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1

Derek, Marsh, ed. Phospholipid bilayers: Physical principles and models. New York: Wiley, 1987.

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2

H, Templer Richard, Leatherbarrow Robin y Royal Society of Chemistry (Great Britain). Biophysical Chemistry Group., eds. Biophysical chemistry: Membranes and proteins. Cambridge, UK: Royal Society of Chemistry, 2002.

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3

von, Heijne Gunnar, ed. Membrane protein assembly. Austin: R.G. Landes, 1997.

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4

Xu, Weihua. Design and development of a pervaporation membrane separation module. Ottawa: National Library of Canada, 2001.

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5

1932-, Osa Tetsuo y Atwood J. L, eds. Inclusion aspects of membrane chemistry. Dordrecht: Kluwer Academic Publishers, 1991.

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6

Frishman, Dmitrij. Structural bioinformatics of membrane proteins. Wien: Springer, 2010.

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7

Norbert, Latruffe, Federation of European Biochemical Societies. y Centre national de la recherche scientifique (France), eds. Dynamics of membrane proteins and cellular energetics. Berlin: Springer-Verlag, 1988.

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8

NATO Advanced Study Institute on Physical Methods on Biological Membranes and Their Model Systems (1982 Altavilla Milicia, Italy). Physical methods on biological membranes and their model systems. New York: Plenum Press, 1985.

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9

Elena, Eizenberg, ed. Membranes and other extendons (p-branes). Singapore: World Scientific, 1995.

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10

Gao, Fei. Proton exchange membrane fuel cells modeling. London: ISTE, 2011.

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11

Zellin, Göran. Growth factors and bone regeneration: Implications of barrier membranes. Göteborg, Sweden: Department of Oral Biochemistry, Faculty of Dentistry, Göteborg University, 1998.

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12

sc, Müller Eberhard Dr y Dahse Ingo, eds. Vom Organismus zum Molekül: Physiologische Prozesse, ihre Modellierung und Beeinflussbarkeit auf verschiedenen Ebenen : Festschrift für Eberhard Müller zum 60. Geburtstag. Jena: Friedrich-Schiller-Universität, 1992.

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13

Lipowsky, Reinhard. The Structure and Conformation of Amphiphilic Membranes: Proceedings of the International Workshop on Amphiphilic Membranes, Jülich, Germany, September 16-18, 1991. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992.

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14

C, Pozrikidis, ed. Modeling and simulation of capsules and biological cells. Boca Raton, Fla: Chapman & Hall/CRC, 2003.

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15

Azarov, I͡A B. Matematicheskai͡a modelʹ vozbudimosti v sisteme ionnykh kanalov aksona kalʹmara. Pushchino: Nauch. t͡sentr biologicheskikh issledovaniĭ AN SSSR, 1990.

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16

Păun, Andrei. Computability of the DNA and cells: Splicing and membrane computing. Choudrant, La: SBEB Pub., 2008.

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17

Ribatti, Domenico. The chick embryo chorioallantoic membrane in the study of angiogenesis and metastasis. Dordrecht: Springer, 2010.

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18

Dimitrievski, Kristian. Monte Carlo simulations of supported biomembranes and protein folding. Göteborg: Göteborg University, Department of Physics, 2006.

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19

Zakrzewska-Trznadel, Grażyna. Procesy membranowe w technologiach jądrowych: Wybrane zagadnienia modelowania transportu masy oraz projektowania systemów rozdzielania. Warszawa: Instytut Chemii i Techniki Jądrowej, 2006.

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20

Jacob, Abraham. Nonlinear, finite strain, finite element model of the mechanics of the posterior tracheal membrane. [Downsview, Ont.]: University of Toronto, Institute for Aerospace Studies, 2003.

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21

Zakrzewska-Trznadel, Grażyna. Procesy membranowe w technologiach jądrowych: Wybrane zagadnienia modelowania transportu masy oraz projektowania systemów rozdzielania. Warszawa: Instytut Chemii i Techniki Jądrowej, 2006.

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22

Lockheed Martin Astronautics (Firm). Flight Systems Division. y United States. National Aeronautics and Space Administration., eds. Membrane Transport Phenomena (MTP): Semi-annual technical progress report, November 1996 - May 1997, contract number: NAS8-40633. [Denver, Colo.]: Lockheed Martin, 1997.

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23

Lockheed Martin Astronautics (Firm). Flight Systems Division. y United States. National Aeronautics and Space Administration., eds. Membrane Transport Phenomena (MTP): Semi-annual technical progress report, May 1996 - November 1996, contract number: NAS8-40633. [Denver, Colo.]: Lockheed Martin, 1997.

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24

Lockheed Martin Astronautics (Firm). Flight Systems Division. y United States. National Aeronautics and Space Administration., eds. Membrane Transport Phenomena (MTP): Semi-annual technical progress report, May 1996 - November 1996, contract number: NAS8-40633. [Denver, Colo.]: Lockheed Martin, 1997.

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25

Lockheed Martin Astronautics (Firm). Flight Systems Division. y United States. National Aeronautics and Space Administration., eds. Membrane Transport Phenomena (MTP): Semi-annual technical progress report, June 1997-October 1997 : contract number NAS8-40633. [Denver, Colo.]: Lockheed Martin, 1997.

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26

National Institute of Child Health & Human Development Research Planning Workshop (1987 Bethesda, Md.). The onset of labor: Cellular and integrative mechanisms : a National Institute of Child Health & Human Development Research Planning Workshop, November 29-December 1, 1987. Ithaca, NY, U.S.A: Publisher and sole distributor, Perinatology Press, 1988.

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27

1941-, Beyreuther K. y Schettler Gotthard, eds. Molecular mechanisms of aging. Berlin: Springer-Verlag, 1990.

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28

Rautenbach, Robert. Procesy membranowe: Podstawy projektowania modu¿o w i instalacji. Warszawa: Wydawn. Naukowo-Techn., 1996.

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29

Cholesterol in membrane models. Boca Raton, Fla: CRC Press, 1992.

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30

Heimburg, Thomas. Thermal Biophysics of Membranes. Wiley-VCH, 2007.

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31

Heimburg, Thomas. Thermal Biophysics of Membranes. Wiley & Sons, Limited, John, 2007.

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32

Heimburg, Thomas. Thermal Biophysics of Membranes. Wiley & Sons, Incorporated, John, 2008.

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33

Bacterial outer membranes as model systems. New York: Wiley, 1987.

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34

Berkowitz, Max L. Biomembrane Simulations. Taylor & Francis Group, 2019.

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35

Berkowitz, Max L. Biomembrane Simulations: Computational Studies of Biological Membranes. Taylor & Francis Group, 2014.

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36

Berkowitz, Max L. Biomembrane Simulations. Taylor & Francis Group, 2021.

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37

Biomembrane Simulations: Computational Studies of Biological Membranes. Taylor & Francis Group, 2019.

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38

Protein secretion: A critical analysis of the vesicle model. New York: J. Wiley, 1985.

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39

(Editor), B. U. Raess y G. Tunnicliff (Editor), eds. The Red Cell Membrane: A Model for Solute Transport (Contemporary Biomedicine). Humana Press, 1990.

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40

(Editor), R. H. Templer y R. J. Leatherbarrow (Editor), eds. Biophysical Chemistry: Membrane and Proteins (Biotechnology Intelligence Unit, 283). Royal Society of Chemistry, 2003.

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41

Osa, T. y J. L. Atwood. Inclusion Aspects of Membrane Chemistry. Springer, 2012.

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42

Osa, T. y J. L. Atwood. Inclusion Aspects of Membrane Chemistry. Springer Netherlands, 2012.

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43

Thompson, Gary O. Flexible membrane wave barrier. 1991.

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44

Cattran, Daniel C. y Heather N. Reich. Membranous glomerulonephritis. Editado por Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0064_update_001.

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It has been clear for several decades from comparison with the rodent model disease Heymann nephritis that membranous glomerulonephritis (MGN) is an immune condition in which antibodies, usually autoantibodies, bind to targets on the surface of podocytes. However, the antigen in Heymann nephritis, megalin, is not present on human podocytes. The first potential antigen was identified by studying rare examples of maternal alloimmunization, leading to congenital membranous nephropathy in the infant caused by antibodies to neutral endopeptidase. More recently, the target of autoantibody formation in most patients with primary MGN has been identified to be the phospholipase A2 receptor, PLA2R. Genome-wide association studies identify predisposing genetic loci at HLADQ and at the locus encoding the autoantigen itself. So antibodies to at least two different molecular targets can cause MGN, and it seems likely that there may be other targets in secondary types of MGN, and possibly haptenized or otherwise modified molecules are implicated in drug- and toxin-induced MGN. Once antibodies are fixed, animal models and human observations suggest that complement is involved in mediating tissue damage. However, immunoglobulin G4, not thought to fix complement, is the predominant isotype in human MGN, and the mechanisms are not fully unravelled. Podocyte injury is known to cause proteinuria. In MGN, antibody fixation or cell damage may stimulate production of extracellular matrix to account for the increased GBM thickness with ‘podocyte type’ basement membrane collagen isoforms, and ultimately cell death and glomerulosclerosis.
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45

Hollow Fiber Membrane Contactors: Module Fabrication, Design and Operation, and Potential Applications. Taylor & Francis Group, 2020.

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46

Sirkar, Kamalesh K., Anil Kumar Pabby, Sumith Ranil Wickramasinghe y Ana Maria Sastre Requena. Hollow Fiber Membrane Contactors: Module Fabrication, Design and Operation, and Potential Applications. Taylor & Francis Group, 2020.

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47

Sirkar, Kamalesh K., Ana Maria Sastre, S. Ranil Wickramasinghe y Anil Kumar Pabby. Hollow Fiber Membrane Contactors: Module Fabrication, Design and Operation, and Potential Applications. Taylor & Francis Group, 2022.

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48

Sirkar, Kamalesh K., Anil Kumar Pabby, Sumith Ranil Wickramasinghe y Ana Maria Sastre Requena. Hollow Fiber Membrane Contactors: Module Fabrication, Design and Operation, and Potential Applications. Taylor & Francis Group, 2020.

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49

Sirkar, Kamalesh K., Anil Kumar Pabby, Sumith Ranil Wickramasinghe y Ana Maria Sastre Requena. Hollow Fiber Membrane Contactors: Module Fabrication, Design and Operation, and Potential Applications. Taylor & Francis Group, 2020.

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50

(Editor), Harold E. Layton y Alan M. Weinstein (Editor), eds. Membrane Transport and Renal Physiology. Springer, 2002.

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