Literatura académica sobre el tema "Glycinergic synaptic current"
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Artículos de revistas sobre el tema "Glycinergic synaptic current"
Donato, Roberta y Andrea Nistri. "Differential Short-Term Changes in GABAergic or Glycinergic Synaptic Efficacy on Rat Hypoglossal Motoneurons". Journal of Neurophysiology 86, n.º 2 (1 de agosto de 2001): 565–74. http://dx.doi.org/10.1152/jn.2001.86.2.565.
Texto completoGhavanini, Amer A., David A. Mathers, Hee-Soo Kim y Ernest Puil. "Distinctive Glycinergic Currents With Fast and Slow Kinetics in Thalamus". Journal of Neurophysiology 95, n.º 6 (junio de 2006): 3438–48. http://dx.doi.org/10.1152/jn.01218.2005.
Texto completoMcMenamin, Caitlin A., Laura Anselmi, R. Alberto Travagli y Kirsteen N. Browning. "Developmental regulation of inhibitory synaptic currents in the dorsal motor nucleus of the vagus in the rat". Journal of Neurophysiology 116, n.º 4 (1 de octubre de 2016): 1705–14. http://dx.doi.org/10.1152/jn.00249.2016.
Texto completoTRAN, MY N., MATTHEW H. HIGGS y PETER D. LUKASIEWICZ. "AMPA receptor kinetics limit retinal amacrine cell excitatory synaptic responses". Visual Neuroscience 16, n.º 5 (septiembre de 1999): 835–42. http://dx.doi.org/10.1017/s0952523899165039.
Texto completoTadros, M. A., K. E. Farrell, P. R. Schofield, A. M. Brichta, B. A. Graham, A. J. Fuglevand y R. J. Callister. "Intrinsic and synaptic homeostatic plasticity in motoneurons from mice with glycine receptor mutations". Journal of Neurophysiology 111, n.º 7 (1 de abril de 2014): 1487–98. http://dx.doi.org/10.1152/jn.00728.2013.
Texto completoBuss, Robert R. y Pierre Drapeau. "Synaptic Drive to Motoneurons During Fictive Swimming in the Developing Zebrafish". Journal of Neurophysiology 86, n.º 1 (1 de julio de 2001): 197–210. http://dx.doi.org/10.1152/jn.2001.86.1.197.
Texto completoCROOK, JOANNA D., ORIN S. PACKER y DENNIS M. DACEY. "A synaptic signature for ON- and OFF-center parasol ganglion cells of the primate retina". Visual Neuroscience 31, n.º 1 (27 de noviembre de 2013): 57–84. http://dx.doi.org/10.1017/s0952523813000461.
Texto completoOku, Y., S. Hülsmann, W. Zhang y D. W. Richter. "Modulation of glycinergic synaptic current kinetics by octanol in mouse hypoglossal motoneurons". Pfl�gers Archiv European Journal of Physiology 438, n.º 5 (17 de septiembre de 1999): 656–64. http://dx.doi.org/10.1007/s004240051090.
Texto completoOku, Y., S. Hülsmann, W. Zhang y D. W. Richter. "Modulation of glycinergic synaptic current kinetics by octanol in mouse hypoglossal motoneurons". Pflügers Archiv - European Journal of Physiology 438, n.º 5 (octubre de 1999): 656–64. http://dx.doi.org/10.1007/s004249900089.
Texto completoXie (解瑞立), Ruili y Paul B. Manis. "Glycinergic synaptic transmission in the cochlear nucleus of mice with normal hearing and age-related hearing loss". Journal of Neurophysiology 110, n.º 8 (15 de octubre de 2013): 1848–59. http://dx.doi.org/10.1152/jn.00151.2013.
Texto completoTesis sobre el tema "Glycinergic synaptic current"
Rančić, Vladimir. "Characterization of synaptic circuits changes in ventral horn of embrionic spinal slices cultures from SOD1 G93A mice". Doctoral thesis, Università degli studi di Trieste, 2011. http://hdl.handle.net/10077/4598.
Texto completoAmyotrophic lateral sclerosis (ALS) is a lethal neurodegenerative disease characterized by loss of motoneurons. The discovery of mutations in the gene for the cytosolic Cu/Zn superoxide dismutase in a small proportion of familiar ALS patients led to an animal model in which the human mutant SOD1 is overexpressed in mice (G93A). For this study, we employed the long term spinal cord organotypic cultures developed from G93A embryonic mice and their wild type (WT) littermates, starting from the recent findings emerged from a study by Avossa et al. (2006). These authors reported that G93A organotypic spinal cultures exhibited increased vulnerability to AMPA glutamate receptormediated excitotoxic stress, prior to clear disease appearance, besides showing a significantly increased ratio between inhibitory and excitatory synapses, although they did not express evident morphological differences, when compared to WT ones (Avossa et al., 2006). The primary objective of this study was to investigate this early ALS stage to understand how functional changes can predate morphological alterations. To that aim we monitored spontaneous synaptic activity via patch clamping interneurons both in WT and G93A spinal cultures after 7, 14 and 21 days of in vitro (DIV) growth. At 7 DIV, when synchronous episodes of activity are normally detected in cultured spinal circuits, G93A slices displayed bursting with a higher probability (83%) when compared to controls (54%). Between 14 and 21 DIV, when bursting activity disappear, both in G93A and WT slices, pharmacological dissection of glutamate, GABA and glycine mediated post synaptic currents (PSCs), showed, in G93A, a significant reduction in GABAergic PSCs and mPSCs in respect to WT. Upon pharmacological removal of the GABAergic component, fast glycinergic events were unmasked and these events displayed a similar frequency in both culture groups. Along with in vitro growth, we detected a progressive reduction in the decay time constant of glycinergic PSCs, such process was significantly faster in G93A. Thus, a shift in dynamic communication within spinal networks might be involved in ALS progression.
XXIII Ciclo
1980