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Libros sobre el tema "HCV"

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Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 6 de septiembre de 2023

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1

Thiemann, Lillian. Double jeopardy: The HIV/HCV co-infection handbook. New York: Community Prescription Service, 1999.

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2

Dammacco, Franco, ed. HCV Infection and Cryoglobulinemia. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-1705-4.

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3

Dammacco, Franco. HCV Infection and Cryoglobulinemia. Milano: Springer-Verlag Italia, 2012.

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4

Musharraf, Husain y Population Council (Bangladesh), eds. Prevalence of HIV, HBV, HCV and syphilis markers in pregnant women of Bangladesh. Dhaka, Bangladesh: Population Council, 1997.

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5

Okita, Kiwamu, ed. HCV and Related Liver Diseases. Tokyo: Springer Japan, 1999. http://dx.doi.org/10.1007/978-4-431-68488-6.

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6

Jürgens, Ralf. HIV/AIDS and HCV in prisons: A select annotated bibliography. Ottawa, Ont: International Affairs Directorate, Health Canada, 2005.

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7

Okita, Kiwamu, ed. HCV/Oxidative Stress and Liver Disease. Tokyo: Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-67005-6.

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8

Kiwamu, Okita, ed. HCV/oxidative stress and liver disease. Tokyo: Springer, 2003.

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9

name, No. HCV/oxidative stress and liver disease. Tokyo: Springer, 2003.

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10

Sofia, Michael J., ed. HCV: The Journey from Discovery to a Cure. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-28400-8.

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11

Sofia, Michael J., ed. HCV: The Journey from Discovery to a Cure. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-28207-3.

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12

Dirgahayu, Paramasari. Molecular epidemiology database of HIV, HBV, HCV, HDV, HTLV-1/2, and TTV in Central of Java, Indonesia: Final report international research collaboration and international publication. [Surakarta]: Sebelas Maret Univ., 2010.

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13

Association, Canadian Hospital, ed. Guide to policies for health care facilities and agencies: Bloodborne pathogens, especially Human Immunodeficiency Virus (HIV), Hepatitis B Virus, (HBV) and Hepatitis C Virus (HCV). Ottawa, Ont: Canadian Hospital Association, 1994.

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14

G, Gish Robert, ed. Maximizing the benefits of antiviral therapy for HCV: The advantages of treating side effects. Philadelphia: W.B. Saunders, 2004.

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15

Duncan, Tim. Conquering Hepatitis C and surviving treatment: An essential guide through every step of the HCV treatment process. [Place of publication not identified]: T. Duncan, 2010.

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16

Giulio, Pisani, ed. Nucleic acid amplification technology (NAT) for the detection of Hepatitis C Virus (HCV) in plasma pools: Validation report. Roma: Istituto superiore di sanità, 2000.

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17

Mohamed Hatem Fathi El-Saied Wali. Natural history, factors affecting severity and progression rate of hepatitis c virus (HCV) infection in liver transplanted and non-transplanted patients. Birmingham: University of Birmingham, 2002.

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18

Seminario, di aggiornamento sull'epatite da HCV: diagnosi epidemiologia prevenzione e. terapia (4th 1997 Rome Italy). IV Seminario di aggiornamento sull'epatite da HCV: Diagnosi, epidemiologia, prevenzione e terapia : Istituto superiore di sanità, Roma, 3-4 novembre 1997 : atti. Roma: Istituto superiore di sanità, 1997.

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19

Käser, Elke Beatrice. Der Hallesche HCV-Impfschadensfall 1978/79 und die Verantwortung der Bundesrepublik Deutschland: Gleichzeitig ein Beitrag zu intertemporalkollisionsrechtlichen Fragestellungen auf einfachgesetzlicher und verfassungsrechtlicher Ebene. Leipzig: Leipziger Universitätsverlag, 1999.

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20

Seminario, di aggiornamento sull'epatite da virus HCV e. nuovi virus potenzialmente epatitici: diagnosi epidemiologia prevenzione e. terapia (5th 2000 Rome Italy). V Seminario di aggiornamento sull'epatite da virus HCV e nuovi virus potenzialmente epatitici: Diagnosi, epidemiologia, prevenzione e terapia : Istituto superiore di sanità, Roma, 20-21 dicembre 2000 : atti. Roma: Istituto superiore di sanità, 2000.

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21

Corporation, International Business Machines, ed. I/O configuration using z/OS HCD and HCM. [S.l.]: Vervante, 2010.

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22

Administration, United States Occupational Safety and Health. Enforcement procedures for occupational exposure to hepatitis B virus (HBV) and human immunodeficiency virus (HIV). Washington, D.C: U.S. Dept. of Labor, Assistant Secretary for Occupational Safety and Health, 1990.

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23

United States. Occupational Safety and Health Administration. Enforcement procedures for occupational exposure to hepatitis B virus (HBV) and human immunodeficiency virus (HIV). Washington, D.C: U.S. Dept. of Labor, Assistant Secretary for Occupational Safety and Health, 1990.

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24

United States. Occupational Safety and Health Administration. Enforcement procedures for occupational exposure to Hepatitis B Virus (HBV) and Human Immunodeficiency Virus (HIV). [Washington, D.C.?: U.S. Dept. of Labor, Assistant Secretary for Occupational Safety and Health, 1988.

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25

Kumar, Rajendra. HCI Human Computer Interaction: HCI. 2a ed. India: Laxmi Publications, 2005.

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26

Wilson, Deanna. Hepatitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0035.

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Hepatitis A (HAV) and E (HEV) viruses are spread via the fecal-oral route. Hepatitis B virus (HBV) exposure is via occupational or recreational activities. Hepatitis D virus (HDV; also spread parentally) can only coinfect or superinfect those with chronic HBV. Hepatitis C (HCV) transmission is predominantly parenteral; the highest risk group is injection drug users. Prodromal-period patients with acute hepatitis present with vague constitutional symptoms when serum transaminases peak, with elevated serum bilirubin and varying levels of hepatic protein synthesis impairment; during the icteric phase, patients develop abdominal pain, hepatomegaly, and jaundice. Acute hepatitis has limited therapy; treatment is predominantly supportive. However, most adults with acute phase HAV, HBV, HDV, and HEV spontaneously clear the virus. Most individuals with HCV develop chronic hepatitis. Patients with known HAV, HBV, or HEV exposures may be eligible for post-exposure prophylaxis to reduce their risk of infection.
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27

Keshav, Satish y Palak Trivedi. Viral hepatitis. Editado por Patrick Davey y David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0212.

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Hepatitis means ‘inflammation of the liver’ and is manifest with symptoms that include malaise, anorexia, fever, flu-like symptoms, and pain in the right upper quadrant of the abdomen, with the pain being caused by swelling of the liver and its capsule. Elevations in circulating hepatic enzymes, particularly aspartate transaminase and alanine transaminase, are common, with jaundice occurring some time after the onset of other symptoms and signs. There are five viruses that primarily cause viral hepatitis: hepatitis A, B, C, D, and E viruses, abbreviated HAV, HBV, HCV, HDV, and HEV, respectively. These viruses are all hepatotrophic, in that the liver is the primary site of infection. HAV, HBV, and HEV are usually acute, self-limiting infections that may, nonetheless, cause morbidity and, in the case of HEV, fatality. However, HBV and, more so, HCV can cause chronic carriage of the virus over many years, as well as the development of chronic hepatitis. HDV is only pathogenic in conjunction with HBV. After recovery from acute infection with HAV, individuals have long-lasting immunity against further infection. The same holds true for the majority of individuals with acute HBV infection. There seems to be little natural immunity to HCV infection, and a significant proportion of cases result in chronic hepatitis. Immunity to HEV is not long-lasting, and repeated infections are possible. Many other viruses can cause hepatitis, of which cytomegalovirus, herpes simplex virus, Epstein–Barr virus, and flaviviruses such as dengue and yellow fever are the most important. The liver, however, is not their primary site of replication or cellular damage.
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28

HCV Testing. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2013.

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29

Decision-Making in HIV/HCV Co-Infection. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2016.

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30

Price, Jennifer Cohen, Priyanka Amin y Antoine Douaihy. Hepatitis C and HIV Co-Infection. Editado por Mary Ann Cohen, Jack M. Gorman, Jeffrey M. Jacobson, Paul Volberding y Scott Letendre. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199392742.003.0043.

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Chronic infection with hepatitis C virus (HCV) is a leading cause of end-stage liver disease and is the most common indication for liver transplantation in the United States. Because of shared risk factors, individuals living with HIV infection are disproportionately affected by HCV. Moreover, co-infection with HIV accelerates the natural history of chronic HCV infection, increasing the risk of cirrhosis, hepatocellular carcinoma, hepatic decompensation, and death. Highly effective medications such as direct-acting antivirals (DAA) to cure HCV are now available and have the potential to profoundly improve the health of HIV-HCV-co-infected individuals. However, addressing the many gaps in the HCV care cascade is necessary to fully achieve the benefits of these drugs. This chapter reviews the natural history of HIV-HCV co-infection, the psychiatric comorbidities associated with HCV infection, the evolution of HCV treatment, and the barriers to care that HIV-HCV-co-infected individuals continue to face.
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31

Ashm. Decision Making in HCV. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2016.

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32

Preventing HCV in Scotland. NHS Health Scotland, 2002.

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33

Decision Making in HCV. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2019.

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34

Dammacco, Franco. HCV Infection and Cryoglobulinemia. Springer, 2014.

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35

Decision Making in HCV. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2015.

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36

Decision Making in Hcv. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2012.

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37

Decision-Making in HCV. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2012.

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38

HIV, STIs, HEPATITIS B (HBV) and HEPATITIS C (HCV) ISSUES for MEDICARE LOCALS. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2012.

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39

Decision Making in HCV (Canada). Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2019.

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40

HCV and Related Liver Diseases. Springer, 2012.

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41

Okita, Kiwamu. HCV and Related Liver Diseases. Springer London, Limited, 2012.

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42

Hcv Treatments Quick Reference Tool. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2019.

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43

Decision-Making in Hcv ¿ Portugal. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2018.

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44

Okita, Kiwamu. HCV and Related Liver Diseases. Springer, 2012.

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45

HCV Treatment Quick Reference Tool. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2017.

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46

Hsieh, Meng-Tien. HBV- and HCV-related hepatocarcinogenesis: Mechanisms and implication of treatment. 2005.

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47

Decision-Making in Hcv in Chicago. Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2018.

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48

Okita, K. HCV/Oxidative Stress and Liver Disease. Springer London, Limited, 2013.

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49

HCV TREATMENTS QUICK REFERENCE TOOL (Canada). Australasian Society for HIV, Viral Hepatitis and Sexual Health Medicine (ASHM), 2019.

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50

Okita, K. Hcv/Oxidative Stress and Liver Disease. Springer, 2013.

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