Literatura académica sobre el tema "Microsatellites instability"
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Artículos de revistas sobre el tema "Microsatellites instability"
Wada, C., S. Shionoya, Y. Fujino, H. Tokuhiro, T. Akahoshi, T. Uchida y H. Ohtani. "Genomic instability of microsatellite repeats and its association with the evolution of chronic myelogenous leukemia [see comments]". Blood 83, n.º 12 (15 de junio de 1994): 3449–56. http://dx.doi.org/10.1182/blood.v83.12.3449.3449.
Texto completoWada, C., S. Shionoya, Y. Fujino, H. Tokuhiro, T. Akahoshi, T. Uchida y H. Ohtani. "Genomic instability of microsatellite repeats and its association with the evolution of chronic myelogenous leukemia [see comments]". Blood 83, n.º 12 (15 de junio de 1994): 3449–56. http://dx.doi.org/10.1182/blood.v83.12.3449.bloodjournal83123449.
Texto completoGadgil, Rujuta Yashodhan, Eric J. Romer, Caitlin C. Goodman, S. Dean Rider, French J. Damewood, Joanna R. Barthelemy, Kazuo Shin-ya, Helmut Hanenberg y Michael Leffak. "Replication stress at microsatellites causes DNA double-strand breaks and break-induced replication". Journal of Biological Chemistry 295, n.º 45 (1 de septiembre de 2020): 15378–97. http://dx.doi.org/10.1074/jbc.ra120.013495.
Texto completoYang, Guang, Ru-yi Zheng y Zai-shun Jin. "Correlations between microsatellite instability and the biological behaviour of tumours". Journal of Cancer Research and Clinical Oncology 145, n.º 12 (15 de octubre de 2019): 2891–99. http://dx.doi.org/10.1007/s00432-019-03053-4.
Texto completoHaiduk, Tiffany, Michael Brockmann, Christoph Schmitt, Ramona-Liza Tillmann, Monika Pieper, Jessica Lüsebrink, Oliver Schildgen y Verena Schildgen. "Are Microsatellite Patterns Specific for Tumor Types? A Pilot Investigation". Journal of Molecular Pathology 1, n.º 1 (4 de septiembre de 2020): 3–8. http://dx.doi.org/10.3390/jmp1010002.
Texto completoSchöniger, Sandra y Josef Rüschoff. "Mismatch Repair Deficiency and Microsatellite Instability". Encyclopedia 2, n.º 3 (31 de agosto de 2022): 1559–76. http://dx.doi.org/10.3390/encyclopedia2030106.
Texto completoLong, Dustin R., Adam Waalkes, Varun P. Panicker, Ronald J. Hause y Stephen J. Salipante. "Identifying Optimal Loci for the Molecular Diagnosis of Microsatellite Instability". Clinical Chemistry 66, n.º 10 (24 de septiembre de 2020): 1310–18. http://dx.doi.org/10.1093/clinchem/hvaa177.
Texto completoT, Muthu Venkat, Vijayalakshmi Vijayalakshmi y Pramila Pramila. "Significance of Microsatellite Instability in Colorectal Carcinoma- A Complete Review". Saudi Journal of Pathology and Microbiology 9, n.º 03 (27 de marzo de 2024): 71–74. http://dx.doi.org/10.36348/sjpm.2024.v09i03.003.
Texto completoChen, Sophia, John Hatch, Ashley Luck, Nicole M. Nichols, Emily J. Gleason, Kathryn Martin, Kevin D. Foley, D. Scott Copeland, Sebastian Kraves y Ezequiel Alvarez Saavedra. "Detection of DNA Microsatellites Using Multiplex Polymerase Chain Reaction Aboard the International Space Station". Gravitational and Space Research 9, n.º 1 (1 de enero de 2021): 164–70. http://dx.doi.org/10.2478/gsr-2021-0013.
Texto completoRogge, Ryan, Taylor Patterson, Alicia Navetta y Dhruti Legare. "Abstract 2943: Investigation of microsatellite instability in RNA compared to DNA, using microsatellite targeted, anchored multiplex PCR and NGS". Cancer Research 84, n.º 6_Supplement (22 de marzo de 2024): 2943. http://dx.doi.org/10.1158/1538-7445.am2024-2943.
Texto completoTesis sobre el tema "Microsatellites instability"
Bodo, Sahra. "Induction d'un processus d'instabilité des microsatellites du génome dans des modèles murin et cellulaire : intérêt physiopathologique et clinique". Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066565/document.
Texto completoInactivation of the MMR (mismatch repair) system promotes the oncogenic process of microsatellite instability (MSI). During my PhD, I firstly investigated the role of azathioprine (Aza) in the induction of MSI tumors in mice. Epidemiological studies reported a correlation between the occurrence of late MSI cancers in humans and long-term treatment with this immunosuppressant whose cytotoxicity was shown in vitro to be mediated by MMR activity. Using a dose-response study, I observed the occurrence of rare late-onset MSI lymphomas in wild-type mice treated with Aza, but not with ciclosporin (another immunosuppressant used for comparison). These results established in vivo that long-term Aza exposure is a risk factor for the emergence of MSI tumors. Secondly, I was interested in the CMMRD syndrome (constitutional MMR deficiency), a major and rare predisposition to MSI cancers. Since CMMRD patients are carriers of biallelic germline mutations of a MMR gene, diagnosis is based on constitutional genotyping, a method that was found non-contributory when a variant of unknown significance is detected (30% patients). In this context, I developed a complementary approach for the detection of this syndrome in at-risk patients, based on the hypothesis that two functional features of MMR-deficient tumor cells, i.e. the MSI phenotype and the tolerance to genotoxic agents such as Aza, can be demonstrated in non-neoplastic tissues of CMMRD patients. We provided a sensitive and specific method that may constitute a valuable tool when diagnosis of CMMRD could not be confirmed by genetic testing
Wright, C. M. "The prognostic significance of microsatellite instability in sporadic stage C colorectal cancer". Thesis, The University of Sydney, 2008. https://hdl.handle.net/2123/28955.
Texto completoGreene, Malorie. "Étude des conséquences génomiques et fonctionnelles de l'instabilité des microsatellites dans le cancer colorectal". Thesis, Paris 6, 2017. http://www.theses.fr/2017PA066592/document.
Texto completoSince the discovery of a link between mismatch repair (MMR) deficiency and cancer, microsatellite instability (MSI) is thought as a process underlying cell transformation and tumour progression and invasion. MSI tumours are a subset of frequent human neoplasms, both inherited and sporadic, associated with several primary locations (colon, stomach, endometrium…). In MMR-deficient cells, MSI generates hundreds of frameshift mutations in genes (MSI Target Genes, MSI-TGs) containing coding microsatellite sequences (e.g. -1/+1 bp, insertions/deletions, i.e. indels). Some of these mutations affect genes with a role in human carcinogenesis and are thus expected to promote the MSI-driven tumorigenic process. During my PhD, I aimed to decipher the role of MSI in colon tumorigenesis. I exploited exome-sequencing data available in my lab that were generated from the analysis of a series of 47 human MSI primary colorectal cancer (CRC). Through biostatistics analysis and mathematical models that we designed to interpret mutation rates in the context of the high background for instability characterizing MSI in CRC, we identified a few microsatellites containing genes coding mutations that were negatively selected in MSI colon tumours (N=13). Under the hypothesis that these events may have a negative impact in colon tumorigenesis, I demonstrated that the silencing of these MSI target genes (siRNA/shRNA) was deleterious for MSI cancer cells using in vitro and in vivo models (impairment of proliferation and/or migration and/or response to chemotherapy and/or tumour growth) (Jonchère*, Marisa*, Greene* et al., submitted)
Micelli, Lupinacci Renato. "Caractérisation anatomo-clinique et phénotypique des adénocarcinomes canalaires du pancréas avec instabilité des microsatellites". Thesis, Paris 6, 2017. http://www.theses.fr/2017PA066311/document.
Texto completoPancreatic ductal adenocarcinoma (PDAC) is a major health problem in France and around the world. PDAC is developed mainly from two precursor lesions: pancreatic intraepithelial neoplasia and intraductal papillary mucinous neoplasm (IPMN). There are several molecular mechanisms underlying pancreatic oncogenesis. Particularly, we were interested in the MSI (MicroSatellite Instability) which is due to a defective DNA Mismatch Repair (MMR) system, which normally functions to recognize and repair erroneous insertions, deletions, and mis-incorporation of bases that can arise during DNA replication and recombination. The MSI phenotype was first described in the familial cancer condition known as Lynch syndrome (LS), where the MMR genes MLH1, MSH2, MSH6 or PMS2 harbor germline mutations. Interest in MSI tumors has recently increased after studies have highlighted the concomitant expression of multiple active immune checkpoint (ICK) markers including PD-1 and PD-L1 and the role of the MSI status to predict clinical benefit from immune checkpoint blockade. A Our results indicate that the MSI phenotype occurs in PDAC with a frequency of 1-2%. Our data showed that IHC using antibodies against the four MMR proteins was more sensitive for the assessment of MSI status than PCR-based methods. In addition, we demonstrate for the first time a statistically significant positive association between MSI and IPMNs in PDAC. MSI PDAC, including IPMN, are unlikely to be sporadic since they display molecular features that are usually observed in LS-related neoplasms. Also, our results highlight that an MSI-driven immunogenic pathway to cancer is active in MSI PDACs but suggest that MSI-driven somatic events may be tissue-specific. We observed a stronger lymphocytic tumor infiltration by activated TCD8 cells in MSI PDAC compared to MSS PDAC and found a positive association between PD-L1 expression and MSI status, suggesting that MSI PDAC could be responsive to ICK blockade therapy
Cohen, Romain. "Caractérisation phénotypique et clinique des cancers colorectaux métastatiques avec instabilité des microsatellites Clinical and molecular characterization of hereditary and sporadic metastatic colorectal cancer harbouring microsatellite instability/DNA mismatch repair deficiency Association of primary resistance to immune checkpoint inhibitors in metastatic colorectal cancer with misdiagnosis of microsatellite instability or mismatch repair deficiency status". Thesis, Sorbonne université, 2018. http://www.theses.fr/2018SORUS313.
Texto completoMicrosatellite instability (MSI) is a tumor phenotype linked to somatic or germline inactivating alterations of DNA mismatch repair (MMR) genes. MSI is observed in approximately 5% of metastatic colorectal cancers (mCRC) and has recently emerged as a major positive predictive biomarker for the efficacy of immune checkpoint inhibitors (ICKi) amongst mCRC patients. The objectives of my work was to clinically and molecularly characterize MSI mCRC, to evaluate the accuracy of MSI screening methods and the response to immunotherapy in the context of ICKi clinical trials. Fist, I show that sporadic and inherited MSI mCRC display distinct natural history (Cohen et al., Eur J Cancer 2017). In a second work, I show that MSI testing in routine practice is associated with almost 10% of false positives due to misinterpration of IHC and PCR assays. Moreover, these false-positives are the main cause of mCRC primary resistance to ICKi observed in clinical trials (Cohen*, Hain* et al., JAMA Oncol. 2018). After summarizing the literature concerning MSI, its consequences on CRC and immunotherapy, I present the results of the nosologic and diagnostic works developed during this doctoral thesis. Then I will go on perspectives in the context of MSI cancer
El-Murr, Nizar. "Étude de l'impact des microARNs sur la carcinogenèse des cancers colorectaux instables sur les séquences répétées microsatellites du génome". Phd thesis, Université Pierre et Marie Curie - Paris VI, 2014. http://tel.archives-ouvertes.fr/tel-00990895.
Texto completoBoulagnon-Rombi, Camille. "Etude du récepteur d’endocytose LRP1 dans les adénocarcinomes coliques : caractéristiques cliniques, pathologiques et moléculaires associées et valeur pronostique". Thesis, Reims, 2017. http://www.theses.fr/2017REIMM203/document.
Texto completoLRP1 (low-density lipoprotein receptor–related protein 1), a multifunctional endocytic receptor, has recently been identified as a hub in a biomarker network for multi-cancer clinical outcome prediction. Its role in côlon cancer has not been characterized. Here, we investigate the relationship between LRP1 and colon cancer.LRP1 mRNA expression was determined in colon adenocarcinoma and paired colon mucosa samples, and in stromal and tumoral cells obtained after laser capture microdissection. The clinical potential was further investigated by immunohistochemistry in a population-based colon cancer series (n = 307). LRP1 methylation, mutation and miR-205 expression were evaluated and compared to LRP1 expression levels.LRP1 mRNA levels are significantly decreased in colon adenocarcinoma cells compared to colon mucosa and stromal cells. Low LRP1 immunohistochemical expression in adenocarcinomas was associated with higher age, right-sided tumor, loss of CDX2 expression, Annexin A10 expression, CIMP-H, MSI-H and BRAFV600E mutation. Low LRP1 expression correlates with poor clinical outcome, especially in stage IV patients. LRP1 expression was downregulated by LRP1 mutation. LRP1 expression was slightly modified by miR-205 expression. LRP1 promoter was never methylated.Loss of LRP1 expression is associated with peculiar clinocopathological and molecular characteristics and with worse colon cancer outcomes
Vaysse-Zinkhöfer, Wilhelm. "Mécanismes de réparations d’une cassure double-brin et résection au sein d’un microsatellite humain". Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS477.
Texto completoMicrosatellites are tandem repeats of a motif between one and nine base pairs. These repeats are found ubiquitously in all organisms and are particularly abundant in eukaryotic organisms. All these repeats are capable of forming secondary structures in vitro and possibly in vivo. Some microsatellites are prone to expansion, leading to many neurodegenerative diseases in humans such as myotonic dystrophy type 1 (DM1), the most frequently transmitted neurodegenerative disease. The onset and severity of symptoms are positively correlated with the number of repeats located in the 3'UTR of the DMPK gene. In previous work in the laboratory, a TALE nuclease (TALEN) was developed to introduce a double-strand break into a microsatellite (GTC)n from a DM1 patient. Understanding the mechanisms leading to repeat contraction in yeast is necessary to understand the mechanisms in humans. Thus, experiments were conducted in cells with altered CBD repair systems showing that RAD51, POL32 and DNL4 were not required for CBD repair within microsatellites. Only RAD50 and RAD52 appear to be required, indicating that the cell repairs CBDs in repeated regions by single-strand annealing. The objective of this thesis was to study the role of several genes (MRE11, EXO1, SGS1, DNA2, SAE2, RIF1 and RIF2), involved in the resection and repair of a single CBD within a CTG repeat region, in yeast
Palassin, Pascale. "Etude du rôle du corégulateur transcriptionnel RIP140 dans le contrôle de l'instabilité microsatellitaire des cancers colorectaux héréditaires". Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT054/document.
Texto completoThe transcriptional coregulator RIP140 is an ubiquitous cofactor playing a major role in the regulation of many physiopathological processes. It can either act as a coactivator or as a corepressor of signaling pathways depending on its recruitment on target genes. It has been shown that RIP140 is a good prognostic marker in sporadic intestinal tumorigenesis. This work focuses on its role in familial colorectal cancers and particularly in relation to the Lynch syndrome (LS). Lynch syndrome is a hereditary cancer predisposition, mostly colorectal, characterized by a defect in the Mismatch Repair (MMR) system, due to a first germline mutation of one gene of this system. Loss of MMR function induces a microsatellite instability (MSI) phenotype. However, there are some MSI familial colorectal cancers, where neither germinal nor somatic alteration of one MMR gene is found. They are referred to as Lynch like Syndrome (LLS) and their overall management is identical to that of LS. Murine models and colorectal cell lines, harboring modulations of RIP140 expression, allowed us to demonstrate the positive transcriptional regulation of the MMR genes, MSH2 and MSH6 by RIP140. Functional validation of this regulation was explored by microsatellite instability and sensitivity to various cytotoxic drugs analyses. A positive correlation has been confirmed between RIP140 and MSH2 and MSH6 gene expression in a cohort of 396 patients. Moreover, the transcriptional regulation by RIP140 of a specialized translesional DNA polymerase, the Polκ polymerase, has been investigated. Polκ ensures microsatellite sequences replication. We have demonstrated that RIP140 positively stimulates the expression of the POLK gene in our cell models and which appears correlated with that of RIP140 in human colorectal tumors. Finally, by sequencing different cell lines, we found a frameshift mutation of RIP140, generating a truncated protein with loss of the last two repression domains. High-throughput sequencing allowed us to look for this mutation in patient MSI colorectal tumor samples. This mutation was found in 19% of these tumors, especially LLS (16,2%), where it has been associated with lower overall survival. This mutation affects the antiproliferative and transrepressive properties of RIP140, as well as the positive regulation of the MSH2, MSH6 and POLK gene. Development of a specific antibody for this mutation would be extremely useful in following the expression of this mutated form within tumors and first tests have been already carried out. In conclusion, RIP140 controls expression of major genes involved in genome integrity maintenance and a mutation of this transcriptional coregulator could be responsible for microsatellite instability of some tumors where alterations of MMR genes are not found. Clinical studies on larger cohorts will be necessary to validate its interest as a marker usable in patient management
Warusavitarne, Janindra. "Analysis of the factors that influence the biological behaviour and response to chemotherapy in sporadic colorectal carcinomas with microsatellite instability". Thesis, The University of Sydney, 2005. https://hdl.handle.net/2123/27920.
Texto completoLibros sobre el tema "Microsatellites instability"
Millar, Anna L. Frequency estimation of endometrial cancer associated with microsatellite instability and mismatch repair gene defects. Ottawa: National Library of Canada, 1999.
Buscar texto completoGryfe, Robert. Colorectal cancer microsatellite instability. 2001.
Buscar texto completoUmar, A. Lynch Syndrome (HNPCC) and Microsatellite Instability. Ios Pr Inc, 2004.
Buscar texto completoUmar, A. Lynch Syndrome (HNPCC) and Microsatellite Instability Analysis Guidelines, Part 2. IOS Press, 2006.
Buscar texto completoLeahy, Dermot T. Clinicopathological significance of p53, bcl-2 and microsatellite instability in colorectal carcinoma. 1998.
Buscar texto completoCurran, Bernie. Microsatellite instability and loss of heterozygosity distinguish two phenotypes in colorectal carcinoma and identity a subgroup with poor prognosis. 1996.
Buscar texto completoTumor microsatellite instability, mismatch repair deficiency and response to chemotherapy: A molecular reanalysis of randomized adjuvant chemotherapy trials in colon cancer. Ottawa: National Library of Canada, 2001.
Buscar texto completoCapítulos de libros sobre el tema "Microsatellites instability"
Patel, Nisha R., Michael L. Wong, Anthony E. Dragun, Stephan Mose, Bernadine R. Donahue, Jay S. Cooper, Filip T. Troicki et al. "Microsatellite Instability". En Encyclopedia of Radiation Oncology, 502. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_211.
Texto completoAhlquist, Terje C. y Ragnhild A. Lothe. "Microsatellite Instability". En Encyclopedia of Cancer, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_3731-2.
Texto completoAhlquist, Terje C. y Ragnhild A. Lothe. "Microsatellite Instability". En Encyclopedia of Cancer, 2842–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_3731.
Texto completoAhlquist, Terje y Ragnhild A. Lothe. "Microsatellite Instability". En Encyclopedia of Cancer, 2305–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3731.
Texto completoPrasad, Meena A. y Barbara Jung. "Microsatellite Instability and Intestinal Tumorigenesis". En Intestinal Tumorigenesis, 29–53. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19986-3_2.
Texto completoLi, Jinhong y Fan Lin. "Lower Gastrointestinal Tract and Microsatellite Instability". En Handbook of Practical Immunohistochemistry, 423–33. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-8062-5_25.
Texto completoChen, Guoli, Jianhong Li, Zongming Eric Chen, Jinhong Li y Fan Lin. "Lower Gastrointestinal Tract and Microsatellite Instability (MSI)". En Handbook of Practical Immunohistochemistry, 701–14. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-83328-2_29.
Texto completoElfimova, Natalia, Wafa Amer y Margarete Odenthal. "Analysis of Microsatellite Instability by Microfluidic-Based Electrophoresis". En Methods in Molecular Biology, 287–96. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-029-8_25.
Texto completoViana-Pereira, Marta, Chris Jones y Rui Manuel Reis. "Pediatric High-Grade Glioma: Role of Microsatellite Instability". En Pediatric Cancer, Volume 3, 205–10. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-4528-5_22.
Texto completoLiu, Yuqing, Weiwen Wang, Chuan-Xian Ren y Dao-Qing Dai. "MetaCon: Meta Contrastive Learning for Microsatellite Instability Detection". En Medical Image Computing and Computer Assisted Intervention – MICCAI 2021, 267–76. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-87237-3_26.
Texto completoActas de conferencias sobre el tema "Microsatellites instability"
Pirini, Francesca, Luigi Pasini, Sara Ravaioli, Gianluca Tedaldi, Emanuela Scarpi, Giorgia Marisi, Chiara Molinari, Daniele Calistri, Alessandro Passardi y Paola Ulivi. "Abstract 5296: Instability of non standard microsatellites in metastatic colorectal cancer patients treated with a bevacizumab based chemotherapy". En Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-5296.
Texto completoSingh, Sukhi, Janez Kokosar, Jan Otonicar y Luka Ausec. "179 Profiling microsatellite instability using RNA sequencing data". En SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0179.
Texto completoPaulose, Assoc Prof Roopa, Prof Roopa Paulose, Divya Ail, Shital Biradar y K. Sundaram. "PTU-054 Microsatellite instability in stage II colorectal carcinoma". En British Society of Gastroenterology, Annual General Meeting, 4–7 June 2018, Abstracts. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-bsgabstracts.395.
Texto completoKIM, Jee Hung, Chang-gon Kim, Joong Bae Ahn, MinKyu Jung, Seung Hoon Beom, Joo Hoon Kim, Soo Jin Heo y Sang Joon Shin. "Abstract 808: Microsatellite instability in metastatic colorectal cancer (mCRC)". En Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-808.
Texto completoSamara, Katerina D., Eirini Neofytou, Nikolaos Tzanakis, Alexandros D. Karatzanis, Dimitra Papandrinopoulou, Nikolaos Siafakas y Eleni G. Tzortzaki. "Perforin Expression In COPD Patients With Microsatellite DNA Instability". En American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3879.
Texto completoBond, Catherine E., Aarti Umapathy, Ron L. Buttenshaw, Leesa Wockner, Barbara A. Leggett y Vicki LJ Whitehall. "Abstract 106: Chromosomal instability inBRAFmutant, microsatellite stable colorectal cancers". En Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-106.
Texto completoWang, Huilin, Jiang Zhu, Guanbiao Liang, Yongyong Wang, Jianji Guo, Min Zheng, Qin Fu y Weiwei Shi. "Abstract 4316: Frequent microsatellite instability in Chinese solid tumor patients". En Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-4316.
Texto completoSussman, Daniel A., Shivali Berera, Peter Hosein, Feng Miao, Tulay Koru-Sengul, Jacob McCauley, Erin Kobetz-Kerman, Olveen Carrasquillo y Maria T. Abreu. "Abstract B45: Microsatellite instability among disenfranchised minority colorectal cancer patients". En Abstracts: Sixth AACR Conference: The Science of Cancer Health Disparities; December 6–9, 2013; Atlanta, GA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7755.disp13-b45.
Texto completoAbbas, Basman F., Ahmed S. Abood, Ghufran H. Abed, Nidhal A. Mohammad y Alaa D. Kadhim. "BAT26 marker as detector of microsatellite instability in breast tumors". En 1ST SAMARRA INTERNATIONAL CONFERENCE FOR PURE AND APPLIED SCIENCES (SICPS2021): SICPS2021. AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0121227.
Texto completoKurata, K., M. Kubo, H. Mori, H. Kawaji, Y. Motoyama, L. Kuroki, M. Yamada, K. Kaneshiro, M. Kai y M. Nakamura. "Abstract P1-06-11: Microsatellite instability in triple negative breast cancers". En Abstracts: 2018 San Antonio Breast Cancer Symposium; December 4-8, 2018; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p1-06-11.
Texto completoInformes sobre el tema "Microsatellites instability"
Baranovskaya, Svetlana. Microsatellite and Chromosomal Instability in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, julio de 2004. http://dx.doi.org/10.21236/ada430384.
Texto completoBaranovskaya, Svetlana. Microsatellite and Chromosomal Instability in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, julio de 2003. http://dx.doi.org/10.21236/ada418690.
Texto completoMichelmore, Richard, Eviatar Nevo, Abraham Korol y Tzion Fahima. Genetic Diversity at Resistance Gene Clusters in Wild Populations of Lactuca. United States Department of Agriculture, febrero de 2000. http://dx.doi.org/10.32747/2000.7573075.bard.
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