Bibliografías temáticas / Immunity control / Artículos de revistas

Artículos de revistas sobre el tema "Immunity control"

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Publicado: 4 de mayo de 2024

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1

Bendelac, Albert y Douglas T. Fearon. "Innate immunity Innate pathways that control acquired immunity". Current Opinion in Immunology 9, n.º 1 (febrero de 1997): 1–3. http://dx.doi.org/10.1016/s0952-7915(97)80151-3.

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2

Busslinger, M. y A. Tarakhovsky. "Epigenetic Control of Immunity". Cold Spring Harbor Perspectives in Biology 6, n.º 6 (1 de junio de 2014): a019307. http://dx.doi.org/10.1101/cshperspect.a019307.

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3

Busslinger, M. y A. Tarakhovsky. "Epigenetic Control of Immunity". Cold Spring Harbor Perspectives in Biology 6, n.º 7 (1 de julio de 2014): a024174. http://dx.doi.org/10.1101/cshperspect.a024174.

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4

Tracey, Kevin J. "Reflex control of immunity". Nature Reviews Immunology 9, n.º 6 (junio de 2009): 418–28. http://dx.doi.org/10.1038/nri2566.

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5

Gamboa, Lena, Ali H. Zamat y Gabriel A. Kwong. "Synthetic immunity by remote control". Theranostics 10, n.º 8 (2020): 3652–67. http://dx.doi.org/10.7150/thno.41305.

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6

ALVAREZ, MARÍA E., FLORENCIA NOTA y DAMIÁN A. CAMBIAGNO. "Epigenetic control of plant immunity". Molecular Plant Pathology 11, n.º 4 (1 de junio de 2010): 563–76. http://dx.doi.org/10.1111/j.1364-3703.2010.00621.x.

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7

Chen, Huihui, Xiaohan Ning y Zhengfan Jiang. "Caspases control antiviral innate immunity". Cellular & Molecular Immunology 14, n.º 9 (10 de julio de 2017): 736–47. http://dx.doi.org/10.1038/cmi.2017.44.

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8

Bachère, Evelyne. "Shrimp immunity and disease control". Aquaculture 191, n.º 1-3 (noviembre de 2000): 3–11. http://dx.doi.org/10.1016/s0044-8486(00)00413-0.

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9

Shanker, Anil. "Adaptive control of innate immunity". Immunology Letters 131, n.º 2 (julio de 2010): 107–12. http://dx.doi.org/10.1016/j.imlet.2010.04.002.

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10

Kiberstis, P. A. "Oncogene control of antitumor immunity". Science 352, n.º 6282 (7 de abril de 2016): 183. http://dx.doi.org/10.1126/science.352.6282.183-d.

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11

Fric, Jan, Teresa Zelante, Alicia Y. W. Wong, Alexandra Mertes, Hong-Bing Yu y Paola Ricciardi-Castagnoli. "NFAT control of innate immunity". Blood 120, n.º 7 (16 de agosto de 2012): 1380–89. http://dx.doi.org/10.1182/blood-2012-02-404475.

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Abstract The calcineurin/nuclear factor of activated T cells (NFAT) signaling pathway mediates multiple adaptive T-cell functions, but recent studies have shown that calcineurin/NFAT signaling also contributes to innate immunity and regulates the homeostasis of innate cells. Myeloid cells, including granulocytes and dendritic cells, can promote inflammation, regulate adaptive immunity, and are essential mediators of early responses to pathogens. Microbial ligation of pattern-recognition receptors, such as TLR4, CD14, and dectin 1, is now known to induce the activation of calcineurin/NFAT signaling in myeloid cells, a finding that has provided new insights into the molecular pathways that regulate host protection. Inhibitors of calcineurin/NFAT binding, such as cyclosporine A and FK506, are broadly used in organ transplantation and can act as potent immunosuppressive drugs in a variety of different disorders. There is increasing evidence that these agents influence innate responses as well as inhibiting adaptive T-cell functions. This review focuses on the role of calcineurin/NFAT signaling in myeloid cells, which may contribute to the various unexplained effects of immunosuppressive drugs already being used in the clinic.
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12

Pouikli, Andromachi y Christian Frezza. "Metabolic control of antitumor immunity". Science 381, n.º 6664 (22 de septiembre de 2023): 1287–88. http://dx.doi.org/10.1126/science.adk1785.

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13

Akira, Shizuo y Kazuhiko Maeda. "Control of RNA Stability in Immunity". Annual Review of Immunology 39, n.º 1 (26 de abril de 2021): 481–509. http://dx.doi.org/10.1146/annurev-immunol-101819-075147.

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Posttranscriptional control of mRNA regulates various biological processes, including inflammatory and immune responses. RNA-binding proteins (RBPs) bind cis-regulatory elements in the 3′ untranslated regions (UTRs) of mRNA and regulate mRNA turnover and translation. In particular, eight RBPs (TTP, AUF1, KSRP, TIA-1/TIAR, Roquin, Regnase, HuR, and Arid5a) have been extensively studied and are key posttranscriptional regulators of inflammation and immune responses. These RBPs sometimes collaboratively or competitively bind the same target mRNA to enhance or dampen regulatory activities. These RBPs can also bind their own 3′ UTRs to negatively or positively regulate their expression. Both upstream signaling pathways and microRNA regulation shape the interactions between RBPs and target RNA. Dysregulation of RBPs results in chronic inflammation and autoimmunity. Here, we summarize the functional roles of these eight RBPs in immunity and their associated diseases.
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14

Ansaldo, Eduard, Taylor K. Farley y Yasmine Belkaid. "Control of Immunity by the Microbiota". Annual Review of Immunology 39, n.º 1 (26 de abril de 2021): 449–79. http://dx.doi.org/10.1146/annurev-immunol-093019-112348.

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The immune system has coevolved with extensive microbial communities living on barrier sites that are collectively known as the microbiota. It is increasingly clear that microbial antigens and metabolites engage in a constant dialogue with the immune system, leading to microbiota-specific immune responses that occur in the absence of inflammation. This form of homeostatic immunity encompasses many arms of immunity, including B cell responses, innate-like T cells, and conventional T helper and T regulatory responses. In this review we summarize known examples of innate-like T cell and adaptive immunity to the microbiota, focusing on fundamental aspects of commensal immune recognition across different barrier sites. Furthermore, we explore how this cross talk is established during development, emphasizing critical temporal windows that establish long-term immune function. Finally, we highlight how dysregulation of immunity to the microbiota can lead to inflammation and disease, and we pinpoint outstanding questions and controversies regarding immune system–microbiota interactions.
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15

Collins, Nicholas y Yasmine Belkaid. "Control of immunity via nutritional interventions". Immunity 55, n.º 2 (febrero de 2022): 210–23. http://dx.doi.org/10.1016/j.immuni.2022.01.004.

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16

Machado, João Paulo B., Iara P. Calil, Anésia A. Santos y Elizabeth P. B. Fontes. "Translational control in plant antiviral immunity". Genetics and Molecular Biology 40, n.º 1 suppl 1 (13 de febrero de 2017): 292–304. http://dx.doi.org/10.1590/1678-4685-gmb-2016-0092.

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17

Harjes, Ulrike. "Germline control of anti-tumour immunity". Nature Reviews Cancer 20, n.º 8 (17 de junio de 2020): 414. http://dx.doi.org/10.1038/s41568-020-0282-x.

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18

Lau, Henry Y. K., Vicky W. K. Wong y Ivan S. K. Lee. "Immunity-based autonomous guided vehicles control". Applied Soft Computing 7, n.º 1 (enero de 2007): 41–57. http://dx.doi.org/10.1016/j.asoc.2005.02.003.

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19

Brave, Martina, Dana J. Lukin y Sridhar Mani. "Microbial control of intestinal innate immunity". Oncotarget 6, n.º 24 (3 de julio de 2015): 19962–63. http://dx.doi.org/10.18632/oncotarget.4780.

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20

Mehta, Manan M., Samuel E. Weinberg y Navdeep S. Chandel. "Mitochondrial control of immunity: beyond ATP". Nature Reviews Immunology 17, n.º 10 (3 de julio de 2017): 608–20. http://dx.doi.org/10.1038/nri.2017.66.

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21

Gray-Owen, Scott D. y Richard S. Blumberg. "CEACAM1: contact-dependent control of immunity". Nature Reviews Immunology 6, n.º 6 (junio de 2006): 433–46. http://dx.doi.org/10.1038/nri1864.

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22

Pelgrom, Leonard R. y Bart Everts. "Metabolic control of type 2 immunity". European Journal of Immunology 47, n.º 8 (14 de julio de 2017): 1266–75. http://dx.doi.org/10.1002/eji.201646728.

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23

Jerison, Elizabeth. "Dynamical control of immunity and inflammation". Biophysical Journal 123, n.º 3 (febrero de 2024): 309a. http://dx.doi.org/10.1016/j.bpj.2023.11.1908.

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24

Jin, Hyo Sun, Hyun-Woo Suh, Seong-Jun Kim y Eun-Kyeong Jo. "Mitochondrial Control of Innate Immunity and Inflammation". Immune Network 17, n.º 2 (2017): 77. http://dx.doi.org/10.4110/in.2017.17.2.77.

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25

Banchereau, Jacques y Ralph M. Steinman. "Dendritic cells and the control of immunity". Nature 392, n.º 6673 (marzo de 1998): 245–52. http://dx.doi.org/10.1038/32588.

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26

Lau, H. Y. K. y V. W. K. Wong. "An immunity-based distributed multiagent-control framework". IEEE Transactions on Systems, Man, and Cybernetics - Part A: Systems and Humans 36, n.º 1 (enero de 2006): 91–108. http://dx.doi.org/10.1109/tsmca.2005.859103.

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27

Moehlman, Andrew T. y Richard J. Youle. "Mitochondrial Quality Control and Restraining Innate Immunity". Annual Review of Cell and Developmental Biology 36, n.º 1 (6 de octubre de 2020): 265–89. http://dx.doi.org/10.1146/annurev-cellbio-021820-101354.

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Maintaining mitochondrial health is essential for the survival and function of eukaryotic organisms. Misfunctioning mitochondria activate stress-responsive pathways to restore mitochondrial network homeostasis, remove damaged or toxic proteins, and eliminate damaged organelles via selective autophagy of mitochondria, a process termed mitophagy. Failure of these quality control pathways is implicated in the pathogenesis of Parkinson's disease and other neurodegenerative diseases. Impairment of mitochondrial quality control has been demonstrated to activate innate immune pathways, including inflammasome-mediated signaling and the antiviral cyclic GMP-AMP synthase (cGAS)/stimulator of interferon genes (STING)–regulated interferon response. Immune system malfunction is a common hallmark in many neurodegenerative diseases; however, whether inflammation suppresses or exacerbates disease pathology is still unclear. The goal of this review is to provide a historical overview of the field, describe mechanisms of mitochondrial quality control, and highlight recent advances on the emerging role of mitochondria in innate immunity and inflammation.
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28

Park, Eun Jeong, Motomu Shimaoka y Hiroshi Kiyono. "MicroRNA-mediated dynamic control of mucosal immunity". International Immunology 29, n.º 4 (1 de abril de 2017): 157–63. http://dx.doi.org/10.1093/intimm/dxx019.

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29

Zhao, Ende, Huanbin Xu, Lin Wang, Ilona Kryczek, Ke Wu, Yu Hu, Guobin Wang y Weiping Zou. "Bone marrow and the control of immunity". Cellular & Molecular Immunology 9, n.º 1 (24 de octubre de 2011): 11–19. http://dx.doi.org/10.1038/cmi.2011.47.

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30

Ottenhoff, Tom H. M., Frank A. W. Verreck, Marieke A. Hoeve y Esther van de Vosse. "Control of human host immunity to mycobacteria". Tuberculosis 85, n.º 1-2 (enero de 2005): 53–64. http://dx.doi.org/10.1016/j.tube.2004.09.011.

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31

Quintana, Francisco J. y David H. Sherr. "Aryl Hydrocarbon Receptor Control of Adaptive Immunity". Pharmacological Reviews 65, n.º 4 (1 de agosto de 2013): 1148–61. http://dx.doi.org/10.1124/pr.113.007823.

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32

Lau, Henry Y. K. y Vicky W. K. Wong. "An immunity approach to strategic behavioral control". Engineering Applications of Artificial Intelligence 20, n.º 3 (abril de 2007): 289–306. http://dx.doi.org/10.1016/j.engappai.2006.06.002.

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33

Liu, Juan, Cheng Qian y Xuetao Cao. "Post-Translational Modification Control of Innate Immunity". Immunity 45, n.º 1 (julio de 2016): 15–30. http://dx.doi.org/10.1016/j.immuni.2016.06.020.

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34

Wakelin, D. "Genetic control of immunity to helminth infections". Parasitology Today 1, n.º 1 (julio de 1985): 17–23. http://dx.doi.org/10.1016/0169-4758(85)90101-2.

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35

Potter, Tim y Kat Baxter-Smith. "Modernising BRD control". Livestock 25, n.º 6 (2 de noviembre de 2020): 292. http://dx.doi.org/10.12968/live.2020.25.6.292.

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36

Anandasabapathy, Niroshana, Rachel Feder, Shamim Mollah, Sze-Wah Tse, Maria Paula Longhi, Saurabh Mehandru, Ines Matos et al. "Classical Flt3L-dependent dendritic cells control immunity to protein vaccine". Journal of Experimental Medicine 211, n.º 9 (18 de agosto de 2014): 1875–91. http://dx.doi.org/10.1084/jem.20131397.

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DCs are critical for initiating immunity. The current paradigm in vaccine biology is that DCs migrating from peripheral tissue and classical lymphoid-resident DCs (cDCs) cooperate in the draining LNs to initiate priming and proliferation of T cells. Here, we observe subcutaneous immunity is Fms-like tyrosine kinase 3 ligand (Flt3L) dependent. Flt3L is rapidly secreted after immunization; Flt3 deletion reduces T cell responses by 50%. Flt3L enhances global T cell and humoral immunity as well as both the numbers and antigen capture capacity of migratory DCs (migDCs) and LN-resident cDCs. Surprisingly, however, we find immunity is controlled by cDCs and actively tempered in vivo by migDCs. Deletion of Langerin+ DC or blockade of DC migration improves immunity. Consistent with an immune-regulatory role, transcriptomic analyses reveals different skin migDC subsets in both mouse and human cluster together, and share immune-suppressing gene expression and regulatory pathways. These data reveal that protective immunity to protein vaccines is controlled by Flt3L-dependent, LN-resident cDCs.
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37

Polushin, P. A., O. R. Nikitin y I. R. Dubov. "Quasioptimal control in diversed signal transmission". IOP Conference Series: Materials Science and Engineering 1227, n.º 1 (1 de febrero de 2022): 012003. http://dx.doi.org/10.1088/1757-899x/1227/1/012003.

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Abstract To increase the noise immunity of signal transmission, diversity methods are now widely used, consisting in obtaining and combining several copies of the transmitted signal. In this case, it is possible to perform a combination either before the detection procedure or after it. If you do not take into account the possible use of non-linear types of modulation, then the pre-detector combination always has advantages over the post-detector combination. However, taking into account the nonlinear properties of the transmitted signals, new possibilities appear for increasing the noise immunity in combination and simplifying the processing. In the case of using analog signals, in particular frequency modulation, at certain points in time, the pre-detection combination can lose to the post-detection combination. At the same time, by combining pre-detector and post-detector combining circuits, it is possible to lower the threshold level during demodulation and increase noise immunity. In the case of using digital modes of modulation, it is possible to process only the signals after demodulation without reducing the noise immunity and to eliminate the need for preliminary phasing of the diversity signals before detection.
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38

Watts, Tania H. "Stepping up Th1 immunity to control phagosomal bacteria". Trends in Immunology 42, n.º 6 (junio de 2021): 461–63. http://dx.doi.org/10.1016/j.it.2021.04.008.

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39

Schlosser, Thomas P. "Sovereign Immunity: Should the Sovereign Control the Purse?" American Indian Law Review 24, n.º 2 (1999): 309. http://dx.doi.org/10.2307/20070637.

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40

Carter, Philip B. "Immunity to Parasites: How Animals Control Parasite Infections". American Journal of Tropical Medicine and Hygiene 34, n.º 4 (1 de julio de 1985): 825. http://dx.doi.org/10.4269/ajtmh.1985.34.4.tm0340040825a.

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41

BACON, L. D. y R. R. DIETERT. "Genetic Control of Cell-Mediated Immunity in Chickens". Poultry Science 70, n.º 5 (mayo de 1991): 1187–99. http://dx.doi.org/10.3382/ps.0701187.

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42

Befus, A. Dean, Derek Wakelin y Edward Arnold. "Immunity to Parasites: How Animals Control Parasite Infections". Journal of Parasitology 71, n.º 3 (junio de 1985): 364. http://dx.doi.org/10.2307/3282019.

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43

Schiering, Chris, Emma Wincent, Amina Metidji, Andrea Iseppon, Ying Li, Alexandre J. Potocnik, Sara Omenetti et al. "Feedback control of AHR signalling regulates intestinal immunity". Nature 542, n.º 7640 (febrero de 2017): 242–45. http://dx.doi.org/10.1038/nature21080.

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44

Asaturova, A. V., A. V. Tregubova y D. V. Shushkanova. "Inhibition of immunity control points in ovarian cancer". CLINICAL AND EXPERIMENTAL MORPHOLOGY 9, n.º 1 (2020): 11–19. http://dx.doi.org/10.31088/cem2020.9.1.11-19.

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45

Grant, Audrey V., Christian Roussilhon, Richard Paul y Anavaj Sakuntabhai. "The genetic control of immunity to Plasmodium infection". BMC Immunology 16, n.º 1 (2015): 14. http://dx.doi.org/10.1186/s12865-015-0078-z.

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46

Naik, S., N. Bouladoux, C. Wilhelm, M. J. Molloy, R. Salcedo, W. Kastenmuller, C. Deming et al. "Compartmentalized Control of Skin Immunity by Resident Commensals". Science 337, n.º 6098 (26 de julio de 2012): 1115–19. http://dx.doi.org/10.1126/science.1225152.

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47

Palm, Noah W. y Ruslan Medzhitov. "Pattern recognition receptors and control of adaptive immunity". Immunological Reviews 227, n.º 1 (enero de 2009): 221–33. http://dx.doi.org/10.1111/j.1600-065x.2008.00731.x.

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48

Secher, Thomas, Olivier Gaillot, Bernhard Ryffel y Mathias Chamaillard. "Remote Control of Intestinal Tumorigenesis by Innate Immunity". Cancer Research 70, n.º 5 (28 de febrero de 2010): 1749–52. http://dx.doi.org/10.1158/0008-5472.can-09-3401.

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49

Huang, Lei y Andrew L. Mellor. "Metabolic control of tumour progression and antitumour immunity". Current Opinion in Oncology 26, n.º 1 (enero de 2014): 92–99. http://dx.doi.org/10.1097/cco.0000000000000035.

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50

Wang, Tian. "Flavivirus Immunity in Disease Control and Viral Pathogenesis". Viral Immunology 33, n.º 1 (1 de febrero de 2020): 1–2. http://dx.doi.org/10.1089/vim.2019.29047.tjt.

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