Bibliografías temáticas / Human dendritic cell

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Literatura académica sobre el tema "Human dendritic cell"

Autor: Grafiati
Publicado: 11 de marzo de 2023

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Artículos de revistas sobre el tema "Human dendritic cell"

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Bigley, Venetia, Urszula Cytlak y Matthew Collin. "Human dendritic cell immunodeficiencies". Seminars in Cell & Developmental Biology 86 (febrero de 2019): 50–61. http://dx.doi.org/10.1016/j.semcdb.2018.02.020.

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Collin, Matthew, Naomi McGovern y Muzlifah Haniffa. "Human dendritic cell subsets". Immunology 140, n.º 1 (12 de agosto de 2013): 22–30. http://dx.doi.org/10.1111/imm.12117.

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Tsunoda, Rikiya, Alain Bosseloir, Kikuo Onozaki, Ernst Heinen, Katsuya Miyake, Hiro-oki Okamura, Kazunori Suzuki, Teizou Fujita, Léon J. Simar y Naonori Sugai. "Human follicular dendritic cells in vitro and follicular dendritic-cell-like cells". Cell and Tissue Research 288, n.º 2 (9 de abril de 1997): 381–89. http://dx.doi.org/10.1007/s004410050824.

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Caron, Gersende, Yves Delneste, Edith Roelandts, Catherine Duez, Jean-Yves Bonnefoy, Joel Pestel y Pascale Jeannin. "Histamine Polarizes Human Dendritic Cells into Th2 Cell-Promoting Effector Dendritic Cells". Journal of Immunology 167, n.º 7 (1 de octubre de 2001): 3682–86. http://dx.doi.org/10.4049/jimmunol.167.7.3682.

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Olweus, J., A. BitMansour, R. Warnke, P. A. Thompson, J. Carballido, L. J. Picker y F. Lund-Johansen. "Dendritic cell ontogeny: A human dendritic cell lineage of myeloid origin". Proceedings of the National Academy of Sciences 94, n.º 23 (11 de noviembre de 1997): 12551–56. http://dx.doi.org/10.1073/pnas.94.23.12551.

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Foster, Barbara, Dean D. Metcalfe y Calman Prussin. "Human dendritic cell 1 and dendritic cell 2 subsets express FcεRI". Journal of Allergy and Clinical Immunology 112, n.º 6 (diciembre de 2003): 1132–38. http://dx.doi.org/10.1016/j.jaci.2003.09.011.

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Lin, Chin-Hsien, Hsun Li, Yi-Nan Lee, Ying-Ju Cheng, Ruey-Meei Wu y Cheng-Ting Chien. "Lrrk regulates the dynamic profile of dendritic Golgi outposts through the golgin Lava lamp". Journal of Cell Biology 210, n.º 3 (27 de julio de 2015): 471–83. http://dx.doi.org/10.1083/jcb.201411033.

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Constructing the dendritic arbor of neurons requires dynamic movements of Golgi outposts (GOPs), the prominent component in the dendritic secretory pathway. GOPs move toward dendritic ends (anterograde) or cell bodies (retrograde), whereas most of them remain stationary. Here, we show that Leucine-rich repeat kinase (Lrrk), the Drosophila melanogaster homologue of Parkinson’s disease–associated Lrrk2, regulates GOP dynamics in dendrites. Lrrk localized at stationary GOPs in dendrites and suppressed GOP movement. In Lrrk loss-of-function mutants, anterograde movement of GOPs was enhanced, whereas Lrrk overexpression increased the pool size of stationary GOPs. Lrrk interacted with the golgin Lava lamp and inhibited the interaction between Lva and dynein heavy chain, thus disrupting the recruitment of dynein to Golgi membranes. Whereas overexpression of kinase-dead Lrrk caused dominant-negative effects on GOP dynamics, overexpression of the human LRRK2 mutant G2019S with augmented kinase activity promoted retrograde movement. Our study reveals a pathogenic pathway for LRRK2 mutations causing dendrite degeneration.
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Elahi, Zahra, Paul W. Angel, Suzanne K. Butcher, Nadia Rajab, Jarny Choi, Yidi Deng, Justine D. Mintern, Kristen Radford y Christine A. Wells. "The Human Dendritic Cell Atlas: An Integrated Transcriptional Tool to Study Human Dendritic Cell Biology". Journal of Immunology 209, n.º 12 (15 de diciembre de 2022): 2352–61. http://dx.doi.org/10.4049/jimmunol.2200366.

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Abstract Dendritic cells (DCs) are functionally diverse and are present in most adult tissues, but deep understanding of human DC biology is hampered by relatively small numbers of these in circulation and their short lifespan in human tissues. We built a transcriptional atlas of human DCs by combining samples from 14 expression profiling studies derived from 10 laboratories. We identified significant gene expression variation of DC subset–defining markers across tissue type and upon viral or bacterial stimulation. We further highlight critical gaps between in vitro–derived DC subsets and their in vivo counterparts and provide evidence that monocytes or cord blood progenitor in vitro–differentiated DCs fail to capture the repertoire of primary DC subsets or behaviors. In constructing a reference DC atlas, we provide an important resource for the community wishing to identify and annotate tissue-specific DC subsets from single-cell datasets, or benchmark new in vitro models of DC biology.
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Newman, Rosie, Raina Simpson, Teresa Domagala, Mei Lim, Linda Crofts, Glenn Pilkington y Denese Marks. "Generation of Dendritic Cells from Human Cell Expressed Cytokines." Blood 108, n.º 11 (16 de noviembre de 2006): 5208. http://dx.doi.org/10.1182/blood.v108.11.5208.5208.

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Abstract Traditionally, human cytokines have been expressed in non-human cells, including bacteria, yeast and mouse cells. However, the biological importance of species-specific post-translational modifications, in particular glycosylation, is increasingly being implicated as pivotal to protein function. Glycosylation is important for solubility, resistance to proteolysis, immunogenicity, biological recognition, biological activity, in vivo stability and clearance of glycoproteins including cytokines and growth factors, as up to 75% of their mass may consist of carbohydrate moieties. Clinical trials in cancer immunotherapy have attempted to benefit from the ability of autologous dendritic cells to boost antigen specific T-cell immunity. To date most clinical trials have generated dendritic cells using cytokines expressed in non-human cells. We have purified recombinant human cytokines expressed in modified human 293 cells. Human cell expressed human cytokines including IL-4, GM-CSF and TNF-alpha were used to generate dendritic cells from human peripheral blood mononuclear cells. The resulting cells exhibited a typical mature dendritic cell phenotype including expression of CD209, HLA-DR, CD40, CD80, CD83 and CD86, and were able to generate a T-cell response. In vitro comparisons of the biological activity of human cell expressed cytokines, in particular IL-4, with those expressed in other species has demonstrated that human cell expressed cytokines are more stable. It is proposed that the enhanced biological stability of human cytokines derived from a human cell expression system would make these cytokines ideal for ex vivo derivation of immune cells such as dendritic cells for cancer therapy.
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Bamboat, Zubin M., Jennifer A. Stableford, George Plitas, Bryan M. Burt, Hoang M. Nguyen, Alexander P. Welles, Mithat Gonen, James W. Young y Ronald P. DeMatteo. "Human Liver Dendritic Cells Promote T Cell Hyporesponsiveness". Journal of Immunology 182, n.º 4 (6 de febrero de 2009): 1901–11. http://dx.doi.org/10.4049/jimmunol.0803404.

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Tesis sobre el tema "Human dendritic cell"

1

Morel, Anne-Sophie. "Manipulation of human dendritic cell function". Thesis, Imperial College London, 1999. http://hdl.handle.net/10044/1/11840.

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Jones, Angela. "Human dendritic cell interactions with respiratory syncytial virus". Thesis, University of Sheffield, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.289663.

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Heijstek, Helena Cornelia. "Modulation of human dendritic cell function by therapeutic agents". [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2002. http://dare.uva.nl/document/64240.

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Schäkel, Knut, Claudia Poppe, Elfriede Mayer, Christine Federle, Gert Riethmüller y Ernst Peter Rieber. "M-DC8+ Leukocytes – A Novel Human Dendritic Cell Population". Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-135252.

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Dendritic cells (DC) constitute a heterogeneous leukocyte population having in common a unique capacity to induce primary T cell responses and are therefore most attractive candidates for immunomodulatory strategies. Two populations of blood DC (CD11c+ CD123dim and CD11c– CD123high) have been defined so far. However, their direct isolation for experimental purposes is hampered by their low frequency and by the lack of selective markers allowing large scale purification from blood. Here we describe the monoclonal antibody (mAb) M-DC8, which was generated by immunizing mice with highly enriched blood DC. This mAb specifically reacts with 0.2–1% of blood leukocytes and enables their direct isolation by a one-step immunomagnetic procedure from fresh mononuclear cells. These cells can be differentiated from T cells, B cells, NK cells and monocytes using lineage-specific antibodies. M-DC8+ cells express HLA class II molecules, CD33 and low levels of the costimulatory molecules CD86 and CD40. Upon in vitro culture M-DC8+ cells spontaneously mature into cells with the phenotype of highly stimulatory cells as documented by the upregulation of HLA-DR, CD86 and CD40; in parallel CD80 expression is induced. M-DC8+ cells display an outstanding capacity to present antigen. In particular, they proved to be excellent stimulators of autologous mixed leukocyte reaction and to activate T cells against primary antigens such as keyhole limpet hemocyanin. Furthermore, they induce differentiation of purified allogeneic cytotoxic T cells into alloantigen-specific cytotoxic effector cells. While the phenotypical analysis reveals similarities with the two known blood DC populations, the characteristic expression of Fc=γRIII (CD16) and the M-DC8 antigen clearly defines them as a novel population of blood DC. The mAb M-DC8 might thus be a valuable tool to determine circulating DC for diagnostic purposes and to isolate these cells for studies of antigen-specific T cell priming
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
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Schäkel, Knut, Claudia Poppe, Elfriede Mayer, Christine Federle, Gert Riethmüller y Ernst Peter Rieber. "M-DC8+ Leukocytes – A Novel Human Dendritic Cell Population". Karger, 1999. https://tud.qucosa.de/id/qucosa%3A27632.

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Dendritic cells (DC) constitute a heterogeneous leukocyte population having in common a unique capacity to induce primary T cell responses and are therefore most attractive candidates for immunomodulatory strategies. Two populations of blood DC (CD11c+ CD123dim and CD11c– CD123high) have been defined so far. However, their direct isolation for experimental purposes is hampered by their low frequency and by the lack of selective markers allowing large scale purification from blood. Here we describe the monoclonal antibody (mAb) M-DC8, which was generated by immunizing mice with highly enriched blood DC. This mAb specifically reacts with 0.2–1% of blood leukocytes and enables their direct isolation by a one-step immunomagnetic procedure from fresh mononuclear cells. These cells can be differentiated from T cells, B cells, NK cells and monocytes using lineage-specific antibodies. M-DC8+ cells express HLA class II molecules, CD33 and low levels of the costimulatory molecules CD86 and CD40. Upon in vitro culture M-DC8+ cells spontaneously mature into cells with the phenotype of highly stimulatory cells as documented by the upregulation of HLA-DR, CD86 and CD40; in parallel CD80 expression is induced. M-DC8+ cells display an outstanding capacity to present antigen. In particular, they proved to be excellent stimulators of autologous mixed leukocyte reaction and to activate T cells against primary antigens such as keyhole limpet hemocyanin. Furthermore, they induce differentiation of purified allogeneic cytotoxic T cells into alloantigen-specific cytotoxic effector cells. While the phenotypical analysis reveals similarities with the two known blood DC populations, the characteristic expression of Fc=γRIII (CD16) and the M-DC8 antigen clearly defines them as a novel population of blood DC. The mAb M-DC8 might thus be a valuable tool to determine circulating DC for diagnostic purposes and to isolate these cells for studies of antigen-specific T cell priming.
Dieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich.
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Sampangi, Sandeep. "Autologous human kidney proximal tubule epithelial cells (PTEC) modulate dendritic cell (DC), T cell and B cell responses". Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/82033/1/Sandeep_Sampangi_Thesis.pdf.

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This is a comprehensive study of human kidney proximal tubular epithelial cells (PTEC) which are known to respond to and mediate the pathological process of a range of kidney diseases. It identifies various molecules expressed by PTEC and how these molecules participate in down-regulating the inflammatory process, thereby highlighting the clinical potential of these molecules to treat various kidney diseases. In the disease state, PTEC gain the ability to regulate the immune cell responses present within the interstitium. This down-regulation is a complex interaction of contact dependent/independent mechanisms involving various immuno-regulatory molecules including PD-L1, sHLA-G and IDO. The overall outcome of this down-regulation is suppressed DC maturation, decreased number of antibody producing B cells and low T cell responses. These manifestations within a clinical setting are expected to dampen the ongoing inflammation, preventing the damage caused to the kidney tissue.
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Hu, Yaling. "Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells /". View abstract or full-text, 2008. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202008%20HU.

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Varani, Stefania. "Human cytomegalovirus and dendritic cell interaction : role in immunosuppression and autoimmunity /". Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-505-4/.

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NIZZOLI, GIULIA. "Human dendritic cell subsets: cytokine production and their role in T-cell priming". Doctoral thesis, Università degli Studi di Milano-Bicocca, 2014. http://hdl.handle.net/10281/50066.

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Dendritic cells (DC) have the unique capacities to induce primary T cell responses. In mice, CD8α+DC are specialized to cross-prime CD8+ T-cells and produce IL-12 that promotes cytotoxicity. Human BDCA-3+DC share several relevant characteristics with CD8α+DC, but the capacities of human DC subsets to induce CD8+ T cell responses are incompletely understood. Here we compared CD1c+mDC1, BDCA-3+mDC2 and plasmacytoid DC (pDC) in peripheral blood and lymphoid tissues for phenotype, cytokine production and their capacities to prime cytotoxic T cells. mDC1 were surprisingly the only human DC that secreted high amounts of IL-12p70, but they required combinational Toll-like receptor (TLR) stimulation. mDC2 and pDC produced IFN-λ and IFN-α, respectively. Importantly, mDC1 and mDC2 required different combinations of TLR-ligands to cross-present protein antigens to CD8+ T cells. pDC were inefficient, and also expressed lower levels of MHC- and co-stimulatory molecules. Nevertheless, all DC induced CD8+ memory T-cell expansions upon licensing by CD4+ T cells, and primed naive CD8+ T-cells following appropriate TLR stimulation. However, since mDC1 produced IL-12 they induced the highest levels of cytotoxic molecules. In conclusion, CD1c+mDC1 are the relevant source of IL-12 for naïve T cells, and are fully equipped to cross-prime cytotoxic T cell responses.
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Thacker, Robert I. "Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen)". University of Cincinnati / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1218553202.

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Libros sobre el tema "Human dendritic cell"

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Lekkerkerker, Annemarie Nicolette. Human antibodies to dendritic cells: Generation, analysis and use in vaccination. [S.l: s.n.], 2002.

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Capítulos de libros sobre el tema "Human dendritic cell"

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Lee, Jimmy S. y Neil E. Reiner. "Stable Lentiviral Vector-Mediated Gene Silencing in Human Monocytic Cell Lines". En Macrophages and Dendritic Cells, 287–300. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-396-7_18.

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Svensson, Mattias. "Isolation and Culture of Human Hematopoietic Progenitors for Studies of Dendritic Cell Biology". En Macrophages and Dendritic Cells, 187–202. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-396-7_13.

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Jones, Hannah E., Nigel Klein y Garth L. J. Dixon. "Human Dendritic Cell Culture and Bacterial Infection". En Methods in Molecular Biology, 217–35. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-346-2_14.

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Gunawan, Merry, Laura Jardine y Muzlifah Haniffa. "Isolation of Human Skin Dendritic Cell Subsets". En Methods in Molecular Biology, 119–28. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3606-9_8.

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Gogolak, Peter y Eva Rajnavölgyi. "Genomics and Functional Differences of Dendritic Cell Subsets". En Immunogenomics and Human Disease, 209–47. Chichester, UK: John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/0470034092.ch10.

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Segura, Elodie. "Review of Mouse and Human Dendritic Cell Subsets". En Methods in Molecular Biology, 3–15. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3606-9_1.

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Grouard, G., O. de Bouteiller, C. Barthelemy, S. Lebecque, J. Banchereau y Y. J. Liu. "Regulation of Human B Cell Activation by Follicular Dendritic Cell and T Cell Signals". En An Antigen Depository of the Immune System: Follicular Dendritic Cells, 105–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79603-6_7.

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Alculumbre, Solana y Lucia Pattarini. "Purification of Human Dendritic Cell Subsets from Peripheral Blood". En Methods in Molecular Biology, 153–67. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3606-9_11.

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Mason, Lauren M. K. y Joppe W. R. Hovius. "Investigating Human Dendritic Cell Immune Responses to Borrelia burgdorferi". En Methods in Molecular Biology, 291–99. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7383-5_21.

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Collin, Matthew y Venetia Bigley. "Monocyte, Macrophage, and Dendritic Cell Development: the Human Perspective". En Myeloid Cells in Health and Disease, 79–97. Washington, DC, USA: ASM Press, 2017. http://dx.doi.org/10.1128/9781555819194.ch6.

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Actas de conferencias sobre el tema "Human dendritic cell"

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Vitte, Joana, Delphine Gras, Laure De Senneville, Daniel Ferry, Pierre Bongrand y Pascal Chanez. "Human Bronchial Epithelium Modulates Dendritic Cell Responses To Particulate Matter". En American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3793.

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Rich, Eric N., Ashley N. Desch, Peter M. Henson y Claudia Jakubzick. "Characterizing The Human Orthologues Of Murine Pulmonary Dendritic Cell Populations". En American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2837.

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Yuan, Song y Qijuan Chen. "Dendritic Cell Algorithm for Anomaly Detection in Unordered Data Set". En 2012 4th International Conference on Intelligent Human-Machine Systems and Cybernetics (IHMSC). IEEE, 2012. http://dx.doi.org/10.1109/ihmsc.2012.69.

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O'Sullivan, Mary P., Ruth C. Ryan y Joseph M. Keane. "Mycobacterium Tuberculosis Infection Induces Non-Apoptotic Cell Death Of Human Monocyte-Derived Dendritic Cells". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3266.

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O'Sullivan, M., D. Triglia Van Nierop, K. Gogan y J. Keane. "Immunometabolism Directs Human CD1c-Positive Myeloid Dendritic Cell Responses to BCG Infection". En American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4237.

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Verhamme, Fien M., Ken R. Bracke, Geert Van Pottelberge, Guy Joos y Guy G. Brusselle. "Expression Of Interferon Regulatory Factor 8 In Human Lung Dendritic Cell Subsets". En American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1074.

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Metcalf, J. P., V. I. Patel y J. L. Booth. "Phagocytosis by Human Airway Macrophage and Dendritic Cell Subsets Is Impaired by Anthrax Toxins". En American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7725.

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NAKAMURA, Shin, Yukio SUZUKI, Takayuki HARADA, Shigeru MORIKAWA, Shunichiro KAWABATA y Sadaaki IWANAGA. "TISSUE FACTOR OF A HUMAN CELL LINE, RET-1: ITSPRODUCTION, PURIFICATION AND PROPERTIES." En XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643287.

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A dendritic cell-like cell line, HIN-Ret-1(RET-1), was found to produce the highest level of tissue factor (TF) among several established cell lines from human lymphoma or leukemia. The TF level expressed by this cell line exceeded 5.6 times that expressed by a monocyte-like cell line, U-937. Unlike other TF producing cell line, i.e. RET-2, HL-60, ML-3, and U-937, the TF expression by RET-1 was spontaneous and unaffected with TP A, PHA, LPS, or MAF. The TF activity of RET-1 was markedly inhibited by Con A as well as that produced by LPS-stimulated monkey monocytes, whereas the TF activity of monkey brain and lung was hardly inhibited by the lectin. Hence, the RET-1 cell lysate solubilized in Triton X-100 was subjected to affinity chromatography on a Con A-Sepharose column, and TF-apoprotein (TF-Apo) was completely bound to the column and eluted with TBS containing 0.15 M α(-methylglucoside and 0.1 % Triton X-100. Further purification of this material was performed with combination of FPLC on a DEAE-5PW column and affinity chromatography using a factor VII-Sepharose column. By these methods, TF-Apo preparation with purification-fold of 9,400 and over-all yield of 7 % was obtained. Its apparent molecular weight was estimated to be 120 kDa by gel filtration in TBS containing 0.1 % Triton X-100. SDS-PAGE gave the value of 47 kDa, which was almost compatible with that of TF-Apo from brain or placenta. TF-Apo frcm the monocytes also bound to the lectin column, suggesting that the apoprotein of these macrophage-related cells has an oligosaccharide chain interacting with the lectin. RET-1 TF-Apo was unstable under acidic condition (<pH 4.0) or in organic solvents such as isopropanol (>18 %) and acetonitrile (>8 %).
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Toma, Marieta I., Rebekka Wehner, Anja Kloß, Kati Erdmann, Susanne Fuessel, Barbara Seliger, Dorothee Brech et al. "Abstract 1278: Accumulation of tolerogenic human 6-sulfo LacNAc+ dendritic cells in renal cell carcinoma is associated with poor prognosis". En Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-1278.

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Kwiecień, Iwona, Elżbieta Rutkowska, Rafał Sokołowski, Joanna Bednarek, Agata Raniszewska, Karina Jahnz-Różyk, Piotr Rzeepecki y Joanna Domagała-Kulawik. "Immunosuppressive properties of human PD-1+ PDL-1+ dendritic cell from Lymph Nodes Aspirates of NSCLC patients". En ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1656.

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Informes sobre el tema "Human dendritic cell"

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Chung, David J. Evaluation of Immune Responses Mediated by Listeria-Stimulated Human Dendritic Cells: Implications for Cancer Vaccine Therapy. Fort Belvoir, VA: Defense Technical Information Center, julio de 2014. http://dx.doi.org/10.21236/ada612542.

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Chung, David J. Evaluation of Immune Responses Mediated by Listeria-Stimulated Human Dendritic Cells: Implications for Cancer Vaccine Therapy. Fort Belvoir, VA: Defense Technical Information Center, julio de 2013. http://dx.doi.org/10.21236/ada581991.

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También puede estar interesado en las bibliografías ampliadas sobre el tema "Human dendritic cell" para tipos de fuentes particulares:
Artículos de revistas Tesis
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