Literatura académica sobre el tema "Metaflammation"
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Artículos de revistas sobre el tema "Metaflammation"
Wei, Lisha, Yan-Yan Zheng, Jie Sun, Pei Wang, Tao Tao, Yeqiong Li, Xin Chen et al. "GGPP depletion initiates metaflammation through disequilibrating CYB5R3-dependent eicosanoid metabolism". Journal of Biological Chemistry 295, n.º 47 (10 de septiembre de 2020): 15988–6001. http://dx.doi.org/10.1074/jbc.ra120.015020.
Texto completoEmbgenbroich, Maria, Hendrik J. P. van der Zande, Leonie Hussaarts, Jonas Schulte-Schrepping, Leonard R. Pelgrom, Noemí García-Tardón, Laura Schlautmann et al. "Soluble mannose receptor induces proinflammatory macrophage activation and metaflammation". Proceedings of the National Academy of Sciences 118, n.º 31 (29 de julio de 2021): e2103304118. http://dx.doi.org/10.1073/pnas.2103304118.
Texto completoHotamisligil, Gökhan S. "Inflammation, metaflammation and immunometabolic disorders". Nature 542, n.º 7640 (febrero de 2017): 177–85. http://dx.doi.org/10.1038/nature21363.
Texto completoFuruhashi, Masato, Shutaro Ishimura, Hideki Ota y Tetsuji Miura. "Lipid Chaperones and Metabolic Inflammation". International Journal of Inflammation 2011 (2011): 1–12. http://dx.doi.org/10.4061/2011/642612.
Texto completoMuskiet, Frits A. J., Pedro Carrera-Bastos, Leo Pruimboom, Alejandro Lucia y David Furman. "Obesity and Leptin Resistance in the Regulation of the Type I Interferon Early Response and the Increased Risk for Severe COVID-19". Nutrients 14, n.º 7 (26 de marzo de 2022): 1388. http://dx.doi.org/10.3390/nu14071388.
Texto completoEgger, Garry y John Dixon. "Obesity and chronic disease: always offender or often just accomplice?" British Journal of Nutrition 102, n.º 8 (18 de mayo de 2009): 1238–42. http://dx.doi.org/10.1017/s0007114509371676.
Texto completoTufanli, Ozlem, Pelin Telkoparan Akillilar, Diego Acosta-Alvear, Begum Kocaturk, Umut Inci Onat, Syed Muhammad Hamid, Ismail Çimen, Peter Walter, Christian Weber y Ebru Erbay. "Targeting IRE1 with small molecules counteracts progression of atherosclerosis". Proceedings of the National Academy of Sciences 114, n.º 8 (30 de enero de 2017): E1395—E1404. http://dx.doi.org/10.1073/pnas.1621188114.
Texto completoNapitupulu, Rosalia E., Anna Meiliana y Andi Wijaya. "Correlation of Progranulin, Granulin, Adiponectin and Vaspin with Metaflammation (hs-CRP) in Indonesian Obese Men". Indonesian Biomedical Journal 5, n.º 2 (1 de agosto de 2013): 107. http://dx.doi.org/10.18585/inabj.v5i2.59.
Texto completoPaccoud, Romain, Céline Saint-Laurent, Enzo Piccolo, Mylène Tajan, Alizée Dortignac, Ophélie Pereira, Sophie Le Gonidec et al. "SHP2 drives inflammation-triggered insulin resistance by reshaping tissue macrophage populations". Science Translational Medicine 13, n.º 591 (28 de abril de 2021): eabe2587. http://dx.doi.org/10.1126/scitranslmed.abe2587.
Texto completoKanbay, Mehmet, Aslihan Yerlikaya, Alan A. Sag, Alberto Ortiz, Masanari Kuwabara, Adrian Covic, Andrzej Wiecek, Peter Stenvinkel y Baris Afsar. "A journey from microenvironment to macroenvironment: the role of metaflammation and epigenetic changes in cardiorenal disease". Clinical Kidney Journal 12, n.º 6 (18 de septiembre de 2019): 861–70. http://dx.doi.org/10.1093/ckj/sfz106.
Texto completoTesis sobre el tema "Metaflammation"
Delépine, Chloé. "L’Interleukine 34 produite dans le pancréas, régule l’inflammation locale". Electronic Thesis or Diss., Angers, 2023. https://dune.univ-angers.fr/documents/dune17687.
Texto completoInterleukin 34 (IL-34) is a cytokine described as a second ligand for the M-CSF receptor, CD115. IL-34 has been identified as predominantly produced in the skin and brain, where it is involved in the maintenance of Langerhans cells and microglia. IL-34 has been identified as overexpressed in several inflammatory pathologies. In our study, we observed a constitutive expression of IL-34 in human and murine Langerhans islets. Analysis of Il34 gene invalidation effects in β cells in a mouse model (IL-34Δpdx) highlighted a role for the cytokine in local immunity.As they aged, IL-34Δpdx mice showed impaired glucose tolerance associated with more pronounced local inflammation than control mice. This phenotype is exacerbated when mice are subjected to a high-fat diet. Insulin secretion from the islets of control mice is impaired in the presence of pro-inflammatory cytokines. The addition of IL-34 restores insulin release. Moreover, IL-34 production is increased in type 2 diabetic patients.These results support the idea of a protective role for IL-34 locally in pancreatic islets in a chronic inflammatory context
Capítulos de libros sobre el tema "Metaflammation"
"9 Metaflammation: eine Entzündung ausgelöst durch Nährstoffspeicherungen". En Orale Präventivmedizin, editado por René B. A. Sanderink, Heinz H. Renggli y Ulrich P. Saxer. Stuttgart: Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/b-0042-184319.
Texto completoActas de conferencias sobre el tema "Metaflammation"
Yoshimura, Tania M., Ilka T. Kato, Alessandro M. Deana y Martha S. Ribeiro. "Treating metabolic syndrome’s metaflammation with low level light therapy: preliminary results". En SPIE BiOS, editado por Michael R. Hamblin, James D. Carroll y Praveen Arany. SPIE, 2014. http://dx.doi.org/10.1117/12.2040901.
Texto completoGhadimi, D., A. Nielsen, M. Farghaly Yoness Hassan, R. Fölster-Holst, M. de Vrese y KJ Heller. "Modulation of GSK – 3β/β – catenin cascade by commensal bifidobateria plays an important role for the inhibition of metaflammation-related biomarkers in response to LPS or non-physiological concentrations of fructose: An in vitro study". En 47. Jahrestagung der Gesellschaft für Gastroenterologie in Bayern e.V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688867.
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