Literatura académica sobre el tema "IL-10 deficiency"
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Artículos de revistas sobre el tema "IL-10 deficiency"
Asadullah, K., K. Stephanek, M. Leupold, D. Jasulaitis, H. Audring, H. D. Volk, W. D. Döcke y W. Sterry. "Relative IL-10 deficiency and effects of IL-10 therapy in psoriasis". Journal of Dermatological Science 16 (marzo de 1998): S30. http://dx.doi.org/10.1016/s0923-1811(98)83173-3.
Texto completoWan, Xin, Wen Juan Huang, Wen Chen, Hong-Guang Xie, Pan Wei, Xin Chen y Chang-Chun Cao. "IL-10 Deficiency Increases Renal Ischemia-Reperfusion Injury". Nephron Experimental Nephrology 128, n.º 1-2 (31 de octubre de 2014): 37–45. http://dx.doi.org/10.1159/000366130.
Texto completoHennessy, A., H. L. Pilmore, L. A. Simmons y D. M. Painter. "A Deficiency of Placental IL-10 in Preeclampsia". Journal of Immunology 163, n.º 6 (15 de septiembre de 1999): 3491–95. http://dx.doi.org/10.4049/jimmunol.163.6.3491.
Texto completoZigmond, Ehud, Biana Bernshtein, Gilgi Friedlander, Catherine R. Walker, Simon Yona, Ki-Wook Kim, Ori Brenner et al. "Macrophage-Restricted Interleukin-10 Receptor Deficiency, but Not IL-10 Deficiency, Causes Severe Spontaneous Colitis". Immunity 40, n.º 5 (mayo de 2014): 720–33. http://dx.doi.org/10.1016/j.immuni.2014.03.012.
Texto completoJones, Steven P., Steven D. Trocha y David J. Lefer. "Cardioprotective actions of endogenous IL-10 are independent of iNOS". American Journal of Physiology-Heart and Circulatory Physiology 281, n.º 1 (1 de julio de 2001): H48—H52. http://dx.doi.org/10.1152/ajpheart.2001.281.1.h48.
Texto completoGunnett, Carol A., Donald D. Heistad, Daniel J. Berg y Frank M. Faraci. "IL-10 deficiency increases superoxide and endothelial dysfunction during inflammation". American Journal of Physiology-Heart and Circulatory Physiology 279, n.º 4 (1 de octubre de 2000): H1555—H1562. http://dx.doi.org/10.1152/ajpheart.2000.279.4.h1555.
Texto completoWang, B., L. Zhuang, H. Fujisawa y D. N. Sauder. "Effect of IL-10 deficiency on the migration of epidermal Langerhans cells in IL-10 null mice". Journal of Dermatological Science 16 (marzo de 1998): S90. http://dx.doi.org/10.1016/s0923-1811(98)83537-8.
Texto completoZhang, Guag-Xian, Fang Zhou, Bogoljub Ciric, Hongmei Li, Yaping Yan, Ke Li, Melissa Cullimore et al. "Ability of LPS to regulate expression of tolerance-related molecules on dendritic cells is blocked by IL-10 deficiency (48.18)". Journal of Immunology 188, n.º 1_Supplement (1 de mayo de 2012): 48.18. http://dx.doi.org/10.4049/jimmunol.188.supp.48.18.
Texto completoPérez-de Puig, Isabel, Francesc Miró, Angélica Salas-Perdomo, Ester Bonfill-Teixidor, Maura Ferrer-Ferrer, Leonardo Márquez-Kisinousky y Anna M. Planas. "IL-10 Deficiency Exacerbates the Brain Inflammatory Response to Permanent Ischemia without Preventing Resolution of the Lesion". Journal of Cerebral Blood Flow & Metabolism 33, n.º 12 (11 de septiembre de 2013): 1955–66. http://dx.doi.org/10.1038/jcbfm.2013.155.
Texto completoMarco, Roberto Di, Ming Xiang, Paola Zaccone, Camilla Leonardi, Sabrina Franco, Pierluigi Meroni y Ferdinando Nicolettl. "Concanavalin A-induced Hepatitis in Mice is Prevented by Interleukin (IL)-10 and Exacerbated by Endogenous IL-10 Deficiency". Autoimmunity 31, n.º 2 (enero de 1999): 75–83. http://dx.doi.org/10.3109/08916939908994050.
Texto completoTesis sobre el tema "IL-10 deficiency"
Riquelme, Neira Roberto Andrés. "Influencia de la deficiencia de Il-10 sobre el fenotipo mastocitario, la microbiota y la respuesta inmune intestinal en un modelo murino". Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/664251.
Texto completoMast cells (MC) can participate in the response to microorganisms by various pattern recognition receptors (PRRs). After their activation through these receptors, MC can orchestrate a response by secreting immunological mediators such as cytokines. Among these, interleukin 10 (IL-10) is an important cytokine due to its immunomodulatory characteristics, as well as its ability to regulate the expression of MC proteases. Additionally, thanks to the existence of genetically modified murine models, such as IL-10 deficient (IL-10-/-) mice that develop colitis spontaneously, it is possible to investigate the potential role of IL-10 in MC response to activation with antigens of different microorganisms. On the other hand, the use of this animal model allows investigating the influence of this cytokine on the composition of the intestinal microbiota. This work has explored the functional role of IL-10 in differentiated MC in vitro, as well as the effects of IL-10 deficiency on the composition of the microbiota and the expression of factors related to the immune response, before (6 weeks) and at the onset (20 weeks) of colitis. For this purpose, the effect of IL-10 deficiency has been characterized on MC of different phenotype and after its activation via PRRs. Additionally, the effect produced by the lack of IL-10 on the microbiota composition, the expression of TLRs and proinflammatory cytokines, as well as the production of luminal IgA, in the same stages and after antibiotics treatment was evaluated. The results obtained indicated that the IL-10 deficiency produced different effects depending on the MC phenotype, age and type of PRR ligand. Thus, in the absence of IL-10, mucosal-like MC (MLMC) showed lower expression of TLR4 and NOD2 at week 6 and TLR7 at week 20. In addition, both MC phenotypes (mucosa and connective), showed a lower secretion of IL-6 and TNFα after TLR2 activation. The TLR4 and TLR7 activation in MLMC generated a lower secretion of IL-6 at week 6, while MLMC secreted less TNFα at week 20. Finally, after NOD2 stimulation, no cytokine secretion was observed in any of the MC phenotypes. On the other hand, it was observed that in IL-10-/- animals there are factors that potentially favor the development of colitis. Thus, IL-10-/- mice at week 6 showed representatives of Verrucomicrobia phylum and a lower relative abundance of Rikenellaceae and Lachnospiraceae taxa. Meanwhile at week 20 in IL-10-/- mice, microorganisms of the phylum TM7 were observed, as well as, a lower expression of IL-1β, IL-6, TLR6, -7 and -8, and an increase of TNFα and IgA. Additionally, the use of antibiotics before the development of colitis induced a decrease in diversity and a restructuring of the microbiota, together with a decrease in TLRs and cytokines expression, and a lower production of luminal IgA. In summary, these findings provide new insights on the role of MC and IL-10 in the host-microorganism interaction. They show how the IL-10 deficiency can affect the microbiota composition and the expression of factors associated with the immune response. And they suggest that early modification of the microbiota through the use of antibiotics in genetically susceptible individuals could alter the colitis progression.
Arrieta, Mendez Marie Claire. "The role of small intestinal permeability in the pathogenesis of colitis in the interleukin-10 gene deficient mouse". Phd thesis, 2011. http://hdl.handle.net/10048/1848.
Texto completoExperimental Medicine
Tso, Victor Key. "Nuclear Magnetic Resonance metabolomic fingerprint of the Interleukin 10 gene deficient mouse model of Inflammatory Bowel Disease". Master's thesis, 2010. http://hdl.handle.net/10048/1450.
Texto completoExperimental Medicine
Capítulos de libros sobre el tema "IL-10 deficiency"
Jyonouchi, Harumi y Lee Geng. "Associations between Monocyte Cytokine Profiles and Co-Morbid Conditions in Autism Spectrum Disorders". En Autism Spectrum Disorder - Profile, Heterogeneity, Neurobiology and Intervention. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95548.
Texto completoActas de conferencias sobre el tema "IL-10 deficiency"
Zhang, Wanying, Jie Liao, Haonan Li, Hua Dong, Han Bai, Allison Liao Yang, Bruce D. Hammack y Guang-Yu Yang. "Abstract 1006: Soluble epoxide hydrolase gene deficiency attenuates colitis-induced carcinogenesis in IL-10 knockout mice". En Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1006.
Texto completoLendermon, E., X. Wang, I. Popescu, A. Salaney, M. Helfers, M. Brown, K. Noda, J. Yu y J. F. McDyer. "IL-10 Deficiency in Mice Results in Accelerated Airway Rejection but Is Not Required for Costimulation Blockade-Induced Lung Allograft Acceptance". En American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1005.
Texto completoMcGuire, John K., Cliff Rims y Xiaodong Zhu. "Delayed Recovery After Acute P. Aeruginosa Pneumonia In CD103-Null Mice Is Associated With Deficiency Of IL-10 Producing T Cells In BAL And Lung". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3694.
Texto completoSantos, Daniel Oliveira, CHRISTIAN WALLACE SANTOS MENESES, MELISSA VIEIRA GOMES y LARISSA EMILY OGANDO DE JESUS SENA. "AS RESPOSTAS DA IMUNIDADE INATA NO PACIENTE COM CANDIDÍASE MUCOCUTÂNEA CRÔNICA". En II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6752.
Texto completoInformes sobre el tema "IL-10 deficiency"
Ficht, Thomas, Gary Splitter, Menachem Banai y Menachem Davidson. Characterization of B. Melinensis REV 1 Attenuated Mutants. United States Department of Agriculture, diciembre de 2000. http://dx.doi.org/10.32747/2000.7580667.bard.
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