Bibliografías temáticas / Lymphocyte precursors, chronic inflammation, autoimmune diseases

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Literatura académica sobre el tema "Lymphocyte precursors, chronic inflammation, autoimmune diseases"

Autor: Grafiati

Publicado: 11 de marzo de 2023

Última modificación: 31 de julio de 2025

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Artículos de revistas sobre el tema "Lymphocyte precursors, chronic inflammation, autoimmune diseases"

Martin Calderon, L., and J. Pope. "AB0721 From Undifferentiated Connective Tissue Disease to Identifiable Disease: Precursors of Systemic Sclerosis and Systemic Lupus Erythematosus." Annals of the Rheumatic Diseases 81, Suppl 1 (2022): 1487.3–1488. http://dx.doi.org/10.1136/annrheumdis-2022-eular.4241.

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BackgroundThe pathogenesis of systemic lupus erythematosus and systemic sclerosis is characterized by derangements of the innate and adaptive immune systems, and inflammatory pathways leading to autoimmunity, chronic cytokine production, and chronic inflammation. Diagnosis is rooted in meeting established criteria. However, in pre-clinical states criteria is not fulfilled but biochemical and autoimmune derangements are present. Understanding the underlying processes responsible for disease pathogenesis in pre-clinical states, which place patients at increased risk for the development of establ

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Sesti-Costa, Renata, Carolina Lanaro, Dulcinéia Martins de Albuquerque, Sara T. Olalla Saad, and Fernando Ferreira Costa. "Sickle Cell Disease Patients Have Altered Number and Function of Dendritic Cells." Blood 134, Supplement_1 (2019): 3569. http://dx.doi.org/10.1182/blood-2019-129854.

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Dendritic cells (DCs) are the sentinels of the immune system able to recognize pathogen- and damage-associated molecular patterns (PAMPs and DAMPs), promoting a bridge between the innate and adaptive immune systems. They form a heterogeneous group of cells with different development, phenotype and functions, and they are mainly classified in conventional DCs 1 (cDC1) and 2 (cDC2), plasmacytoid DCs (pDC) and inflammatory DCs. Changes in the development of DCs, in the ratio of the subsets or in the maturation and activation can impair immunity or tolerance, inducing susceptibility to infections,

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Weinberg, J. Brice, David J. DiLillo, Yohei Iwata, et al. "Chronic Lymphocytic Leukemia Shares a Common Cellular Origin with Regulatory B10 Cells." Blood 118, no. 21 (2011): 286. http://dx.doi.org/10.1182/blood.v118.21.286.286.

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Abstract Abstract 286 Background: The cell of origin of CLL is unknown. Researchers have proposed various B cell subsets as the normal counterparts based on surface marker similarities or Ig gene utilization comparisons of normal and CLL cells. Regulatory B lymphocytes (“B10” cells), with the capacity to produce IL-10, negatively regulate T cell, B cell, and mononuclear phagocyte function. CLL patients are immunosuppressed with abnormalities in both humoral and cellular immunity. B10 cells have a phenotype similar to CLL cells (CD24hiCD27+CD5+CD19+). B10 cells are increased in autoimmune mice

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Gray, Kathryn J., and Julie E. Gibbs. "Adaptive immunity, chronic inflammation and the clock." Seminars in Immunopathology 44, no. 2 (2022): 209–24. http://dx.doi.org/10.1007/s00281-022-00919-7.

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Abstract The adaptive arm of the immune system facilitates recognition of specific foreign pathogens and, via the action of T and B lymphocytes, induces a fine-tuned response to target the pathogen and develop immunological memory. The functionality of the adaptive immune system exhibits daily 24-h variation both in homeostatic processes (such as lymphocyte trafficking and development of T lymphocyte subsets) and in responses to challenge. Here, we discuss how the circadian clock exerts influence over the function of the adaptive immune system, considering the roles of cell intrinsic clockwork

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Tomczyńska, Małgorzata, and Joanna Saluk-Bijak. "The mutual cooperation of blood platelets and lymphocytes in the development of autoimmune thyroid diseases." Acta Biochimica Polonica 65, no. 1 (2018): 17–24. http://dx.doi.org/10.18388/abp.2017_2321.

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Autoimmune thyroid diseases include several distinct clinical entities and mainly concern Graves` disease and Hashimoto's thyroiditis. An incompetent immune response directed against the body’s own tissues and the production of antibodies against specific cell antigens, accompanied by chronic inflammation occur in autoimmune thyroid diseases. The autoimmune process is induced by difficult to identify genetic and environmental factors, and generates the development of concomitant diseases of other systems. The inflammatory mediators, high level of thyroid hormones, lymphocyte activation and oth

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Meller, Agnieszka, Wioletta Pawlukowska, Karolina Machowska-Sempruch, and Masztalewicz Marta. "Spectrum of Autoimmune Diseases—Chronic Lymphocytic Inflammation with Pontine Perivascular Enhancement Responsive to Steroids (CLIPPERS)—Clinical Case." Medicina 59, no. 3 (2023): 549. http://dx.doi.org/10.3390/medicina59030549.

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Chronic lymphocytic inflammation with pontine perivascular enhancement responsive to steroids (CLIPPERS) syndrome is a rare inflammatory disease of an undetermined aetiology. The condition is characterised by a range of clinical manifestations generally associated with damage to brainstem structures, the cerebellum, with characteristic magnetic resonance imaging (MRI) findings. The main feature is a good clinical and radiological response to glucocorticosteroid (GCS)-based immunosuppressive treatment. The diagnosis of CLIPPERS is difficult and requires extensive differential diagnosis. A speci

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Hwang, Il-Young, Chung Park, Kathleen Harrison, and John H. Kehrl. "TLR4 signaling augments B lymphocyte migration and overcomes the restriction that limits access to germinal center dark zones." Journal of Experimental Medicine 206, no. 12 (2009): 2641–57. http://dx.doi.org/10.1084/jem.20091982.

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B lymphocyte–intrinsic Toll-like receptor (TLR) signals amplify humoral immunity and can exacerbate autoimmune diseases. We identify a new mechanism by which TLR signals may contribute to autoimmunity and chronic inflammation. We show that TLR4 signaling enhances B lymphocyte trafficking into lymph nodes (LNs), induces B lymphocyte clustering and interactions within LN follicles, leads to sustained in vivo B cell proliferation, overcomes the restriction that limits the access of nonantigen-activated B cells to germinal center dark zones, and enhances the generation of memory and plasma cells.

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Wongkar, Martini, Handoko Lowis, Sarah M. Warouw, Julius Lolombulan, and Stefanus Gunawan. "Blood count to determine chronic inflammation severity in obese adolescents." Paediatrica Indonesiana 60, no. 1 (2020): 6–12. http://dx.doi.org/10.14238/pi60.1.2020.6-12.

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Background Obesity is a growing public health problem of rapidly increasing prevalence in developing countries. Chronic low-grade inflammation plays a key role in the pathophysiology of obesity. Blood count values and ratios have been used as markers of inflammatory diseases. These parameters may be useful to determine the severity of chronic inflammation in obese children. Objective To determine if red blood cell distribution width (RDW), neutrophil-to-lymphocyte ratio (NLR), mean platelet volume (MPV), platelet distribution width (PDW), and platelet-to-lymphocyte ratio (PLR) can be use

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Emmanuel K, Mugisha. "Adaptive Immunity and Autoimmune Disease: Mechanisms, Pathogenesis, and Therapeutic Approaches." NEWPORT INTERNATIONAL JOURNAL OF BIOLOGICAL AND APPLIED SCIENCES 5, no. 3 (2024): 44–48. https://doi.org/10.59298/nijbas/2024/5.3.444811.

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Autoimmune diseases arise when the adaptive immune system mistakenly targets self-antigens, leading to chronic inflammation and tissue damage. This review explores the mechanisms by which adaptive immunity, particularly T and B lymphocyte responses, contributes to the development and persistence of autoimmune diseases. It examines the underlying factors that influence autoreactivity, including genetic susceptibility, environmental triggers, and breakdowns in immune tolerance. Additionally, we discuss the role of T cells, B cells, and autoantibodies in various autoimmune diseases, with emphasis

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Conti, Pio, Luisa Stellin, Alesssandro Caraffa, et al. "Advances in Mast Cell Activation by IL-1 and IL-33 in Sjögren’s Syndrome: Promising Inhibitory Effect of IL-37." International Journal of Molecular Sciences 21, no. 12 (2020): 4297. http://dx.doi.org/10.3390/ijms21124297.

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Sjögren’s syndrome (SS) is a chronic autoimmune inflammatory disease that affects primarily older women and is characterized by irreversible damage of the exocrine glands, including tear (xerophthalmia) and salivary glands (xerostomia). Secretory glands lose their functionality due to the infiltration of immune cells, which produce cytokines and cause inflammation. Primary SS is characterized by dry syndrome with or without systemic commitment in the absence of other pathologies. Secondary SS is accompanied by other autoimmune diseases with high activation of B lymphocytes and the production o

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Tesis sobre el tema "Lymphocyte precursors, chronic inflammation, autoimmune diseases"

francesca, moretta. "ANALYSIS OF A “NOVEL” SUBSET OF CD34+ HAEMATOPOIETIC PRECURSORS IN PERIPHERAL BLOOD OF PATIENTS WITH CHRONIC INFLAMMATORY DISEASES." Doctoral thesis, 2019. http://hdl.handle.net/11562/995037.

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Background: in chronic inflammatory disorders, both infectious and non infectious, a novel population of CD34+ hemopoietic precursors is detectable in high proportions in peripheral blood (PB). These precursors display peculiar phenotypic characteristics, namely the expression of DNAM-1 (bright), an activating receptor expressed by natural killer (NK) cells and CXCR4, a chemokine receptor that suggests their recent exit from bone marrow (BM). In healthy donors (HD), these precursors are virtually undetectable in PB while they represent 10-15% of BM CD34+ cells. It is conceivable that, in chron

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