Literatura académica sobre el tema "INTERFERONOPATIA"
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Artículos de revistas sobre el tema "INTERFERONOPATIA"
Souza, Victória Cabral de, Roberto de Barros Silva y Caique Antunis. "INTERFERON-I COMO ALVO TERAPÊUTICO NO TRATAMENTO DO LÚPUS ERITEMATOSO SISTÊMICO: USO DOS ANTICORPOS MONOCLONAIS ANIFROLUMABE, RONTALIZUMABE E SIFALIMUMABE". Revista Ibero-Americana de Humanidades, Ciências e Educação 9, n.º 1 (31 de enero de 2023): 717–31. http://dx.doi.org/10.51891/rease.v9i1.8303.
Texto completoTommasini, Alberto, Irene Bruno, Maria Elisa Morelli y Loredana Lepore. "Le interferonopatie di tipo I". Medico e Bambino 40, n.º 8 (23 de septiembre de 2021): 509–14. http://dx.doi.org/10.53126/meb40509.
Texto completoPayet, Cloé A., Axel You, Odessa-Maud Fayet, Nadine Dragin, Sonia Berrih-Aknin y Rozen Le Panse. "Myasthenia Gravis: An Acquired Interferonopathy?" Cells 11, n.º 7 (4 de abril de 2022): 1218. http://dx.doi.org/10.3390/cells11071218.
Texto completoBriggs, Tracy A., Anindita Paul, Gillian Rice y Ariane L. Herrick. "RNASEH2B Related Adult-Onset Interferonopathy". Journal of Clinical Immunology 39, n.º 6 (31 de julio de 2019): 620–22. http://dx.doi.org/10.1007/s10875-019-00673-w.
Texto completoRodero, Mathieu P., Marie-Louise Frémond, Gillian I. Rice, Bénédicte Neven y Yanick J. Crow. "JAK inhibition in STING-associated interferonopathy". Annals of the Rheumatic Diseases 75, n.º 12 (12 de octubre de 2016): e75-e75. http://dx.doi.org/10.1136/annrheumdis-2016-210504.
Texto completoManoussakis, Menelaos N., Clio P. Mavragani, Adrianos Nezos, Evangelia Zampeli, Anastassios Germenis y Haralampos M. Moutsopoulos. "Type I interferonopathy in a young adult". Rheumatology 56, n.º 12 (14 de agosto de 2017): 2241–43. http://dx.doi.org/10.1093/rheumatology/kex316.
Texto completoKrieger, Ben Zion, Theresa Calvelli, Anna Kadish y Arye Rubinstein. "1042 INTERFERONOPATHY IN THE BARE LYMPHOCYTE SYNDROME". Pediatric Research 19, n.º 4 (abril de 1985): 284A. http://dx.doi.org/10.1203/00006450-198504000-01072.
Texto completoPiotto, Daniela Gerent Petry, Kátia Tomie Kozu y Sandro Félix Perazzio. "Quando suspeitar de doenças autoinflamatórias na infância?" Reumatologia Pediátrica, n.º 2021 jan-mar;20(1) (31 de marzo de 2021): 16–26. http://dx.doi.org/10.46833/reumatologiasp.2021.20.1.16-26.
Texto completoCrow, Yanick, Nandaki Keshavan, Jacques Patrick Barbet, Geanina Bercu, Vincent Bondet, Charlotte Boussard, Nathalie Dedieu et al. "Cardiac valve involvement in ADAR-related type I interferonopathy". Journal of Medical Genetics 57, n.º 7 (26 de noviembre de 2019): 475–78. http://dx.doi.org/10.1136/jmedgenet-2019-106457.
Texto completoKozlova, A. L., E. S. Romanenko, V. I. Burlakov, E. V. Deripapa, Sv P. Khomyakova, A. N. Remizov, G. V. Tereshenko y A. Yu Shcherbina. "CLINICAL CASE OF TYPE I INTERFERONOPATHY: AICARDI–GOUTIERES SYNDROME". Pediatria. Journal named after G.N. Speransky 98, n.º 3 (10 de junio de 2019): 257–65. http://dx.doi.org/10.24110/0031-403x-2019-98-3-257-265.
Texto completoTesis sobre el tema "INTERFERONOPATIA"
König, Nadja. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-225727.
Texto completoViengkhou, Barney. "Delineating the pathogenetic cellular response in type I interferonopathic brain disease". Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/24137.
Texto completoLucas, Nadja [Verfasser], Min Ae [Akademischer Betreuer] [Gutachter] Lee-Kirsch y Axel [Gutachter] Roers. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene / Nadja König ; Gutachter: Min Ae Lee-Kirsch, Axel Roers ; Betreuer: Min Ae Lee-Kirsch". Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-225727.
Texto completoKönig, Nadja [Verfasser], Min Ae [Akademischer Betreuer] [Gutachter] Lee-Kirsch y Axel [Gutachter] Roers. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene / Nadja König ; Gutachter: Min Ae Lee-Kirsch, Axel Roers ; Betreuer: Min Ae Lee-Kirsch". Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://d-nb.info/1135907390/34.
Texto completoAhmed, S. A. Abu Tayeh. "Constitutive RIG-I activation causes skin lesion resembling psoriasis in transgenic mice". Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263796.
Texto completoPayet, Cloé. "Study of Interferon type I in Myasthenia Gravis". Thesis, Sorbonne université, 2021. http://www.theses.fr/2021SORUS517.
Texto completoMyasthenia gravis (MG) is an autoimmune disease mediated by autoantibodies against the acetylcholine receptor (AChR). The thymus of patients is the effector organ and is characterized by chronic overexpression of interferon (IFN)-β. My PhD aimed to understand the implication of IFN-I in MG. First, I demonstrated that no IFN-I signature is detected in serum and PBMC of patients. IFN-I signature is specific to the thymus and I investigated the cause of this overexpression. IFN-I is produced in response to pathogen infection but also in response to endogenous nucleic acids (eNA) in diseases, such as interferonopathies. I demonstrated that molecules mimicking eNA, such as double-stranded DNA or RNA induced the overexpression of IFN-β and of AChR in human thymic epithelial cells (TEC) or in the thymus of mice. As I was suspecting eNA to be released by necrotic cells, I induced thymocyte necrosis in in vitro or in vivo models. I then showed an increased IFN-I signature and the expression of AChR in TEC and in the thymus of mice treated with dexamethasone. In addition, I observed in the MG thymus a decrease in thymic macrophages, cells responsible for the clearance of apoptotic cells. In mice, depletion of thymic macrophages led to an increase in necrotic thymocytes associated with IFN-I and AChR expression. Consequently, I hypothesize that in the MG thymus, a decrease in the number of macrophages may alter the processing of apoptotic cells leading to the release of eNA from necrotic thymocytes. These eNA would activate innate immunity signaling pathways leading to the IFN-β signature which would induce thymic changes self-sensitization against AChR
Actas de conferencias sobre el tema "INTERFERONOPATIA"
Schorling, D. C., R. Kaur, S. Donkervoort, C. G. Bönnemann, A. Lee-Kirsch, J. Kirschner, M. Eckenweiler y W. G. Janzarik. "Phenotypic Spectrum of PNPT1: Interferonopathy or Not?" En Abstracts of the 46th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1739628.
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