [Akademischer Betreuer] Brochers. "The analysis of Cav1α function in Xenopus motoneuron outgrowth and neuromuscular integrity / Marlen Breuer ; Betreuer: Annette Brochers". Marburg : Philipps-Universität Marburg, 2021. http://d-nb.info/1227580274/34.
Texto completoHannah, Ricci. "The influence of sex-differences and exercise on knee joint neuromuscular function: implications for dynamic joint stabilisation". Thesis, Nottingham Trent University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.657623.
Texto completoHenaghan, Sharon M. "The effects of moderate sleep loss on sleepiness levels and neuromuscular function in healthy males a thesis submitted to Auckland University of Technology in partial fulfillment of the degree of Master of Health Science, April 2004". Full thesis. Abstract, 2004. http://puka2.aut.ac.nz/ait/theses/HenaghanS.pdf.
Texto completoRohadhia, Wafina. "Cardio-metabolic and neuromuscular adaptations following whole-body eccentric resistance training in older adults". Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2019. https://ro.ecu.edu.au/theses/2203.
Texto completoDwyer, Daniel y na. "Serotonin as a Mediator of Fatigue During Exercise and Training". Griffith University. School of Health Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20040521.130535.
Texto completoDwyer, Dan. "Serotonin as a Mediator of Fatigue During Exercise and Training". Thesis, Griffith University, 2004. http://hdl.handle.net/10072/366705.
Texto completoThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Health Sciences
Full Text