Literatura académica sobre el tema "MiR-204"
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Artículos de revistas sobre el tema "MiR-204"
Huang, Zhengbiao y Tianling Deng. "miR-204-3p Regulates Glioma Cell Biological Behaviors via Targeting Protein Kinase B (AKT1)". Journal of Biomaterials and Tissue Engineering 12, n.º 12 (1 de diciembre de 2022): 2395–400. http://dx.doi.org/10.1166/jbt.2022.3188.
Texto completoSun, Xueqin, Shan Su, Guoxiang Zhang, Hong Zhang y Xiaohui Yu. "MiR-204 suppresses cell proliferation and promotes apoptosis in ovarian granulosa cells via targeting TPT1 in polycystic ovary syndrome". Biochemistry and Cell Biology 97, n.º 5 (octubre de 2019): 554–62. http://dx.doi.org/10.1139/bcb-2019-0019.
Texto completoWang, Zhiguo, Zehui Fang, Runzhang Lu, Hongli Zhao, Tiejun Gong, Dong Liu, Luojia Hong, Jun Ma y Mei Zhang. "MicroRNA-204 Potentiates the Sensitivity of Acute Myeloid Leukemia Cells to Arsenic Trioxide". Oncology Research Featuring Preclinical and Clinical Cancer Therapeutics 27, n.º 9 (23 de septiembre de 2019): 1035–42. http://dx.doi.org/10.3727/096504019x15528367532612.
Texto completoDu, Youyou, Guanghui Liu, Luosha Zhao y Rui Yao. "Protective Effect of miR-204 on Doxorubicin-Induced Cardiomyocyte Injury via HMGB1". Oxidative Medicine and Cellular Longevity 2020 (19 de noviembre de 2020): 1–16. http://dx.doi.org/10.1155/2020/8819771.
Texto completoSong, Rui, Yufeng Zhai, Lihua Ao, David A. Fullerton y Xianzhong Meng. "MicroRNA-204 Deficiency in Human Aortic Valves Elevates Valvular Osteogenic Activity". International Journal of Molecular Sciences 21, n.º 1 (20 de diciembre de 2019): 76. http://dx.doi.org/10.3390/ijms21010076.
Texto completoSu, Qunxue, Hao Shen, Bei Gu y Ning Zhu. "miR-204-5p Hampers Breast Cancer Malignancy and Affects the Cell Cycle by Targeting PRR11". Computational and Mathematical Methods in Medicine 2022 (27 de enero de 2022): 1–10. http://dx.doi.org/10.1155/2022/4010947.
Texto completoLi, Liang-Qing, Dun Pan, Qun Chen, Sheng-Wei Zhang, Di-Ya Xie, Xue-Lan Zheng y Hui Chen. "Sensitization of Gastric Cancer Cells to 5-FU by MicroRNA-204 Through Targeting the TGFBR2-Mediated Epithelial to Mesenchymal Transition". Cellular Physiology and Biochemistry 47, n.º 4 (2018): 1533–45. http://dx.doi.org/10.1159/000490871.
Texto completoEstephan, Leonard E., Michael V. Genuardi, Chad M. Kosanovich, Michael G. Risbano, Yingze Zhang, Nancy Petro, Annie Watson et al. "Distinct plasma gradients of microRNA-204 in the pulmonary circulation of patients suffering from WHO Groups I and II pulmonary hypertension". Pulmonary Circulation 9, n.º 2 (28 de marzo de 2019): 204589401984064. http://dx.doi.org/10.1177/2045894019840646.
Texto completoQu, Liangliang, Zhongqiu Li y Pinduan Liu. "mir-204-5p Acts as a Tumor Suppressor by Targeting DNM2 in Osteosarcoma Cells". Journal of Healthcare Engineering 2022 (9 de febrero de 2022): 1–7. http://dx.doi.org/10.1155/2022/8944588.
Texto completoCheng, Yuan, Dandan Wang, Feng Wang, Jing Liu, Baorui Huang, Maria Angeles Baker, Jianyong Yin et al. "Endogenous miR-204 Protects the Kidney against Chronic Injury in Hypertension and Diabetes". Journal of the American Society of Nephrology 31, n.º 7 (2 de junio de 2020): 1539–54. http://dx.doi.org/10.1681/asn.2019101100.
Texto completoTesis sobre el tema "MiR-204"
Bhat, Rajeshwari Subray. "Study of the role of miR-204 in photoreceptor development". Thesis, Open University, 2016. http://oro.open.ac.uk/48207/.
Texto completoMigliore, Chiara Maria. "RNA-sequencing based identification of microRNA-204 targets". Doctoral thesis, Università degli studi di Trieste, 2011. http://hdl.handle.net/10077/4595.
Texto completoWith the completion of the sequencing and annotation of hundreds of genomes, and the accumulation of data on the mammalian transcriptome, greater emphasis has been placed on elucidating the function of non-coding DNA and RNA sequences. It is well known that the non-coding portion of the genome can transcribe functional RNAs. Several categories of non-coding RNAs (ncRNAs) have been defined, such as transport RNAs (tRNAs) ribosomal RNAs (rRNAs), small nuclear RNAs (snRNAs) and small nucleolar RNAs (snoRNAs). A larger group of ncRNAs comprises the so-called microRNAs (miRNAs) and long non-coding RNAs serving key regulatory roles. It has been shown that miRNAs directly target a large number of genes, thus affecting significantly major pathways. In my project, I focused on miR-204, a microRNA that is highly conserved from zebrafish to human and located in the sixth intron of the human TRPM3 gene. I sought to identify mir-204 targets by using the Medaka fish (Oryzias latipes), where mir-204 is expressed at very low levels in the nervous system, as a model for perturbation of the mir-204 network. Transient transgenic Medaka fish were produced to knock down and over-express mir-204. Next-generation sequencing was used to sequence the Medaka transcriptome, dissect the putative targets of miR-204, and thus gain further insight about its function. Potential target genes of mir-204 were selected by choosing genes, which presented lower expression in the wild-type (wt) fish than in the knock down, a lower expression in the over-expression than in the wt and, finally, a higher expression in the knock down than in the over-expression. At the same time, I collected a list of putative miR-204 mouse and human targets using the prediction softwares miRanda, PicTar and TargetScan, obtained the Medaka orthologues and verified that the selected genes in Medaka had a statistically significant enrichment in miR-204 targets as compared to the complete set of genes obtained from the RNA-Sequencing approach. The combined RNA-Sequencing and bioinformatics analysis revealed 147 predicted targets of mir-204, which showed a significant enrichment for the axon guidance pathway. In order to confirm this data, real time quantitative PCR has been performed on total RNA from wt and morphant fish. Results showed a higher expression in the knock down fish for 15 out of 25 putative targets (Neo1, Trim71, Ddx3y, Prkar1a, MyoX, Sema3B, Sema3F, Ptprg, Slit2, Epha4, Epha7, Amot, Lpp, Odz4, Jarid2). I further validated these genes by both Q-PCR and luciferase assays. To this aim, I cloned five putative target sequences into the 3’UTR of a luciferase reporter vector (pGL3-TK-luc Promega) to use them in luciferase assays: co-transfection with miR-204 reduced the luciferase activity of Sema3F, belonging to the class of receptors involved upstream of the axon guidance pathway. These results indicate that mir-204 directly targets key genes involved in the axon guidance pathway such as Sema3F in the nervous system. Further validation of the disruption of axon guidance in the transgenic fish has been undertaken in vivo by our collaborators: the experiment demonstrated a clear role of this microRNA in axon path finding during retinal development.
XXII Ciclo
1981
Libros sobre el tema "MiR-204"
Music, Alfred. Cantata No. 204 -- Ich Bin in Mir Vergnugt: Soprano Solo. Alfred Publishing Company, Incorporated, 1985.
Buscar texto completoBach, Johann Sebastian. Cantatas Nos. 204, Ich bin in mir vergnught (G) and 205 Zerreisset, Zersprenget: Miniature Score, Kalmus Edition. Alfred Publishing Company, 1985.
Buscar texto completoCapítulos de libros sobre el tema "MiR-204"
"Abkürzungen". En Das Geschlecht in mir, editado por Gerhard Schreiber, XXIII—XXIV. Berlin, Boston: De Gruyter, 2019. http://dx.doi.org/10.1515/9783110614626-204.
Texto completoActas de conferencias sobre el tema "MiR-204"
Ryan, Jacqueline M., Amanda Tivnan, Isabella Bray, Joanna Fay, Andrew M. Davidoff, Lorraine Tracey y Raymond Stallings. "Abstract 130: MiR-204 acts as a tumor suppressor in neuroblastoma through down-regulation of the neurotrophic receptor TrkB". En Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-130.
Texto completoKoga, T., K. Migita, M. Umeda, F. Nonaka, S.-Y. Kawashiri, N. Iwamoto, K. Ichinose et al. "FRI0620 MIR-204-3P inhibits the production of TLR4-related cytokines in familial mediterranean fever by targeting the PIK3 signaling pathway". En Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2813.
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