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Libros sobre el tema "METABOLISMO BASALE"

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Publicado: 11 de marzo de 2023

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1

Baik, Sonya A. Catecholamines and basal metabolism in the myocardium. Ottawa: National Library of Canada, 1986.

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2

Reeves, Sue L. Biological variation in basal metabolic rate and energy metabolism. Oxford: Oxford Brookes University, 1997.

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3

Schofield, W. N. Basal metabolic rate: Review and prediction, together with an annotated bibliography of source material. London: J. Libbey, 1985.

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4

Measuring metabolic rates: A manual for scientists. Oxford: Oxford University Press, 2008.

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5

Abbott, Maude E. The determination of basal metabolism by the "respiratory-valve and spirometer method" of indirect calorimetry with an observation on a case of polyeythaemia with splenomegaly / by Maude E. Abbott. [Canada?: s.n., 1996.

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6

Human energetics in biological anthropology. Cambridge: Cambridge University Press, 1995.

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7

S, Oh Man, ed. Water, electrolyte, and acid-base metabolism: Diagnosis and management. 2a ed. Philadelphia: J.B. Lippincott, 1989.

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8

Michael, Tepper James, Abercrombie Elizabeth D y Bolam J. P, eds. GABA and the basal ganglia: From molecules to systems. Amsterdam: Elsevier, 2007.

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9

Johnson, Jan. Metabolic balancing: Organizational worksbook : an individual approach of a personal health maintenance program. Palo Alto, Calif: Bottom Line Books, 1988.

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10

Garcia, Oz. The balance: Your personal prescription for super metabolism, renewed vitality, maximum health, instant rejuvenation. New York: Regan Books, 2000.

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11

Garcia, Oz. The balance: Your personal prescription for super metabolism, renewed vitality, maximum health, instant rejuvenation. New York: Regan Books, 2000.

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12

Cholesterol metabolism, LDL, and the LDL receptor. San Diego: Academic Press, 1990.

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13

Extreme measures: The ecological energetics of birds and mammals. Chicago: The University of Chicago Press, 2012.

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14

P, Riederer y Wesemann W, eds. Parkinson's disease: Experimental models and therapy. Wien: Springer-Verlag, 1995.

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15

Kjell, Fuxe y Wenner-Grenska samfundet, eds. Trophic regulation of the basal ganglia: Focus on dopamine neurons. Oxford, OX, UK: Pergamon, 1994.

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16

Norbert, Heisler, ed. Mechanisms of systemic regulation: Acid-base regulation, ion-transfer, and metabolism. Berlin: Springer, 1995.

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17

Siegal, Sanford. Is Your Thyroid Making You Fat. New York: Grand Central Publishing, 2009.

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18

Miller, Robert, 1943 Aug. 29- y Wickens J, eds. Brain dynamics and the striatal complex. Amsterdam, Netherlands: Harwood Academic, 2000.

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19

J, O'Donovan D., ed. Nutritional and acid-base aspects of amino acid metabolism: 7th International Ammoniagenesis Workshop, Galway, May 20-23, 1996. Basel: Karger, 1997.

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20

Adrogué, Horacio J. Acid-base. Houston, Tex: Libra & Gemini Publications, 1991.

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21

E, Wesson Donald, ed. Acid-base. Boston: Blackwell Scientific Publications, 1994.

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22

Is your thyroid making you fat: 28 days to a life-changing diagnosis. New York: Warner Books, 2000.

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23

Willatts, Sheila M. Lecture notes on fluid and electrolyte balance. 2a ed. Oxford: Blackwell Scientific, 1987.

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24

Brown, Jessy M. y Francesco Serra. Imparare a Massimizzare il Vostro Metabolismo: Perdere Peso Accelerando la Combustione Di Calorie, Perdere Peso Velocemente con un Metabolismo Basale Ultra-Potente. Independently Published, 2019.

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25

Baiamonte, Salvatore. Dieta Del Dr. Salvatore Baiamonte: Il Famoso Metodo Del Dr. Baiamonte, Che Ha Come Base Principale il Metabolismo Basale, Trattato in Maniera Semplice. Independently Published, 2017.

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26

Harris, James Arthur. Biometric Study of Basal Metabolism in Man. Creative Media Partners, LLC, 2018.

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27

Harris, James Arthur. Biometric Study of Basal Metabolism in Man. Creative Media Partners, LLC, 2018.

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28

Harris, James Arthur. Biometric Study of Basal Metabolism in Man. Creative Media Partners, LLC, 2018.

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29

Harris, James Arthur. Biometric Study of Basal Metabolism in Man. Creative Media Partners, LLC, 2018.

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30

Harris, James Arthur. A Biometric Study of Basal Metabolism in Man. Franklin Classics Trade Press, 2018.

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31

Harris, James Arthur. A Biometric Study of Basal Metabolism in Man. Franklin Classics Trade Press, 2018.

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32

Harris, James Arthur. A Biometric Study of Basal Metabolism in Man. Franklin Classics, 2018.

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33

Hatfield, Anthea. Metabolism. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199666041.003.0024.

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This chapter tells you how homeostasis in the body is achieved. Contributing factors such as stress, hormones, and the automatic nervous system are integrated into the discussion in a thoughtful way. The problem of cold postoperative patients is thoroughly referenced to modern investigation. Diabetes, how surgery destabilizes diabetics, and how to use insulin is explained. Malignant hyperthermia, thyroid storm, and acid—base disorders are all problems that can occur in the recovery room and guidelines for the management of these patients are outlined. Hydrogen ions affect haemoglobin and biochemical reactions and can cause acidosis and alkalosis—this chapter outlines how to interpret the blood gas results. How to distinguish between respiratory and metabolic causes of acid—base disorders is simply and clearly explained.
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34

Lighton, John R. B. Measuring Metabolic Rates: A Manual for Scientists. Oxford University Press, 2021.

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35

Lighton, John R. B. Measuring Metabolic Rates: A Manual for Scientists. Oxford University Press, 2019.

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36

Lighton, John R. B. Measuring Metabolic Rates: A Manual for Scientists. Oxford University Press, Incorporated, 2008.

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37

Tatro, Victoria Kingston. The effects of aerobic training on basal metabolic rate. 1988.

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38

Evans, Pamela Sue. The effects of exercise training on basal metabolic rate. 1986.

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39

Tatro, Victoria Kingston. The effects of aerobic training on basal metabolic rate. 1988.

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40

Evans, Pamela Sue. The effects of exercise training on basal metabolic rate. 1986.

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41

Almeida, Rita y Jessy M. Brown. APRENDER A MAXIMIZAR O SEU METABOLISMO: PERDER PESO ACELERANDO A QUEIMA DE CALORIAS, PERDER PESO RAPIDAMENTE COM UM METABOLISMO BASAL ULTRA-PODEROSO. Independently Published, 2019.

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42

Bolam, J. P., J. M. Tepper y E. D. Abercrombie. GABA and the Basal Ganglia. Elsevier Science & Technology Books, 2007.

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43

Brown, Jessy M. Aprende a Aumentar Al Máximo Tu Metabolismo: Baja de Peso Acelerando la Quema de Calorías, Adelgaza Rápidamente con un Metabolismo Basal Ultra Poderoso. Independently Published, 2019.

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44

Reproducibility of the resting metabolic rate. 1987.

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45

Reproducibility of the resting metabolic rate. 1985.

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46

Reproducibility of the resting metabolic rate. 1987.

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47

Reproducibility of the resting metabolic rate. 1987.

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48

Validity of the doubly labeled water method of measuring energy expenditure with rest and exercise in non-human primates. 1989.

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49

Sedel, Frédéric y Carla E. M. Hollak. Disorders of Thiamine Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0028.

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Thiamine is a water-soluble vitamin acting in the mitochondria as a cofactor for energy metabolism and, in the cytoplasm, in the pentose phosphate biosynthetic pathway. Its transport through the plasma membrane requires two transporters with overlapping functions: THTR1 encoded by SLC19A2, and THTR2 encoded by SLC19A3. Thiamine is transformed into its active form, thiamine pyrophosphate (TPP) by a kinase encoded by the TPK1 gene. Then it may enter the mitochondria through a TPP transporter encoded by SLC25A19. Mutations in SLC19A2 cause thiamine-responsive megaloblastic anemia (TRMA). Mutations in SLC19A3 cause biotin/thiamine–responsive basal ganglia disease. Mutations in SLC25A19 may cause early microcephaly with death in infancy (also called Amish microcephaly) or a later-onset bilateral striatal necrosis with progressive peripheral neuropathy. Recently, mutations in the TPK1 gene have been associated with recurrent encephalopathy with mild lactic acidosis.
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50

Tuschl, Karin, Peter T. Clayton y Philippa B. Mills. Disorders of Manganese Metabolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0045.

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Manganese is an essential trace metal for numerous metalloenzymes. Manganese homeostasis requires tight regulation in vivo and disruption of this balance can lead to manganese overload and subsequent accumulation of manganese in brain, liver, and blood. Mutations in SLC30A10, a cell surface-localized manganese efflux transporter, cause an autosomal recessive hypermanganesemia syndrome with two distinct phenotypes: childhood onset dystonia and adult onset Parkinsonism, associated with chronic liver disease, polycythemia and features of iron depletion. MRI brain appearances are characteristic of Mn deposition with hyperintense basal ganglia on T1-weighted images. Chelation therapy with disodium calcium edetate and iron supplementation effectively lower blood manganese levels, halt liver disease progression and improve neurological symptoms.The inherited form of hypermanganesemia can be distinguished from acquired causes of manganese overload including environmental overexposure and acquired hepatocerebral degeneration in cases of end stage liver disease.
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