Literatura académica sobre el tema "IL-4"
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Artículos de revistas sobre el tema "IL-4"
Usha, Prof. "IL-4 and IL-6 in Bronchial Asthma Does IL-6 Plays More Important Role than IL-4? A Preliminary Study". Journal of Medical Science And clinical Research 05, n.º 04 (18 de abril de 2017): 20446–50. http://dx.doi.org/10.18535/jmscr/v5i4.115.
Texto completoKelly-Welch, A., E. M. Hanson y A. D. Keegan. "Interleukin-4 (IL-4) Pathway". Science Signaling 2005, n.º 293 (12 de julio de 2005): cm9. http://dx.doi.org/10.1126/stke.2932005cm9.
Texto completoBorish, Larry. "IL-4 and IL-13 Dual Antagonism". American Journal of Respiratory and Critical Care Medicine 181, n.º 8 (15 de abril de 2010): 769–70. http://dx.doi.org/10.1164/rccm.201002-0147ed.
Texto completoSchindler, C., H. Kashleva, A. Pernis, R. Pine y P. Rothman. "STF-IL-4: a novel IL-4-induced signal transducing factor." EMBO Journal 13, n.º 6 (marzo de 1994): 1350–56. http://dx.doi.org/10.1002/j.1460-2075.1994.tb06388.x.
Texto completoGause, W. C., T. Takashi, J. D. Mountz, F. D. Finkelman y A. D. Steinberg. "Activation of CD 4-, CD 8- thymocytes with IL 4 vs IL 1 + IL 2." Journal of Immunology 141, n.º 7 (1 de octubre de 1988): 2240–45. http://dx.doi.org/10.4049/jimmunol.141.7.2240.
Texto completoNAKANISHI, KENJI. "Interleukin cascade of IL-4,IL-5 and IL-2." Japanese Journal of Clinical Immunology 13, n.º 5 (1990): 438–40. http://dx.doi.org/10.2177/jsci.13.438.
Texto completoAtamas, S. P., J. Choi, V. V. Yurovsky y B. White. "An alternative splice variant of human IL-4, IL-4 delta 2, inhibits IL-4-stimulated T cell proliferation." Journal of Immunology 156, n.º 2 (15 de enero de 1996): 435–41. http://dx.doi.org/10.4049/jimmunol.156.2.435.
Texto completoNoben-Trauth, N., L. D. Shultz, F. Brombacher, J. F. Urban, H. Gu y W. E. Paul. "An interleukin 4 (IL-4)-independent pathway for CD4+ T cell IL-4 production is revealed in IL-4 receptor-deficient mice". Proceedings of the National Academy of Sciences 94, n.º 20 (30 de septiembre de 1997): 10838–43. http://dx.doi.org/10.1073/pnas.94.20.10838.
Texto completoHo, S. N., R. T. Abraham, A. Nilson, B. S. Handwerger y D. J. McKean. "Interleukin 1-mediated activation of interleukin 4 (IL 4)-producing T lymphocytes. Proliferation by IL 4-dependent and IL 4-independent mechanisms." Journal of Immunology 139, n.º 5 (1 de septiembre de 1987): 1532–40. http://dx.doi.org/10.4049/jimmunol.139.5.1532.
Texto completoReich, Adam, Justyna Szczęch y Dominik Samotij. "Biologics for Itch: IL-4/IL-13, IL-31, IL-17, and IL-23 Antagonists". Current Dermatology Reports 6, n.º 4 (17 de octubre de 2017): 263–72. http://dx.doi.org/10.1007/s13671-017-0204-7.
Texto completoTesis sobre el tema "IL-4"
Dawson, Charlotte Helen. "STAT 6 and IL-4 signalling". Thesis, University of Sheffield, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245716.
Texto completoNieuwenhuizen, Natalie. "Immune and allergic responses to Anisakis pegreffii, with focus on the roles of IL-4, IL-13 and the IL-4 receptor alpha". Doctoral thesis, University of Cape Town, 2007. http://hdl.handle.net/11427/3115.
Texto completoThe fish-parasitizing nematode Anisakis pegreffii induces gastrointestinal disease and allergy when ingested by humans, and can cause occupational allergy in seafood processing workers. The present study examines immune and allergic responses to A. pegreffii in wildtype and gene deficient mice, with special focus on interleukin(IL)-4, IL-13, and the IL-4 receptor alpha (IL-4Rα). Experimental murine models of Anisakis infection, Anisakis-induced anaphylaxis and Anisakis-induced dermatitis were established in order to gain insight into the immune responses generated against Anisakis and unravel mechanisms of allergic disease.
Oksuz, Samet. "Targeting IL-4 locus for epigenetic reprogramming". University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1423581203.
Texto completoGovender, Melissa. "Investigating the role of IL-4/IL-13 signalling through the IL-4 receptor alpha (IL-4Rα) on keratinocytes in murine models of Leishmania major and Schistosoma mansoni". Doctoral thesis, University of Cape Town, 2017. http://hdl.handle.net/11427/24890.
Texto completoМозгова, Юлія Анатоліївна, Юлия Анатольевна Мозговая y Yuliia Anatoliivna Mozghova. "Динаміка вмісту IL-4 та IL-6 при хронічному тонзиліті в дітей". Thesis, Сумський державний університет, 2013. http://essuir.sumdu.edu.ua/handle/123456789/32359.
Texto completoPizzuti, Lucas. "Síntese de 4-(fur-2-il)- e 4-(tien-2-il)-pirimidinas a partir de β-Alcoxivinil trifluormetil cetonas". Universidade Federal de Santa Maria, 2005. http://repositorio.ufsm.br/handle/1/10401.
Texto completoThe cyclocondensation of the 1,1,1-trifluoro-4-methoxy-4-(2-heteroaryl)-3-buten-2-ones (heteroaryl = furyl and thienyl) with urea and amidines (acetamidine, benzamidine, guanidine, 1H-pyrazole-1-carboxamidine and 2-methyl-2-thiopseudourea) for synthesis of two 4-(2-heteroaryl)-6-trifluoromethylpyrimidinones and a series of ten 4-(2-heteroaryl)-6-trifluoromethylpyrimidines is reported. The reaction of the 1,1,1-trifluoro-4-methoxy-4-(2-heteroaryl)-3-buten-2-ones with urea was carried out in the presence of boron trifluoride etherate as a catalyst, at 50°C for 20 hours. The reactions of the same substracts with amidines were carried out in the presence of a 1 M solution of sodium hydroxide at r.t.-50°C for 1 hour. Under this conditions, only aromatic pyrimidines were obtained in 48-67%.
Este trabalho relata a síntese e isolamento de duas 4-(2-heteroaril)-6-trifluormetilpirimidinonas e uma série de dez 4-(2-heteroaril)-6-trifluormetilpirimidinas (heteroaril = furil e tienil), a partir da ciclocondensação de 1,1,1-trifluor-4-metoxi-4-(2-heteroaril)-3-buten-2-onas com uréia e amidinas (acetamidina, benzamidina, guanidina, 1Hpirazolil-1-carboxamidina e 2-metil-2-tiopseudouréia). A reação de 1,1,1-trifluor-4-metoxi-4-(2-heteroaril)-3-buten-2-onas com uréia ocorreu na presença de BF3.Et2O como catalisador, à 50°C por 20 horas. As reações dos mesmos substratos com amidinas ocorreu na presença de uma solução 1 M de NaOH à t.a.-50°C por 1 hora. Nestas condições, foram obtidas somente pirimidinas aromáticas com rendimentos entre 48-67%.
Cirocco, Robert E. "Cytokine analisys in atlantic bottlenose dolphins: molecular characterization of IL-4, IL-6, and IL-10". FIU Digital Commons, 2001. http://digitalcommons.fiu.edu/etd/2370.
Texto completoVale, Mariana Lima. "Atividade analgesica das interleucinas 4, 10 e 13 (IL-4, IL-10 e il-13) na dor inflamatoria experimental : papel de celulas residentes e citocinas". reponame:Repositório Institucional da UFC, 2000. http://www.repositorio.ufc.br/handle/riufc/2569.
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The release of cyclo-oxigenase products and sympathomimetics amines, the final mediators of inflammatory pain, is preceded by the generation of cytokines by resident cells. In recent years a number of cytokines such as IL-4, IL-10, IL-13, IL-6, TGF-β e IFN-α have been described to inhibit the production of TNF-α, IL-1β, IL-6 and IL-8 (cytokines regarded as pró-inflammatory) and possibly to exert their modulatory effect on macrophages and mast cells. Since it is known the capacity of those cytokines to inhibit the production of pro-inflammatory cytokines and the pivotal role of resident cells in the development of inflammatory pain we have decided to test the possibility of IL-4, IL-13 and IL-10 to modulate inflammatory pain. In short, IL-4 (1 – 5ng/animal), IL-13 (0.4 - 2.5ng/animal) or IL-10 (0.4 - 10ng/animal) was given 30 min before acetic acid (AAc) or zymosan (Zym) administration in the writhing model. IL-4 (2.5 e 5 ng/animal), IL-13 (1 e 2.5 ng/animal) or IL-10 (2 e 10 ng/animal) were injected, ip, 30 min before Zym (1 mg/animal; intra-articular) in the rat knee joint incapacitation test up to the 4th hour (the number of leukocytes was determined in the articular exsudate 6 hours later). Doses of those cytokines that exerted maximum effect in the writhing test were also injected 30 min before the hot plate test. These same doses were injected ip before naloxone administration in the AAc-induced writhing model in mice. TNF-α and IL-1β were determined in the supernatant of a macrophage culture which were collected from peritoneal fluid of mice treated with Zym and pre-treated with the cytokines under test. Our results show that interleukins 4, 13 and 10 inhibit writhing response in mice induced by AAc or Zym up to 58.7, 89.2 and 52%, and up to 62.6, 61.7 and 74.4%, respectively (p<0.05). Similar results were observed in the rat knee joint incapacitation test induced by Zym: 49.2, 56.6, 69,9% of inhibition (p<0.05). The same interleukins were able to inhibit Zym-induced leukocyte influx into articular cavity (53.8, 92.1 e 62% of inhibition, respectively - p<0,05). The analgesic activity of IL-4, IL-13 and IL-10 seems to be peripheral, since these cytokines presented no effect in the reaction time of the animals on hot plate test. This antinociceptive effect seems to have no relation with endogen opioid release since naloxone (opioid receptor antagonist) had no effect in reverting the antinociceptive effect of cytokines in the AAc-induced writhing in mice. However, IL-4 and IL-10 inhibit the release of TNF-α (42 e 41.2%, respectively - p<0.05) and of IL-1β (61.9 e 80.9%, respectively - p<0,05) by macrophages stimulated in vivo by Zym, indicating that their antinociceptive activities may be due to the inhibition of those cytokines release by resident cells.
Já está estabelecido que a liberação de produtos da cicloxigenase e aminas simpatomiméticas, mediadores finais da dor inflamatória é precedido pela geração, por células residentes, de uma cascata de citocinas. Recentemente dados do nosso laboratório demonstraram que no modelo de contorções abdominais (CA) a ativação dessa cascata é dependente também da presença de células residentes como macrófagos e mastócitos. Dados da literatura apontam algumas citocinas capazes de modular negativamente a função dessas células: IL-4,. IL-10, IL-13, IL-6, TGF-β e IFN-α . Com base nesses dados, o objetivo do presente trabalho foi estudar uma possível atividade analgésica de três citocinas: IL-4, IL-13 e IL-10. Para tanto injetou-se, via ip, IL-4 (1–5ng/animal), IL-13 (0.4-2.5ng/animal) ou IL-10 (0.4-10ng/animal) 30 min antes da administração de zymosan (Zym) ou ácido acético (AAc) para o teste de CA. IL-4 (2.5 e 5ng/animal), IL-13 (1 e 2.5ng/animal) ou IL-10 (2 e 10ng/animal) foi injetada, ip, 30 min antes do Zym (1 mg/animal; intra-articular) e logo após foi medida a incapacitação articular (IA) até a 4ª hora e na 6ª hora foi feita a contagem de leucócitos no fluido articular. As interleucinas estudadas também foram administradas (30 min antes) na dose que melhor inibiu as CA no teste da placa quente (PQ) e em camundongos que haviam recebido ou não a naloxona previamente ao estímulo (AAc) no teste de CA. IL-4 (5 ng/animal) ou IL-10 (10 ng/animal) foi injetada ip 30 min antes do Zym (ip) e após 15 min os animais foram sacrificados e o exsudato peritoneal foi colhido e posto em cultura para a dosagem de IL-1β e TNF-α no sobrenadante. No presente trabalho ficou demonstrado que as interleucinas-4, 13 e 10 são analgésicas tanto no modelo de CA induzidas por AAc (58.7, 89.2, 52% de inibição, efeito máximo, respectivamente, p<0.05) ou Zym (62.6, 61.7, 74.4% de inibição, efeito máximo, respectivamente, p<0.05) como também no modelo de IA induzido por Zym (49.2, 56.6, 69,9% de inibição, efeito máximo, respectivamente, p<0.05). As citocinas estudadas foram capazes de inibir o influxo de leucócitos para a cavidade articular (53.8, 92.1 e 62%, respectivamente - p<0,05). Foi demonstrado que o efeito analgésico parece ser de domínio periférico visto que nenhuma das citocinas modificou o tempo de reação na PQ, teste algesimétrico sensível apenas para drogas que exercem efeito central. Também foi demonstrado que a atividade analgésica das interleucinas testadas não depende da liberação de opióides endógenos, visto que o pré-tratamento com naloxona não foi capaz de reverter a atividade analgésica de nenhuma das interleucinas no modelo de CA. Contudo essa atividade analgésica parece depender da inibição da liberação de citocinas por células residentes visto que IL-4 e IL-10 foram capazes de diminuir a liberação de TNF-α (42 e 41.2% de inibição respectivamente - p<0.05) e IL-1β (61.9 e 80.9% de inibição respectivamente - p<0,05) por macrófagos peritoneais residentes.
Toor, Iqbal Singh. "Investigating the role of eosinophils in cardiac remodelling following myocardial infarction". Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31187.
Texto completoDheda, Keertan Unkha Jairam. "The expression and role of IL-4 and IL-4(delta)2 in tuberculosis with and without HIV co-infection". Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1445411/.
Texto completoLibros sobre el tema "IL-4"
Samolot bombowy DB-3/Ił-4. Warszawa: Dom Wydawniczy Bellona, 2006.
Buscar texto completoMutin, U. G. Lichnai︠a︡ voĭna mekhanika Il-4. Staryĭ Oskol: Rosa, 2012.
Buscar texto completo1948-, Spits Hergen, ed. IL-4: Structure and function. Boca Raton, FL: CRC Press, 1992.
Buscar texto completoKotelnikov, Vladimir. Il-4: "vozdushnye kreĭsera" Stalina. Moskva: I︠A︡uza, 2009.
Buscar texto completoLazara, Silvia Ferri de, Giulia D'Amaro Valle y Enoch Battagin. Cos'è il contemporaneo? 4: What is the contemporary? 4. Padova: CLEUP, 2012.
Buscar texto completoTrevisi, Giuseppe. Il delitto Fanin: 4 novembre 1948. Bologna: Il Mulino, 1998.
Buscar texto completoIl gastigamatti: Commedia in 4 atti. Firenze: FirenzeLibri, 2010.
Buscar texto completoGiuseppe, Verdi. Il trovatore: Dramma in 4 parti. Roma: Gremese Editore, 1988.
Buscar texto completoGiuseppe, Verdi. Il Trovatore: Dramma in 4 parti. Roma: Gremese Editore, 1988.
Buscar texto completoNatacha, Godeau, ed. Shrek 4: Il était une fin. [Paris]: Hachette jeunesse, 2010.
Buscar texto completoCapítulos de libros sobre el tema "IL-4"
Ranasinghe, Charani, Sreeja Roy, Zheyi Li, Mayank Khanna y Ronald J. Jackson. "IL-4 and IL-13 Receptors". En Encyclopedia of Signaling Molecules, 2549–57. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101978.
Texto completoRanasinghe, Charani, Sreeja Roy, Zheyi Li, Mayank Khanna y Ronald J. Jackson. "IL-4 and IL-13 Receptors". En Encyclopedia of Signaling Molecules, 1–8. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6438-9_101978-1.
Texto completoPelaia, Girolamo, Alessandro Vatrella y Rosario Maselli. "Anti-IL-4/IL-13 Biologics". En Asthma: Targeted Biological Therapies, 67–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46007-9_6.
Texto completoBorish, Larry, John W. Steinke, Marion Kasaian y Samuel J. Goldman. "IL-4- and IL-13-directed approaches". En New Drugs and Targets for Asthma and COPD, 115–21. Basel: KARGER, 2010. http://dx.doi.org/10.1159/000320808.
Texto completoSchroeder, John T. "Diagnostic Components: T Helper Cell Cytokines (IL-4, IL-5, IL-9, IL-10, IL-13, IL-17)". En Encyclopedia of Medical Immunology, 221–26. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_298.
Texto completoKeegan, Achsah, Keats Nelms y William E. Paul. "The IL-4 Receptor — Signaling Mechanisms". En Advances in Experimental Medicine and Biology, 211–15. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-0987-9_21.
Texto completoClair, R. P. "Saturday, September 4, 2010—Chicago, IL". En Zombie Seed and the Butterfly Blues, 323–25. Rotterdam: SensePublishers, 2013. http://dx.doi.org/10.1007/978-94-6209-308-9_60.
Texto completoClair, R. P. "Saturday, September 4, 2010—Chicago, IL". En Zombie Seed and the Butterfly Blues, 327. Rotterdam: SensePublishers, 2013. http://dx.doi.org/10.1007/978-94-6209-308-9_61.
Texto completoShirokaze, J., K. Yanagida, K. Shudo, K. Konomoto, K. Kamiya y K. Sagara. "IL-4 Production Using Macroporous Microcarrier". En Animal Cell Technology: Developments Towards the 21st Century, 877–81. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0437-1_141.
Texto completoKhan, Manzoor M. "Regulation of IL-4 and IL-5 Secretion by Histamine and PGE2". En Advances in Experimental Medicine and Biology, 35–42. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1891-4_5.
Texto completoActas de conferencias sobre el tema "IL-4"
Kalinauskaitė-Žukauskė, Virginija, Andrius Januškevičius, Ieva Janulaitytė y Kęstutis Malakauskas. "IL-4, IL-8, IL-13, IL-17, IL-33 serum levels changes in acute allergic asthma model". En Abstracts from the 17th ERS Lung Science Conference: ‘Mechanisms of Acute Exacerbation of Respiratory Disease’. European Respiratory Society, 2019. http://dx.doi.org/10.1183/23120541.lungscienceconference-2019.pp237.
Texto completoMuchtar, R. Masri. "294 Polymorphism promotor gene of il-4 and il-4 level at graves disease patients". En LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.294.
Texto completoHaque, S. Jaharul, Rikhia Chakraborty, Pankaj Sharma, Suzy Comhair, Sudakshina Ghosh, Weiling Xu, Manoj M. Veleeparambil et al. "Antioxidant-Mediated Negative Regulation Of IL-4/IL-13 Signaling". En American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2794.
Texto completoKalinauskaitė-Žukauskė, Virginija, Andrius Januškevičius, Ieva Janulaitytė y Kęstutis Malakauskas. "LSC - 2019 - IL-4, IL-8, IL-13, IL-17, IL-33 serum levels changes in acute allergic asthma model". En ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4083.
Texto completoButova, Tatiana, Mykhailo Kuzhko, Dmytro Butov y Nataliya Piriatinskaa. "Association between IL-2,IL-4 gene polymorphisms and pulmonary MDRTB". En ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa3035.
Texto completoYadav, Aravind, Rakesh Naidu y Gopala Krishnan Govindasamy. "IL-4, IL-13 And IL-4RA Gene Polymorphisms And The Risk For Developing Idiopathic Nonallergic Rhinitis". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4191.
Texto completoPunia, Navneet, Jennifer Thomson, Irene Babirad y Roma Sehmi. "IL-4 And IL-13 Prime The Transmigrational Responses Of Hemopoietic Progenitor Cells". En American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4034.
Texto completoWirth, F., A. Lubosch, C. Zöller, K. Huck y I. Nakchbandi. "Osteoblasten stimulieren die Hämatopoese durch IL-4 Produktion". En Jahreskongress DVO OSTEOLOGIE 2021. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0040-1722115.
Texto completoLe Floc’H, Audrey, Jeanne Allinne, Kirsten Nagashima, George Scott, Dylan Birchard, Seblewongel Asrat, Matthew Sleeman, Andrew Murphy y Jamie Orengo. "Il-4 and IL-13 act independently and synergistically to drive type 2 inflammation". En ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa5206.
Texto completoBlauvelt, Andrew y Susanne Kammerer. "Novel IL-4/IL-13 blocker shows high efficacy with only modest conjunctivitis signal". En 2022 American Academy of Dermatology Annual Meeting, editado por Peter van de Kerkhof. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/8aee17f2.
Texto completoInformes sobre el tema "IL-4"
Cao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang y Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, abril de 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.
Texto completoGilroy, Jill M. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, octubre de 2001. http://dx.doi.org/10.21236/ada403390.
Texto completoGooch, Jennifer L. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, julio de 1999. http://dx.doi.org/10.21236/ada390684.
Texto completoGooch, Jennifer. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Fort Belvoir, VA: Defense Technical Information Center, julio de 2000. http://dx.doi.org/10.21236/ada384372.
Texto completoSachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, junio de 2001. http://dx.doi.org/10.21236/ada396609.
Texto completoSachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, junio de 2002. http://dx.doi.org/10.21236/ada407504.
Texto completoSachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, junio de 2003. http://dx.doi.org/10.21236/ada418308.
Texto completoLiu, Miao, Hongan Wang, Jing Lu, Zhiyue Zhu, Chaoqun Song, Ye Tian, Xinzhi Chen et al. Vitamin D supplementation in the treatment of Myasthenia Gravis A protocol for a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, septiembre de 2022. http://dx.doi.org/10.37766/inplasy2022.9.0129.
Texto completoShomer, Ilan, Louise Wicker, Uzi Merin y William L. Kerr. Interactions of Cloud Proteins, Pectins and Pectinesterases in Flocculation of Citrus Cloud. United States Department of Agriculture, febrero de 2002. http://dx.doi.org/10.32747/2002.7580669.bard.
Texto completoElmann, Anat, Orly Lazarov, Joel Kashman y Rivka Ofir. therapeutic potential of a desert plant and its active compounds for Alzheimer's Disease. United States Department of Agriculture, marzo de 2015. http://dx.doi.org/10.32747/2015.7597913.bard.
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