Literatura académica sobre el tema "Id2 inhibiteur de E2A"
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Artículos de revistas sobre el tema "Id2 inhibiteur de E2A"
Nigten, Jeannet, Gorica Nikoloski, Theo De Witte, Bert A. Van der Reijden y Joop H. Jansen. "Id1 and Id2 Are Retinoic Acid Responsive Genes and Induce a G0/G1 Arrest in Acute Promyelocytic Leukemia Cells." Blood 104, n.º 11 (16 de noviembre de 2004): 2029. http://dx.doi.org/10.1182/blood.v104.11.2029.2029.
Texto completoGonda, Hiroyuki, Manabu Sugai, Yukiko Nambu, Tomoya Katakai, Yasutoshi Agata, Kazuhiro J. Mori, Yoshifumi Yokota y Akira Shimizu. "The Balance Between Pax5 and Id2 Activities Is the Key to AID Gene Expression". Journal of Experimental Medicine 198, n.º 9 (27 de octubre de 2003): 1427–37. http://dx.doi.org/10.1084/jem.20030802.
Texto completoKim, Wonil, Kimberly D. Klarmann y Jonathan R. Keller. "Impaired Erythropoiesis Of Gfi-1 Null Hematopoietic Progenitor Cells Is Rescued By Reducing Id2 Levels". Blood 122, n.º 21 (15 de noviembre de 2013): 737. http://dx.doi.org/10.1182/blood.v122.21.737.737.
Texto completoPortis, Toni y Richard Longnecker. "Epstein-Barr Virus LMP2A Interferes with Global Transcription Factor Regulation When Expressed during B-Lymphocyte Development". Journal of Virology 77, n.º 1 (1 de enero de 2003): 105–14. http://dx.doi.org/10.1128/jvi.77.1.105-114.2003.
Texto completoYamamoto, Hideyuki, Fumihiko Hayakawa, Takahiko Yasuda, Yuka Minamikawa, Naoyuki Tange, Daiki Hirano, Yuki Kojima et al. "ZNF384-Fusion Proteins Have High Affinity to EP300, Which Increases Their Transcriptional Activities". Blood 132, Supplement 1 (29 de noviembre de 2018): 1554. http://dx.doi.org/10.1182/blood-2018-99-114414.
Texto completoCondorelli, G., L. Vitelli, M. Valtieri, I. Marta, E. Montesoro, V. Lulli, R. Baer y C. Peschle. "Coordinate expression and developmental role of Id2 protein and TAL1/E2A heterodimer in erythroid progenitor differentiation". Blood 86, n.º 1 (1 de julio de 1995): 164–75. http://dx.doi.org/10.1182/blood.v86.1.164.bloodjournal861164.
Texto completoBoos, Markus D., Yoshifumi Yokota, Gerard Eberl y Barbara L. Kee. "Mature natural killer cell and lymphoid tissue–inducing cell development requires Id2-mediated suppression of E protein activity". Journal of Experimental Medicine 204, n.º 5 (23 de abril de 2007): 1119–30. http://dx.doi.org/10.1084/jem.20061959.
Texto completoJi, Ming, Huajie Li, Hyung Chan Suh, Kimberly D. Klarmann, Yoshifumi Yokota y Jonathan R. Keller. "Id2 intrinsically regulates lymphoid and erythroid development via interaction with different target proteins". Blood 112, n.º 4 (15 de agosto de 2008): 1068–77. http://dx.doi.org/10.1182/blood-2008-01-133504.
Texto completoYan, W., A. Z. Young, V. C. Soares, R. Kelley, R. Benezra y Y. Zhuang. "High incidence of T-cell tumors in E2A-null mice and E2A/Id1 double-knockout mice." Molecular and Cellular Biology 17, n.º 12 (diciembre de 1997): 7317–27. http://dx.doi.org/10.1128/mcb.17.12.7317.
Texto completoMasson, Frederick, Margherita Ghisi, Joanna R. Groom, Axel Kallies, Cyril Seillet, Ricky W. Johnstone, Stephen L. Nutt y Gabrielle T. Belz. "Id2 represses E2A-mediated activation of IL-10 expression in T cells". Blood 123, n.º 22 (29 de mayo de 2014): 3420–28. http://dx.doi.org/10.1182/blood-2014-03-561456.
Texto completoTesis sobre el tema "Id2 inhibiteur de E2A"
Lacroix, Aurélie. "Etude de la localisation de l’herpèsvirus humain de type 6 (HHV-6) dans les tissus hodgkiniens : recherche de l’expression de l’oncogène viral DR7 et de ses conséquences pour la cellule". Limoges, 2008. https://aurore.unilim.fr/theses/nxfile/default/45243d16-ed38-4721-8146-f2c5dd68586b/blobholder:0/2008LIMO4001.pdf.
Texto completoThere are two variants, A and B, of human lymphotropic herpesvirus type 6 (HHV-6). The B variant is associated with Hodgkin’s lymphoma (HL). However this association is contreversial in the literature because it only exists in rare cases and not in Reed-Sternberg cells (RS), the characteristic cells of HL. In the first part of our study quantitative PCR demonstrated the existence of HHV-6 alone or concommitantly with Epstein-Barr virus, known to be associated with HL, in a cohort of HL patients. We then used an anti-HHV-6 DR7B antibody produced in our laboratory to determine the cellular localization of HHV-6 and to detect its presence in large quantities in RS cells from 73. 7% of HL patients positive only for HHV-6. Furthermore, double labeling studies in patients from the cohort positive for both herpesviruses demonstrated the colocalization, notably in RS cells, of DR7B from HHV-6 and LMP1 from EBV oncoproteins, indicating a probable viral interaction. Finally, a stable transfection system was realised to adjust DR7B expression in mature B cells which are close to RS cells. In the presence of DR7B, a transcriptionnal and translational expression of Id2, an inhibitor of E2A wich is a transcription factor implicated in phenotypical reprogramming of RS cells, similar to RS cells in HL, was observed