Literatura académica sobre el tema "Mccadden"

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Artículos de revistas sobre el tema "Mccadden"

1

Espino Martín, Javier. "Carlos J. McCadden y Miguel del Castillo Negrete, La clase media en México: [reseña]". Estudios: filosofía, historia, letras 14, n.º 119 (2016): 216. http://dx.doi.org/10.5347/01856383.0119.000272132.

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Beuchot, Mauricio. "Carlos J. McCadden y José Manuel Orozco Garibay, Decir y contradecir : el principio más firme de la filosofía primera : [reseña]". Estudios: filosofía, historia, letras 18, n.º 133 (2020): 177. http://dx.doi.org/10.5347/01856383.0133.000299237.

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3

Wolff, Dj. "Jennifer McCadney, Former Trade Counsel, US House of Representatives Committee on Ways & Means". Global Trade and Customs Journal 6, Issue 5 (1 de mayo de 2011): 249–52. http://dx.doi.org/10.54648/gtcj2011033.

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Cummings, Jeffrey L. "Commentary on: L-Methylfolate, Methylcobolamine, and N-Acetylcysteine in the Treatment of Alzheimer's Disease-Related Cognitive Decline". CNS Spectrums 15, S1 (enero de 2010): 7. http://dx.doi.org/10.1017/s1092852900027607.

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Drs. McCaddon and Hudson provide a thorough review of the multiple ways in which vitamin B12, vitamin B6, folate, and homocysteine (Hey) are implicated in the pathogenesis of Alzheimer's disease (AD). They noted that Hey is more often elevated in AD and in mild cognitive impairment (MCI) than in cognitively healthy elderly; phosphatases needed to limit tau hyperphosphoryalation and neurofibrillary tangle formation require methylation and are dependent on folate and methylation status; cerebrospinal fluid (CSF) tau levels correlated with markers of methylation status; reduced folate and B12 levels lead to increase β-secretase and pesenilin 1 (PS1) actions leading to greater amyloid-β production in in vitro models; elevated Hey levels in rates are associated with increased PS1 activity and spatial memory deficits that are reversed following treatment with B12 and folate; raised Hey levels in vitro increase amyloid-β protein neurotoxicity; methylation impacts transmitters and transmitter function relevant to AD; in cultured neurons, Hey induces injury in DNA and stimulates cell death pathways. B12 deficiency leads to accumulation of methyl malonic acid, which inhibits mitochondrial function and may compromise energy generation and impair maintenance of synaptic plasticity. Methylation abnormalities result in excessive generation of reactive oxygen species that contribute importantly to cell injury. Biomarkers of oxidative injury, such as isoprostanes, are elevated in AD and suggest excess oxidation. Thus, there are multiple pathways through which deficient methylation may contribute to AD. In some cases, the observations are derived from models with B12 or folate deficiency and some in vitro observations have not been tested in in vivo models. There are no biomarkers specific to some of the pathways implicated and the magnitude of the impact of the deficiency or its treatment has not been established for all the relationships. Two open-label experiments in early- and late-stage AD patients have suggested benefit.
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Jahnson, Christa. "Charles Dickens, Bleak House. Original + Ungekürzt. Englische Originalfassung. Vollständige Lesung + PDF-Textdatei. Sprecher: Robert Whitfield: 2 mp3-CDs, 1973 Minuten (Berlin: Bertz + Fischer, 2009). € 19.90. E.M. Forster: A Room with a View. Original + Ungekürzt. Englische Originalfassung. Vollständige Lesung. Sprecherin: Wanda McCaddon. 1 mp3-CD, 393 Minuten (Berlin: Bertz + Fischer, 2009). € 19.90. James Joyce: Dubliners. Original + Ungekürzt. Englische Originalfassung. Vollständige Lesung. Sprecher: Ralph Cosham. 1 mp3-CD, 369 Minuten (Berlin: Bertz + Fischer, 2009). € 19.90. Great American Stories by S. Crane, O. Henry, S.O. Jewett, E.A. Poe, M. Twain. Original + Ungekürzt + PDF-Textdatei. Englische Originalfassung. Vollständige Lesung. Sprecher: Ralph Cosham. 1 mp3-CD, 369 Minuten (Berlin: Bertz + Fischer, 2009). € 19.90." Archiv für das Studium der neueren Sprachen und Literaturen, n.º 2 (20 de diciembre de 2010). http://dx.doi.org/10.37307/j.1866-5381.2010.02.59.

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Libros sobre el tema "Mccadden"

1

Dietz, William C. McCade for hire. New York: Berkley Books, 2004.

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Dietz, William C. McCade on the run. New York: Ace Books, 2005.

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3

Lusby, Jim. Flashback (Carl McCadden Mysteries). Orion Media, 2003.

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4

Lusby, Jim. Flashback (A Carl McCadden Mystery). Orion mass market paperback, 1997.

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5

Lusby, Jim. Making the Cut (Carl McCadden Mysteries). Orion Books Limited, 2003.

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6

Lusby, Jim. Crazy Man Michael. Orion Books Limited, 2003.

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7

Lusby, Jim. Crazy Man Michael. Orion, 2000.

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8

Lusby, Jim. Crazy Man Michael. Orion, 2000.

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9

Crazy man Michael. London: Gollancz, 2000.

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10

A waste of shame. London: Orion, 2001.

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Capítulos de libros sobre el tema "Mccadden"

1

Davis, Charles T. y Henry Louis Gates. "The Slave Narratives as History". En The Slave’s Narrative, 35–154. Oxford University PressNew York, NY, 1991. http://dx.doi.org/10.1093/oso/9780195066562.003.0002.

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Abstract One of the most significant research projects generated by the Depression was the Federal Writers Project’s Slave Narrative Collection. Drawing upon precedents established by Lawrence Reddick and John McCade, the F.W.P. collected thousands of oral narratives during the nineteen thirties from black men and women who had once been enslaved. Used as sources of folklore by Benjamin Botkin in his collection Lay My Burden Down ( 1945), the worth of these narratives as historical data has remained in question until the last decade. Sterling Brown’s memorandum to that project’s interviewers reveals the care encouraged by the project’s directors to make those data collected of potential use to historians, linguists, and to literary scholars. One such literary scholar, John Widman, compares the use of language in these narratives to that used by Charles Chesnutt in his fictions. Essays by historians are sustained responses to the published edition of these oral tales, edited by the historian, George P. Rawick, and published as a “composite history” in over forty-volumes entitled The American Slave.
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