Literatura académica sobre el tema "Instestin"
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Artículos de revistas sobre el tema "Instestin"
Rodrigues, H. de Oliveira, J. Julio Vicente y Delir Corrêa Gomes. "Strongyloides ferreirai sp.n. (Nematoda, Rhabdiasoidea) parasito do roedor Kerodon rupestris (Wied.) no Brasil". Memórias do Instituto Oswaldo Cruz 80, n.º 4 (diciembre de 1985): 407–10. http://dx.doi.org/10.1590/s0074-02761985000400005.
Texto completoHorny, H. P. y H. A. Horst. "Lymphoreticular Infiltrates in Adenocarcinoma of the Large Instestine". Pathology - Research and Practice 182, n.º 2 (abril de 1987): 222–27. http://dx.doi.org/10.1016/s0344-0338(87)80108-5.
Texto completoMoreno-Osset, Eduardo. "Ser y no ser del síndrome del instestino irritable". Medicina Clínica 117, n.º 7 (enero de 2001): 257–59. http://dx.doi.org/10.1016/s0025-7753(01)72079-9.
Texto completoLiu, Guanwen, Bing Pang, Na Li, Han Jin, Junjun Li, Wanqin Wu, Chongyang Ai, Chunmei Jiang y Junling Shi. "Therapeutic effect of Lactobacillus rhamnosus SHA113 on intestinal infection by multi-drug-resistant Staphylococcus aureus and its underlying mechanisms". Food & Function 11, n.º 7 (2020): 6226–39. http://dx.doi.org/10.1039/d0fo00969e.
Texto completoFerreira de Morais, Fernanda, José Diego Marques Santos, Samuel Oliveira de Vera, Raiane Gomes Araújo Oliveira, Elaine Maria Leite Rangel Andrade y Sarah Nilkece Mesquita Araújo. "Management of instestinal stomas: knowledge of the caregiver". O Mundo da Saúde 42, n.º 4 (20 de diciembre de 2018): 823–44. http://dx.doi.org/10.15343/0104-7809.20184204823844.
Texto completoEide, Tor J. "A morphometrical analysis of dysplasia in small adenomas of the large instestine". Virchows Archiv A Pathological Anatomy and Histopathology 410, n.º 2 (1987): 119–24. http://dx.doi.org/10.1007/bf00713515.
Texto completoKonarska, L. y L. Tomaszewski. "Studies on l-arginase in developing rat small instestine, brain, and kidney". Biochemical Medicine and Metabolic Biology 35, n.º 2 (abril de 1986): 156–69. http://dx.doi.org/10.1016/0885-4505(86)90070-8.
Texto completoChiocchetti, Gabriela Matuoka, Dinoraz Vélez Pacios y Vicente Devesa Pérez. "Instestinal toxicity of a subchronic exposure to inorganic arsenic". Toxicology Letters 280 (octubre de 2017): S179. http://dx.doi.org/10.1016/j.toxlet.2017.07.502.
Texto completoNeves, José de Souza, José Eduardo de Aguilar Nascimento, Maria Helena Gaiva Gomes da Silva, Alberto Salomão Bicudo, Mariana Nascimento y Rubens Nochi Junior. "Influência da glutamina na mucosa do instestino de ratos submetidos à enterectomia extensa". Revista do Colégio Brasileiro de Cirurgiões 30, n.º 6 (diciembre de 2003): 406–15. http://dx.doi.org/10.1590/s0100-69912003000600002.
Texto completoAssis Rodrigues, Maria Luiza, Sirlene Souza Rodrigues Sartori, Priscila Izabel Santos Totaro y Sérgio Luis Pinto da Matta. "Hystometric evaluation of nickel chronic exposure effects on large instestine of adult Wistar male rats". Revista de Ciencias Agrícolas 36, E (16 de octubre de 2019): 21–30. http://dx.doi.org/10.22267/rcia.1936e.103.
Texto completoTesis sobre el tema "Instestin"
Rabahi, Soraya. "Interleukin-22 : Functional analysis in zebrafish". Electronic Thesis or Diss., Université Paris sciences et lettres, 2023. http://www.theses.fr/2023UPSLS061.
Texto completoCytokines promote gut defense and homeostasis. Among key gut cytokines, interleukin-22 (IL-22) is produced by immune cells and primarily targets epithelial cells. IL-22 protects the gut from pathogens by inducing anti-microbial peptides expression and promoting tissue repair. Dysregulation of this cytokine can lead to inflammatory bowel disease and cancer. During development, the post-embryonic gut is colonized by commensal microorganisms that promote maturation of the gut and its immune system. Nevertheless, whether and how cytokines such as IL-22 play a role in gut organ development and maturation during this critical time window remains unclear. The zebrafish allows us to visualize and manipulate physiological processes in vivo since early development due to its external development and transparency.During my PhD, I wanted to decipher the function of il22 in the developing gut. My data show that il22 is expressed in larval gut epithelial cells before il22-expressing lymphocytes appear in the gut. I identified enteroendocrine cells (EECs), a gut epithelial cell subtype, as the main source of il22 in larvae. Furthermore, I revealed conservation of the IL-22 signaling pathway, its transcriptional regulation by microbe sensing, and its antibacterial function in the gut. My latest data suggest that il22 expression can also be induced by the activation of Trpa1, a receptor known to recognize tryptophan metabolites in zebrafish and mice. Finally, I found a novel role of IL-22 in modulating gut motility in zebrafish. Mechanistically, dysbiosis was observed in il22-/-, along with potential defects in the production of bacteria-derived metabolites in the gut. Surprisingly, we found that the microbiota from wild-type larvae was able to restore the gut motility impairment of il22-/-, highlighting the important role of the microbiota in il22-mediated regulation of this process. Furthermore, an impairment was found in EECs function, which are known to be sensitive and respond to microbial cues. The dysregulation of EECs was characterized by abnormal hormone expression, specifically reduced levels of serotonin (5-HT), a critical hormone regulating gut motility in both zebrafish and mammals. External administration of 5-HT successfully rescued the gut motility phenotype of il22-/-, indicating that 5-HT is sufficient to restore proper gut motility. Finally, I found conservation of the gut motility defect in early life mice, suggesting the conservation of this novel IL-22 function in mammals. Altogether, this project contributes to a better understanding of how cytokines orchestrate gut development and maturation during the early stages of vertebrate life
Morais, Renata Alexandra Martins de. "Manifestações extra-intestinais da doença inflamatória instestinal". Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/21132.
Texto completoOliveira, Lucília de Jesus Guimarães. "Manifestações extra-intestinais da doença inflamatória instestinal". Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/21152.
Texto completoMorais, Renata Alexandra Martins de. "Manifestações extra-intestinais da doença inflamatória instestinal". Dissertação, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/21132.
Texto completoOliveira, Lucília de Jesus Guimarães. "Manifestações extra-intestinais da doença inflamatória instestinal". Dissertação, Instituto de Ciências Biomédicas Abel Salazar, 2008. http://hdl.handle.net/10216/21152.
Texto completoJacob, Jean-Marie. "Role of subepithelial fibroblasts in the instestinal barrier". Thesis, Université de Paris (2019-....), 2019. http://www.theses.fr/2019UNIP7081.
Texto completoMesenchymal cells (MCs) are non-immune, non-epithelial and non-endothelial cells present in all organs. They contribute to the organ’s structure by producing extracellular matrix (ECM). In addition to their role as ECM-producing cells, increasing evidence suggests that they play an active role in intestinal homeostasis. However, the lack of specific markers and tools to investigate MCs hindered a deeper understanding of their function(s). In the intestine, a major fraction of MCs expresses podoplanin (also known as gp38) a marker for MCs in lymphoid organs. Using new markers and reporter mice to study MCs that express the Lymphotoxin-ß Receptor (LTßR), a receptor involved in the crosstalk with immune cells, we identified distinct subsets of gp38+ MCs with specific functions in intestinal homeostasis and immunity. Inducible lineage tracing of gp38+LTßR+ MCs identifies a specific lineage of PDGFRα+ mesenchymal cells located closely to epithelial cells. We show that the lineage of PDGFRα+ subepithelial fibroblasts derived from LTßR+ progenitors (LTßR-SF) has a unique function in the first few weeks after birth. Indeed, this specific lineage develops around weaning independently of microbial colonization, and promotes maturation of the epithelial barrier, including differentiation of Paneth and goblet cells, involved in bacterial protection, and development of CD103+CD11b+ dendritic cells, a specific subset of immune cells involved in intestinal tolerance. LTßR-SF overexpress a set of genes essential for epithelial homeostasis and immune regulation, including bmp4, sfrp3, dll1, vcam, aldh1a3 and cox1. We further show that PDGFRα signaling in LTßR-SF is required to imprint both epithelial and immune function. Taken together, our results show that a subset of gp38+ MCs derived from LTßR+ progenitors plays an essential role in intestinal barrier development at weaning, by coordinating immune and epithelial maturation through PDGFRα signaling
Soares, Fábio Alves. "Análise da freqüência de anorretocele em mulheres adultas com evacuação obstruída, comparando com a paridade e idade, utilizando cinedefecografia e eletromanometria anorretal". reponame:Repositório Institucional da UFC, 2006. http://www.repositorio.ufc.br/handle/riufc/7314.
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The aim of this study is to analyse the frequence of anorectocele in adult women with obstructed defecation accordind to parity and age by means of cinedefaecography and anorectal eletromanometry. Forty-five adult women complaining of obstructed defecation were evaluated, with mean age of 46.3 years (23-72) and mean SCCC-C score of 13.3 points (6-23). Fifteen (33.3%) patients were nulliparous, seven (15,6%) primiparous and 23 (51,1%) multiparous, with mean parity per patient of 2.8 (0-11), considering only vaginal deliveries. Eighteen (60%) had a history of episiotomy, fourteen (46,7%) delivered macrossomic children and two (6,7%) had history of forceps-assisted delivery. Anal hipertony was verified in fourteen (31,1%) patients, while anal hipotony was present in eight (17,8%). Anismus was identified in thirteen (28,9%) patients. Anorecoceles were demonstrated in 34 (75,6%) patients, with mean size (TAR) of 24,8 mm (0-64). Thirty-six (80%) patients presented excessive perineal descent (DPM), rectal mucosal prolapse (PM) in 17 (37,8%) and rectoanal intussusception (IRA) in twelve (26,7%). There were no correlations between anorectocele and anal hipertony (p = 0,7171), anismus (p = 0,4666), IRA (p= 0,6991), PM (p = 0,2279), parity comparing nulliparous and multiparous patients (p = 1,000), episiotomy (p = 1,0000), forceps assistance (p = 1,0000) and delivery of macrossomic children (p = 1,0000). There were also no correlation between TAR and PMR (p =0,0883), PVM (p = 0,7327), parity (p = 0,4987) or age (p = 0,8603). There were correlations between anorectocele and DPM (p = 0,0275), score of SCCC-C (p =0,0082) and anal hipotony (p = 0,0141). In conclusion, anorectocele frequence is high and doesn’t correlate to parity, age but correlates to anal hipotony, DPM andconstipation.
O objetivo é avaliar a freqüência e o tamanho de anorretocele em mulheres adultas com evacuação obstruída, correlacionando-os com paridade, idade e parâmetros clínicos, utilizando cinedefecografia e eletromanometria. Foram avaliadas 45 mulheres adultas, com idade média de 46,3 anos (23-73) e sintomas de evacuação obstruída, com escore médio de 13,3 (6-23) pontos, segundo o Sistema de Classificação da Cleveland Clinic para Constipação (SCCC-C). Os parâmetros avaliados foram idade, dados obstétricos, escore do, SCCC-C, dados manométricos e achados de cinedefecografia.. Quinze (33,3%) pacientes eram nulíparas, 7 (15,6%) primíparas e 23 (51,1%) multíparas, com média de 2,8 (0-11) partos vaginais por paciente. Dezoito (60,0%) pacientes haviam sido submetidas a parto vaginal com episiotomia, sendo verificado feto macrossômico em 14 (46,7%) e aplicação de fórcipe em duas (6,7%) . Foi observada hipertonia esfincteriana em 14 (31,1%) e hipotonia em 8 (17,8%) pacientes. Foi identificado anismus em 13 (28,9%) pacientes. Foram demonstradas anorretoceles em 34 (75,6%) pacientes com tamanho (TAR) médio de 24,8 mm (0 - 64). Foram verificados descenso perineal móvel acentuado (DPM) em 36 (80%) pacientes, prolapso mucoso (PM) em 17 (37,8%) e intussuscepção reto-anal (IRA) em doze (26,7%). Não houve correlação entre anorretocele e hipertonia esfincteriana (p = 0,7171), anismus (p = 0,4666), IRA (p = 0,6991), PM (p= 0,2279), paridade comparando-se nulíparas e multíparas (p =1,000), episiotomia (p = 1,0000), uso de fórcipe (p = 1,0000), parto de feto macrossômico (p = 1,0000). Não houve correlação entre TAR e PMR (p = 0,0883), PVM (p = 0,7327), paridade (p = 0,4987) e idade (p = 0,8603). Houve correlação entre anorretocele e DPM (p = 0,0275), escore de constipação do SCCC-C (p = 0,0082) e hipotonia esfincteriana (p = 0,0141). Conclui-se que a freqüência de anorretocele foi elevada, não se correlacionou com paridade e idade, associando-se com hipotonia esfincteriana, DPM e constipação.
Royo, Aznar Ana. "Factores predictivos de la reconstrucción instestinal tras la intervención de Hartmann". Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/458134.
Texto completoIntroduction. Hartmann´s procedure is still a valid alternative in the treatment of pathologies of the left colon or rectum in patients having an ASA score IV, feculent peritonitis, malnutrition, immunosuppression or with hemodynamic instability. Hartmann´s procedure is mainly indicated in cases of high risk of anastomotic leakage, local tumor recurrence or anal incontinence. However, the factors related to the decision to restore intestinal continuity are not well established. The main objective of this study was to determine predictive factors of Hartmann´s reversal. The analysis of the morbidity of those interventions and the identification of predictive factors of intestinal transit reconstruction could ease an accurate selection of patients and give individualized preoperative counselling providing information on the most likely outcomes of the intervention. Material and methods. A retrospective observational study in which all consecutive patients that underwent Hartmann´s procedure from January 1999 to December 2014 in a tertiary University Hospital were included. No patient with a possible intestinal continuity restoration was excluded. The data collected were classified into 1) patient-specific: age, sex, body mass index, ASA score, Charlson index, anal incontinence; 2) disease-specific: type of disorder (benign vs malignant), main diagnosis, tumor stage, degree of peritoneal contamination, 3) treatment-specific: period of years of surgery, indication of Hartmann´s procedure, perioperative and postoperative transfusion, main surgical procedure, type of surgery (elective vs urgent), type of surgeon (general vs colorrectal), length of the rectal stump, Clavien-Dindo classification, readmission rate, causes of nonreversal Hartmann´s procedure. A descriptive analysis was performed. The 2 test or the Fisher exact test were used for categorical variables. Comparisons between groups were made using Mann-Whitney U test or Kruskal-Wallis test for continuous variables, where appropriate. Univariate and multivariate binary logistic regression model were used. Further, a classification and regression tree was performed. Finally, COR curves of each model were elaborated and compared with the DeLong test. Results. A total of 533 consecutive patients underwent Hartmann’s procedure. 110 (20,8%) patients underwent Hartmann´s reversal procedure. Mean age was 71,7 years. Multivariate analysis showed that the independent predictors of higher probability of intestinal transit reversal were age lower than 69 years, ASA grade I or II, indication of HP for anastomotic leak and the rectal stump above or at the sacral promontory. However, the independent factors related to a reduced probability of intestinal reconstruction following HP were anal incontinence, stage IV, postoperative transfusion or elective Hartmann's intervention. From the classification tree it is deduced that a patient below 69 years of age who presents low comorbidity, with a rectal stump at or above the promontory and that did not require perioperative transfusion would have 85% of probability of intestinal transit reconstruction. Discussion. Identification of predictive factors of intestinal continuity restoration may help surgeons to inform the patient and to choose the better option, both before performing a Hartmann procedure, and at the time of indicating reconstruction of intestinal continuity. Conclusion. Age, ASA, indication of Hartmann’s procedure, length of rectal stump, anal incontinence, tumor stage, postoperative transfusion and elective surgery can predict Hartmann’s reversal.
Loganathan, Arunan. "Relationship between Human Instestinal Permeability and Potassium Channel Function during Metabolic Stress". Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.507636.
Texto completoMelo, Janaina Viana de. "Caracterização ultra-estrutural dos efeitos citopatológicos causados por toxinas de Bacillus sphaericus no instestino de larvas de Culex quinquefasciatus". reponame:Repositório Institucional da FIOCRUZ, 2008. https://www.arca.fiocruz.br/handle/icict/3906.
Texto completoO Bacillus sphaericus (Bsp) é uma bactéria entomopatógena eficiente para o controle de Culex quinquefasciatus, um importante vetor da filariose e arboviroses. O fator larvicida do Bsp é a toxina binária (Bin) e a sua ação em C. quinquefasciatus depende da ligação ao receptor Cqm1. A ausência deste receptor no epitélio intestinal é o principal mecanismo de resistência à toxina Bin. Larvas resistentes a esta toxina, são susceptíveis ao Bsp IAB59 que, além da Bin, produz as toxinas Cry48Aa e Cry49Aa. O principal objetivo deste estudo foi caracterizar os efeitos de toxinas do Bsp nas células do epitélio intestinal de C. quinquefasciatus, utilizando como modelos larvas de uma colônia susceptível a todas as toxinas estudadas (S), de uma colônia resistente à toxina Bin (R2362) e de uma colônia resistente à Bin e Cry48Aa/Cry49Aa (RIAB59). Na primeira etapa, larvas não tratadas das colônias S e R2362 disseccionadas 30 min, 4, 6 e 48 h após a muda para o 4° estádio foram fixadas e processadas para microscopia eletrônica de transmissão (MET). A avaliação morfológica do epitélio intestinal mostrou que células de larvas R2362, ao final do 4° estádio, são caracterizadas por um intenso acúmulo de inclusões lipídicas, sugerindo que a ausência da a-glicosidase Cqm1 pode estar envolvida com alterações no metabolismo. Para caracterizar os efeitos causados pelas toxinas no epitélio intestinal, as larvas foram disseccionadas 1 e 6 h após o tratamento e processadas para MET. A avaliação ultra-estrutural da ação da toxina Bin nas células do epitélio intestinal mostrou que os principais efeitos em larvas S foram a vacuolização citoplasmática e destruição de microvilosidades. Estes foram observados exclusivamente em células que possuem o receptor Cqm1, demonstrando que esta molécula é essencial para mediar a ação da toxina Bin. Em células de larvas das colônias S e R2362, susceptíveis à Cry48Aa/Cry49Aa, o principal efeito destas toxinas foi a vacuolização mitocondrial, e este parece estar associado à Cry48Aa que possui estrutura de 3 domínios típica de toxinas da família Cry. Efeitos similares aos da toxina Bin também foram observados e parecem resultantes da ação da Cry49Aa que possui homologia com toxinas do tipo binária. Os dados mostram que a Cry48Aa/Cry49Aa possui uma ação complexa nas células e seu sítio de ligação é diferente do Cqm1. Combinações da toxina Bin com as toxinas Cry11Aa e Cyt1Aa do Bti também provocaram alterações em células de larvas R2362, desprovidas do receptor Cqm1. A combinação Bin/Cry11Aa causou efeitos similares aos induzidos pela toxina Cry48Aa/Cry49Aa, e sugerem que as toxinas de 3 domínios, Cry11Aa e Cry48Aa, podem mediar os efeitos das toxinas Bin e Cry49Aa, respectivamente, nos modelos estudados. A combinação Bin/Cyt1Aa provocou efeitos drásticos como a perda precoce de microvilosidades e a lise celular, característica da toxina Cyt1Aa que apresenta ação citolítica. Os resultados deste trabalho contribuem para o entendimento do modo de ação de toxinas do Bsp, bem como do efeito sinergístico de suas toxinas com aquelas do Bti
Libros sobre el tema "Instestin"
Igan to daichōgan. Tōkyō: Iwanami Shoten, 1999.
Buscar texto completoCocina Para Celiacos/ Cooking for Your Instestines. Atlantida, 2006.
Buscar texto completoCapítulos de libros sobre el tema "Instestin"
Vieira, Kérima D’ Israel y Rúbia Carla Oliveira. "A RELAÇÃO DO DESEQUÍLIBRIO DA MICROBIOTA INSTESTINAL E TRANSTORNO DEPRESSIVO: UMA REVISÃO DE LITERATURA". En Abordagens em medicina: ciência e prática, 177–82. Amplla Editora, 2023. http://dx.doi.org/10.51859/amplla.amc256.1123-21.
Texto completoActas de conferencias sobre el tema "Instestin"
Šalig, Sanela, Izabela Kranjčec y Gordana Jakovljević. "320 Immune thrombocytopenia and instestinal parasitosis – is there a causal link?" En 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.320.
Texto completo"TL 756 PAPEL DE MASTOCITOS EN LA EXPRESIÓN EPITELIAL INTESTINAL DE B-CELL LYMPHOMA-3 (BCL-3) Y LA PROTEÍNA DE UNIÓN ESTRECHA (UE) ZÓNULA OCCLUDEN-1 (ZO-1) EN EL SÍNDROME DE INSTESTINO IRRITABLE (SII)". En XLIX Congreso Chileno de Gastroenterología. Editorial Iku Limitada, 2022. http://dx.doi.org/10.46613/congastro2022-65.
Texto completoSilva, Isabella Maria Da, Ingrid Morselli Santos y Fábio Henrique De Oliveira. "DIAGNÓSTICO DE LINFOMA DE BURKITT POR MEIO DE COLONOSCOPIA: A IMPORTÂNCIA DO RASTREIO". En II Congresso Brasileiro de Hematologia Clínico-laboratorial On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/hematoclil/153.
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