Literatura académica sobre el tema "Immunosuppression – Maladies"
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Artículos de revistas sobre el tema "Immunosuppression – Maladies"
Aziz, Haya, Marc Bienz, Abdullah Esmaiel y George Thanassoulis. "Primary Staphylococcus aureus Pericarditis". Canadian Journal of General Internal Medicine 15, n.º 2 (29 de abril de 2020): 39–41. http://dx.doi.org/10.22374/cjgim.v15i2.361.
Texto completoRolland, Ossibi Ibara Bienvenu, ,. Obengui, Damba Banzouzi Bébene, Ossou-Nguiet Paul Macaire, Paka Herdan, Boumandouki Paul Jean Claude y Puruenhce Marie Francke. "Affections Neuroméningées Au Cours Du VIH Dans Le Service Des Maladies Infectieuses Du CHU De Brazzaville : Prévalence Et Facteurs Associés Au Décès". European Scientific Journal, ESJ 12, n.º 33 (30 de noviembre de 2016): 177. http://dx.doi.org/10.19044/esj.2016.v12n33p177.
Texto completoKadir, Ibrahim Mamadou Abdoul, Amadou Oumarou y Udou-Daouda Moussa. "Traitement de la maladie de Vogt-Koyanagi-Harada : Une revue narrative de la littérature". Annales Africaines de Medecine 15, n.º 1 (30 de enero de 2022): e4482-e4491. http://dx.doi.org/10.4314/aamed.v15i1.9.
Texto completoGhizlene, Laachir, Najioullah Dounia, Kabajj Najat y El Quessar Abdeljalil. "ENCEPHALITE DE RASMUSSEN CAUSE RARE DE LEPILEPSIE CHEZ LENFANT A PROPOS DUN CAS". International Journal of Advanced Research 9, n.º 08 (31 de agosto de 2021): 61–64. http://dx.doi.org/10.21474/ijar01/13230.
Texto completoFest, Thierry, Delphine Rossille, Mélanie Gressier, Delphine Maucort-Boulch, Diane Damotte, Céline Pangault, Steven Le Gouill, Karin Tarte, Thierry Lamy y Noel Milpied. "Blood Soluble PD-L1 Protein In Aggressive Diffuse Large B-Cell Lymphoma Impacts patient’s Overall Survival". Blood 122, n.º 21 (15 de noviembre de 2013): 361. http://dx.doi.org/10.1182/blood.v122.21.361.361.
Texto completoAhmed, S. G. y U. A. Ibrahim. "A review of the role of infections in the aetiology of haemolysis in patients with sickle cell diseases: pathogenesis, management, and prevention". African Journal of Clinical and Experimental Microbiology 23, n.º 4 (23 de octubre de 2022): 345–57. http://dx.doi.org/10.4314/ajcem.v23i4.3.
Texto completoBecker, William J., Prakash S. Nagarkatti y Mitzi Nagarkatti. "Allogeneic skin transplantation triggers unique miRNA signature profiles in CD4+ T cells with potential therapeutic implications." Journal of Immunology 196, n.º 1_Supplement (1 de mayo de 2016): 140.39. http://dx.doi.org/10.4049/jimmunol.196.supp.140.39.
Texto completoChow, Justin, Umair Iftikhar, Victoria Weaver, Kwadwo Mponponsuo y Amy Bromley. "Sudden Cardiac Death in a Patient with Systemic Lupus Erythematosus and Cytomegalovirus Myocarditis". Canadian Journal of General Internal Medicine 15, n.º 4 (18 de noviembre de 2020): 36–40. http://dx.doi.org/10.22374/cjgim.v15i4.400.
Texto completoBeliavsky, Alina, Sigmund Krajden, Anita Chae, Barbara Newton y Greg Sue-A-Quan. "Pasteurella multocida Intramuscular Chest Abscess in a Healthy Man". Canadian Journal of General Internal Medicine 16, n.º 3 (21 de septiembre de 2021): e78-e80. http://dx.doi.org/10.22374/cjgim.v16i3.510.
Texto completoLiman, H. M., M. A. Makusidi, A. M. Sakajiki, H. J. Ishaku, Y. A. Shehu, A. A. Yusuf y S. Yusuf. "Challenges in the management of kidney transplant recipients in a centre without a kidney transplant program: A single centre experience". Research Journal of Health Sciences 9, n.º 1 (7 de abril de 2021): 2–7. http://dx.doi.org/10.4314/rejhs.v9i1.1.
Texto completoTesis sobre el tema "Immunosuppression – Maladies"
Viau, Muriel. "Effets des superantigènes B in vitro et in vivo dans un modèle de souris transgèniques humanisées". Paris 13, 2004. http://www.theses.fr/2004PA132031.
Texto completoChanson, Laura Bohne Wolf. "Impact des troubles anxio-dépressifs sur les tissus de la cavité buccale". [S.l.] : [s.n.], 2008. http://castore.univ-nantes.fr/castore/GetOAIRef?idDoc=52216.
Texto completoDumetz, Fabien. "Les antigènes de surface de Flavobacterium psychrophilum : approche protéomique et caractérisation de deux protéines (OmpA/P60 et OmpH/P18)". Bordeaux 2, 2006. http://www.theses.fr/2006BOR21363.
Texto completoFlavobacterium psychrophilum is a Gram negative bacteria responsible for fish infection. We used a proteomic approach to identify some outer membrane components such as putative adhesins, proteins involved in iron acquisition or in efflux systems, a HtrA homologue and some other molecules with unknown function. Several major antigens have been identified in the outer membrane including the two components OmpH/P18 and OmpA/P60. They are surface-exposed since they were completely digested by in situ proteinase K treatment and the two monospecific sera were bacteriostatic/bactericidal. Vaccination trials showed that both proteins can induce a high titter of specific antibodies which are protective. Collectively, these results indicate that these two proteins could be used in future vaccine development as promising candidate antigens
Lefevre, Camille. "Exploration fonctionnelle des cellules souches du tissu adipeux dans l'émergence des maladies de l'obésité". Electronic Thesis or Diss., Lyon, 2020. https://n2t.net/ark:/47881/m6319v9s.
Texto completoObesity induces a chronic inflammation responsible for complications (diabetes, cardiovascular diseases) where adipose tissue plays a central role. At the onset of obesity, inflammation is at low grade suggesting the control by immunosuppressive mechanisms that decrease with obesity, promoting complications. Adipose stem cells (ASC) support the development and the homeostasis of adipose tissue in subcutaneous and visceral adipose depots. Inflammatory stimuli induce immunosuppressive functions in ASC in vitro, but published data report that ASC isolated from inflammatory adipose tissues in established obesity have lost these properties. The role of ASC in the immune homeostasis of adipose tissue is poorly known. We made the hypothesis that immunosuppressive properties of ASC are induced since the onset of obesity and that their alterations contribute to complications. To address this question, I performed two protocols of diet induced obesity in mice and I explored the functions of ASC isolated from subcutaneous (S-ASC) and visceral (V-ASC) adipose depots. My results show major differences in the proliferation and the differentiation potential of ASC from distinct adipose depots, according to published data. We reveal that these differences correlate with distinct metabolomes, V-ASC having lower mitochondrial and higher glycolytic activity than S-ASC. Using this model, we studied the immunosuppressive functions of ASC in early obesity in both adipose depots. To this end we performed a kinetic of 6, 10 and 14 weeks of high fat diet (HF) in C57Bl/6 mice. This timing covered low grade inflammation progress from glucose intolerance with 6 weeks of diet in the absence of adipose tissue inflammation, to insulin resistance at 14 weeks of diet associated with visceral, but not subcutaneous, adipose tissue inflammation. My results show that at 6 weeks of HF diet, V-ASC attracted macrophages and inhibited the pro-inflammatory M1 polarization of these cells. At the same time, S-ASC completely suppressed the proliferation of activated T lymphocytes and strongly inhibited their migration. This study shows (i) that CSA from both adipose depots are activated by HF diet, independently of inflammation and diet time, (ii) that the induced immunosuppressive properties target distinct immune cells, (iii) that they are maintained with resistance to insulin. The analysis of adipose tissue composition showed that the ASC population decreased and had lower proliferation rate with HF diet. This indicate that at the onset of obesity, intrinsic properties of ASC were maintained in vitro but their environment altered their maintenance in both adipose depots. In a second protocol, I explored the consequences of maternal overnutrition during lactation on the properties of ASC in the adulthood. It has been reported that these conditions favorize the development of metabolic diseases in the offspring. We wondered whether ASC are targeted by maternal diet and develop long term alterations. I isolated S- and V-ASC from the offspring 6 weeks after weaning and I explored the influence of a HF post-weaning diet. RNA seq analysis showed that maternal diet altered the transcriptome of ASC in the adulthood and induced glucose intolerance, even in animal fed with a standard diet after weaning. Gene expression altered in ASC support cell death, metabolic stress, transcription, protein synthesis. Some genes are only affected by the mother’s diet. For genes associated with cell death, differential expressions induced by HF diet are increased when cumulated with mother’s HF diet. These results show that alterations of ASC precede the complications in the adult offspring. ASC can thus be programmed by maternal overnutrition and are the probable vectors of later adipose tissue dysfunctions
Paul, Carle. "Développement des inhibiteurs de la calcineurine pour le traitement des maladies inflammatoires cutanées : de l' immunosuppression systémique à l'immunomodulation topique". Paris 7, 2005. http://www.theses.fr/2005PA077076.
Texto completoYedikardachian, Christine. "Prévention et traitement de la varicelle chez l'enfant immunodéprimé". Paris 5, 1988. http://www.theses.fr/1988PA05P138.
Texto completoEscaffre, Olivier. "Bases moléculaires du pouvoir pathogène et développement d’une méthode de quantification différentielle des quatre brins génomiques chez l’Avibirnavirus de la bursite infectieuse". Rennes 1, 2011. http://www.theses.fr/2011REN1S005.
Texto completoInfectious Bursal Disease (IBD) is an infectious disease of young chickens (Gallus gallus) caused by a bisegmented (A & B) double-stranded RNA genome virus (IBDV, family Birnaviridae, genus Avibirnavirus). The virus replication cycle is not fully described regarding the time and place of strand synthesis. In a first part of this study, four real-time quantitative RT-PCR methods, able to quantify specifically each strand of the IBDV genome, were developed. Implementation of these methods on purified IBDV particles and on a kinetic study of in-vitro strand production provided interesting data as to the possible genomic content of virus particles and to the replication model, respectively. These results suggest that negative strand synthesis occurs after equimolar positive strand (A & B) packaging inside nascent virus particle. Molecular basis of the pathogenicity have been investigated in a second study. Very virulent IBDV strain (vvIBDV) can induce important clinical sign and mortality rates but no reliable molecular marker has been definitely identified yet. This study was carried out on a field strain (94432) which, in spite of being phylogenetically related to vvIBDVs, does not induce any mortality and only low morbidity in chickens. Using a reverse genetic system associated to 94432, several recombinant derived-94432 viruses were characterized in-vivo. Results demonstrated an important contribution of the polymerase encoded-segment B, especially of one amino acid at position 276, in the reduced pathogenicity of 94432. The biological function of amino acid 276 remains unknown
Kerestedjian, Jean-Jacques. "Traitement de l'infection expérimentale à "Rhodococcus equi" chez la souris nude". Paris 5, 1992. http://www.theses.fr/1992PA05P111.
Texto completoMenager-Marcq, Ingrid. "Etude du potentiel immunosuppresseur des lymphocytes t régulateurs CD8+CD28- dans un modèle murin de maladie inflammatoire chronique intestinale". Toulouse 3, 2006. http://www.theses.fr/2006TOU30176.
Texto completoLanaya, Hanane. "Rôle des cellules myéloïdes immatures GR1+CD11b+ dans le rejet du mastocytome P815". Doctoral thesis, Universite Libre de Bruxelles, 2008. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210372.
Texto completoThe aim of our work was to define the mechanisms by which a single dose of cyclophosphamide (CTX), a chemical agent commonly used in chemotherapy treatment, induces the rejection of established P815 mastocytoma.
Our data show that CTX treatment leads to the selective loss of GR1medCD11b+ splenic myeloid cell producing TGF-â, a cytokine which is known to suppress antitumoral response. Furthermore, injection of CTX causes a decrease in the number of naturally occurring regulatory T cells (CD4+CD25+Foxp3+) in the spleen and the tumor. Finally, CTX treatment induces the differentiation of GR1highCD11b+ splenic myeloid cells into mature GR1highCD11b+CD11c+ (possibly dendritic cells?) which express high levels of CD11c, MHC class II and CD86 molecules. Of note, these cells are mainly detected in tumour necrosis areas.
Collectively, these results suggest that CTX prevents suppressive mechanisms and induces a population of CD11c+ myeloid cells which may present tumor antigens and activate T lymphocytes, an hypothesis in line with the requirement for CD4+ cells in CTX-induced long term resistance.
Doctorat en Sciences
info:eu-repo/semantics/nonPublished
Libros sobre el tema "Immunosuppression – Maladies"
W, Thomson Angus, ed. The Molecular biology of immunosuppression. Chichester: Wiley, 1992.
Buscar texto completo1922-, Good Robert A. y Lindenlaub E, eds. The Nature, cellular, and biochemical basis and management of immunodeficiencies: Symposium Bernried, West Germany, 21st - 25th September, 1986. Stuttgart: F.K. Schattauer Verlag, 1987.
Buscar texto completoed, Gonzalez Norberto C. y Lindenlaub E. ed, eds. The Nature, cellular, and biochemical basis and management of immunodeficiencies: Symposium Bernried, West Germany, 21st - 25th September, 1986. Stuttgart: F.K. Schattauer Verlag, 1987.
Buscar texto completoHäusermann, Peter, Jürg Steiger y Jakob Passweg. Transplantation dermatology. Basel: Karger, 2012.
Buscar texto completoGood, Robert A. The Nature, Cellular, and Biochemical Basis and Management of Immunodeficiencies (Symposia Medica Hoechst, No 21). Wiley-Liss, 1988.
Buscar texto completoImmunopharmacology. Springer Verlag, 1990.
Buscar texto completoActas de conferencias sobre el tema "Immunosuppression – Maladies"
Fricain, M., P. Weidmann, Y. Roche y J. C. Fricain. "Vitiligo labial associé à une pathomimie". En 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603003.
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