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Libros sobre el tema "Mechanism of action of electroconvulsive therapy"

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Publicado: 14 de marzo de 2023

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1

International Conference on Tumor Necrosis Factor and Related Cytokines (2nd 1989 Napa, Calif.). Tumor necrosis factor: Structure, mechanism of action, role in disease and therapy. Editado por Bonavida Benjamin y Granger Gale. Basel: Karger, 1990.

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2

Hannan, J. M. Abdul. Studies on the efficacy and mechanism of action of tropical plants for diabetes therapy. [S.l: The Author], 2004.

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3

Godfrey, Tunnicliff, Eison Arlene S y Taylor Duncan P, eds. Buspirone: Mechanisms and clinical aspects. San Diego: Academic Press, 1991.

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4

Godfraind, T. Calcium channel blockers. Boston, MA: Birkhauser Verlag, 2003.

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5

A, Hickman John y Tritton Thomas R, eds. Cancer chemotherapy. Oxford: Blackwell Scientific Publications, 1993.

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6

Calcium channel blockers. Basel: Birkhäuser Verlag, 2004.

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7

Lithium: Actions and mechanisms. Washington, DC: American Psychiatric Press, 1996.

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8

N, Herrington Reginald, ed. Biological treatments in psychiatry. Oxford: Oxford University Press, 1990.

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9

Biological treatments in psychiatry. 2a ed. Oxford: Oxford University Press, 1996.

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10

1944-, Yaksh T. L., ed. Anesthesia: Biologic foundations. Philadelphia: Lippincott-Raven, 1998.

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11

A, Montgomery S. y Corn Timothy H, eds. Psychopharmacology of depression. Oxford: Oxford University Press, 1994.

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12

International Symposium on Molecular Aspects of Chemotherapy. (3rd 1991 Gdańsk, Poland). Molecular aspects of chemotherapy: Proceedings of the Third International Symposium on Molecular Aspects of Chemotherapy, Gdansk, Poland, June 19-21, 1991. Berlin: Springer-Verlag, 1992.

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13

L, Faingold Carl y Fromm Gerhard H, eds. Drugs for control of epilepsy: Actions on neuronal networks involved in seizure disorders. Boca Raton, FL: CRC Press, 1992.

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14

1950-, Morstyn George, Foote MaryAnn y Lieschke Graham J, eds. Hematopoietic growth factors in oncology: Basic science and clinical therapeutics. Totowa, N.J: Humana Press, 2004.

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15

C, Dale David y SpringerLink (Online service), eds. Hematopoietic Growth Factors in Oncology. Boston, MA: Springer Science+Business Media, LLC, 2011.

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16

Godfrey, Tunnicliff y Raess B. U, eds. GABA mechanisms in epilepsy. New York: Wiley-Liss, 1991.

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17

M, Pinedo H. y Schornagel J. H, eds. Platinum and other metal coordination compounds in cancer chemotherapy 2. New York: Plenum Press, 1996.

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18

International Symposium on Platinum and Other Metal Coordination Compounds in Cancer Chemotherapy (6th 1991 San Diego, Calif.). Platinum and other metal coordination compounds in cancer chemotherapy. New York: Plenum Press, 1991.

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19

1960-, Ozawa H., Saito Toshikazu, Takahata Naohiko 1932-, Nihon Seishin Shinkei Gakkai y Symposium on Affective Disorders and Neuronal Signal Transduction (1996 : Sapporo-shi, Japan), eds. Signal transduction in affective disorders. Tokyo: Springer, 1998.

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20

Fever and antipyresis: The role of the nervous system. Cambridge: Cambridge University Press, 1995.

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21

W, Thomson Angus, ed. The Molecular biology of immunosuppression. Chichester: Wiley, 1992.

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22

J, Garland C. y Angus James A, eds. The pharmacology of vascular smooth muscle. Oxford: Oxford University Press, 1996.

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23

1946-, Stephens David N., ed. Anxiolytic [Beta]-carbolines: From molecular biology to the clinic. Berlin: Springer-Verlag, 1993.

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24

Ross, Lisa. Electroconvulsive Therapy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190495756.003.0029.

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The anesthetic management of patients who receive electroconvulsive therapy (ECT) for various psychiatric conditions in both the inpatient and the outpatient settings must take into account a number factors, such as its associated physiologic responses, existing comorbidities, medication management, monitoring, complications, and contraindications in order for it to remain a safe procedure. This chapter reviews the indications for ECT, the preprocedure anesthetic evaluation; theories regarding the therapeutic mechanism of action leading to the efficacy of ECT; cerebrovascular, cardiovascular, and neuroendocrine responses and monitoring standards. Furthermore, it discusses the selection of medications for induction, inhalational agents, and muscle relaxants as well as common drug interactions and premedication practices. The chapter culminates with an assessment of the morbidity and mortality associated with ECT both anesthetic and nonanesthetic related.
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25

(Editor), Kenneth S. Korach, Alexander Hillisch (Editor) y K. H. Fritzemeier (Editor), eds. New Molecular Mechanisms of Estrogen Action and Their Impact on Future Perspectives in Estrogen Therapy. Springer, 2004.

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26

Tunnicliff, Godfrey y Arlene S. Eison. Buspirone: Mechanisms and Clinical Aspects. Academic Pr, 1991.

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27

Tunnicliff, Godfrey y Arlene S. Eison. Buspirone: Mechanisms and Clinical Aspects. Academic Pr, 1991.

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28

Cognitive Enhancing Drugs (Milestones in Drug Therapy). Birkhäuser Basel, 2004.

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29

(Editor), Gale Granger, ed. Tumor Necrosis Factor: Structure, Mechanism of Action, Role in Disease and Therapy : 2nd International Conference on Tumor Necrosis Factor and Relat. S. Karger AG (Switzerland), 1990.

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30

Lunenfeld, B. GnRH Analogues: The State of the Art at the Millennium. Informa Healthcare, 1999.

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31

Török, M. Estée, Fiona J. Cooke y Ed Moran. Antiparasitic therapy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0005.

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This chapter provides a systematic summary of antiparasitic agents, grouped by class and mechanism of action. Each summary provides information on the mode of action, resistance mechanisms, pharmacology, and clinical use.
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32

Hematopoietic Growth Factors in Oncology (Cancer Drug Discovery and Development). Humana Press, 2004.

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33

El-Mallakh, Rif S. Lithium: Actions and Mechanisms. Progress in Psychiatry, No. 50. American Psychiatric Press, 1996.

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34

Shugar, David y Wojciech Rode. Molecular Aspects Of Chemotherapy. Springer, 1993.

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35

Shugar, David. Molecular Aspects of Chemotherapy. Springer, 2012.

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36

Molecular Aspects of Chemotherapy. Springer Verlag, 1992.

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37

Borowski, Edward, David Shugar y Wojciech Rode. Molecular Aspects of Chemotherapy: Proceedings of the Third International Symposium on Molecular Aspects of Chemotherapy Gdańsk, Poland June 19-21 1991. Springer London, Limited, 2013.

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38

Morstyn, George, MaryAnn Foote y Graham J. Lieschke. Hematopoietic Growth Factors in Oncology: Basic Science and Clinical Therapeutics. Humana Press, 2012.

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39

Morstyn, George, MaryAnn Foote y Graham J. Lieschke. Hematopoietic Growth Factors in Oncology: Basic Science and Clinical Therapeutics. Humana Press, 2004.

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40

Platinum and Other Metal Coordination Compounds in Cancer Chemotherapy 2. Springer, 1996.

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41

Dale, David C., Gary H. Lyman y Gary Lyman. Hematopoietic Growth Factors in Oncology. Springer, 2012.

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42

Fallon, Marie T. y Nathan I. Cherny. Opioid therapy: optimizing analgesic outcomes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0094.

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Treatment with analgesic drugs is the mainstay of cancer pain management. The major group of drugs used in cancer pain management is the opioid analgesics. During the last 30 years, there has been a dramatic increase in our knowledge of the sites and mechanism of action of the opioids. The development of analytical methods has also been of great importance in facilitating pharmacokinetic studies of the disposition and fate of opioids in patients. More recently, advances in genomic research have indicated the potential importance of pharmacogenetic factors in the response to opioid analgesics. These studies have begun to offer us a better understanding of some of the sources of variation between individuals in their response to opioids and to suggest ways of minimizing some of their adverse effects. This chapter presents a comprehensive discussion of the pre-clinical pharmacology and clinical aspects of opioid analgesia and the principles of opioid administration.
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43

Hedqvist, Per, Sven-Erik Dahlen y Bengt Samuelsson. Leukotrienes As Mediators of Asthma and Inflammation: Basic and Applied Research (Advances in Prostaglandin, Thromboxane, and Leukotriene Research). Lippincott Williams & Wilkins, 1995.

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44

Rauch, Sheila A. M. y Israel Liberzon. Mechanisms of Action in Psychotherapy. Editado por Israel Liberzon y Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0019.

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Therapy at its core is based on learning, and learning at its core is biological. Experience that is not in some way encoded in the brain and/or body is lost. This chapter provides a discussion of mechanisms of therapy research in PTSD in which the goal is to understand how PTSD therapy works. First, the chapter reviews what a mechanism is and how therapeutic mechanisms are examined. It then discusses the importance of therapeutic mechanisms research within the broader realm of mental health research. It focuses on prolonged exposure (PE) therapy for PTSD as an example of application of mechanisms research methodology and begins with the presentation of a theoretical model that builds on previous theory and mechanisms research to date. While much of this model is theoretical, the goal is to show how mechanisms research may apply to clinical practice to improve precision, efficiency, and efficacy.
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45

Wijdicks, Eelco F. M. y Sarah L. Clark. Antimicrobial Therapy for Central Nervous System Infections. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190684747.003.0011.

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Broad-spectrum antibiotics—those that are bactericidal and able to penetrate the blood–brain barrier—must be administered to any patient with a suspected infection of the central nervous system (CNS). Antimicrobials should be selected with consideration of spectrum of activity, pharmacokinetics, mechanism of action, and need to be administered in a timely fashion. This chapter discusses antibiotics that provide good penetration of the central nervous system, and appropriate doses for these infections are outlined. Antiviral therapy for CNS infections is limited but complex. Antimicrobial therapy for fungal meningitis is well defined but rarely indicated. Understanding the spectrum of antimicrobials and dosing is essential knowledge in caring for patients in the neurosciences intensive care unit with CNS infections.
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46

Shorter, Edward y Max Fink. L’Envoi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190881191.003.0014.

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The long history of catatonia is presently resolved into a systemic syndrome, identified by symptoms and signs, verified by tests, and fully resolved by defined means. Our skills in relieving catatonia are as successful as our treatment for neurosyphilis. Catatonia is best seen as a systemic medical, not a psychiatric disorder. Unresolved fear is a core feature in its pathophysiology. There are difficulties distinguishing catatonia from melancholia; the prompt resolution of both with induced seizures presents a challenge in understanding them. Are they related? Melancholia’s neuroendocrine abnormalities offer direction to neuroscience studies of catatonia and of the mechanism of electroconvulsive therapy (ECT).
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47

Garland, C. J. The Pharmacology of Vascular Smooth Muscle. Oxford University Press, 1996.

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48

Stephens, David N. Anxiolytic B-Carbolines: From Molecular Biology to the Clinic (Psychopharmacology Series). Springer-Verlag Telos, 1993.

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49

Foo, Joanne, Benazir Saleem y Philip G. Conaghan. Analgesics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0078.

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Pain is one of the commonest presenting symptoms of musculoskeletal disorders and may be one the hardest to treat successfully. The available analgesic options provide different modes of action and their ranks continue to expand with new agents, some with multiple target action. This chapter reviews currently available analgesics (paracetamol and opioids) used for managing musculoskeletal pain and the agents used for neuropathic pain, including their mechanism of action, pharmacokinetics and side effects. The role of neuroleptic agents is reviewed, and a brief outline of some newer therapies for the treatment of pain such as tapentadol, and a potential therapy, anti-nerve growth factor monoclonal antibodies.
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50

TENS-like devices. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199673278.003.0011.

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TENS-like devices deliver electrical currents across the intact surface of the skin using pulse generators with technical output specifications that differ from a standard TENS device. Technological advances have resulted in reductions in the size and cost of electrotherapeutic devices with increasing varieties of self-administered hand-held TENS-like devices available to practitioners and the general public. The diversity of TENS-like devices available on the market makes synthesizing evidence difficult. The purpose of this chapter is to categorize TENS-like devices and briefly overview the characteristics, mechanism of action, and effectiveness of various TENS-like devices. The chapter covers high-voltage pulsed (Galvanic) current, microcurrent electrical therapy, low-intensity transcutaneous cranial electrical stimulation, transcutaneous spinal electroanalgesia, transcutaneous piezoelectric current, non-invasive interactive neurostimulation, action potential simulation and H-wave therapy, and transcutaneous electrical acupoint stimulation.
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