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Libros sobre el tema "Ischemia Etiology"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 29 de enero de 2023

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1

Vukovic, Dmitry. Myocardial ischemia: Causes, symptoms and treatment. New York: Nova Biomedical Books, 2010.

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2

1938-, Rosen Michael R. y Palti Yoram, eds. Lethal arrhythmias resulting from myocardial ischemia and infarction: [proceedings of the Second Rappaport Symposium]. Boston: Kluwer Academic Publishers, 1989.

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3

Coronary circulation in nonsmokers and smokers. New York: Nova Science Publishers, 2008.

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4

Zoppo, Gregory J. Del. Innate inflammation and stroke. Editado por New York Academy of Sciences. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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5

1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Basel: Birkhäuser, 2001.

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6

International, Symposium on Mechanisms of Secondary Brain Damage (7th 2003? Mules Italy). Mechanisms of secondary brain damage from trauma and ischemia: Recent advances of our understanding : [proceedings of 7th International Symposium on Mechanisms of Secondary Brain Damage, Mauls, Italy]. Wien: Springer, 2004.

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7

A, Baethmann, ed. Mechanisms of secondary brain damage from trauma and ischemia: Recent advances of our understanding : [proceedings of 7th International Symposium on Mechanisms of Secondary Brain Damage, Mauls, Italy]. Wien: Springer, 2004.

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8

Kirkham, Lindsay Jack. The epidemiologic characteristics of a century of end stage atherosclerotic deaths: Ischemic heart disease and cerebral thrombosis : a surprise, a disappointment, and new etiologic concept. Washington, DC: Morris Publishing, 1999.

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9

Kirkham, Lindsay Jack. The epidemiologic characteristics of a century of end stage atherosclerotic deaths: Ischemic heart disease and cerebral thrombosis : a surprise, a disappointment, a new etiologic concept. Kearney, NE: Morris Publishing, 1999.

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10

D, Silver Malcolm, ed. Sudden death in ischemic heart disease: An alternative view on the significance of morphologic findings. New York: Springer-Verlag, 1995.

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11

Michael, Piper Hans, ed. Pathophysiology of severe ischemic myocardial injury. Dordrecht: Kluwer Academic Publishers, 1990.

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12

Gorō, Mimura, Yamazaki Seiichirō 1919- y Hsu C. -J, eds. Lipids and ischemic heart diseases: Proceedings of the Symposium on Lipids and Ischemic Heart Diseases, Naha, 5-6 December 1983. Amsterdam: Excerpta Medica, 1985.

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13

1943-, Hartmann A., Kuschinsky Wolfgang 1944- y Hoyer S. 1933-, eds. Cerebral ischemia and dementia. Berlin: Springer-Verlag, 1991.

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14

Mehta, Jawahar, ed. Thrombosis & Platelets In Myocardial Ischemia. F.A. Davis Company, 1987.

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15

Dmitry, Vukovic y Kiyan Vladimir, eds. Myocardial ischemia: Causes, symptoms and treatment. Hauppauge, N.Y: Nova Science, 2009.

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16

1934-, Conti C. Richard y Mehta Jawahar L, eds. Thrombosis and platelets in myocardial ischemia. Philadelphia: F.A. Davis Co., 1987.

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17

Myocardial Ischemia and Lipid Metabolism. Springer, 2012.

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18

Ferrari, R. Myocardial Ischemia and Lipid Metabolism. Springer London, Limited, 2012.

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19

Ferrari, R. Myocardial Ischemia and Lipid Metabolism. Springer, 2012.

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20

Myocardial Ischemia and Lipid Metabolism. Plenum Pub Corp, 1985.

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21

Pluta, Ryszard. Ischemia-Reperfusion Pathways in Alzheimer's Disease. Nova Science Pub Inc, 2007.

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22

Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction (Developments in Cardiovascular Medicine). Springer, 1988.

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23

(Editor), A. Baethmann, N. Plesnila (Editor), F. Ringel (Editor) y J. Eriskat (Editor), eds. Current Progress in the Understanding of Secondary Brain Damage from Trauma and Ischemia: Proceedings of the 6th International Symposium: Mechanisms of ... 1998 (Acta Neurochirurgica Supplementum). Springer, 1999.

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24

(Editor), Bo K. Siesjo y Tadeusz Wieloch (Editor), eds. Cellular & Molecular Mechanisms of Ischemic Brain Damage. Lippincott Williams & Wilkins, 1996.

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25

Siesjo, Bo K. Cellular and Molecular Mechanisms of Ischemic Brain Damage. Raven P., 1996.

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26

Arnim, Th Von. Predisposing Conditions for Acute Ischemic Syndromes. Springer, 1989.

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27

(Editor), A. Baethmann, J. Eriskat (Editor), J. Lehmberg (Editor) y N. Plesnila (Editor), eds. Mechanisms of Secondary Brain Damage from Trauma and Ischemia: Recent Advances of Our Understanding (Acta Neurochirurgica Supplementum). Springer, 2004.

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28

(Editor), Egan O'Connor, ed. Radiation from Medical Procedures in the Pathogenesis of Cancer and Ischemic Heart Disease: Dose-Response Studies with Physicians per 100,000 Population. Committee Nuclear Responsibility, 1999.

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29

(Editor), Egan O'Connor, ed. Radiation from Medical Procedures in the Pathogenesis of Cancer and Ischemic Heart Disease: Dose-Response Studies with Physicians per 100,000 Population. Committee Nuclear Responsibility, 1999.

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30

Pisklakov, Sergey, Haitham Ibrahim y Ingrid A. Fitz-James Antoine. Elevated ICP. Editado por David E. Traul y Irene P. Osborn. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850036.003.0023.

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Perioperative management of a patient with elevated intracranial pressure (ICP) is of paramount importance in neuroanesthesiology. Should this clinical emergency remain unaddressed, disability and death will ensue. Suboptimal care of a patient with elevated ICP is associated with avoidable morbidity and predictable mortality unless timely medical interventions, a focused history, targeted physical findings and a high degree of clinical suspicion confirmed by selective imaging result in medical stabilization and more definitive neurosurgical intervention. This may require interinstitutional transport. Understanding the physiologic and pathologic concepts that underlie elevated ICP permit anticipatory interventions to avert inexorable deterioration. The etiology of elevated intracranial pressure is often multifactorial. The deleterious effects of rising ICP demand a clear understanding of the relationship between ICP, mean arterial pressure (MAP), cerebral perfusion pressure (CPP), and cerebral autoregulation. Maintaining optimal CPP to prevent cerebral ischemia is the neuroanesthesiologist’s ultimate goal while managing a patient with an elevated ICP.
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31

Dodds, Jodi, Aaron I. Loochtan y Cheryl D. Bushnell. Ischemic Stroke Management in Pregnancy. Editado por Emma Ciafaloni, Cheryl Bushnell y Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0014.

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Ischemic stroke during pregnancy is relatively rare, but when it occurs, the risk is highest in the postpartum period. This is a condition that requires immediate recognition and evaluation for acute management to potentially prevent devastating neurological consequences. Determining an etiology while considering physiological changes during and after pregnancy is also important. Post-stroke care including implementing secondary stroke prevention via pharmacological and non-pharmacological methods is regular practice. Consideration of physical, occupational, and speech therapy strategies as well as lifestyle modification and evaluation and treatment of co-morbid psychiatric conditions is also paramount. Postpartum care and consideration of future pregnancies and hormonal changes that may occur is also important.
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32

F, Pérez-Gómez, Prentice C. R. M y Meyer Jürgen MD, eds. Coronary thrombosis: Intracardiac thrombosis. New York, NY: Raven Health Care Communications, 1994.

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33

Perez-Gomez, F., C. R. M. Prentice y Jurgen Meyer. Coronary Thrombosis: Intracardiac Thrombosis. Raven Health Care Communications, 2001.

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34

Ringer, Andrew. Surgical and Radiologic Intervention for Prevention of Ischemic Stroke. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0104.

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The etiology of ischemic strokes can be broadly categorized into several subtypes: cardioembolic, extracranial atherosclerotic, intracranial atherosclerotic, lacunar, traumatic (e.g., dissections), inflammatory (e.g., Moyamoya, vasculitis), and last cryptogenic. This chapter focuses on those etiologies that are treated or prevented by cerebrovascular specialist, including strokes due to extracranial athero-occlusive disease, intracranial athero-occlusive disease, arterial dissections, and Moyamoya disease. Both medical and surgical approaches are discussed.
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35

Chong, Ji Y. y Michael P. Lerario. Puff of Smoke. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0023.

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Moyamoya is a rare cause of stroke caused by progressive distal carotid occlusion. Children and adults may be affected, but it is more common in Asian populations, particularly in patients presenting with intracranial hemorrhage. Angiographic findings of distal internal carotid artery stenosis with lenticulostriate or choroidal neocollaterals are important in the diagnosis of moyamoya disease. A similar pathophysiological state to moyamoya disease is associated with several clinical conditions, some of which are acquired and some of which have a genetic etiology. Treatment is with surgical revascularization, which can reduce both ischemic and hemorrhagic complications.
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36

Goeral, Katharina, Ali Fatemi y Michael V. Johnston. Neonatal Brain Injuries. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0072.

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Neonatal brain injuries (NBIs) are important causes of motor and neurocognitive disabilities in children worldwide. This chapter discusses the most common types of NBIs in developed countries, namely neonatal encephalopathy associated with birth asphyxia in term infants, perinatal arterial ischemic stroke, perinatal white matter injury, and germinal matrix hemorrhage and neonatal hydrocephalus. Epidemiologic studies suggest that prenatal stressors, including intrauterine inflammation, maternal drug/toxin exposure, and placental pathologies are key players in the etiology of NBIs, whereas genetic contributors are yet to be identified. Hypothermia has become a standard neuroprotective strategy in neonatal encephalopathy, and novel therapeutics are currently being evaluated.
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37

Transient Amnesia: Clinical and Neuropsychological Aspects (Major Problems in Neurology). W.B. Saunders Company, 1991.

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