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Libros sobre el tema "Human t-cell leukemia viru"

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Publicado: 11 de marzo de 2023

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1

Vogt, Peter K., ed. Human T-Cell Leukemia Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70113-9.

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2

1932-, Vogt P. K., ed. Human T-cell leukemia virus. Berlin: Springer, 1985.

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3

Essex, Myron. Selected abstracts on viral etiology of human cancer.: Human T-cell leukemia viruses. Bethesda, MD: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1986.

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4

Kenton, Charlotte. Human T-cell leukemia/lymphoma virus (HTLV), January 1982 through September 1984, 234 citations. [Bethesda, Md.]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1985.

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5

Symposium, Takamatsu no Miya Hi Gan Kenkyū Kikin International. Retroviruses in human lymphoma/leukemia: Proceedings of the 15th International Symposium of the Princess Takamatsu Cancer Research Fund, Tokyo, 1984. Tokyo: Japan Scientific Societies Press, 1985.

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6

C, Román Gustavo, Vernant Jean-Claude, Osame Mitsuhiro, Texas Tech University. Dept. of Neurology., Meynard Hospital. Dept. of Neurology. y International Symposium on HTLV-I and the Nervous System (1968 : Meynard Hospital., eds. HTLV-I and the nervous system: Proceedings of an international meeting organized by the Departments of Neurology of Texas Tech University and La Meynard Hospital, held in Fort-de-France, Martinique, French Antilles, April 15-16, 1988. New York: Liss, 1989.

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7

Jean, Otter y American Association of Blood Banks., eds. Current status of HTLV-III testing. Arlington, Va: American Association of Blood Banks, 1986.

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8

E, Menitove Jay, Kolins Jerry, American Association of Blood Banks. Committee on Technical/Scientific Workshops. y AIDS Technical Workshop (1986 : San Francisco, Calif.), eds. AIDS. Arlington, Va: American Association of Blood Banks, 1986.

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9

Vogt, Peter K. Human T-Cell Leukemia Virus. Brand: Springer, 2011.

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10

Vogt, P. K. Human T-Cell Leukemia Virus. Springer London, Limited, 2012.

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11

Human T-Cell Leukemia Virus. Springer, 2011.

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12

Human T-Cell Leukemia Virus. Springer Verlag, 1985.

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13

Moriuchi, Hiroyuki. Human T-cell Lymphotropic Virus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0010.

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Human T-cell lymphotropic virus type 1 (HTLV-1), a human retrovirus that infects an estimated 10–20 million people worldwide, has endemic foci in Japan, West and Central Africa, the Caribbean, Central and South America, and Melanesia. Also, it is the etiological agent of a lymphoproliferative malignancy, adult T-cell leukemia/lymphoma (ATLL), as well as chronic inflammatory diseases such as HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP). HTLV-1 can be transmitted vertically, sexually, or by blood-borne transmission. ATLL occurs in approximately 5% of carriers who are infected during early childhood, and primary prevention is the only strategy likely to reduce this fatal disease. Children born to carrier mothers acquire the virus predominantly from breastfeeding. In endemic areas, mother-to-child transmission (MTCT) can be significantly reduced by screening pregnant women for the HTLV-1 antibody, followed by replacing breastfeeding with exclusive formula feeding. Indications for serological screening and recommendations for prevention of perinatal transmission are reviewed in this chapter.
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14

Sugamura, Kazuo, ed. Two Decades of Adult T Cell Leukemia and HTLV-1 Research (Gann Monographs on Cancer Research). Karger, 2003.

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15

Modeling the risk of adult T-cell leukemia/lymphoma in persons infected with human T-lymphotropic virus Type I. [Bethesda, Md.?]: National Cancer Institute, Office of Cancer Communications, 1988.

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16

Roman, Eve, Alexandra Smith y Lorelei Mucci. Leukemias. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0028.

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Leukemias are a diverse group of acute and chronic haematological malignancies, that account for 2% to 3% of cancers globally. Recent advances in molecular biology and therapy have transformed the landscape for several leukemia subtypes changing some, but by no means all, from rapidly fatal diseases to treatable conditions with a good prognosis. In general, however, this progress has not been matched by new aetiological insights. Albeit accounting for a relatively small proportion, genetic predisposition syndromes such as neurofibromatosis, Li-Fraumeni and Trisomy 21, have the biggest impact in children and young adults. At older ages, established chemical, physical and biological risk factors, which explain only a small proportion of the total disease burden, include chemotherapy for a preceding cancer, ionizing radiation, and the viral infections human T-cell lymphotropic virus type 1 (HTLV-1) which causes the rare adult T-cell leukemia/lymphoma (ATLL) and the human immunodeficiency virus (HIV) which is associated with an increased risk of acute lymphoid leukaemias. Workplace exposures to potential carcinogens such as benzene, butadiene, and styrene have also been linked to increased risk of leukemia.
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17

Franceschi, Silvia, Hashem B. El-Serag, David Forman, Robert Newton y Martyn Plummer. Infectious Agents. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0024.

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Eleven infectious agents (seven viruses, three parasites, and one bacterium) have been classified by the International Agency for Research on Cancer as carcinogenic to humans for one or more cancer sites: hepatitis B virus; hepatitis C virus; thirteen types of human papillomavirus (HPV); human immunodeficiency virus type 1 (HIV-1); human T-cell leukemia virus type 1; Epstein-Barr virus; Kaposi sarcoma herpesvirus; Helicobacter pylori; Opisthorchis viverrini; Clonorchis sinensis; and Schistosoma haematobium. Other infectious agents, such as Merkel cell polyomavirus, Plasmodium falciparum, and cutaneous HPVs, have been classified as “probably carcinogenic” or “possibly carcinogenic.” Accurate biomarkers of chronic infection have been essential for estimating risk and ascribing a causal role to infectious agents in cancer. Of the 14 million cases of cancer estimated to have occurred worldwide in 2012, 2.2 million were caused by infectious agents. Vaccination and screen-and-treat programs have the potential for greatly reducing the burden of cancer caused by infections.
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18

Human T-cell Leukemia Vir (Current Topics in Microbiology and Immunology). Springer, 1985.

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